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Ectropion


Pathophysiology: Why does the eyelid turn out?






Congenital Ectropion






Congenital ectropion is very rare. When present, it is due to



a vertical shortage of skin. This may be seen in congenital




skin diseases such as icthyosis. In this condition the lower lid




skin keratinization process is faulty which subsequently leads




to tight scaly skin that gradually pulls the lower lid over.




by ○
Blepharophimosis syndrome is a congenital disorder with


David R. Jordan ptosis, phimosis, telecanthus and lateral lower lid ectropion




(secondary to tight lower lid skin). Both conditions are very

M.D., F.R.C.S.(C), F.A.C.S.



rare.


Professor of Ophthalmology


University of Ottawa


Ottawa, ON, Canada

Acquired Ectropion




Involutional Ectropion – is by far the commonest form of



Introduction


ectropion. [Figure 1] The etiology is similar to involutional



entropion with laxity of the medial and lateral canthal tendons



Ectropion refers to an outward turning of the eyelid and may


and thinning of the lower lid retractors (help stabilize the lower

be unilateral or bilateral [Figure 1]. It primarily involves the



lower lid and only very rarely the upper lid. As the eyelid lid in the vertical position). The orbicularis muscle however




does not ride upward as it does in entropion. In ectropion the

turns out, the eye often begins to tear since the punctal


openings move out of position and are unable to collect orbicularis has a normal or decreased tone. In addition ectropic


lids have tarsal plate heights that are greater in vertical height


tears properly. With further outward turning the conjunctiva



becomes red, dry and irritated. If untreated it may cause than normal lids. Thus, in the aging lax lid there is loosening


or thinning of the medial and lateral canthal tendon tissue as



persistent eye irritation, recurrent tearing and discharge.


well as the lower lid retractors. Whether the eyelid turns in or



Etiology




Ectropion may be classified as congenital or acquired.





Congenital ectropion is uncommon. Acquired ectropion



may be divided into involutional, paralytic, cicatricial,





inf lammatory or mechanical types [Table 1].









Table 1 – Ectropion Classification







• very rare

Congenital







Acquired • involutional


paralytic



• cicatricial



inflammatory

• mechanical


Figure 1 – Involutional ectropion of the lower lid.







1


out often depends upon the tone of the orbicularis muscle as





well as the height of the tarsus. A weak orbicularis (e.g. seen



as a result of aging or in 7th nerve palsy) will allow the aging




lid to fall over. A higher tarsal plate in the face of a weak




orbicularis will also have a tendency to fall outward whereas




shorter tarsal plates will have a tendency to turn inward




(providing the orbicularis tone is active – as is the case with



entropion).






Paralytic ectropion – follows temporar y or permanent




seventh nerve palsy. When the orbicularis muscle loses its tone



the lid loses an important support structure and as a result




the lid falls outward. Poor blinking ability and incomplete lid




closure, lead to corneal exposure, tearing, chronic eye




irritation and corneal ulceration.





Cicatricial ectropion – involves a scarring process of the skin



Figure 2 –


with resultant shortening of the skin. It may be seen secondary



Cicatricial


to thermal/chemical burns or trauma [Figure 2].




ectropion –


Inf lammatory ectropion – is associated with inf lammation



secondary to


of the skin as seen in acute allergic reactions to topical drops


or ointment, atopic dermatitis, eczema or occasionally rosacea ○ a scar, pulling


the lid over.



[Figure 3]. Each of these conditions may cause tightening of


the lid skin which may then pull the lid outward. If the process


becomes chronic the eyelid will remain permanently turned





out due to tight lower lid skin. In the early phase however it


may be reversible with anti-inf lammatory steriod creams.





Mechanical ectropion – bulky tumors of the eyelid may pull





the lid over. Similarly, poorly fitting eye glasses sitting on a lax


lid may pull the eyelid outward.









How is Ectropion treated?




Ectropion treatment for the most part is surgical. The only


types that may be amenable to temporizing measures are the





inf lammatory variety and the paralytic form (secondary to


7th ner ve palsy). If there is active inf lammation of the skin





(ex atopic dermatitis, allergy) a steroid cream may settle the


inf lammation and allow the ectropic lid to return to a normal





position. In the 7th nerve palsy patients, the use of lubricating


drops and ointment combined with taping the involved lids




closed at night may be successful in keeping the eye


comfortable for a short time period while working on recovery


of function. If the 7th nerve function recovers the lid should



Figure 3 – Inflammatory ectropion – secondary to severe


return to normal.

atopic dermatitis of the lower lid.




Most ectropions, however, will require surgery. Surgery is




generally done as an outpatient under local anesthesia. The




surgery involves tightening the lower lid and turning the lid


margin back to its normal position. If one eyelid is operated


Complications

on, patients are usually patched overnight. If both eyelids are



Ectropion surgery is successful in over 95% of patients.


involved, cool compresses over each eye rather than eye




patches are used. Antibiotic drops and ointment are required Recurrence occasionally occurs over 2-10 years but is

uncommon. If the ectropion does recur, surgery can always


for about 7-10 days post surgery. Pain is not a major factor.


be done again. Occasionally an infection occurs at the site


Most people will have some mild discomfort in the first one

to two days. Patients can return to their usual daily activities where sutures are placed, oral antibiotics and war m


within 2-3 days. compresses are required if this occurs.






If you have any questions regarding the topics of this newsletter, or requests for future topics of “InSight”, please contact Dr. David R. Jordan
by telephone (613) 563-3800, fax (613) 563-1576 or e-mail at drjordan@magma.ca • Check our Web Site for previous issues of InSight – www.drjordan.on.ca

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