Beruflich Dokumente
Kultur Dokumente
Theories of Leadership
Great Man Theory
Argues that few people are born to be great leaders
who are well rounded & simultaneously instrumental
& supportive also of the premise that leaders are
born not made, which suggests that leadership
cannot be developed
Styles of Leadership
Autocratic
involves centralized decision making, with the
leader making the decision & using power to
command & control others
Democratic
Management Process
Planning Organizing
Sources of Power
Expert power it is derived from the knowledge &
skills one possess
Legitimate power is derived from the position one
holds in a group & indicates authority but not
sufficient as ones only source of power
Referent power derived from respect & trust
coming from any individual group or organization
Reward power it comes from the ability to
recognize others for complying
Coercive power is based on fear of punishment if
one fails to conform
Connection power it comes from coalition &
interpersonal relationship
Great Man
Leaders are born and not made
Great leaders will arise when there is a great need
Ex. King of Spain
Behavioral Theory
Successful leadership is based in definable, learnable
behavior
Situational theory
The best action of leader depends on range of
situational factors
*motivation
*capability of followers
performance of leader and follower
attitudes, needs and expectations
Frederick W. Taylor (1856 1917)
Father of Scientific Management
If workers could be taught the one best way to
accomplish a task, productivity would increase.
4 overriding principles of scientific management:
1. Replace rule-of-thumb work methods with methods
based on a scientific study of the tasks.
2.
Scientifically select, train, and develop each
employee rather than passively leaving them to train
themselves.
3. The spirit of cooperation between the management
and workers for accomplishing the job.
4.
Divide work equally between managers and
workers, so that the managers apply scientific
management principles to planning the work and the
workers actually perform the tasks.
Managers need to think of new ways to do traditional
tasks so that work is more efficient.
The amount of and effort each employee expends to
produce a unit of output can be reduced by
increasing specialization and the division of labor
Bureaucratic Model
Max Weber (1864 1920)
Stressed the need for a strictly defined hierarchy
governed by clearly defined regulations and lines of
authority
Studied large organizations to determine what made
some more efficient than the others
Saw the need for legalized, formal authority and
consistent rules and regulation for personnel in
different positions
Proposed bureaucracy as organizational design
Dimensions of Bureaucracy
Performance Appraisal
Fractures:
Immobilize joint above and below fracture
Cover open fracture with cleanest material available
Check temperature, color, sensation, capillary refill
distal to fracture
Close reductionmanually manipulate bone or use
traction
Bucks Traction
Use to relieve muscle spasm of leg and back
If used for muscles spasms only, they can turn to either side.
If used for fracture treatment, only can turn to unaffected
side.
Use 8-20 lbs of weight, if used for scoliosis will use 40 lbs of
weight.
Elevate head of bed for countertraction or foot bed
Place pillow below leg not under heel or behind knee.
Russells Traction
Sling is used
Check for popliteal pulse
Place pillow below lower leg and heel off the bed
Dont turn from waist down
Lift patient, not the leg
Cervical Tongs
Never lift the weights
No pillow under head during feedings
Balanced Suspension Traction
For femur realignment
Maintain weights hanging free and not on floor
Fractured Hip
Assessments
Leg shortened
Adducted
Externally rotated
Implementation
Care after a total hip replacement
Abduction pillows
Crutch walking with 3-point gait
Dont sleep on operated side
Dont flex hip more than 45-60 degrees
Dont elevate head of the bed more than 45
degrees
Amputations
Guillotine (open)
Flap (closed)
Delayed prosthesis fitting
Residual limb covered with dressing and elastic
bandage (figure eight)
Guillain-Barre Syndrome
- GBS often preceded by a viral infection as well as
immunizations/vaccinations
- Intervention is symptomatic
- Acute phase: Steroids, plasmapheresis, aggressive
respiratory care; prevent hazards of immobility, maintain
adequate nutrition; physical therapy; pain-reducing
measures; eye care, prevention of complications (UTI,
aspiration); psychosocial support
Parkinsons disease
- Activities should be scheduled for late morning when energy
level is highest and patient wont be rushed
- Symptoms: tremors, akinesia, rigidity, weakness, motorized
propulsive gait, slurred monotonous speech, dysphagia,
drooling, mask-like expression.
