Alzheimer's Disease

Written by Lyle MacWilliam

Wednesday, 16 March 2011 20:08 - Last Updated Friday, 08 April 2011 16:10

Alzheimers Disease

and the Protective Effects of Nutrients

scientific research summaries

To assist those readers searching for scientific evidence supporting the use of natural ways to
prevent the onset of senile dementia, NutriSearch has compiled a series of research reports on
nutrients and Alzheimers disease. Each report is a compilation of research summaries from
scientific studies gleaned from leading scientific and medical journals around the world. These
research abstracts provide hard scientific evidence of the power of supplementation in disease
prevention.

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Alzheimer's Disease
Written by Lyle MacWilliam
Wednesday, 16 March 2011 20:08 - Last Updated Friday, 08 April 2011 16:10

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Alzheimers disease (AD) is the leading cause of dementia in the elderly and the fourth leading
cause of non-accidental death after heart disease, cancer and stroke. In the United States, five
percent of the population over 65 suffers from severe dementia and a further ten percent suffer
from mild-to-moderate dementia, the majority of which is expressed as AD.

The incidence of this dreaded disease doubles every five years after the age of 60. Up to
two-thirds of those in their 90s havesome level of AD, a disease that is currently incurable. The
mean life expectancy following diagnosis is around seven years.In 2006, there were 26.6
million people affected worldwide; the disease is projected to affect 1 in 85 people globally by
2050. 1

Alzheimers disease attacks the nerves that form the memory centers of the brain, deep within
the cerebral cortex. Characterized by a progressive loss of short-term memory, dramatic
personality changes, disorientation and an inexorable decline of body functions, death comes
not from the disease but with it. As it progresses, AD robs its victims of their memory, their
identity and their dignity. While current therapies cannot halt AD, they can slow its progress.

A Protein Folding Disease

In essence, Alzheimers disease is a consequence of a malfunction in the synthesis of protein

structures critical to the memory-forming functions of the neuron. In fact, most neurologists now
contend that the deposition of damaged beta amyloid proteins may be the factor most
responsible for kick-starting

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Alzheimer's Disease
Written by Lyle MacWilliam
Wednesday, 16 March 2011 20:08 - Last Updated Friday, 08 April 2011 16:10

2
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level
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Environmental
Triggers
is
Several
toxins,
development
the
long-term
increase
a brain.
causative
factors,
the
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consumption
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body
of
factor
AD.
including
burden
to
It
or
is
aluminum
ainduced
of
known
consequence
severe
antacids,
of
aluminum;
that
oxidative
through
head
food
AD
trauma,
of
however,
sufferers
stabilizers
drinking
damage
the
disease
exposure
uniformly
whether
water,
to
and
brain
is
non-dairy
still
antiperspirants,
to
accumulation
tissues
express
aluminum
an
open
creamers
have
high
question.
and
been
of
levels
cookware
other
the
is
implicated
known
metal
of
environmental
aluminum
and
into
the
the
inThis
brain
in If

3
Protection
through
Diet
vegetables,
collectively
consumption
to
whole
of
reduce
red
grains,
the
is
risk
The
olive
associated
of
components
AD.
fish
Supporting
with
and
of
reduced
red
the
wine,
evidence
Mediterranean
risk
have
of
confirms
been
the
disease.
found
diet,
that
which
individually
light
to
includes
moderate
and
fruit
and
In
of
vitamins.
leading
multi-nutrient
the
elderly,
North
to
In
the
fact,
American
deficiency.
cognitive
postulate
many
adult
cases
function
population
AD
chronic
may
is
directly
simply
deficient
nutrient
related
be
the
deficiency
in
one
end
nutritional
or
game
more
mimic
of
status,
of
ain
the
the
chronic
symptoms
critical
with
sub-clinical
athe
significant
B-complex
of
Alzheimers,
portion
Studies
folic
neurotransmitter,
acetylcholine
have
acid
been
show
can
documented
each
in
that
the
acetylcholine,
nutrient
produce
brain.
with
Positive
deficiencies
AD-like
thiamine
involved
clinical
symptoms.
in
in
vitamin
results
memory,
In
E,
in
fact,
improving
magnesium,
and
thiamine
can
potentiate
memory
(vitamin
vitamins
retention
the
B1)
B1,
effects
mimics
B3,
in
and
AD
ofthe
B12,
patients
critical
and
Summary
Alzheimers
consequence
consequently,
and
For
includes
exposures,
natural
plant-based
disease
these
exposure
health
process
reducing
reasons,
consumption
disease
anti-inflammatory
of
to
products,
prevention
our
environmental
before
the
itwine
rapidly
promises
is
bodys
fundamental
itthat
including
of
can
remains
a
aging
toxic
low
take
nutrients.
tooil,
toxins
become
glycemic
vitamins,
demographic
burden
paramount.
root.
for
all
everyone
After
Together,
play
the
through
and
minerals,
next
all,
fundamental
Oxidative
largely
profile.
its
to
global
fresh
these
consider
all
antioxidants,
vegetarian
Once
about
air,
stress,
measures
epidemic
roles
regular
established,
appropriate
choice.
chronic
diet,
and
in
the
can
exercise,
the
aetiology
and
the
help
systemic
coming
lifestyle
AD
wide
staunch
avoidance
is
judicious
incurable;
variety
decades,
inflammation
changes.
the
the
disease.
of
of
use
a
toxic
This
of
The
by
provide
Alzheimer's
leading
research
evidence
scientific
disease:
reports
supporting
and
listed
medical
below
the
use
journals
are
of
asupplementation.
selectednutrients
compilation
throughout
of
the
summaries
world.
in
the
These
prevention
from
research
recent
and
studies
abstracts
mitigation
published
of
2nd
Reference
(1)
http://en.wikipedia.org/wiki/Alzheimer's_disease.
(2)
Treatment.
(3)
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NOW!
Alzheimer's
Murray
ed.
Roseville,
Expanded
List
MT,
Pizzorno
disease.
Disease.
CA:
3rd
Prima
wikipedia
ed.
JP.
In:
Publishing;
Segala
Hollywood,
Alzheimer's
org
M,
2011;Available
editor.
1998.
FL:
Disease.
Life
Life
p.
Accessed
Extension
221-31.
Extension
Encyclopedia
at:
URL:
February
Media;
Disease
of
2000.
Natural
18,
Prevention
2011.
p.
Medicine.
27-33.
and
Revised

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