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This document lists various stressors and causes of intimal vessel wall injury, including mechanical stress from high blood pressure and procedures like PCI, chemical influences from conditions like hypercholesterolemia and smoking, immunologic factors such as infections, and genetic and biologic alterations like aging. Mechanical sheer stress, chemical influences on vessels from conditions and substances, immunologic antigen-antibody responses, and natural genetic and biologic changes can all potentially cause injury to the intimal layer of blood vessels.
This document lists various stressors and causes of intimal vessel wall injury, including mechanical stress from high blood pressure and procedures like PCI, chemical influences from conditions like hypercholesterolemia and smoking, immunologic factors such as infections, and genetic and biologic alterations like aging. Mechanical sheer stress, chemical influences on vessels from conditions and substances, immunologic antigen-antibody responses, and natural genetic and biologic changes can all potentially cause injury to the intimal layer of blood vessels.
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This document lists various stressors and causes of intimal vessel wall injury, including mechanical stress from high blood pressure and procedures like PCI, chemical influences from conditions like hypercholesterolemia and smoking, immunologic factors such as infections, and genetic and biologic alterations like aging. Mechanical sheer stress, chemical influences on vessels from conditions and substances, immunologic antigen-antibody responses, and natural genetic and biologic changes can all potentially cause injury to the intimal layer of blood vessels.
Copyright:
Attribution Non-Commercial (BY-NC)
Verfügbare Formate
Als PDF, TXT herunterladen oder online auf Scribd lesen
Source and Effect Sheer stress from blood flow at vessel branches/bifurcation due to HTN; injury secondary to PCI procedures Hypercholesterolemia; homocystinemia; hydrocarbons, nicotine, and carbon monoxide from cigarettes; free fatty acids, oxygen radicals, angiotensin, hormones; oxidized lipids; oxidant stress caused by cigarette smoking, hypertension, and advanced glycation end products in diabetes mellitus; estrogen deficiency; vasoactive amines Exposure to antigenantibody complexes; virally induced injuries; exposure to bacteria; activated macrophages; infectious microorganisms such as herpesviruses, Chlamydia pneumoniae Alterations Advancing age