Labs
HbA1c (4.5-7.6%)
- indicates overall glucose control for the previous 120 days
Serum Amylase / Somogyl (60-160 u/dL)
- elevated in acute pancreatitis
Erythrocyte Sedimentation Rate (ESR)
-
Men (1-15)
Women (1-20)
Rate at which RBCs settle out of unclotted blood in one hour
Indicates inflammation/neurosis
Hematocrit (Hct)
- Men (40-45) u/mL
- Women (37-45) u/mL
- Relative volume of plasma to RBC
Men (12-70)
Women (10-55)
Enzyme specific to brain, myocardium, and skeletal muscles
Indicates tissue necrosis or injury
Serum Glucose
- 60-110 mg/dL
Sodium (Na+)
- 135-145 mEq/L
- Hypernatremia
o Dehydration and insufficient water intake
Chloride (Cl-)
- 95-105 mEq/L
Potassium (K+)
- 3.5-5.0 mEq/L
Bicarbonate (HCO3)
- 22-26 mEq/L
- Decreased levels seen with starvation, renal failure, diarrhea.
Blood, Urea, Nitrogen (BUN)
- 6-20 mg
- Elevated levels indicate rapid protein catabolism, kidney
dysfunction, dehydration
Labs
HbA1c (4.5-7.6%)
- indicates overall glucose control for the previous 120 days
Serum Amylase / Somogyl (60-160 u/dL)
- elevated in acute pancreatitis
Erythrocyte Sedimentation Rate (ESR)
-
Men (1-15)
Women (1-20)
Rate at which RBCs settle out of unclotted blood in one hour
Indicates inflammation/neurosis
Hematocrit (Hct)
-
Lithium
- targeted blood level: (1-1.5 mEq/L)
Tofranil and AnafranilOCD medications
Pick physical needs over psychosocial needs!!!!!!!!!!!!!!!
MI
Implementation for MI
Increased ICP
Assessments
Altered LOC (Earliest Sign)
Glasgow coma scale <7
indicates coma
Confusion
Restlessness
Pupillary changes
Vital sign changes
WIDENING PULSE PRESSURE
Implementations
corticosteroids
Electrolytes
Potassium:
3.5-5.0 mEq/L
Sodium:
135-145 mEq/L
Calcium:
4.5-5.2 mEq/L
Hypokalemia Implementations
Hyperkalemia Assessments
K+ >5.0 mEq/L
EKG changes
Paralysis
Diarrhea
Nausea
Potassium Supplements
Dont give > 40 mEq/L into
peripheral IV or without
cardiac monitor
Increase dietary intake
oranges, apricots, beans,
potatoes, carrots, celery,
raisins
Hyperkalemia Implementations
Restrict oral intake
Kayexalate
Calcium Gluconate and Sodium
Bircarbonate IV
Peritoneal or hemodialysis
Diuretics
Hyponatremia Assessments
Na+ < 135 mEq/L
Nausea
Muscle cramps
Confusion
Increased ICP
Hypernatremia Assessments
Na+ >145 mEq/L
Disorientation, delusion,
hallucinations
Thirsty, dry, swollen tongue
Sticky mucous membranes
Hypotension
Tachycardia
Hypocalcemia Assessments
Hypercalcemia Assessments
Ca+> 5.2 mEq/L
Sedative effects on CNS
Muscle weakness, lack of
coordination
Constipation, abdominal pain
Hyponatremia Implementations
I&O
Daily weight
Increase oral intake of sodium rich
foods
Water restriction
IV Lactated Ringers or 0.9% NaCL
Hypernatremia Assessments
I&O
Daily Weight
Give hypotonic solutions:
0.45% NaCl or 5% Dextrose in
water IV
Hypocalcemia Implementations
Oral calcium supplements with
orange (maximizes absorption)
Calcium gluconate IV
Seizure precautions
Meet safety needs
Hypercalcemia Implementations
0.4% NaCl or 0.9% NaCl IV
Encourage fluids (acidic drinks:
cranberry juice)
Diuretics
Calcitonin
Mobilize patient
Surgery for hyperparathyroidism
Hypomagnesemia
Implementations
Monitor cardiac rhythm and
reflexes
Test ability to swallow
Seizure precautions
Increase oral intakegreen
vegetables, nuts, bananas,
oranges, peanut butter,
chocolate
