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TABLE OF CONTENTS
Introduction..2
Nursing Assessment.2
Patients Profile. 2
Patients History.2
Assessment tool..3
Pathophysiology5
List of Prioritized Nursing Problems7
Nursing Care Plans...9
Acute pain.. 9
Decreased cardiac output..11
Ineffective tissue perfusion...13
Anxiety..15
Reference.17
II.
INTRODUCTION
This is the case of Mrs. B with a clinical condition that involves the renal and cardiovascular systems, two
systems which are closely interrelated. These topics interest me the most due to dynamic changes that can occur
because of how one impaired system can affect the functions of the other system, which eventually progresses to a
more critical stage if not given prompt medical and nursing interventions. These clinical conditions also require the
nurse to be more critical and alert in observing life-threatening manifestations in a glimpse of an eye, thus
promotes the enhancement of skills in critical care. I have only handled the patient for 3 days, January 3-5, 2013,
where the prevalent manifestation she had was chest pains and hypertension. Although she has a kidney problem,
obvious manifestations like oliguria, ascites and edema were not present during assessment; however, she
manifests personality changes at times due to increased circulating metabolic wastes in the blood. Concepts
involved in this case analysis include the relationship of the cardiovascular functions and the kidney functions, the
complications that entail them, and the nursing interventions that should be provided to the patient which include
cardiovascular care like monitoring for vital signs, specifically the blood pressure and pulse rate, monitoring for
signs and symptoms if impending myocardial infarction, and monitoring for peripheral tissue perfusion and its
effects to the neurologic system. For the renal system, strict fluid and electrolyte balance must be monitored
III.
NURSING ASSESSMENT
Patients Profile
Demographic data
NAME: Mrs. B
GENDER: Female
AGE: 60 y/o
RELIGION: Roman Catholic
BIRTHDAY: May 2, 1952
CIVIL STATUS: Married
OCUPATION: Businesswoman in a contraction company subcontractor
Admission Details
DATE ADMITTED: December 31, 2012
TIME ADMITTED: 7:10 A,M
ROOM: Medical ward, 205A
CHIEF COMPLAINS: Epigastric pain and bloatedness
IMPRESSION DIAGNOSIS: Acid related disorder rule out acute coronary syndrome, chronic kidney disease stage V
secondary to hypertensive nephrosclerosis, coronary artery disease, chronic stable angina rule out community acquired
pneumonia, hypertension, hyperuricemia
A. History of Present Illness
Four days prior to consultation, patient felt bloatedness and epigastric pain rated as 8/10, non-radiating.
It was with associated dyspnea, restlessness, irritability and difficulty sleeping. There were no associated
vomiting, fever, and LBM. The patient was maintained on Pantoprazole and TUMS due to acid related
disorder, although claims that she was not relieved by these medications. Persistence of epigastric pain
prompted consultation, hence admission.
1. Past Medical History
10 years ago, she was diagnosed with hypertension and takes unrecalled maintenance
medications. According to her, she was repeatedly admitted in thei and other institutions due
to
hypertension.
2 years ago, the patient was diagnosed with chronic kidney disease stage V, and was advised to
undergo dialysis, but patient refused.
2 weeks prior to admission, patient was admitted in this institution due to dyspnea, epigastric pain and
right flank pain. Patients condition improved with a diagnosis of chronic kidney disease stage V,
hypertension, anemia and acid related disorder, with home medications of Twynsta, Metoprolol and
Clopidogrel. The patient has no known food or drug allergies.
2. Socio-cultural history
The patient lives in a non-congested neighborhood with her family. She does not drink alcoholic
beverages, and she was a smoker, with 1 pack year. She works as a businesswoman in a subcontraction
firm and is managing her employees.
3. Heredofamilial history
Comprehensive Case Analysis | 2
The patient has a family history of hypertension on both paternal and maternal side and bronchial
asthma on paternal side. The patient has no renal disease, PTB, DM, and cancer history.
