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Definition:
Incedence
Osteomyelitis affects about 2 in
10,000 people.
Acute osteomyelitis is rare in
children. It's estimated that one in
every 1,000
children under the age of one will
develop acute osteomyelitis.
The condition is even less
common in children over one, with
one in every 5,000
children affected.
For example, it's estimated that
between 30% and 40% of people
with diabetes,
who experience a puncture injury
to the foot, will develop
osteomyelitis. One in
every 200 people with sickle cell
anaemia (a hereditary blood
condition) will
develop osteomyelitis in any given
year.
Risk Factors
Trauma
Diabetes
Hemodialysis
Splenectomy
Advanced age
- Immune function
Poor circulation
Manifestation
Pain and/or tenderness in the
infected area
Swelling and warmth in the
infected area
Fever
Nausea, secondarily from being ill
with infection
General discomfort, uneasiness,
or ill feeling
Drainage of pus through the skin
Additional symptoms that may be
associated with this disease
include:
Excessive sweating
Chills
Lower back pain (if the spine is
involved)
Swelling of the ankles, feet, and
legs
Changes in gait (walking pattern
that is a painful, yielding a limp)
Types
There are three main types of
osteomyelitis:
Acute osteomyelitis, where the
bone infection develops within two
weeks
of an initial infection, injury or the
onset of an underlying disease.
Sub-acute osteomyelitis,
where the bone infection develops
within one ot two months of
an initial infection, injury or onset
of an
underlying disease.
Stages of Osteomyelitis
PATHOPHYSIOLOGY OF
OSTEOMYELITIS
SEKELETAL
SYSTEM
Risk Factors:
-trauma
-diabetes
-hemodialysis
-splenectomy
-advanced age
Changes:
Vascular congestion
Small vessel thrombosis
Edema
inflammation
if compensation fails
great losses
Molecular Changes:
exudation of
polymorphonuclear
leukocytes:
increased erythrocytes
increased WBC
ETIOLOGY/CAUSE:
1. Newborns (younger than 4
mo):
S. aureus, Enterobacter
species, and group A and B
Streptococcus species
2. Children, adolescents (aged
4 y to adult):
S. aureus (80%),
group A Streptococcus
species, H.
influenzae, and
Enterobacter species
Enterobacter or
Streptococcus
Bacterial invasion (
hematogenous, loval
extension , trauma)
Pathophysiologic Manifestation on
Effect on Bodily Function:
cardiac output
impaired cellular metabolism
ischemic necrosis
systemic infection
lysis of the bone cell
Clinical Manifestations ( S/S ) :
Increased CRP compliment
reactive protein
Increased erythrocyte
sendimentatiion rate ESR
Drainage of pus through the skin
Pain and/or tenderness in the
infected area
Swelling and warmth in the
infected
area
Fever
Nausea, secondarily from being ill
with infection
Laboratory Exams:
CBC
Bone scan
ESR
Pathophysiology
Osteomyelitis tends to
occlude local blood vessels,
which causes bone necrosis
and local spread of infection.
Infection may expand
through the bone cortex and
spread under the periosteum,
with formation of
subcutaneous abscesses that
may drain spontaneously
through the skin.
In vertebral osteomyelitis,
paravertebral or epidural
abscess can develop.
If treatment of acute
osteomyelitis is only partially
successful, low-grade chronic
osteomyelitis develops.
Diagnostic Studies
Management
Nursing Diagnoses
Nursing Intervention
References:
Medical-Surgical Nursing by Josie
Quiambao-Udan, RN, MAN
http://www.nlm.nih.gov/medlineplus/ency/i
magepages/9712.htm
http://www.nhs.uk/conditions/Osteomyeliti
s/Pages/Introduction.aspx
www.merck.com
Lemone, Page 1267
Professional Guide to Diseases (Eighth
Edition), 2005
Edited by-Belinda Josephine,Aseana
College of Health Sciences