Klver-Bucy Syndrome Another form of visual agnosia is the psychic blindness

syndrome described by Klver and Bucy in 1939. They reported the syndrome
originally in monkeys with bilateral temporal lobectomies, but similar symptoms
develop in humans with bilateral temporal lesions (Trimble et al., 1997). An animal
may inappropriately try to eat or mate with objects or fail to show customary fear
when confronted with a natural enemy. Human Klver-Bucy patients manifest visual
agnosia and prosopagnosia as well as memory loss, language deficits, and changes
in behavior such as placidity, altered sexual orientation, and excessive eating.
Cases of the human Klver-Bucy syndrome have been reported with bitemporal
damage from surgical ablation, herpes simplex encephalitis, and dementing
conditions such as Pick disease. Patients with Klver-Bucy syndrome appear to have
no major deficits of primary visual perception, but connections appear to be
disrupted between vision and memory and limbic structures, so visual percepts do
not arouse their ordinary associations.