Blue Bloaters
Pink Puffers
Weakness
Change in postured day and
hs (dont sleep laying down,
have to stay erect)
Use of accessory muscles of
breathing
COPD Implementations
Assess airway clearance
Listen to breath sounds
Administer low-flow oxygen (12 L, not too much because your
trying to prevent CO2 narcosis)
Encourage fluids
Small frequent feedings
Use metered dose inhalers
(MDI)
Dyspnea
Cough
Adventitious breath sounds
Left-Side CHF
Right-Side CHF
Dyspnea, orthopnea
Cough
Pulmonary edema
Weakness/Changes in mental
status
Dependent edema
Liver enlargement
Abdominal pain/Nausea/Bloating
Coolness of extremities
CHF Implementations
Anemia Assessments
(reduction in hemoglobin
amount/erythrocytes)
Palpitations
Dyspnea
Diaphoresis
Chronic fatigue
Sensitivity to cold
Anemia Implementations
Identify cause
Frequent rest periods
High protein, high iron,
high vitamin diet
Protect from infection
Spoon fingernails
Impaired cognition
Pernicious Anemia Assessments
(gastric mucosa fail to secrete
enough intrinsic factor for stomach
to absorb)
Vitamin B12IM
Rest of life cant be absorbed
PO
Schillings Test
Fatigue
Sore, red tongue
Paresthesia in hands and feet
Pain /Swelling/Fever
Schlerae jaundiced
Cardiac murmurs
Tachycardia
Parkinsons Disease
Assessments
Deficiency of dopamine
Tremors, rigidity, propulsive
gait
Parkinsons Disease
Implementations
Teach ambulation modification:
goose stepping walk
(marching), ROM exercises
MedicationsArtane, Cogentin,
Monotonous speech
Mask like expression
O,A,B,AB
A,O
A,AB
B,O
B,AB
AB
O,A,B,AB
AB
Autologous Transfusion:
Collected 4-6 weeks before surgery
Contraindicatedinfection, chronic disease, cerebrovascular
or cardiovascular
disease
Hypotonic
Solution
NS
(0.45%
Saline)
Isotonic Solution
Hypertonic Solution
0.9% NaCl
(Normal Saline)
5% D/W
(Dextrose in
Water)
Lactated Ringers
5% D/ NS (5%
Dextrose in
0.225% Saline )
5% Sodium Bicarbonate
Anti-Depressants
Monoamine
Oxidase Inhibitors
(MAO)
Action:
Causes
increases
concentration of
neurotransmitter
s
Used for:
AntiDepressants
(Monoamine
Oxidase
Inhibitors)
Medications:
Marplan
Nardil
Parnate
Depression
Chronic pain
Anti-Depressants
Selective
Serontonin
Anti-Depressants
(Monoamine Oxidase
Inhibitors)
Side effects:
Hypertensive Crisis
(Sudden headache,
diaphoretic, palpitations,
stiff neck, intracranial
hemorrhage) with food
that contain Tyramine
Nursing Considerations:
Avoid foods containing
Tyramine: Aged cheese,
liver, yogurt, herring, beer
and wine, sour cream,
bologna, pepperoni,
salami, bananas, raisins,
and pickled products
Monitor output
Takes 4 weeks to work
Dont combine with
sympathomometics
vasoconstrictors, and cold
medications
AntiDepressants
Selective
Anti-Depressants
Selective Serontonin
Reuptake Inhibitors (SSRI)
Reuptake
Inhibitors (SSRI)
Serontonin
Reuptake
Inhibitors
(SSRI)
Action:
Inhibits CNS
uptake of
serotonin
Used for:
Medications:
Paxil
Prozac
Zoloft
Depression
ObsessiveCompulsive
Disorder
Bulimia
Anti-Depressants
(Tricyclics)
AntiDepressants
(Tricyclics)
Action:
Inhibits
reuptake of
neurotransmitter
s
Used for:
Medications:
Norpramin
Elavil
Tofranil
Depression
Sleep apnea
Antipsychotic
Agents
Side effects:
Anxiety
GI upset
Change in appetite and
bowel function
Urinary retention
Nursing Considerations:
Suicide precautions
Takes 4 weeks for full effect
Take in a.m.
