Beruflich Dokumente
Kultur Dokumente
ECG
Representation of Electrical activity of heart
ECG Leads
12 lead ECG 6 limb leads: Lead I, II, III
aVL, aVR, aVF
ECG paper
Speed 25mm/s 1 large square= 5 small square [5mm] Voltage 10mm =1mV
Appearance of waves
Positive deflection [upward] If electrical impulses flowing towards that lead
Origin of waves
P wave PR interval
Atrial depolarization
T wave
QT interval U wave
Ventricular repolarization
Ventricular depolarization & repolarization ? Interventricular septal repolarization
Systematic approach
The following 14 points should be analyzed carefully in every ECG: Standardization Heart rate Rhythm P waves PR interval QRS voltages QRS interval QT interval Mean QRS axis Precordial R-wave progression Abnormal Q waves ST segments T waves U waves
Standardization
Heart Rate
1500/RR
If HR is irregular Count no. of QRS complexes in 30 large squares= 6 sec Multiply it with 10 HR [per min]
Rate calculation
Memorize the number sequence: 300, 150, 100, 75, 60, 50
Bradycardia: <60/min
Tachycardia: >100/min
Rhythm
Rhythm strip: prolonged recording of Lead II Sinus rhythm ? Each QRS complex preceded by P wave
Regular/ irregular?
Irregular
QRS AXIS
Indicator of overall direction that wave of depolarization takes when passing through ventricles Also called ANGLE Measured in degrees
Photo
Method 1
Most precise method Use of vectors Measure overall height of QRS in lead I & aVF Plot in graph paper Measure the ANGLE of vector
Method 2
Quick method
Method 3
For quick assessment Look at QRS complexes in lead I & II
Predominantly
positive QRS in
lead I
Axis between
-90 to +90
Excludes RAD
Predominantly positive QRS in lead II Axis between -30 to +150 Excludes LAD
Lead I
QRS Positive QRS Positive
Lead II
QRS Positive
Cardiac Axis
Normal Axis
P wave
Present or not? Sinus rhythm If completely absent
Atrial Fibrillation
Hyperkalemia
If intermittently absent
Sinus arrest
Inverted P waves?
Height of P waves
P PULMONALE
P MITRALE
PR Interval
From start of P wave to start of R wave Normally Not <3 small squares Not > 5 small squares Consistent
Short PR Interval
AV junctional rhythm WPW syndrome
Long PR Interval
Denotes delay in conduction through AV node First Degree Block PR prolonged, constant
Mobitz Type II
PR interval normal & fixed, But occasional P waves fail to produce QRS
Third Degree Block [Complete AV Block] No relationship between P waves & QRS complex
Q WAVE
First negative deflection in QRS complex ? Pathological Q waves If >2 small squares deep >1 small square wide >25% of height of the following R wave in depth
QRS complex
Appearance of QRS Complex vary from lead to lead
?incorrect calibration
Progression of R wave
V1: small R wave , large S wave, Gradually R wave increases, S wave decreases
Progression of R wave
LVH
RBBB
Right Bundle Branch Block Broad QRS complex Small r wave in V1, small Q wave in V6 S wave in V1, R wave in V6 R wave in V1, S wave in V6
LBBB
Left Bundle Branch Block Broad QRS Small Q wave in V1, Small r wave in V6 R wave in V1, S wave in V6
RBBB
Ischemic heart
disease
Cardiomyopathy
ASD
Massive pulmonary
Fibrosis
embolism
ST Segment
From end of S wave to start of T Wave Normally: Isoelectric ? Depressed/ elevated
Elevated ST segment Acute MI Prinzmetals angina Pericarditis LV aneurysm High take off
Depressed ST segment Myocardial ischemia Posterior MI Ventricular hypertrophy with Strain Drugs: Digoxin
T Wave
T wave
Inverted? Normal in aVR V1,V2, III Size Normal: not > size of preceeding QRS complex Too small? Too large?
Tall T waves
Hyperkalemia Acute MI
hypothyroidism
Inverted T waves
Normal in few leads: aVR, V1, V2, III MI Myocardial ischemia Ventricular hypertrophy with strain Digoxin toxicity
QT Interval
From start of QRS complex to end of T wave Varies with HR
Corrected QT interval
QTC QTC =QT/RR Normal: 0.35-0.43 sec
Prolonged QTc
If 0.44 sec Hypocalcemia Acute myocarditis Torsades de pointes
U waves
Mostly in anterior chest leads Difficult to identify clearly
Hyperthyroidism
ACUTE MI
Ischemia
HYPERKALEMIA
PERICARDITIS