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1.
JUN
11
one scorer. Definitions 1) Boundaries:Lines on the four sides of the playing field
are called boundaries. These lines should
not be more than 5 cm wide and form part
of the playing field. 2) Lobbies:- Each strip
measuring 1 metre in width on the sides of
the playing field is called lobby. 3) Baulk
Line:- Each line parallel to the centre or
middle line drawn at a specified distance
from it is called a baulk line. 4) Cant:Repeated sounding of word Kabaddi in
one breathe is called cant. 5) Raider:- A
player who enters the opponent's court
with cant is called a raider. 6) Anti:- A
player in whose court raid is made is
called an anti. 7) Raid:- When a raider
enters the court of opponent with cant, a
raid is made. 8) Struggle:- When an anti
touches or holds a raider or raider touches
an anti, a struggle begins. 9) Successful
external examiner
ACKNOWLEDGEMENT
I would like to express my sincere gratitude to my Physical
Education teacherMr. Shashi Bhushan Sharma Sir for his
vital support, guidance and encouragement without which this
project would not have come forth from my side.
Special thanks of mine goes to my colleagues
Ujjwal Kr. & Md. Arman who helped me a lot in completing
the project by giving interesting ideas, thoughts & made this
project easy and accurate. I wish to thanks my parents for
their undivided support & interest who inspired me &
encouraged me to go my own way, without which I would be
unable to complete my project. And at last but by no means
the least I would thank to God who made all the things
possible.
INTRODUCTION
The word Kabaddi is derived from the Tamil, Kannada,
and Malayalam word "kai"(hand), "pidi"(catch), which
could be translated into "Holding Hands."
Two teams occupy opposite halves of a field and take
turns sending a "raider" into the other half, in order to
win points by tackling members of the opposing team.
The raider then tries to return to his own half, holding his
CONTENTS
HISTORY OF KABADDI
FORMS OF KABADDI
o Amar
o Sanjeevni
o Gaminee
KABADDI GROUND
RULES OF KABADDI
o Kabaddi ground
o Rules of play
o Rules of matches
SOME DEFINITIONS
FUNDAMENTAL SKILLS OF THE GAME
o Raiding Skills
o Defensive Skills
KABADDI RELATED TERMINOLOGIES
IMPORTANT TOURNAMENTS
IMPORTANT PERSONALITIES
ARJUNA AWARD WINNER
History
The origin of Kabaddi can be traced to pre-historic times when man learned
how to defend in groups against animals or attack weaker animals individually
or in groups for survival and food. Though kabaddi is primarily a South Asian
game, it has originated from Punjab (India). There is however concrete
evidences that the game is over 4,000 years old. In India, kabaddi was played in
a very crude form. Its followers in Satara tried to refine this game in 1918.
Rules of this game were framed in the beginning of the 20th century by the
Deccan Gymkhana. In 1923, Hind Vijay Gymkhana, Baroda, published the rules
of this game and an All India Competition was held in the same year in Baroda
in which these rules were applied. In 1934, Akhil Maharashtra Sharirik Parishad
published the revised rules of this game.
The state of Maharashtra took keen interest in this game but Kabaddi obtained
national status only in 1952, when the National Kabaddi Federation of India
was formed. It organized the first National Kabaddi Championship in that year.
In the year1955, competitions for women were also included in the national
championship held at Calcutta.
Although kabaddi is a simple game and can be played in a small area without
any equipment, it has not become very popular India. Only Bangladesh,
Pakistan,
Sri Lanka and Myanmar play this game.
Forms of Kabaddi
Amar
In the 'Amar' form of Kabaddi, whenever any player is touched (out), he does not go out of the court, but
stays inside, and one point is awarded to the team that touched him. This game is also played on a time
basis, i.e. the time is fixed. This form of Kabaddi is played in Punjab, Canada, England, New Zealand, USA,
Pakistan and Australia. In the Amar form of Kabaddi, each team consists of 56 stoppers and 45 raiders.
At one time, only 4 stoppers are allowed to play on the field. Every time a stopper stops the raider from
going back to his starting point, that stoppers team gets 1 point. On the other hand, every time the raider
tags one of the stoppers and returns to his starting point, his team gets one point. At one time, only one of
the stoppers can try. The dimensions are 10m x 12.50m.
