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E v en t s

DiseasesofPoultry

Ou r S h op

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ByIvanDinev,DVM,PhD

BookContents

BacterialDiseases

FOWLCHOLERA

ESCHERICHIACOLIINFECTIONS
SALMONELLOSES
PARATYPHOIDINFECTIONS
FOWLCHOLERA
RIEMERELLAANATIPESTIFERINFECTIONS
MYCOPLASMA
NECROTICENTERITIS
CHOLANGIOHEPATITISINBROILERCHICKENS
GANGRENOUSDERMATITIS
BOTULISM
AVIANTUBERCULOSIS

ViralDiseases
VIRALINCLUSIONBODYHEPATITIS
HAEMORRHAGICENTERITISOFTURKEYS
EGGDROPSYNDROME1976
ADENOVIRUSGROUPIASSOCIATEDINFECTIONS
INFECTIOUSBURSALDISEASE(GUMBORO)
INFECTIOUSBRONCHITIS(IB)
LARYNGOTRACHEITIS
SWOLLENHEADSYNDROME
INFECTIOUSENCEPHALOMYELITIS
NEWCASTLEDISEASE
FOWLPOX
REOVIRUSINFECTIONS

NeoplasticDiseasesinPoultry
VIRUSINDUCEDNEOPLASTICDISEASESMAREK'SDISEASE
LYMPHOIDLEUKOSIS
MYELOCYTOMATOSIS
68. 69.. Fowl cholera is an infectious disease in domestic fowl, waterfowl and other
ERYTHROBLASTOSIS
ADENOCARCINOMATOSIS
avianspecies.Itismanifestedeitherinacutesepticaemicformwithahighmorbidity

ParasiticDiseases
COCCIDIOSIS
HISTOMONOSIS
ASCARIDIOSIS
RAILLIETINOSIS
KNEMIDOKOPTOSIS

Partners
Petersime

anddeathratesoraschroniclocalforms(independentlyorsecondarytoacuteones).
Acute fowl cholera. The sudden and unexpected death could be the first sign of the
disease.Inthisform,thelesionsarepredominantlyrelatedtovascularinjuries.

rlconsulting

Biomin

MycosesandMycotoxicoses
ASPERGILLOSIS
ASPERGILLUSGRANULOMATOUSDERMATITIS
AFLATOXICOSIS
CANDIDIASIS
FUSARIOTOXICOSES

DeficiencyDiseases
VITAMINB2DEFICIENCY
VITAMINBiDEFICIENCY
VITAMINEDEFICIENCY
FATTYLIVERHAEMORRHAGICSYNDROME
70. Commonly observed signs are anorexia, ruffled feathers, oral and nasal mucus
SLIPPEDTENDON(PEROSIS)

discharge, cyanosis and white or greenish watery mucoid diarrhoea. Frequently,


subserous petechial or ecchymosed haemorrhages in the anterior part of the small
PULMONARYHYPERTENSION(ASCITIS)SYNDROMEINBROILERCHICKENS
intestine,thegizzardortheabdominalfatarediscovered.
Other

AMYLOIDOSIS
DEEPPECTORALMYOPATHY

RUPTUREOFTHEGASTROCNEMIUSTENDONINBROILERBREEDERS
CAGELAYERFATIGUE
DYSCHONDROPLASIA
THYPERKERATOSIS
GOUT

http://www.thepoultrysite.com/publications/6/diseasesofpoultry/181/fowlcholera

CIDLINES

Novogen
ECOAnimalHealth
JansenPoultryEquipment
ABVista
PasReform
VencomaticGroup
Halamid
HyLineInternational
Hubbard

SeasonalPicks
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FOWLCHOLERADiseasesofPoultryThePoultrySite

HYPERANDROGENISMINBROILERCHICKENS
EFFECTOFSTICKYWILLY(GALIUMAPARINE)SEEDSONHEALTHYSTATUSANDPRODUCTIONTRAITSIN
BROILERCHICKENS
GIZZARDIMPACTIONINTURKEYPOULTS
ROUNDHEARTINTURKEYS(DILATEDCARDIOMYOPATHY)
ACUTESELENIUMINTOXICATION
ACUTEPROPANEBUTANEINTOXICATION
SPONTANEOUSRUPTUREOFTHECAUDIALRENALARTERYINTURKEYS
SUBCUTANEOUSEMPHYSEMA
GASTROINTESTINALIMPACTION
MALFORMATIONS

71.72.Congestion(overfillingofbloodvesselswithredbloodcells)oftheliverasan
initialmanifestationofE.colisepticaemiainabroilerchicken.H/E,Bar=40m.

73.In layers (commercial or breeders), acute oophorites with regressing follicles and
consequently,diffuseperitonitesarecommonlyobserved.

74. Chronic fowl cholera. It is characterized by local inflammations. The periorbital


sinusesarefrequentlyaffectedbyaserofibrinousinflammation.

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FOWLCHOLERADiseasesofPoultryThePoultrySite

75. 76. 77. Another local form is the injury of wattles that are strongly distended
because of their filling with fibrinous caseous content. The flocks that recuperated
from fowl cholera continue to carry and shed Pasteurella multocida. The carriers
store the organism in nasal choanas and contaminate the forage, water and the
environmentwithoraldischarges.Wildbirdsandsomemammals(swine)couldalso
carrytheagentandintroduceitintopoultryflocks.Cannibalismisanessentialroute
ofspreadingtheinfection.

78. In turkeys a common finding is the unilateral or bilateral croupous


pleuropneumonia

79. the fibrinous caseous exuate accumilated in wattles sometimes leads to


gangreneofthecoveringskin.

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80. 81. possibly be spread from sinuses to adjacent airfilled skull bones with
subsequent necrosis and onset of neurological signs (opisthotonus and torticolis).
Thediagnosisismadeonthebasisofdiseasehistory,clinicalsigns,thelesionsand
theresultsofbacteriologicalstudies.Fowlcholerashouldbedifferentiatedfromacute
E. coli septicaemia, erysipeloid, fowl typhoid etc. The immunization of birds at the
ageof812weeksgivesverypromisingresults.Manyantibioticsandsulfonamides
could lower death rate, but at discontinuation of the treatment, the disease could
recur.Sulfonamidesareappropriatefortreatment,buttheyinhibitegglaying.

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inpart,inanyformat(electronic,photocopiesetc.)withoutthepriorconsent,inwriting,ofcopyrightholders
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