May urine to pinkish-red or
Pinkish-brown
Can be taken with meals
Anti-Depressants
(Tricyclics)
Side Effects:
Sedation/Confusion
Anticholinergics affects
Postural Hypotension
Urinary retention
Nursing Considerations:
Suicide precautions/2-6
weeks to work
Take at hs/Dont
abruptly halt
Avoid alcohol/OTC
/Photosensitivity
Antipsychotic
Antipsychotic
Action:
Agents
Agents
Blocks
dopamine
receptors in
basal ganglia
Used for:
Medications:
Side Effects:
Acute and
Chronic
psychoses
Haldol
Thorazine
Mellaril
Stelazine
Akathisia
(inability to
sit still)
Dyskinesia
Dystonias
Parkinsons
syndrome
Tardive
dyskinesias
Leukopenia
Nursing
Considerations:
Check CBC
Monitor vital
signs
Avoid alcohol
and caffeine
Atypical
Antipsychotic
Agents
Action:
Interferes
with binding
of dopamine
in the brain
Used for:
Acute and
Chronic
psychoses
Atypical
Antipsychotic
Atypical
Antipsychotic
Medications:
Side Effects:
Clozaril
Risperdal
Extrapyramida
l effects
Anticholinergic
Sedative
Orthostatic
hypotension
Nursing
Considerations:
Monitor blood
Change
positions
slowly
Use sunscreen
Cardiac
Glycosides
Cardiac
Glycosides
Action:
Medication:
Increases
Lanoxin
force of
myocardial (Digoxin)
contraction,
slows rate
Used for:
Left-sided
CHF
Cardiac Glycosides
Side Effects:
Bradycardia
Nausea
Vomiting
Visual
disturbances
Nursing
Considerations:
Take apical pulse
Notify physician
if adult <60,
child <90-110,
<70 in older
children
Monitor
potassium level
Dose: 0.5-1
milligram IV or
PO over 24 hr
period
Average: 0.25
mg
Hyperkalemia on Electrocardiogram:
Tall, peaked T waves; prolonged PR interval;
widening QRS complex
THE LAW
This defines the minimum ethical standards in a given area of practice. For
example, deceptive advertising is illegal and violators of this law are liable to large
fines, to arrest and / loss of good will.
TYPE OF LAWS:
Nursing practices subject to:1. Statutory laws. (Nurse Practice Act)
2. Regulatory laws. ( PNC pass Rules & Regulation and administrator laws)
3. Common laws. ( Informed Consent, Clients Rights To Refuse treatment)
4. Criminal laws.
(On Server Offense Imprisonment or Death)
5. Civil laws.
(Protect Individual)
ELEMENTS OF THE CODE
Delegations
PCAs
-skin care, feeding, toileting, vital signs (not initials), height, weight, IOs, ROM exercises,
ambulation, transporting, grooming, and hygiene meaures of stable clients.
