COMMUNICATING PATHOPHYSIOLOGY

PREDISPOSING FACTORS: PRECIPITATING

-Genetics FACTORS:
- Age - NONE
CSF forms in the Choroid
plexus of the lateral ventricles

CSF flows from the Foramen of
Monro to the third ventricle

Impaired Absorption
Obstruction of the CSFof
in the aqueduct
in the Arachnoid space
Sylvius due to incomplete
formation of the lateral and
medial foramina

Dilatation of the third and

lateral ventricles

Obstruction of CSF flow Increased intra-

through the ventricular system ventricular pressure

Continued enlargement
disrupts the ventricular
lining and then the
underlying white matter
 Increase in water content due
to cerebral edema of CSF Bulging and protrusion of the
from elevated intra- eyes or sunset eyes
ventricular and intracranial
pressure

Expansion of the
Axonal and myelin skull and thinning
destruction and atrophy

Contraction of the
Enlarged head cerebral blood
volume

Alteration of
cerebral circulation

CSF circulation is
Irritability
altered

PROGNOSIS

IF TREATED: -Motor Skill IF UNTREATED:

disabilities
-Learning disabilities -Vision problems -Decompensatory mechanisms may continue to
occur:
-Memory deficits -Hearing difficulties,
Decreased Cerebral Perfusion ,Decreased PO2
-Psychological deficits -Seizures, and leading to Hypoxia and Brain Damage
-Hormonal imbalances

DEATH