Beruflich Dokumente
Kultur Dokumente
Chapter 21
o
- Pain assessment
o The goal is to provide accurate info on location and intensity of
pain; vital signs not a reliable assessment of pain in children
o Pain assessment scales for Newborns
Neonatal Infant Pain Scale
Chapter 26
- Pediatric Differences
o Fetal Circulation
Blood flows from the placenta to the fetus through the
umbilical vein. The blood then flows through thte ductus
venosus allowing the blood to enter the right atrium of the
heart. The foramen ovale allows the blood to flow to the
left atrium then left ventricle the to the aorta and into
systemic circulation.
Next blood returns to the right atrium through the superior
vena cava and flows to the right ventricle where its
pumped into the pulmonary artery. Majority of the blood
passes though to the ductus arteriosus, the fetal vascular
channel between the pulmonary artery and the descending
aorta, to enter systemic circulation. Eventually the blood
returns to the placenta through the umbilical arteries.
o Pulmonary Circulation
Once the umbilical cord is cut the baby must adapt quickly
to receciving oxygen in the lungs. Pressure in the left
atrium increases as blood flow is return from the lungs
through the pulmonary veins.
Systemic vascular resistance increases and pressure in the
left atrium stimulates closure of the foramen ovale, and
fibrin seals it.
Ductus arteriousus constricts na closes within 10 to 15
hours after birth, permanent closure 10-21 days after birth.
o Cardiovascular Changes
Right ventricle larger at birth due to high pulmonary
resistance during fetal life but it reverses over time as high
systemic vascular pressure forces left ventricle to develop
quickly
Neonates with conditions like alveolar hypoxia, acidosis,
and hypothermia may develop prolonged pulmonary
vasoconstriction
o Oxygenation
Oxygen bound to hemoglobin is transported to the tissues
by the systemic circulation
Desaturated blood results when oxygenated and
unoxygenated blood mix due to a congenital heart defect
as well as a hematocrit of 50%+ because their bodies
attempt to increase available oxygen to the tissues by
increasing RBC production; increased risk of
thromboembolism
o Cardiac functioning
Cardiac output is the volume of blood ejected from the left
ventricle each minute controlled by
HR, Preload, contractility, afterload
Infant lack of compliance decreases the hearts ability to
increase stroke volume so they become tachycardic in
stressful situations to increase cardiac output
- Congenital Heart Disease
o Defect in the heart or great vessels, persistence of fetal structure
after birth
Most occur during during first 8 weeks of gestation where
they are most vulnerable to teratogens due to a combined
effect of environmental and genetic facors
Increased maternal age, prematurity, drugs, alcohol,
smoikking, viral infections, genetic factors (25%)
o S/S
Murmur usually first sign of congenital heart defect; blood
flowing with higher pressure than normal to get through a
narrow valve
Neworn may be initially asymptomatic but develop
symptoms in the first few days of life
Other signs: chest pain, exercise intolerance, arrhythmias,
syncope, and sudden death.
o Treatment
-
- Nursing management of child undergoing cardiac catheterization
o NPO severeal hours before except for meds, arrive 1-2 hours
before procedure, void and take oral sedative
o Physio assessment: vital signs, H&H, cap refill, baseline skin
temp/color,
o Psych assessment: focus on parents knowledge deficit, anxiety,
and child separation from parents
o Nurse planning
Educate and prepare parents and child on procedure,
maybe a tour of the cath lab with the child
Child stays on bedrest 4 to 6 hours to keep leg straight,
activity limited for 24 hours
Small amounts of clear liquid with steady increasing
amounts as the child can tolerate
- Congenital Heart Defects that INCREASE pulmonary blood flow
o A connection occurs between the left and right side of the heart,
increasing the blood that is pumped to the lungs
Increased pulmonary blood flow increases pulmonary
vascular resistance to reduce flow causing right ventricular
hypertrophy delivering more blood to the lungs
With the pulmonary system overloaded with blood, you may see dyspnea,
tachypnea, intercostal retractions, and periorbital edema
o Treatment
Surgery to correct defects should happen early to prevent
irreversible pulmonary artery hypertension which usually
leads to full recovery
Conservative treatment includes waiting for symptoms to
appear before performing any procedure; sometimes a
defect may fix itself like a ventricular septal defect closing
on its own
o Nursing Management Prior to surgery
Physiologic assessment: assess growth (should be normal,
any lack of growth could an increased metabolic rate and
poor consumption of appropriate calories) and look for CHF
signs (increased resp. effort, poor feeding, diaphoresis,
fatigue, recurrent pulmonary infections).