Sanjeevni
In Sanjeevni form of Kabaddi, one player is revived against one player of the opposite team who is out, one
out, one in. The duration, the number of players, dimensions of the court, etc. have been fixed by the
Kabaddi Federation of India. This form of Kabaddi is the closest to the present game. In this form of
Kabaddi, players are put out and revived and the game lasts 40 minutes with a 5-minute break in between.
There are nine players on each side. The team that puts out all the players on the opponent's side scores
four extra points for a 'Lona'. The winning team is the one that scores most points after 40 minutes. The
field is bigger in this form of Kabaddi and the 'chant' is diferent in various regions. Modern Kabaddi
resembles this form of Kabaddi especially with regard to 'out & in system' and 'Lona'. The present form of
Kabaddi is a synthesis of all these forms with change in the rules.
Gaminee
This is played with nine players on either side, in a field of no specific measurements. The characteristic is
that a player put out has to remain out until all his team members are out. The team that is successful in
putting out all the players of the opponent's side secures a point. This is akin to the present system of
'Lona'. After all the players are put out, the team is revived and the game continues. The game continues
until five or seven 'Lona' are secured. This form of Kabaddi has no fixed game time. The main disadvantage
of Gaminee is that the player is not in position to give his best performance since he is likely to remain out
for the better part of the match until a Lona is scored.
Kabaddi Ground 1
Rules of Kabaddi
Kabaddi ground:Kabaddi is played on a soft ground, rectangular in shape. The ground should measure 12.50
metres for men. In case of women and men below 50 Kg weight, it should measure 11 metres
by 8 metres. Each ground should be divided by a middle line into two halves. Other markings
should be marked as under:a) A strip called Lobby, 1 metre in width, should be marked on the sides of the play ground.
b) A Baulk Line should be drawn through the entire width in each half of the court, at a distance
of 3.75 metres in case of men and 3 metres in case of women and men under 50 Kg weight.
The Baulk Line should not be extended into the Lobbies.
All lines marked in the playing field should not be more than 5 cm in width.
Rules of play:1) The side winning a toss has the right to decide about the choice of the court (Side) or the
Raid.
In the second half, the courts should be changed and the game should be resumed by the
side that had not sent their raider first at the start of the match. The game should be resumed
with the same number of players that were playing at the end of the first half.
2) A player gets out if any part of his body touches the ground outside the boundaries.
However, during a struggle, a player I not out if any part of his body touches the
ground or the player inside the boundaries.
3) A player is declared out if he goes out of the playing field during play.
4) The playfield includes the Lobbies when struggle begins. The players, who enter a Lobby
during a struggle, can enter their courts, by using the Lobbies, after struggle.
5) The word Kabaddi should be used as Cant.
6) A raider should start the cant before he touches the opponents court.
7) When a raid is over or a raider is out in the opponents court, the opponent should send their
raider within 5 seconds.
8) A raider, who escapes to his court after an attempt to hold him, should not be pursued.
9) A raider losing his cant in the opponents court shall be out. If a raider is held by his
opponents, his cant should not be stifled.
10) Only one raider should enter the opponents court at a time. If more than one raider enters
the opponents court, then the side loses their turn of raiding and any opponent touched in
such a raid is declared not out.
11) A raider or anti should not push an opponent out of the boundary intentionally.
12) Antis are allowed to start a raid only when a raider has reached his own court.
13) A raider or an anti should not be held by any part of his body other than his limbs and trunk.
14) Payers should be revived in the same order in which they were put out.
15) When a team puts out all the opponents, it scores a Lona and two additional points are
awarded to this team.
16) When one or two players of a team are left on the court, the captain of that team, in order to
get the full team on court, may declare them out. In such a case the opponent score as many
points as the number of players left in addition to the points won for a Lona.
Rules of matches:1) A Kabaddi team consists of 12 players, out of which 7 form the playing team and the
remaining 5 serves as reserves.
2) The duration of a match should be two halves of 20 minutes each for men and 15 minutes
each for of women and men below 50 Kg weight. The interval for rest between halves should
be 5 minutes. The side that scores the highest number of points at the end wins the
match.