B. ASSESSMENT: MS TOOL
ACTIVITY/REST
The patient is a businesswoman in a contraction firm. She usually spends leisure time by watching and
talking to her relatives. Upon assessment, she has a stooped posture, and was able to walk with a normal gait,
but with slowed movements and phasing. Her activity level is sedentary because at work as well as at home,
she often sits down. She has a normal muscle tone and strength, but her anginal episodes impose immobility,
weakness and breathlessness. Due to her hospitalization, she developed a feeling of dissatisfaction and a
feeling of cannot concentrate due to inability to manage her job as a businesswoman. Her usual hours of sleep
range from 7-8 hours, but due to her chest pains, it was decreased to 4-5 hours. Medications to facilitate sleep
include Alprazolam. Every after getting up to void, she complains of chest pains, mild dizziness, and blood
pressure elevation of 160/90, tremors are also observed in both hands while having anginal episodes. Care
concerns include activity intolerance and risk for injury
CARDIOVASCULAR ASSESSMENT & PAIN/ DISCOMFORT
Upon the rotational period, she mostly complained of chest pain/heaviness with a rate that ranges from
7/10 to 8/10, which also radiates at the scapular and nape area. Physical exertion and low-lying position
aggravates her condition, while medications like antianginal, specifically Isordil, and analgesics like Ketorolac,
Tramadol and Norgesic relieve the pain felt by the patient. Cardiovascular assessment also includes report of
palpitations during the second day of the care provided. During anginal episodes, apical pulse increased up to
103bpm. BP revealed 160/100 and respiratory rate was 20cpm. Peripheral tissue perfusion include capillary
refill of 2s, and pale nail beds and conjunctiva. Peripheral pulses revealed a pulse rate of 82bpm, moderate (+2)
with skip beats. Diagnostic tests include an X-ray as of December 31, 2012 that revealed a mild cardiomegaly
probably multichamber, and an atherosclerotic aortic knob. A 12 lead ECG was also done upon admission with
results of sinus bradycardia to consider old anteroseptal wall infarction, prominent U waves, and left atrial
abnormality; however, 12 lead ECG as of January 4 and 5, 2013 revealed premature ventricular contractions
with bigeminys. Trop I has a normal result of .168 ng/mL but CK-MB has an increased result of 26.4 u/L.
Medical diagnoses are coronary artery disease, Chronic stable angina rule out community acquired pneumonia
and hypertension. Medications given for the carsiovascular system include Antihypertensives (Amvasc,
Twynsta, Metoprolol, Clonidine) Antianginal/Anti ischemia (Isordil/Isoket, Trimetazidine, Nitroglycerine)
Anticoagulant (Clopidogrel) Analgesics (Tramadol, Ketorolac, Norgesic Forte, Ketoprofen/Fastum Gel), and
Antiarrhythmias (Amiodarone). Care concerns are acute pain, decreased cardiac output, ineffective
myocardial tissue perfusion, ineffective peripheral tissue perfusion, disturbed sleep pattern, and activity
intolerance.
GASTROINTESTINAL TRACT/ FOOD & FLUID/ INTEGUMENTARY
The patients usual food intake is 100% but upon hospitalization, it decreased to 50-75%, and takes in
2-3 snacks per day. Her prescribed diet was Full, low cholesterol diet. She usually takes in more than 1 liter of
fluids per day, including caffeine and sodas. Her bowel movement during her hospitalization reached to 3-4
times per day. The patients history upon admission revealed epigastric pain and bloatedness, but upon the time
of assessments, the patient never complained of epigastric pain and bloatedness; however, she vomited twice
during the first day and the second day, characterized as previously ingested food and whitish secretions,
respectively. She verbalized that intake of certain medications, like Lactulose and Maalox, causes increased
borborygmi which is succeeded by vomiting. Her lips are cracked and dry, with dry ashen gray skin and good
turgor, mucous membranes are moderately moist. Diagnostic tests include 12 Lead ECG, which revealed sinus
bradycardia to consider old anteroseptal wall infarction, prominent U waves, and left atrial abnormality. This
diagnostic test was conducted to determine if the pain originates directly from the gastrointestinal tract or just a
radiating pain from an underlying heart condition. With a diagnosis of acid related disorder, the patient is given
Itopride HCl, Hyoscine N-Butylbromide, Lactulose, Maalox, Pantoprazole, and Calcium Carbonate. Due to the
vomiting episodes; care concerns include Risk for aspiration and risk for fluid volume deficit.