EANs
-physiologically stable clients with predictable outcomes
-dressings, suctionings, urinary catheterization, med administrations (only oral, subcutaneous, and
intramuscular), no rectal or IV meds
RNs
-assessment/planning care, initiating teaching, IV meds
RN can not delegate these tasks:
-initial assessments of clients
-evaluation of client data
-nursing judgement
-client/family educatoin/evaluation
-nsg diagnosis
ACID/BASE BALANCE
pH- 7.35-7.45
PCO2- 35-45 mmHg
PO2- 80-100 mmHg
HCO3 22-27 mEq/L
ACIDOSIS
-decrease pH
-Potassium increases
AKALOSIS
-increase pH
-Potassium decreases
ROME
respiratory oppossite metabolic equals
-----------------------------------------------------------------------
RESPIRATORY ACIDOSIS
Causes of Respiratory Acidosis (mostly airways/lungs related)
-Asthma: spasms causing the brochioles to constrict
-Atelectasis: excess mucus collection
-Brain trauma: excessive pressure on the respirtory center
-Bronchiectasis: bronchi become dilated as a result of inflammation
-COPD
-Emphysema: loss of elasticity of alveolar sacs, restricting airflow
-Hypoventilation: Carbon dioxide is retained
-Pulmonary Edema: accumulation of fluid in acute CHF
-Medications
Assessment for Respiratory Acidosis
-headache
-restlessness
-drowiness/confusion
-visual disturbances
-diaphoresis
-cyanosis as the hypoxia become acute
-hyperkalemia
-rapid, irregular pulse
-dysrhythmias leading to VFib.
Interventions for Respiratory Acidosis
RESPIRATORY ALKALOSIS
Causes of Respiratory Alkalosis
-Fever (increases metabolism)
-Hyperventilation
-Hypoxia
-Hysteria
-Overventilation by mechanical ventilators
-Pain
-Aspirin
Clinical Manifestations of Respiratory Alkalosis/Assessment
-headache
-tachypnea (initial but decreases) (abnormal rapid respiration)
-paresthesias (tingling of fingers and toes)
-tetany
-vertigo
-convulsions
-hypokalemia
-hypocalcemia
Interventions for Respiratory Alkalosis
-encourage appropriate breathing patterns
-assist with breathing techniques and breathing aids as prescribed (voluntary holding of breath, use
of rebreathing mask, carbon dioxide breaths)
-no deep breathing???? (not sure but please look it up in your book and let me know)
-administer calicum gluconate for tetany as prescribed.
-----------------------------------------------------------------------
METABOLIC ACIDOSIS
Causes of Metabolic Acidosis
-Diabetes Mellitus or Diabetic Ketoacidosis
METABOLIC ALKALOSIS
Causes of Metabolic Alkalosis
-diuretics
-excessive vomitting or gastrointestinal suctioning
-hyperaldosteronism: increased rental tubular reabsorption of sodium occurs, with the resultant
loss of hydrogen ions.