3)
4)
5)
6)
7)
In case of a tied score at the end of two halves, two extra periods of 5 minutes duration
should be played immediately with the same number of players in the court at the end of
second half. If the tie is still not broken then the team that scores the first leading point at the
end of these 2 extra halves, wins the match.
The toss should decide about the choice of raid or court by a team.
One point is awarded to a team for every opponent put out. A team that puts out the whole
team, scores 2 extra points for a Lona.
A maximum number of 3 players can be substituted with the permission of a referee. A
substituted player cannot be replaced.
A team may start a match with one or two players less in team but when all the players of the
side are out, the absentees shall also be counted as out and Lona be scored against that
team.
A match should be supervised by at least two umpires and one scorer.
Definitions
1) Boundaries:Lines on the four sides of the playing field are called boundaries. These lines should not be
more than 5 cm wide and form part of the playing field.
2) Lobbies:Each strip measuring 1 metre in width on the sides of the playing field is called lobby.
3) Baulk Line:Each line parallel to the centre or middle line drawn at a specified distance from it is called a
baulk line.
4) Cant:Repeated sounding of word Kabaddi in one breathe is called cant.
5) Raider:A player who enters the opponent's court with cant is called a raider.
6) Anti:A player in whose court raid is made is called an anti.
7) Raid:When a raider enters the court of opponent with cant, a raid is made.
8) Struggle:When an anti touches or holds a raider or raider touches an anti, a struggle begins.
9) Successful Raid:A successful raid is one in which the raider crosses the baulk line of the defenders at least
once during the raid and reaches his own court safely with cant.
10) Touch:It means a contact made by a raider or anti with clothing, shoes or body of an opponent.
11) Holding a Raider:If the anti or antis hold a raider, without breaking the rules and do not allow him to reach his
own court with the cant, it is called holding a raider.
12) Bonus Line:A line at a distance of 1 metre from the baulk line towards the end line is called a bonus line.
One point is awarded to the raider for crossing this line. If the antis are 5 or less than 5, the
bonus line rule does not apply. Moreover, there is no revival for a bonus point.
i)
One point is awarded to the raider as soon as he crosses the bonus line. If the raider is
caught after crossing this line, he shall be out and opponents shall be awarded a point. One
point will also be awarded to the raider for crossing the bonus line.
ii)
If the antis are 5 or less than 5 then bonus line rule does not apply.
iii)
There will be no revival for a bonus point.
Raid Structure
e)
f)
g)
h)
vi)
vii)
viii)
1)
a)
b)
c)
d)
e)
f)
g)
h)
2)
a)
b)
3)
a)
b)
c)
Aero kick
Swing kick
Mule kick
Toe kick
Withdrawal
Jumping
Falling
Defensive Skills:Holding
Wrist catch
Over the shoulder catch
Trunk catch
Wrist and ankle catch
Singe knee catch
Double knee catch
Ankle catch
Dive and catch
Offensive tactics
Starting raid from one side
Employing pivoting & dodging
Defensive tactics
Chain formation
Pursuing
No cross play
Sometimes, a raider, for safety reasons has to withdraw from attack and pull him back. It is
an important aspect of game plan. Withdrawing is also resorted to in trying to catch a raider.
9) Chain formation:In order to strengthen the defense, the defenders very often hold their hands to catch a raider.
This holding of hands is called chain formation.
10) Guarding:This term refers to the backing provided by the team mates when a defender is cornered or
when an attacker tries to penetrate into the defenders court. This is usually done by forming a
chain and coming around for help from the back.
11) No cross play:This is a game strategy when the defenders try to stop the raider on the baulk line itself and
try to prevent him from crossing the same.
12) Escape:An attempt by a raider to get out of the hold of antis.
Ashan Kumar
Biswajit Patil
B.C. Ramesh
Ram Mehar Singh
Sanjeev Kumar
Sundar Singh
:
:
:
:
:
:
1998
1998
2001
2002
2003
2004
Add a comment
2.
JUN
11
note-- put image wherever required. u have the images down below.
Date of Submission:
(i)
(ii)
..........................................................................
ACKNOWLEDGEMENT
I would like to express my sincere gratitude to my chemistry
mentorsMr.Ram Ratan Singh & Dr.Subhash Sir for their
vital support, guidance and encouragement without which this
project would not have come forth from my side.
I would also like to express my heartly
gratitude to the lab assistants Mr.Ritesh & Mr.Pramod for
their support during the making of this project.