GENITOURINARY TRACT/ RENAL SYSTEM
Due to her chronic hypertension, the kidneys are also affected, leading to a diagnosis of Chronic
Kidney Disease (CKD) stage V. The patient did not manifest any excess fluid volume symptoms like edema
and ascites, however, during the last day of my rotation, the patient manifested a puffy eyelids which could be
caused by excessive fluid volume due to inability of the kidneys to retain albumin, or may be due to lack of
sleep. She also did not manifest oliguria, where urine output for one day was more than 1ooomL. She also did
not manifest any flank pain during the entire shifts of handling her. Her complexion was ashen gray, which is a
symptom of CKD. Diagnostic tests include BUN and Crea which revealed 129.2mg/dL and 1015umol/L,
respectively. This was done to determine the ability of the kidneys to excrete waste products from the body.
Another test done was serum electrolytes particularly that of serum sodium= 128mmol/L and serum
Potassium= 3.49mmol/L, done to determine presence of electrolyte imbalance brought about by the kidney
Comprehensive Case Analysis | 3
problem. CBC also revealed a low hematocrit level of 0.304 and a low hemoglobin level of 101, indicative of
anemia due to decreased erythropoietin production. Lastly, urinalysis revealed turbid urine, a specific gravity of
1.010, and a positive Albumin in the urine (++) or otherwise known as Albuminuria. Because of the impaired
functionality of the kidneys, metabolic wastes accumulate in the body, particularly in the blood stream. Uric
acid is one of the wastes which accumulate in the blood stream, a manifestation otherwise known as
Hyperuricemia. This hyperuricemia makes the client at risk for mental status and personality changes;
Allopurinol therefore was given to facilitate excretion of excessive uric acid in the blood. Medications given
for her CKD include diuretics (Lasix) and Sodium bicarbonate. Iron and folic acid was given to prevent
anemia. Care concerns include electrolyte imbalance, ineffective renal tissue perfusion, risk for acute
confusion, risk for injury, and risk for infection. Due to lack of knowledge regarding dialysis, another
concern is deficient knowledge.
NEUROLOGIC ASSESSMENT
The patient claimed once of a tingling sensation all over her body accompanied by diaphoresis, during
one of her anginal episodes. The patient is oriented to person, time, place and situation. She is also alert,
conversant, and follows commands; but restless during anginal episode attacks. Her Glasgow coma scale has a
total of 15. All cranial nerves are intact and has no significant findings.
RESPIRATION
Respiratory rate is 20-21cpm, regular and deep. The patient experiences dyspnea related to an
underlying cardiovascular problem. She is relieved by rest and oxygenation at 1-2LPM per nasal cannula. She
was a past smoker with 1 pack year.
PSYCHOSOCIAL ASSESSMENT
This patient verbalized that her hospitalization worries her because she cannot perform her job as a
businesswoman. She constantly thinks about it which causes increased blood pressure and chest pains, which
alters her sleeping pattern. Aside from that, the patient, on the last day of assessment, verbalized that she
refuses to undergo dialysis despite her condition because her notion of undergoing dialysis hastens a persons
death, and it increases her anxiety. However, upon explanation that dialysis would only be the last and best
treatment available, the patient then verbally agreed to undergo dialysis. Care concerns therefore include
Ineffective role performance, mild anxiety, fear, sleep pattern disturbance, and deficient knowledge.
IV.