Home care: Focus on growth by encouraging frequent
feeding for no longer than 30 minutes, especially
breastfeeding with all its benefits
o Nursing Management during Surgery
Assessment and Diagnosis
Take a careful history and physical exam to detect
presence or potential development of any acute
illness
Critical Care
Postop child ill be in ICU to be monitored with
invasive hemodynamic techniques, labs, and physical
examination (basically full head to toe)
Transfer to general nursing unit
Focus assessment on surgical complications like the
infection, rrhytmias, impaired tissue perfusion
Monitor all vital signs and inspect incision site, full
head to toe, I&O
ICU
Child is intubated and on a ventilator, suction is
performed, IVS placed, fever prevented, antibiotics
given prophylaxis
Pain: IV until able to take PO
Take deep breaths, cough, do incentive spirometry,
splint to promote respiratory function
Fluids/Nutrition
Encourage fluid intake while monitoring I&O
Have parents bring favorite foods to motivate eating
and promote bowel movement after surgery
Encourage gradual increase of activity every day,
provide diversional activities for pain and anxiety
Discharge
Info on diagnosis/surgery, meds, wound care,
nutrition
o Spread over a few days
Reassure parents of the should have no
complications and promote return to normal living
- Defects causing DECREASED pulmonary blood flow and mixed defects
o Obstruction causing decreased blood flow to the lungs to be
oxygenated, a septal opening between atria and ventricles lead
to right to left shunting
o Kidneys produce erythropoietin to stimulate RBC production, too
much RBC (polycythemia) can lead to sluggish blood flow
increasing risk of thromboembolism in cerebral and pulmonary
vessels
o Infants may wake with cyanosis and experience a sudden
decrease in systemic vascular resistance and pulmonary blood
flow triggering a hypercyanotic episode
Combine this with increased cardiac output and venous
return from a stress reaction and the brain overreacts
causing increased resp. effort further increasing cardiac
output contributing to a life threatening decline unless
rapid intervention is successful
o MIXED DEFECTS
Mixing of oxygen saturated and desaturated blood results
in a general desaturated systemic blood flow and cyanosis.
Pulmonary congestion occurs because of increased
pulmonary blood flow and obstruction of systemic flow
o S/S
Cyanosis shortly after birth that doesnt respond to 02,
dyspnea, and a loud murmur
Severe obstruction leads to hypercyanotic episodes
Treatment: squat to relieve dyspnea because it
reducse cardiac output by decreasing venous return
and increasing systemic vascular resistance
S/s: increased rate and depth of respirations,
increased cyanosis, pallor and poor tissue perfusion,
increased HR, diaphoresis, irritability and crying,
seizures and LOC
o Collaborative Care (Mixed Defects)
Diagnostics: hypoxia test when <93%,
Treatment: corrective surgery in newborns, palliative
procedure (atrial balloon septostomy) to preserve life first
then corrective surgery,
Closure of the ductus arteriosus: PGE1 reopens it
providing time for newborn to be transferred for
diagnostic evaluation and surgical intervention
o Adverse effects of PGE1: respiratory depression
and apnea
Hypercyanotic episodes: oral propranolol, knee-chest
position,
Long term therapy: children with complex congenital
heart defects require lifelong assessment and med
care
o Nursing Management
Focus on PGE1 therapy, treating hypercyanotic episodes,
and providing postsurgical care
Prior to surgery
At risk for CHF so monitor tachycardia, crackles,
tachypnea, secretions, low urine output and
pulmonary edema,
Monitor all vital signs and assess growth and cardiac
deterioration
Following surgery
Assess all vitals, I&O
Home care of the child before surgery
Referalls to community based early intervention
programs so that specialists can set goals for the
child
Treat child as normally as possible without adjusting
activity too much
Observe signs of worsening cyanosis, especially in
the morning
o Know baseline sp02 to know bad changes
o Call 911 and stay calm, place child in knee
chest position
Report signs of illness to the physician asap, fever
increases metabaolic rate and causes further stress
on the heart
Antipyretic meds and fluid volume replacement
necessary
- Defects OBSTRUCTING systemic blood flow
o An anatomic stenosis causes obstruction of blood flow,
increasing pressure on the left ventricle with decreased cardiac
output and potential shock
o The blood cant move past the obstruction backing up into the
left atrium and the lungs leading to CHF and pulmonary edema
- Congestive Heart Failure
o Cardiac output inadequate to support the bodys circulatory and
metabolic needs
Causes: congenital heart defect that increases pulmonary
blood flow or obstruction of systemic outflow tract
S/s: early -infant tires easily especially in feeding, weight loss or lack of
normal weight gain, diaphoresis, irritability, and frequent respiratory
infections, later tachypnea, tachycardia, pallor, cyanosis, nasal flaring,
grunting, retractions, cough, or crackles, cardiomegaly enlargement of
the heart with the heart compensating to maintain cardiac output