Special thanks of mine goes to my colleagues
Ujjwal Kr. who helped me a lot in completing the project by
giving interesting ideas, thoughts & made this project easy
and accurate. I wish to thanks my parents for their undivided
support & interest who inspired me & encouraged me to go
my own way, without which I would be unable to complete
my project. And at last by no means the least I would thank to
God who made all the things possible.
..................................................................................................
Adulterants in Food
put word art named 'CERTIFICATE'
Name: Azizur Rahman
Class: XII-B
School: D.A.V. Public School, WALMI Complex
Year: 2011-2012
Certified to be the bonafide work done by
Mr. / Miss AZIZUR RAHMAN of class ______________XII B____________
in the________CHEMISTRY LAB_____________ during the year 2011-2012
Date____________________________________ P.G.T. in
CHEMISTRY
D.A.V. PUBLIC SCHOOL, Walmi Complex, Patna-801505
CONTENTS
S.No.
I.
II.
III.
IV.
V.
VI.
VII.
VIII.
IX.
X.
XI.
XII.
XIII.
XIV.
Contents
Introduction to project
Students Info
Acknowledgement
Certificate
Content
Objective
Theory
Experiment 1
Experiment 2
Experiment 3
Observetion Table
Conclusion
Bibliography
Image
II Page No.
1
2
3
4
5
6
7
8
9
10
11-12
13
14
15
(v)
.................................................................................................................................................
OBJECTIVE
The Objective of this project is to study some of the common food adulterants
present in different food stuffs.
Adulteration in food is normally present in its most crude form; prohibited
substances are either added or partly or wholly substituted. Normally the
contamination/adulteration in food is done either for financial gain or due to
carelessness and lack in proper hygienic condition of processing, storing,
transportation and marketing. This ultimately results that the consumer is either
cheated or often become victim of diseases. Such types of adulteration are quite
common in developing countries or backward countries. It is equally important
for the consumer to know the common adulterants and their effect on health.
...................................................................................................................................................
THEORY
The increasing number of food producers and the outstanding amount of import foodstuffs
enables the producers to mislead and cheat consumers. To differentiate those who take
advantage of legal rules from the ones who commit food adulteration is very difficult. The
consciousness of consumers would be crucial. Ignorance and unfair market behavior may
endanger consumer health and misleading can lead to poisoning. So we need simple
screening, tests for their detection.
In the past few decades, adulteration of food has become one of the serious problems.
Consumption of adulterated food causes serious diseases like cancer, diarrhea, asthma, ulcers,
etc. Majority of fats, oils and butter are paraffin wax, castor oil and hydrocarbons. Red chilli
powder is mixed with brick powder and pepper is mixed with dried papaya seeds. These
adulterants can be easily identified by simple chemical tests.
Several agencies .have been set up by the Government of India to remove adulterants from
food stuffs.
AGMARK acronym for agricultural marketing.this organization certifies food products
for their quality. Its objective is to promote the Grading and Standardization of agricultural
and allied commodities.
......................................................................................................................................................
.
EXPERIMENT 1
AIM:
To detect the presence of adulterants in fat, oil and butter.
REQUIREMENTS:
Test-tube, acetic anhydride, conc. H2SO4, acetic acid, conc. HNO3.
PROCEDURE:
Common adulterants present in ghee and oil are paraffin wax, hydrocarbons, dyes and
argemone oil. These are detected as follows:
(i ) Adulteration of paraffin wax and hydrocarbon in vegetable gheeHeat small amount of vegetable ghee with acetic anhydride. Droplets of oil floating on the
surface of unused acetic anhydride indicates the presence of wax or hydrocarbons.
(ii) Adulteration of dyes in fatHeat 1mL of fat with a mixture of 1mL of conc. sulphuric acid and 4mL of acetic acid.
Appearance of pink or red colour indicates presence of dye in fat.
(iii) Adulteration of argemone oil in edible oils-
To small amount of oil in a test-tube, add few drops of conc. HNO3 and shake. Appearance of
red colour in the acid layer indicates presence of argemone oil.
......................................................................................................................................................
........
EXPERIMENT 2
AIM:
To detect the presence of adulterants in sugar.
REQUIREMENTS:
Test-tubes, dil. HCl.