-ingestion of excess sodium bicarbonate/antacids
-massive transfusion of whole blood
Assessment of Metabolic Alkalosis
-nausea, vomiting, diarrhea
-restlessness
-numbness and tingling in the extremities
-twitching in the extremities
-hypokalemia
-hypocalcemia
-dysrhythmias: tachycarida
PATHOPHYSIOLOGY OF ENDOMETRIOSIS
RISK FACTORS:
ETIOLOGY:
UNKNOWN
If Shedding
the egg is of
not
Thickening
of
the
MENSTRUAL
CYCLE:
fertilized,lining
it
endometrial
endometrial
lining
Regurgitation
disintegrates
the
form
offor
ESTROGEN
in in
preparation
Levels
fromofthe
causing
the
menstrual
blood
LEVEL
the
fertilization
of
Estrogen
and
fallopian
tubes
hormones
to
drop
RETROGRADE
HORMONAL
MENSTRUATION
INFLUENCE
Vascular
/
dissemination
of
the endometrial
tissue
LYMPHATIC OR
VASCULAR SPREAD
Endometrial cells
deposited outside
the uterus implant
on structures within
the cavity
Endometrium
build up
Continuously
responds to
menstrual cycle
stimulation
More cells attach
to pelvic
structures
BLEEDING
INFLAMMATION
SCARRING
ADHESION
PHARMACOLOGIC
MNGT:
Nonsteroidal antiinflammatory
drugs or NSAIDs
Gonadotropinreleasing hormone
analogs (GnRH
analogs)
Danazol (Danocrine)
PAIN
S/SX:
Dysmenorrhea
Chronic pelvic
pain
Dyspareunia
Dysuria
Infertility
Florence
the environment
Identified
interventions needed
1. Breathing normally
2. Eating and drinking adequately
3. Eliminating body wastes
4. Moving and maintaining desirable position
5. Sleeping and resting
6. Selecting suitable clothes
7. Maintaining body temperature within normal range
8. Keeping the body clean and well-groomed
9. Avoiding dangers in
the environment
10.
11.
12.
Working in such a way that one feels a sense of
accomplishment
13.
14.
Learning, discovering or satisfying the curiosity that leads
to normal development and health and using available health
facilities.
Nursing
5 basic assumptions:
1.
1. The human being is a unified whole, possessing individual
integrity and manifesting characteristics that are more than and
different from the sum of parts.
2. The individual and
Identified 4 phases of
relationship:
1. Orientation - individual/family has a felt need and seeks
professional assistance from a nurse (who is a stranger). This is
the problem identification phase.
2. Identification - where the patient begins to have feelings of
belongingness and a capacity for dealing with the problem,
creating an optimistic attitude from which inner strength ensues.
Here happens the selection of appropriate professional
assistance.
3. Exploitation -
to offer services to
Carative Factors:
1.
1. The promotion of a humanistic-altruistic system of values
2. Instillation of faith-hope
3. The cultivation of sensitivity to ones self and others
4. The development and acceptance of the expression of positive
and negative feelings.
5. The systemic use of the scientific problem-solving method for
decision making
6. The promotion of interpersonal teaching-learning
7. The provision for supportive, protective and corrective mental,
physical, socio-cultural and spiritual environment
8. Assistance with the gratification of human needs
9. The allowance for existential phenomenological forces
A hernia occurs when the contents of a body cavity bulge out of the area where they are normally
contained. These contents, usually portions of intestine or abdominal fatty tissue, are enclosed in
the thin membrane that naturally lines the inside of the cavity
A hernia is an abnormal protrusion of part of the intestines through a weak part of the
abdomen, that is, part of the guts is poking through the abdominal wall. Normally, the front of
the abdomen has several layers comprising of skin then fat then muscles and broad
ligaments.Underneath all these lie the intestines (guts). If, for any reason, there is a weak point in
the muscles or ligaments, part of the intestines push through. You can then feel a soft lump under
the skin.
TYPE
Femoral hernia : The femoral canal is the path through which the femoral artery, vein, and nerve
leave the abdominal cavity to enter the thigh.
Umbilical hernia : These common hernias (10%-30%) are often noted at birth as a protrusion at
the bellybutton (the umbilicus). This is caused when an opening in the abdominal wall, which
normally closes before birth, doesn't close completely
Incisional hernia : Abdominal surgery causes a flaw in the abdominal wall. This flaw can create
an area of weakness in which a hernia may develop.
Spigelian hernia : This rare hernia occurs along the edge of the rectus abdominus muscle, which
is several inches to the side of the middle of the abdomen.
Obturator hernia : This extremely rare abdominal hernia develops mostly in women. This hernia
protrudes from the pelvic cavity through an opening in the pelvic bone (obturator foramen).
Epigastric hernia : Occurring between the navel and the lower part of the rib cage in the midline
of the abdomen, epigastric hernias are composed usually of fatty tissue and rarely contain
intestine.
