Risk Factors Change in the condition Thrombolyti

of the plaque in the c Therapy

Glycoprotei
coronary artery n IIB/IIIA
receptor
Aspirin antagonist
Activation of
Antiplate ECG
Platelets
let Lab.
CT-Scan,
aggregat Test
Formation of MRI
es
thrombus Elevate
d ST
Ischemia of tissue in the segmen Elevated
Nitrat region supplied by the tQ CK-MB,
Altered repolarization of wave
es artery Elevated
the myocardium appears myoglobi
n,
Coronary blood supply
Troponin
Beta less than demand Release of lysosomal T,Toponi
-Block enzyme nI
ers
Myocardial cell death Anaerobic Glycolysis Lactic Acid
Production
Nitroglyc
Myocardial irritability

Oxyge
n Decreased contractility
Angi
na

Stimulation of the Dysrhyt

sympathetic nervous mias
system

Antidysrhyt
hmias
 Ventricular Remodeling

Heart Failure

Increa Decreased left ventricular

sed function Angiotens
heart in DEATH
rate convertin
Increased g enzyme
Oxygen needs inhibitors

Decreased
Increase
cardiac output
Increase preload Cardiogenic
afterload Shock
Nitrat
es Increa Ndx:
Decre
sed Decreased
ased
Vasoconstri CVP Cardiac output
LV
Increa related to
ejectio
sed negative inotopic
n
PCWP changes in the
Fluid fractio
heart secondary
Restricti n
to myocardial
on ischemia

Legend :

Treatment Clinical Manifestations Complications Pathophysiology Diagnostic

Exam Nursing Diagnosis