PROCEDURE:
Sugar is usually contaminated with washing soda and other insoluble substances which are
detected as follows :
(i) Adulteration of various insoluble substances in sugar
Take small amount of sugar in a test-tube and shake it with little water. Pure sugar dissolves
in water but insoluble impurities do not dissolve.
(ii) Adulteration of chalk powder, washing soda in sugar
To small amount of sugar in a test-tube, add few drops of dil. HCl. Brisk effervescence of
CO2 shows the presence of chalk powder or washing soda in the given sample of sugar.
To detect the presence of adulterants in samples of chilli powder, turmeric powder .
.............................................................................................................................................
EXPERIMENT 3
AIM:
To detect the presence of adulterants in chilli powder, turmeric powder and pepper.
REQUIREMENTS:
Test-tubes, conc. HCl, dil. HNO3, KI solution
PROCEDURE:
Common adulterants present in chilli powder, turmeric powder and pepper are red coloured
lead salts, yellow lead salts and dried papaya seeds respectively. They are detected as follows:
(i) Adulteration of red lead salts in chilli powder
To a sample of chilli powder, add dil. HNO3. Filter the solution and add 2 drops of potassium
iodide solution to the filtrate. Yellow ppt. indicates the presence of lead salts in chilli powder.
(ii) Adulteration of yellow lead salts to turmeric powder
To a sample of turmeric powder add conc. HCl. Appearance of magenta colour shows the
presence of yellow oxides of lead in turmeric powder.
(iii) Adulteration of brick powder in red chilli powder
Add small amount of given red chilli powder in beaker containing water. Brick powder settles
at the bottom while pure chilli powder floats over water.
(iv) Adulteration of dried papaya seeds in pepper
Add small amount of sample of pepper to a beaker containing water and stir with a glass rod.
Dried papaya seeds being lighter float over water while pure pepper settles at the bottom.
.....................................................................................................................................................
OBSERVATION TABLE
PROCEDURE
EXPERIMENT
Adulteration of
paraffin wax and
hydrocarbon in
vegetable ghee
Adulteration of dyes
in fat
Adulteration of
argemone oil in edible
oils
Adulteration of
various insoluble
substances in sugar
Adulteration of chalk
powder, washing soda
in sugar
Adulteration of
yellow lead salts to
turmeric powder
Adulteration of red
lead salts in chilli
powder
Adulteration of brick
powder in chilli
powder
Adulteration of dried
papaya seeds in
Pepper
OBSERVATION
Appearance of oil
floating on the
surface.
Appearance of pink
colour.
No red colour
Observed.
Pure sugar
dissolves in water
but insoluble
impurities do not
dissolve.
No brisk
effervescence
observed.
Appearance of
magenta colour.
No yellow ppt.
..........................................................................................................................................
CONCLUSION
Selection of wholesome and non-adulterated food is essential for daily
life to make sure that such foods do not cause any health hazard. It is
not possible to ensure wholesome food only on visual examination
when the toxic contaminants are present in ppm level. However,
visual examination of the food before purchase makes sure to ensure
absence of insects, visual fungus, foreign matters, etc. Therefore, due
care taken by the consumer at the time of purchase of food after
thoroughly examining can be of great help. Secondly, label
declaration on packed food is very important for knowing the
ingredients and nutritional value. It also helps in checking the
freshness of the food and the period of best before use. The consumer
should avoid taking food from an unhygienic place and food being
prepared under unhygienic conditions. Such types of food may cause
various diseases. Consumption of cut fruits being sold in unhygienic
conditions should be avoided. It is always better to buy certified food
from reputed shop.
(xiii)
............................................................................................................
..........
BIBLIOGRAPHY
1) ENCARTA ENCYCLOPEDIA 2009/2010/2011
2) www.wikipedia.com
3) www.answers.com
4) www.icbse.com
5) www.google.com
6) GUTENBERG e-BOOK
(xiv)
View comments
3.
JUN
11
external examiner
Date: -
ACKNOWLEDGEMENT
I would like to express my sincere gratitude to my chemistry mentors Mr. K.K.Pandey
Sir, Deptt. of Biology for his vital support, guidance and encouragement without which this
project would not have come forth from my side.
