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If the arteries become blocked, autoregulatory mechanisms help maintain cerebral circulation until collateral circulation develops to deliver blood to the affected area. If the compensatory mechanisms become overworked or cerebral blood flow remainsimpaired for more than few minutes, oxygen deprivation leads to infarction of brain tissue. The brain cells cease to function because they can neither store glucose or glycogen for use nor engage in anaerobic metabolism.
If the arteries become blocked, autoregulatory mechanisms help maintain cerebral circulation until collateral circulation develops to deliver blood to the affected area. If the compensatory mechanisms become overworked or cerebral blood flow remainsimpaired for more than few minutes, oxygen deprivation leads to infarction of brain tissue. The brain cells cease to function because they can neither store glucose or glycogen for use nor engage in anaerobic metabolism.
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If the arteries become blocked, autoregulatory mechanisms help maintain cerebral circulation until collateral circulation develops to deliver blood to the affected area. If the compensatory mechanisms become overworked or cerebral blood flow remainsimpaired for more than few minutes, oxygen deprivation leads to infarction of brain tissue. The brain cells cease to function because they can neither store glucose or glycogen for use nor engage in anaerobic metabolism.
Copyright:
Attribution Non-Commercial (BY-NC)
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Als DOC, PDF, TXT herunterladen oder online auf Scribd lesen
There is a deprivation of oxygen and nutrients to Medimap
the tissues of the brain. If the arteries become blocked, autoregulatory mechanisms help maintain cerebral circulation until collateral circulation develops to deliver blood to the affected area. If the compensatory mechanisms become overworked or cerebral blood flow remainsimpaired for more than few minutes, oxygen deprivation leads to infarction of brain tissue. The brain cells cease to function because they can neither store glucose or glycogen for use nor engage in anaerobic metabolism. Signs and Symptoms Diagnostic Tests Unilateral limb weakness Carotid ultrasound – to detect carotid stenosis Speech difficulties CT Scan – to determine cause and location of stroke Numdness on one side Cerebranangiography – to determine extent of Headache Cerebrovascular accident cerebrovacular insufficiency Vision disturbance PET, MRI may be done to localize ischemic damage Dizziness Anxiety Altered level of consciousness Nursing Intervention Treatment Maintain patent airway oxygenation Monitoring and support of vital functions Check vital signs and neurologic status Prevention of decubitus ulcers, thrombosis and pneumonia Maintain fluid and electrolyte balance Rehabilitation Ensure adequate nutrition Physical therapy Manage GI problems Occupational therapy Position the patient and align his extremities correctly Gait training to improve ambulation Assist the patient with exercise Cognitive therapy to improve memory Assess for voluntary or involuntary movements tone of muscles, Speech therapy presence of deep tendon reflexes Drugs Monitor bladder and bowel function Anticoagulant Give medications as ordered Antihyperensive Establish and maintain communication with the patient Diuretics Provide psychological support Anticonvulsants Long term aaspirin to prevent future stroke Surgery Evacuation of hematoma or clot Placement of intracranial pressure monitor Endarterectomy to remove atherosclerotic plaques