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Muscular pathology Hamstring muscle strain Pectoralis minor tightness Tendoachilles rupture Ligament pathology ACL rupture Ankle lateral ligament sprain Dorsal dislocation of PIPJ of hand Pathology from traumatic contact Anterior lower leg contusion Olecranon bursitis
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function
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Aetiology
Risk Factors
Modifiable Risk Factors:
Explosive sports (Ropiak & Bosco, 2012) E.g. Running, kicking, jumping, and hurdling High level of muscle strain High force eccentric contractions Slow-speed stretching (Asking & Tengvar , 2007) E.g. dancing, water skiing and gymnastics Extensive hip flexion and knee extension Hamstring muscles stretched beyond their
[Image] Retrieved from:http://www.coreperformance.com/knowledge/ injury-pain/hamstring-strain.html
Increased neural tension Strength imbalances Poor lumbar spine posture Non-modifiable Risk Factors: Muscle compositions Age Race Previous injuries
(Verrall & Slavotinek, 2001)
limits
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Impingement of L5 nerve root (Orchard & Farhart, 2004) L5 nerve can be easily compressed by
lumbosacral ligament
Correlation between entrapment of L5 nerve
Grade II mild to moderate swelling, pain and tenderness, some loss of Grade III severe pain and immediate swelling, complete loss of
(Gokaraju, Garikipati & Ashwood, 2008)
Recovery time frame Grade I 2 -5 days for tenderness and discomfort to subside Grade 2 Symptoms may last up to a few weeks or a month.
[Image] Retrieved from: http://cspringsphysio.com.au/ conditions/back-pain.html
hamstring strain MRI scans and specific examination procedures can help in differential diagnosis
Management Strategies
Non-operative
(Ali & Leland 2011; Kishan,2008)
RICE (Rest, Ice, Compression, Elevation Therapeutic stretches Massage Exercise NSAIDS (Non-steroidal anti-inflammatory drugs) Operative
(Ali & Leland, 2011)
bruising in the thigh, as this injury causes a persistent and remarkable intramuscular hematoma and edema. (Abebe,
Moorman & Garrett, 2012)
02 weeks: Toe-touch weight bearing (10% of weight) 24 weeks: 25% weight bearing 46 weeks: 50% weight bearing 6+ weeks: weight bearing as tolerated
(Ali & Leland, 2012)
tuberosity
Aetiology
Poor posture over prolonged periods Muscle imbalance (Cortiva, 2006) Dysfunctional breathing Repetitive overuse or stress injury to muscle Trauma (Lewis, & Valentine, 2007)
[Image] Retrieved from: http://thedailyjog.com/ wp-content/uploads/2012/09/bad-computerposture-dude5.jpeg
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Risk factors
Intrinsic risk factors Age Gender More common in women Genetics Anatomical abnormalities Previous injuries (Lewis, & Valentine, 2007) Extrinsic risk factors Desk-based occupations Athletes involved in sports with an overhead action e.g. swimmers, baseball pitchers and rowers (Rondeau, Padua, Thigpen & Harrington, 2009)
[Image] Retrieved from: http://www.drdobbin.co.uk/triathlon-swim-sessions-forautumn-2011
Pathophysiological Processes
Neuroplasticity Poor posture over prolonged periods Muscle imbalance Weak antagonist muscles Altered reciprocal inhibition (Lewis, & Valentine, 2007) Dysfunctional breathing Quiet inspiration using chest/neck muscles
[Image] Retrieved from: rothbartsfoot.es/PectoralisMinor_Syndrome.html
Injury to pectoralis minor e.g. Scar tissue (CliftonSmith, & Rowley, 2011)
Decreased pain Increased pectoralis minor length (Williams, Laudner & McLoda, 2013)
Shoulder impingement Scapular winging Permanent forward head posture Further decline of antagonist muscles Decline in pectoralis minor length Possible development of tightness in neck muscles
i.e. sternocleidomastoid
(Lewis, & Valentine, 2007)
Regain functional use of antagonist muscles Optimized posture Reduced risk for other muscular and joint complications in back region (Williams, Laudner & McLoda, 2013)
Management
Manual therapy Passive gross stretching of
(home or at work)
Use of mirror to correct posture Resistance exercises for
antagonist muscles
i.e. rhomboids,
Williams, Laudner & McLoda, 2013
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Aetiology
Sudden forceful plantarflexion or push-off outside normal ROM (Nandra,
Tendoachilles Rupture
Muscular pathology Group 2a
tendon