I would also like to express my heartily
gratitude to the lab assistant Mr. Ritesh for his support during the making of this project.
Special thanks of mine goes to my
colleague Ujjwal Kr. and Md. Arman who helped me a lot in completing the project by giving
interesting ideas, thoughts & made this project easy and accurate. I wish to thanks my parents
for their undivided support & interest who inspired me & encouraged me to go my own way,
without which I would be unable to complete my project. And at last but by no means the
least I would thank to God who made all the things possible.
INTRODUCTION
In the present 21st century world, one does not need to say about ultra violet rays, it is the
most ignited topic in this era. From the common man to the Academicians, UV rays hold an
important place in their debate.
UV rays have been discussed for a long time all around the world especially after the discovery of the hole
in the ozone layer in 1990s.The hole meant the approaching slow death of our mother earth. A host of
studies on this topic has been undertaken by many people and organizations such as the United Nations,
Green Peace, etc.
At the United Nations Conference on Environment and Development (UNCED) in
1992, it was declared under Agenda 21 that there should be activities on the efects of UV radiation. In
response to Agenda 21, WHO in collaboration with the United Nations Environment Programme, the World
Meteorological Organization, The International Agency on Cancer Research and the International
Commission on Non-Ionizing Radiation Protection set up Intersun, The Global UV Project.
Even when the whole world only speaks about this great calamity that has befallen on our
mother earth, it would surely be a great injustice on part of the young generation as this slow
killer is growing old with the young generation and they are the one who should live on this
earth with this growing menace. Thus taking into consideration of these points, this project
has been taken up not only to know about UV rays but also how it affects the bio diversity of
our planet and how its effect can be minimized.
Here, this project deals with the above mentioned points. Also the project aims to give a
comprehensive picture on the impending disaster that would take place if we be ignorant on
this issue.
CONTENTS
1) ULTRAVIOLET RAYS
1.1) Sources of UV rays
1.2) UV-A, UV-B & UV-C
2) EFFECTS OF UV RADIATION ON NATURE
2.1) Damage to marine life
2.2) Degradation of polymers, pigments and dyes
3) HARMFUL EFFECTS ON HUMANS
3.1) Effects on Eye
3.2) Effects on Skin
3.3) Genetic effects
3.4) Effects on immune system
Abbreviation
Wavelength range in
nanometers
Energy per
photon
Many polymers used in consumer products are degraded by UV light. The problem appears
as discoloration or fading, cracking and sometimes disintegration of total product, if cracking
has proceeded sufficiently. The rate of attack increases with exposure time and sunlight
intensity. It is known as UV degradation, and is one form of polymer degradation. Sensitive
polymers include thermoplastics, such as polypropylene and polyethylene as well as
special fibres like Aramids.
There are several other considerations:
Ultraviolet levels are over 1,000 times higher at the equator than at the Polar
Regions so it is presumed that marine life at the equator is much better adapted to the higher
environmental UV light radiation than organisms in the Polar Regions. The current concern
of marine biologists is mostly over the more sensitive Antarctic phytoplankton which
normally would receive very low doses of UV. Only one large-scale field survey of Antarctic
phytoplankton has been carried out so far [Smith et.al _Science_1992]; they found a 6-12%
drop in phytoplankton productivity in Antarctica. Since the hole only lasts from 10-12weeks,
this translates into a 2-4% loss overall, a measurable but not yet a catastrophic loss.
Both plants and phytoplankton vary widely in their sensitivity to UV-B. When
over 200 agricultural plants were tested, more than half of them showed sensitivity to UV-B
light. Other plants showed a small increase in vigor or even negligible effects. Even within a
species there were marked of differences. For example, one variety of soybeans showed a
16% decrease in growth while another variety of the same soybean showed no effect [R.
Parson]. An increase in UV-B could cause a shift in population rather than a large die-off of
plants.
increased UV-B exposure. But unrepaired genetic damage of the DNA can lead to skin
cancers. In one common damage event, adjacent thymine bases bond with each other, instead
of across the ladder. This makes a bulge, and the distorted DNA molecule does not function
properly.
EFFECTS ON SKIN
Ultraviolet (UV) radiation present in sunlight is an environmental human
carcinogen. The toxic effects of UV from natural sunlight and therapeutic
artificial lamps are a major concern for human health. The major acute effects of
UV irradiation on normal human skin comprise sunburn, inflammation,
erythema, tanning and local or systemic immune-suppression.
