Beruflich Dokumente
Kultur Dokumente
Consultation
3/15/2012
Shock/Sepsis/MODS?
Shock/Sepsis/MODS
Strategies for Early Recognition
facilitates
Understanding
Continuum
the Terms
of Doom
Sepsis
Trauma
SHOCK
Infection
Burns
Drug RX
:::::)
Surgery
SIRS
MODS
SEPSIS
SIRS + Infection
3/15/2012
SIRS: Pathophysiology
Clinical Presentation
51Rs Criteria
- Temp >100
- Hear rate>
90
- RR >20
- WBC > 12,000 or < 4000
Systemic Impact
- Hypoxemia/ARDS
- Decreased
Urinary
Output
- Hypotension
- Hyperglycemia
MODS
SHOCK
"Equal Opportunist"
- Age, sex, race
Early recognition/intervention
patient outcome
critical to
3/15/2012
Stages of Shock
As reflected in the medical record ...
Pathophysiology
of Shock
Progress Note
Date/Time
2/2/0000
[Early]
Re-assessment performed,
0800
condition ...
vss, no change in
[Compensatory]
1420
[Late]
1900
2215
Shock: Types
Cardiogenic
jBP, Temp
Hypovolemic
Distributive
Cardiogenic Shock
Shock
- Anaphylactic
-Septic
- Neurogenic
3/15/2012
Hypovolemic Shock
Pathophysiology of Anaphylaxis
Fluid Loss
Decrease
intravascu ar
volume
Allergen
Decreased
venous
return
Histamine/leukotriene
Reduced Preload
Decreased stroke volume
Decreased
cardiac
output
Vascular dilation
Decreased Perfusion
MAP
Decrease
"'-
/
Smooth muscle
contraction
Decreased
release
Angioedema
Utica ria
ce u ar
enation
Bronchoconstriction
Fluids shifts
Shock
Septic Shock
Neurogenic Shock
Injury/Insult
Spinal Shock
Flaccid Paralysis
MODS
3/15/2012
Respiratory Distress/Failure
Clinical Picture
cv
120/78
110/70
88
82
100/70
90
100/50
100
88/62
100
RESP
14/96%
14/94%
16/90%
24/90%
24/90%
NEURO
A&O
A&O
"sleepy"
"drowsy"
Hypoxemia
Hypercapnia
hypotension,
cyanosis.
Respiratory
"lethargic"
A&O X 3
RENAL
1000/
1200/400
GI
BS +
BS +
INTEGU-
WNL
WNL
MENTARY
ACidosis
Increased work
of breathing
Dyspnea, exhaustion
2000/500
BS-
Facial
Pale, cool
Flushing
as
of SIRS/Sepsis/Shock
Shock/MODS:
Management
Medical
Management
signs of sepsis
Can be primary
or secondary
Physiological
Characterized
by hypoperfusion
- Symptoms of shock
- Bleeding
- Mental status changes
- Decreased urinary output
- Labs: hypoxia/hypoxemia,
liver enzymes/creatinine,
prolonged pT/pn
insults
are triggers
Normal saline
Lactated Ringers
Drug Therapy
Vasopressors
Sodium Bicarb
Antibiotics
Steroids
Insulin
3/15/2012
pcollins@consultationoncall.com
perfusion)
perfusion]
Nutrition
- Enteral/Parenteral
Labs: *monitoring
and interpretation
References
1. AI-Khafaji, Ali (2010). Multisystem Organ Failure of Sepsis. Emedicine. Retrieved June
15, 2010 from http://emedicine.medscape.com/article/169640
.
2. Burdette, S.D., Parilo, M.A., Bailey, H. (2010). Systemic Inflammatory Response
Syndrome. Emedicine. Retrieved June 15,2010 from
http://emedicine.medscape.com/article/
168943.
3. Chulay, M., Bums, S. M. (2010). AACN: Essentials of Progressive Care Nursing, 2nd Ed.
McGraw Hill, Inc.
4. Chulay, M., Bums, S. M. (2010). AACN: Essentials of Progressive Care Nursing, 2nd Ed.
McGraw Hill, Inc.
5. Keller, J. (2005). The Incredible Impact of a Smile. Retrieved September 5, 2005 from
http://asia.groups.yahoo.com/ group/ha1l8 cheer/message/55.
6. Lewis, S. et al (2007). Medical-Surgical Nursing, Assessment and Management of
Clinical Problems. St. Louis, Mo: Mosby.
7. Lewis, S. et al (2007). Medical-Surgical Nursing, Assessment and Management of
Clinical Problems. St. Louis, Mo: Mosby.
8. Lippincott Manual of Nursing Practice Series, Documentation (2007).
29