Pushing off weight-bearing foot with knee extended Unexpected & violent planterflexion & dorsiflexion
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[Image] Retrieved from http://www.drthefootwithoutthedoc.com/wpcontent/uploads/2012/06/achillerupture.jpg
Risk Factors
Intrinsic factors Poor biomechanics (Nandra, Matharu & Porter, 2011) Age (30-50 years old) Gender (male: female; 10:1) (Brukner & Khan, 2012) Pre-existing conditions (inflammatory & autoimmune diseases
dorsiflexion
2-6cm gap palpable above calcaneal attachment Chronic (4 weeks after injury): Difficulty in activities involving plantarflexion e.g. walking up
Extrinsic factors Recreational athletes (weekend warriors) Use of corticosteroids & fluoroquinolone antibiotics (Hess, 2010) High impact sports i.e. badminton, football, gymnastics, tennis
(Nandra, Matharu & Porter, 2011)
Extreme changes in training, activity level Inappropriate footwear & training techniques (Nandra, Matharu & Porter,
2011)
Differential Diagnosis
Differential Diagnosis
Achilles tendinopathy Inflammation & scar tissue formed as a result of micro tears in
tendon
Cause: overuse Slow onset, with morning pain & stiffness Pain worsens during exercise but daily living activities minimally
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Treatment
Operative Tendons dissected and re-joined via sutures Static splinting in Paris cast in equinus position (20 plantarflexion) 4-6 weeks, gradually moving into plantigrade
Conservative
4 weeks in Paris cast in equinus position Pneumatic walker with heel lift (only when standing & walking) for further 4 weeks Degree of heel lift changed to increase amount of plantarflexion
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Aetiology
Risk Factors
(Papoutsidakis, 2011) Cailliet 1991; Harvard health publications 2010; Micheo et al., 2010; Brukner & Kahn 2012)
Pain (Ingersoll et al., 2008; Petty 2012) Joint tension Subjective experience Instability and knee laxity (Noesberger 1992, Gardinier et al., 2012; Petty
2012)
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Aetiology
calcaneofibular ligament [CFL] and posterior talofibular ligament [PTFL] are the major soft tissue stablisers on the lateral aspect of the ankle (Lynch, 2002)
The ATFL is most commonly injured in a
Risk Factors
stable
GRADE 2: partial tearing of ligament tissue, mild instability of the joint,
joint
(NYU Langone Medical Center, 2013)
Management Strategies
Initial localised pain and swelling due to damage to blood vessels (Orthogate, 2006) Ecchymosis/bruising 24-48 hours later (Lynch, 2002) A rapid decrease in pain and improvement in function in the two weeks
REST: avoid placing pressure on the affected ankle ICE: apply ice or cold pack COMPRESSION: wrap an elastic compression bandage ELEVATION: keep ankle raised above the heart level
(NYU Langone Medical Center, 2013)
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Similar mechanism of injury as a lateral ligament ankle Can occur in conjunction with a ankle sprain but also in
(Judd, 2002)
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Stabilising Factor
Synovial hinge joint
extension
Dorsal dislocation occurs secondary to
Type I (Hyperextension) Type II (Dorsal dislocation) Total volar plate avulsion and complete collateral ligament split Type III (Fracture-dislocation)
(Bindra & Foster, 2009) (Bindra & Foster, 2009)
Laxity of volar plate can lead to hyperextension instability and palmar, dorsal translation (Williams Iv, 2012 )
Clinical Course
Clinical presentation (Gabel, 2008)
Presence of open wound Acute inflammation, deformity
Event:
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Clinical Course
DDx
Direction of dislocation Visually Subjective questioning: obtain symptomatic and psychosocial
(Franko, n.d.)
Management
information
Simple dislocation / Fracture-dislocation Radiography (AP, Lateral, Oblique) Active/Passive ROM Test Dorsally directed stress testing Positive test: Hyperextension deformity
(Chinchalkar & Gan, 2003; Petty, 2011)
Complications
Adhesions Chronic hyperextension PIPJ flexion contracture Mechanical impact on metacarpo-phalangeal joint
(Chinchalkar & Gan, 2003)
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Aetiology
Contusions: commonly known as bruising. (McKeag & Moeller, 2007)
Caused by blunt trauma, does not break the skin. Blood vessels are damaged, resulting in bleeding into the interstitial tissues.