PREDISPOSINGFACTORS:
PRECIPITATING FACTORS:
Age: 60 y/o
Gender: Female
Family history: HPN on both
paternal and maternal side
Lifestyle: sedentary
Stress from work
Past smoker: 1 pack year
Diet: fat; caffeine
intake
age=fat; accumulation of
lipids in the intima
(atherosclerosis)
Potent vasoconstrictor
Vasoconstriction
Total peripheral resistance
Decreased perfusion to other
organs, especially the kidneys
Narrowing of preglomerular
arteries and arterioles
RAAS is stimulated
Progressive glomerular
sclerosis
Hypertensive
nephrosclerosis
Myocardium compensates by
contracting harder and faster
Myocardium is overworked
Further BP elevation
perfusion to tissues
Ineffective Cardiac tissue
perfusion
Thickening/ enlargement of
cardiac muscles
Relaxation period
O2 supply to coronary
arteries
Inability to contract
effectively
Decreased cardiac output
Ischemia
Ineffective Cardiac tissue
perfusion
Easy fatigability
Angina
Activity intolerance
Acute pain
Disturbed sleep
pattern
Legends:
Nursing Diagnoses
Lab results
glomerular filtration
capacity
As of 12.31.12
Serum Na: 128mmol/L
Serum K: 3.9mmol/L
uric acid
Electrolyte
imbalance
Inability to hold
bicarbonates/ act as buffer
system
ABG as of 12.31.12
Ph: 7.37
pCO2: 28.4
paO2: 77.2
HCO3: 18
Erythropoietin
production
RBC production
CBG as of 12.31.12
Hct: 0.304
Hgb: 101
Hyperuricemia
Metabolic acidosis
Anemia
As of 12.31.12
BUN: 129.2 mg/dL
Crea: 1015 umol/L
Urinalysis as of 12.31.12 revealed:
Specific grav.: 1.010
Albumin: positive (++)
CHON
excretion
Ig production (made
up of protein)
Ineffective peripheral
tissue perfusion
Risk for fluid volume
deficit
humoral response
Legends:
Nursing diagnoses
Lab results
REFERENCES
BARE et al. (2012), Brunner and Suddarths Textbook of
Medical-Surgical Nursing10th edition, Volume 2
CORWIN, E. (2008), Handbook of Pathophysiology 3rd
Edition
McCANN (2007), Straight As in Fluid and Electrolytes
V.
RATIONALE
RATIONALE
EXPLANATION OF THE
PROBLEM
Reference:
INTERVENTIONS
RATIONALE
Demonstrate DBE
prevent angina
Diet containing cholesterol and caffeine increases
vasoconstriction which compromises blood supply, causing
anginal episodes.
Evaluation:
Goal unmet
VII. JUSTIFICATION
The goal was unmet because Mrs. Bs pain was recurrent due to poor stress management and non-compliance to some medications like pain relievers. Her condition
also worsened which prompted intensive critical care.
INTERVENTIONS
RATIONALE
Maintain on O2 inhalation
position.
Do passive range-of-motion (ROM)
exercises to unaffected extremity
every 2 to 4 hours
Administer antihypertensives:
Twynsta, Amvasc, Metoprolol and
anti coagulants: Clopidogrel; and
antianginal: nitroglycerine
I.
JUSTIFICATION
The goal was unmet because the patients BP remained increased for 3 days upon assessment due to inability to manage stressors like business problems and
hospitalization concerns. The patient wasnt also compliant to some medications and medical interventions like 12 Lead ECG.
II.
This was a result of incomprehension due to the experienced manifestations like chest pains and narrowed focus during explanations. Therefore, recommendations
include proper health teaching and explanation of all interventions to promote patients compliance to both medical and nursing interventions. Nurses should also be assertive
in providing nursing interventions.
Maintain O2 inhalation
I.
JUSTIFICATION
Goal is partially met since during the time of evaluation, the patients condition improved and manifested good cardiac perfusion, although it was not continuously
improving. At times, the patient still manifests decreased cardiac and renal perfusion.
II.
EXPLANATION OF THE
PROBLEM
O>
With vital signs of
BP=160/100, PR= 82bpm, RR=
22;
restlessness
noted;
trembling of hands noted;
diaphoresis noted on forehead
and back area; moaning
episodes noted at times; has
difficulty
concentrating;
decreased interaction with
people noted; stares blankly at
times
INTERVENTIONS
RATIONALE
REFERENCE:
GULANICK et al., (2007)
Nursing Care
Plans:
Nursing
Diagnosis
and
Intervention.
VI. JUSTIFICATION
Goal was partially met because the patient was able to recognize the signs of anxiety; however, she wasnt able to manage the stressors causing her anxiety.
VIII. REFERENCES
BARE et al. (2012), Brunner and Suddarths Textbook of Medical-Surgical Nursing10 th edition, Volume 2
CORWIN, E. (2008), Handbook of Pathophysiology 3rd Edition
DOENGES, et al. (2010) Nurses Pocket Guide 12th Edition
GULANICK et al., (2007) Nursing Care
Plans: