One way of investigating whether aggression is inherited is by looking at
twin studies. Monozygotic twins (MZ twins) share 100% of their genes and Dizygotic twins (DZ twins) share 50% of their genes. If MZ twins have more similar levels of aggression and therefore a higher concordance rate, then it is more likely that aggression is inherited. McGuffin & Gottesman found concordance rates were 87% for MZ twins and 72% for DZ twins. Because MZ concordance rate is higher than DZ, we can assume there is a genetic influence in aggressive behaviour. Another way of investigating whether aggression is genetic is using adoption studies. If a positive correlation is found between aggressive behaviour is adopted children and their biological parents, then a genetic effect is implied. For example, Hutchings and Mednick (1975) studied 14,000 adoptees in Denmark and found a significant number adopted boys with criminal convictions had biological parents with criminal convictions. Another possible idea is that aggression can be inherited through the MAOA gene. MAOA regulates serotonin levels, and low levels of serotonin are associated with aggression, suggesting this gene regulated aggressive behaviour. Brunner studied males in a Dutch family and found that those who had been involved in arson and rape crimes as abnormally low MAOA in their bodies, suggesting that there may be a gene which can influence aggression. However when conducting such research it is very difficult to establish the role of genes in causing aggression. More than one gene can often contribute to a given behaviour, suggesting there may not be just one gene for aggression. This suggests that it is hard to judge the role of the MAOA gene in causing aggression. Moreover, environmental influences may also contribute in a gene-environment interaction. Caspi et al (2002) studied 500 male children, and found that those with low MAOA who were maltreated as children showed increase aggression, but not those with low MAOA or those who were, maltreated alone. Therefore we cannot rely on the role the MAOA gene in contributing to aggression as there may be other environmental influences. There are also some problems with the way some of this data has been collected. Many studies of aggression such as those conducted by Hutchings and Mednick (1975) rely on parental or self reports of aggressive behaviour. This means that someone may say that they are more aggressive than they actually are in order to bias the results of the study in their favour. This means studies such as these do not give reliable explanations of aggressive behaviour, so in the case of Hutchings and Mednick (1975) we cannot guarantee aggression is genetic.
There is research which supports the idea that aggression can be
inherited. Coccaro claims that 50% variance in aggression in adult twins could be attributed to genetic factors. This demonstrates aggression can be inherited. However this study also suggests that 50% variance in aggression is adult twins isn't attributed to genetic factors. Therefore this suggests research into genetic is biologically deterministic, as it ignores other factors which can be present which can also influence levels of aggression. It may be that factors such as social upbringing also contribute to levels of aggression.
Studying aggression in non-human animals can help us understand
aggression in humans. It is possible to manipulate specific genes which code for aggressive behaviour (similar to the MAOA gene). Young et al claim to have identified a genetic mutation which causes mice to become aggressive, showing violence towards other mice. A counterpart of this gene is present in humans, therefore supporting the idea that there is a genetic basis for aggression. However, using animals causes issues with extrapolation, meaning different results regarding aggression may be found if the study was replicated on non-human animals. Also, there are many ethical issues involved with animal research and genetic engineering, suggesting this is not an ethically viable way of investigating genes which influence aggressive behaviour.