1. Sunburn:
One of the most common effects of UV exposure is erythema also known as sunburn.
Sunburn occurs when skin cells are damaged by the absorption of energy from UV rays. To
compensate for this injury, the skin sends extra blood to the damaged skin in an attempt to
repair it. Thus, accounting for the redness that is associated with sunburn, the amount of time
it takes for sunburn to occur is dependent mostly on the relative amounts of UV rays that are
hitting the skin and on a persons skin type. People with naturally dark skin already have
inherently high levels of melanin, and so are able to spend a longer amount of time in the sun
before burning. Fair-skinned people dont have it quite so easy as burning can occur within a
relatively short amount of time.
2. Sun tan:
As a
defense against UV radiation, the amount of the brown pigment melanin in the skin increases
when exposed to moderate (depending on skin type) levels of radiation. This is commonly
known as sun tan. The purpose of melanin is to absorb UV radiation and dissipate the energy
as harmless heat, blocking the UV from damaging skin tissue. UV-A gives a quick tan that
lasts for days by oxidizing melanin that was already present and triggers the release of the
melanin from melanocytes. UV-B yields a tan that takes roughly 2 days to develop because it
stimulates the body to produce more melanin. The photochemical properties of melanin make
it an excellent photoprotectant.
3.
Photodermatoses:
Photodermat
oses are skin diseases where the skin lesions are caused by light. Such lesions may be itching
papules, whaling of the skin, fierce reddening and peeling etc. The more sensitive patients
cannot even stand one minute of outdoor daylight. In these diseases the UV-B radiation in
sunlight is the predominant causative agent. Loss of adaptation of the skin to light appears to
be a predominant factor for these diseases.
3.
Premature Ageing of
Skin:
Another effect of
ultraviolet rays on the skin is premature ageing of the skin. Recent studies have shown that
many of the symptoms commonly associated with mere ageing (i.e. wrinkles, loosening of
the skin) may instead be related to UV exposure. Even careful tanning kills skin cells,
damages DNA and causes permanent changes in skin connective tissues which lead to
wrinkle formation in later life. UV-A, UV-B and UV-C, all these can damage collagen fibers
and thereby accelerate aging of the skin. Both UV-A and UV-B destroy vitamin A in skin
which may cause further damage.
3.
Skin
Cancer:
9
0% of the skin carcinomas are attributed to UV-B exposure [Wayne] and the chemical
mechanism by which it causes skin cancer has been identified [Tevini]. The above named
carcinomas are relatively easy to treat, if detected in time, and are rarely fatal.
There are various types of skin cancer. One main class is formed by the coetaneous
melanomas, the cancers of the pigment cells. This appears to be a correlation between brief
and high intensity exposures to UV and eventual appearance (as long as 10-20yrs) of
melanoma.
The other main types are basal cell carcinomas and squamous cell carcinomas, cancers of the
epithelial cells. These carcinomas of the skin are sometimes, collectively, called nonmelanoma skin cancers. For the present example we will deal with these non-melanoma skin
cancers. In white Caucasians, the incidence of these cancers ranks high among the various
types of cancer. In some populations it is in fact the highest of all. The incidence is lower in
more pigmented populations, typically by a factor of 10 or even 100. The mortality rate is
low in comparison with that for other types of cancer approximately 1% in areas with good
medical care.
The non-melanoma skin cancers are clearly correlated to sunlight. They occur mostly in lightskinned people and then predominantly on skin areas which are most exposed to sunlight,
such as the face. In people of comparable genetic background, the incidences are higher in
the sunnier geographical areas.
Early experiments showed that white rats exposed to sunlight developed skin cancers, but
similar rats exposed to sunlight filtered through window glass did not. As the window glass
absorbed mainly UV-B radiation, this result indicated that the carcinogenic effect to a large
extent due to the UV-B radiation in sunlight.
In technical terms, carcinogenic effectiveness was defined as the reciprocal value of the daily
dose of radiation at a certain wavelength required for the induction of tumors of 1 mm
diameter in 50% of a group of mice in 300 days. The tumors in these mice were
predominantly squamous cell carcinomas.
