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The following features help to distinguish jugular from carotid artery pulsa
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There are several points to remember prior to beginning the examination of the CVS. These include:
1. Review the blood pressure and heart rate recorded during the General Survey and Vital Signs at the start of the
physical examination.
2. Take the time to measure the blood pressure and heart rate using optimal technique.
3. Systematically organize your examination following these components:
a. The jugular venous pressure
b. The carotid upstrokes and presence or absence of bruits
c. The point of maximal impulse(PMI)and any heaves, lifts, or thrills
d. The first and second heart sounds, S1 and S2
e. Presence or absence of extra heart sounds such as S3 or S4
f. Presence or absence of any cardiac murmurs.
§ Decreased pulsations may be caused by decreased stroke volume, but may also be due to local factors in the artery - During the cardiac examination, remember to correlate your findings w
such as atherosclerotic narrowing or occlusion. carotid pulse. It is also important to identify both the anatomic location
§ Amplitude: Small, thready, or weak pulse in cardiogenic shock; bounding pulse in aortic insufficiency cycle.
§ The contour of the pulse wave, namely the speed of the upstroke, the duration of its summit, and the speed of the § Note the anatomic location of sounds in terms of interspace
downstroke. The normal up- stroke is brisk. It is smooth, rapid, and follows S1 almost immediately. The summit is midclavicular, or axillary lines.
smooth, rounded, and roughly midsystolic. The downstroke is less abrupt than the upstroke. § Identify the timing of impulses or sounds in relation to the c
o Delayed carotid upstroke in aortic stenosis
GRADING OF MURMURS
GRADE DESCRIPTION
1 Very faint, heard only after listener has “tuned in”; may not be
2 Quiet, but heard immediately after placing the stethoscope on
3 Moderately loud
4 Loud, with palpable thrill
5 Very loud, with thrill. May be heard when the stethoscope is pa
6 Very loud, with thrill. May be heard with stethoscope entirely o
shape of murmur
§ Shape
§ Location of Maximal Intensity. This is determined by ⇒ For example, a murmur best heard in the 2nd right
the site where the murmur originates. interspace usually originates at or near the aortic valve.
IMPORTANT SIDE NOTES IMPLICATIONS
Opening snap (diastole) Mitral stenosis
S3 Occurs during diastole as a result of the rapid deceleration of
column of blood against the ventricular wall
S4 Marks the atrial contraction (precedes the S1)
2ND- 3RD, Left parasternal border Splitting (S2)
Tricuspid area at the lower left Splitting (S1)
Wide and fixed S2 splitting ASD, LV dysfunction
Wide S2 splitting Right bundle branch block, mitral regurgitation, pulmonary stenosis
Paradoxycal splitting Narrow in inspiration, wide in expiration à left BBB
Soft S1 Severe LV dysfunction, CHD, severe MR
Decreased S1 1st degree block
Decreased S2 Aortic stenosis
Thrills with harsh, rumbling murmurs AS, PDA, VSD
Upward apical pulse Pregnant
Laterally displaced apical pulse Cardiomegaly due to CHF, cardiomyopathy, ischemic HD
Decreased apical pulse Obesity, tick chest and increased AP diameter
Increased amplitude Normal: young patient after exercise
Abnormal: hyperthyroidism, severe anemia, pressure overload due
to AS and volume overload due to MR
Increased amplitude, but normal duration ASD
Increased amplitude, increased duration PS, pHPN
Palpable S2 at the pulmonic area pHPN
Palpable S2 at the aortic area HPN
Small weak pulses ◦ Decreased stroke volume (e.g. heart failure, hypovolemia,
severe AS)
◦ Increased PVR (e.g. cold and severe CHF)
Large, bounding pulse ◦ Increased SV and decreased PVR or both (e.g. fever,
hyperthyroidism, aortic regurgitation, AV fistula, PDA
◦ Increased SV due to bradycardia and complete heart block
◦ Decreased compliance (e.g. atherosclerosis)
Bisferiens pulse Increased pulse w/ double systolic peak (e.g. pure AR or AR w/ AS)
Pulse alternans LV failure with left- sided S3
Bigeminal pulse Alternating beat w/ premature contraction
Paradoxical pulse Decreased amplitude during quiet inspiration, pericardial
tamponade, constrictive pericarditis and restrictive lung diseases.