Inguinal hernia is An a condition in which intra-abdominal fat or part of the small intestine, also
called the small bowel, bulges through a weak area in the lower abdominal muscles.
CAUSES
Although abdominal hernias can be present at birth, others develop later in life. Some
involve pathways formed during fetal development, existing openings in the abdominal cavity, or
areas of abdominal-wall weakness.
Any condition that increases the pressure of the abdominal cavity may contribute to the
formation or worsening of a hernia. Examples include
obesity,
heavy lifting,
coughing,
Nursing
Interventions
1. Verify
doctors
order.
Inform the
client and
explain
the
purpose of
the
procedure.
2. Check for cross matching and typing. To ensure compatibility
3. Obtain and record baseline vital signs
4. Practice strict asepsis
5. At least 2 licensed nurse check the label of the blood transfusion. Check the following:
o Serial number
o Blood component
o Blood type
o Rh factor
o Expiration date
o Screening test (VDRL, HBsAg, malarial smear) this is to ensure that the blood
is free from blood-carried diseases and therefore, safe from transfusion.
6. Warm blood at room temperature before transfusion to prevent chills.
7. Identify client properly. Two Nurses check the clients identification.
8. Use needle gauge 18 to 19 to allow easy flow of blood.
9. Use BT set with special micron mesh filter to prevent administration of blood clots and
particles.
10. Start infusion slowly at 10 gtts/min. Remain at bedside for 15 to 30 minutes. Adverse
reaction usually occurs during the first 15 to 20 minutes.
11. Monitor vital signs. Altered vital signs indicate adverse reaction (increase in temp,
increase in respiratory rate)
12. Do not mix medications with blood transfusion to prevent adverse effects. Do not
incorporate medication into the blood transfusion. Do not use blood transfusion lines for
IV push of medication.
13. Administer 0.9% NaCl before; during or after BT. Never administer IV fluids with
dextrose. Dextrose based IV fluids cause hemolysis.
14. Administer BT for 4 hours (whole blood, packed RBC). For plasma, platelets,
cryoprecipitate, transfuse quickly (20 minutes) clotting factor can easily be destroyed.
15. Observe for potential complications. Notify physician.
CARDIAC ARRYTHMIAS
1. Terminology
a. QRS depolarization always refer to ventricular (not atrial,
junctional, or nodal)
b. P wave refers to atrial
2. Six rhythms tested on NCLEX
a. Asystole
i. A lack of QRS depolarizations (a straight line)
b. Atrial flutter
i. Rapid Pwave
depolarizations in a sawtooth
(flutter)
c. Atrial fibrillation
i. Chaotic Pwave
depolarizations (lacks any discernable
pattern)
d. Ventricular fibrillation
i. Chaotic QRS depolarizations
e. Ventricular tachycardia
i. Wide, bizarre QRSs
ii. Tachy is always discernable repeating pattern
f. Premature ventricular contractions (PVC)
i. Periodic wide, bizarre QRSs
ii. Generally low to moderate priority. unless everyone else has
a normal rhythm
iii. Be concerned, if:
1. More than 6 per minute
2. 6 in a row
3. PVC falls of T-wave of previous beat
3. Lethal arrhythmias
a. Asystole
b. Vfib
4. Potentially life threatening arrhythmia: V-tach
a. Pulseless vtach
same as asystole and v. fib and would depend on
how long down
b. After 8 mins consider dead
5. Treatment
a. PVCs
i. Lidocaine (Ventricular, lasts longer) , Amiodorone
b. V Tach
i. Lidocaine
c. Supraventricular arrhythmias
i. Adenosine (push fast IV push usually 8s or faster)
ii. Beta-Blockers (lol)
iii. Calcium Channel Blockers
iv. Digoxin (Digitalis) Lanocin
d. VFib
i. Best treatment electrically
ii. Shock = 200 Defibrillate
e. Asystole
i. Epinephrine
ii. Atropine
iii. S/E anticholinergics