DNA DAMAGE
UV-B light can cause direct DNA damage. The radiation excites DNA molecules in skin cells,
causing aberrant covalent bonds to form between adjacent cytosine bases, producing a dimer.
When DNA polymerase comes along to replicate this strand of DNA, it reads the dimer as
AA and not the original CC. This causes the DNA replication mechanism to add a TT
on the growing strand. This is a mutation, which can result in cancerous growths and is
known as a classical C-T mutation. The mutations that are caused by the direct DNA
damage carry a UV signature mutation that is commonly seen in skin cancers.
EFFECTS ON EYES
High intensities of UV-B light are hazardous to the eyes and exposure can cause welders
flash (photokeratitis or arc eye) and may lead to cataracts, pterygium, and pinguecula
formation. Another possible eye damage that can result from high doses of UV light is
particularly to the cornea which is a good absorber of UV light. High doses of UV light can
cause a temporary clouding of the cornea called snow-blindness and chronic doses has been
tentatively linked to the formation of cataracts. High incidences of cataracts are found at high
elevations, Tibet and Bolivia and higher incidences are seen at lower latitudes (approaching
the equator). UV light is absorbed by molecules known as chromophores, which are present
in the eye cells and tissues. Chromophores absorb light energy from the various wavelengths
at different rates a pattern known as absorption spectrum. If too much UV light is absorbed,
eye structures such as the cornea, the lens and the retina can be damaged.
EFFECTS ON IMMUNE SYSTEM
Prolonged exposure can damage the human immune system. Cells or tissue components
which are altered by the radiation may be recognized as foreign material by the immune
system and are removed. Certain functions of the immune system are however, suppressed by
exposure to UV-B radiation. When skin is exposed to more UV-B radiation then it is
accustomed to its ability to adapt.
OZONE DEPLETION AND UV RADIATIONS
Ozone depletion results in an increase of UV-B radiation, but the shorter the wavelength, the
stronger the increase within the UV-B range. 1% decrease in the ozone layer will cause an
estimated 2% increase in UV-B irradiation; it is estimated that this will lead to a 4% increase
in basal carcinomas and 6% increase in squamous-cell carcinomas [Graedel & Crutzen].
Many scientists today believe that this life-protecting stratospheric ozone layer is being
reduced by the chlorofluorocarbon (CFCs) gases released into the atmosphere by different
sources on the earth. Many environment groups are vehemently protesting against the use of
these gases and their use in many places in the world has been banned. Pollution on the earth
has already caused a hole in the ozone layer above the Antarctic.
VULNERABILITIES DUE TO UV RADIATIONS
A diagram produced by WHO based on global burden of diseases from solar ultraviolet
radiation in the year 2006.
CONCLUSION
Thus we can conclude that UV rays have a disastrous impact on our planet. It will lead to
many dangers which would affect not only us but also the entire living community on this
earth. UV rays can lead to many unforeseen disasters which can signal the end of life on this
beautiful planet.
All the dangers attributed to UV rays reaching our earth reside only on man. Man by his
reckless actions has dug graves not only for himself but also for the earth too. Its his only
responsibility to restore the earth back to its healthy mode.
Thus, a concerted effort is needed on the side of mankind to quickly address this danger.
Steps have to be taken from individual to the international level to protect our earth. From
abandoning plastics to decrease the dependence on automobiles, man has to be careful to
ensure that he does not push earth to its death. We also need to ensure that we do not create
UV rays by machines and their usage must be strongly restricted only to the academic
field. Also we need to create awareness among ourselves on the need of the hour to protect
life. Its a pity that we try to blame domestic animals for increasing the levels of methane in
the atmosphere. What we need to understand is that their methane levels are nothing as
compared to our creation of a hole on the ozone layer. Their actions are too small to be
compared to our sins.
Being a part of future generation, the student community also needs to be aware about the
topic which is a prime reason for me selecting this project. We too should take care not to
harm our earth in any way. Always we should remember that we have not inherited the
earth from our fathers but have leased it from our future generations. This alone will
make us responsible and be steadfast in our Endeavour to combat the penetration of UV rays.
BIBLIOGRAPHY
R.Parson, FAQ 111, UV and biological effects of UV.
FDA Consumer Magazine and publications: FDA#87-8272, #81-8149 and
#92-1146.
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