Risk Factors
Subsequent discoloration of the tissue Site of the contusion may also be firm and tender to the touch Restricted movement
Retrieved from http://ennui.org/lj/ leglump.jpg
High Impact Sport Individuals with bleeding disorders Individuals on anti-coagulants Co-morbid conditions
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Takes 2-3 days to fully develop Damaged capillary endothelium releases endothelin, minimizing bleeding Coagulation initiated by von Willebrand factor Discoloration and swelling slowly reduce Change colour from a dark blue or red, to a yellow-green, due to breakdown of
Pressure increase within a muscle compartment Compresses muscles, blood vessels and nerves Develops over a period of hours following a contusion Can lead to permanent injury of the muscle and nerves, or tissue death and amputation
haemoglobin
Lower leg contusions generally heal within 1-2 weeks
If compartment syndrome is suspected, look for the 6 Ps: Pain, parathesia, pallor, paralysis, pulselessness and pressure.
Treatment
RICE
(McKeag & Moeller, 2007)
Rest: Affected limb rested to ensure no further injury. Ice: Apply ice within 24 hours of injury. Compression: May help to reduce swelling in the first 2-3 days. Elevation: To reduce swelling and relieve discomfort. Heat and light massage NSAIDs
Olecranon Bursitis
Traumatic contact pathology Group 2c
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[Image] Retrieved from http:// www.targetmassage.co.uk
Aetiology
[Image] Retrieved from: http://eorif.com/ olecranon-bursitis-72633
Risk factors
Bursitis (Musculoskeletal medicine for medical students, 2013) Injury (American Society for Surgery of the Hand, 2011; Synecal &Leblan, 2001) Pre-existing medical condition (Show us your hands, 2012; Orthoanswer, 2012) Infection (American Society for Surgery of the Hand, 2011; Medical News Today, 2009)
For injury occupations, sport people, some procedures For medical conditions systemic diseases For infection immune compromised people Social determinants (Health People, 2012) physical environment, occupation Individual determinants (Capital Health, 2010) health, patient compliance
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Inflammation process (Loher and Gellman, 2012) Differentiation from osteochondritis dissecans (Harvard Health, 2013; Stubbs, Field,
&Savoie, 2001).
Abebe, E.S., Moorman, C. D., & Garrett, W. E.(2012). Proximal hamstring avulsion injuries: Injury mechanism, diagnosis and disease course. Operative Techniques in Sports Medicine, 20(1), 2-6. doi:http://dx.doi.org/10.1053/j.otsm.2012.03.001 Ali, K., & Leland, J. M. (2012). Hamstring Strains and Tears in the Athlete. Clinics in Sports Medicine, 31(2), 263-272. doi: http://0-dx.doi.org.alpha2.latrobe.edu.au/10.1016/j.csm.2011.11.001.
Conclusion
Question and answer Assessment Strategy http://stm2013-enquiry1.weebly.com
Almekinders, L. & Maffulli, N. (2007). The Achilles Tendon. London: Springer American Academy of Orthopaedic Surgeons. (2011). Elbow (Olecranon) Bursitis. Retrieved from http://orthoinfo.aaos.org/topic.cfm?topic=A00028 American Society for Surgery of the Hand. (2011). Olecranon Bursitis. Retrieved from http://www.assh.org/Public/HandConditions/Pages/Olecranon-Bursitis.aspx Ankle Pain. (2013). Ankle Sprain Symptoms and Categories. Retrieved from http://anklepain.info/ankle-sprain-symptoms-and-categories/ Askling, C, M., Saartok, T. Tengvar, M. Thorstensson, A. (2007). Acute first-time hamstring strains during slow-speed stretching: clinical, magnetic resonance imaging, and recovery characteristics. The American Journal of Sports Medicine.. 35 (10). Retrieved from: http://0-go.galegroup.com.alpha2.latrobe.edu.au/ps/retrieve.do? sgHitCountType=None&sort=RELEVANCE&inPS=true&prodId=EAIM&userGro upName=latrobe&tabID=T002&searchId=R4&resultListType=RESULT_LIST&co ntentSegment=&searchType=AdvancedSearchForm¤tPosition=1&contentSet =GALE|A169614203&&docId=GALE|A169614203&docType=GALE&role= Bahr, R. (2012). The IOC Manual of Sports Injuries: An Illustrated Guide to the Management of Injuries in Physical Activity (1st ed), UK: Wiley-Blackwell. Bahr, R. & Krosshaug, T. (2005). Understanding injury mechanisms: a key component of preventing injuries in sport. British Journal Sports Medicine, 39(6), 324-329
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