PREDISPOSING

Non-Modifiable
AGE (loss of blood vessel
elasticity)
GENDER
FAMILY (hereditary)
Insufficient insulin
Modifiable
secretion/ production of
LIFESTYLE
the pancreas
DIET (high fat, sweet)
Glucose is not metabolize
PRECIPITATING
by the body
Diabetes Mellitus

Accumulation of glucose
in the blood stream

Increase fat deposit Increase viscosity of

on the walls of the blood/ sluggish flow of
blood vessels blood

Narrowing of blood vessel Atherosclesrosis Evaluate level

(coronary artery) of blood
glucose/
Pressure on the wall of blood increase
Decrease blood flow concentration of
through the coronary sugar in blood
S/sx: BP: 140/90 Increase Peripheral Vascular
mmHg resistance
S/sx:
Some fragments joins in circulation CBG
CORONARY ARTERY (EMBOLISM) monitoring:
103-
Goes to small vessels and clog (deep 166mg/dL
Decrease blood
penetrating arteries especially lacunar
flow to the heart
region)

Increase blood Obstruction/ interruption of O2 supply

pressure due to decreased blood supply
HCVD
Lacunar infarct
Severe elevation in BP
w/o progressive target
Decrease oxygen Influences autonomic nervous
supply to basal
Hypertensiv ganglia (L)
e Responsible for cerebellar
INFARCTION function
Prognosis
Also affects motor skills of
chewing & swallowing
(DYSPHAGIA)
If treated: If untreated:
Further Difficulty in chewing &
• Nursing complication swallowing could lead to
Management can occur: decrease appetite
• Diet • Ischemia
Management • Angina Causing weight loss
• Pharmacologic pectoris
Medication: • MI
(Aspirin, ACE • Heart
inhibitor, attack
Nitroglycerin for • CVA
angina)
• Oxygen

Recove Death
ry