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Causes: Multi-factorial
1. Genetics
2. Environmental
3. Maternal Diseases
Diagnosis:
Ultrasound
Physical Assessment
CL is apparent at birth
CP may not be detected without thorough assessment of the mouth
Management: Surgery
Nursing Care:
Incidence:
Clinical Manifestations:
1. Projectile vomiting
* Nonbilous
* Blood Tinged
2. No evidence of pain or discomfort except that of chronic hunger
3. S/s of dehydration -decreased number of stools, weight loss.
4. Distended upper abdomen
5. Readily palpable olive mass in the upper abdomen.
6. Visible peristaltic waves
7. Hyperactive Bowel sounds
Diagnosis:
Management: Surgery
* Esophageal Atresia
- failure of the esophagus to develop a continuous passage
- esophagus ends in a blind pouch with no entry to the stomach
* Tracheoesophageal Fistula
- abnormal opening between the esophagus and the trachea
Incidence:
- rare
- equal sex incidence
- low birth weight
- prematurely
Cause: unknown
Types:
Name Description
A Simple Esophageal atresia with no fistula Upper and lower ends of the esophagus are blind,
(Isolated EA) no connection to the trachea
B EA with proximal TEF Upper end of the esophagus ends into the trachea
C EA with Distal TEF Upper end of esophagus is blind, lower end
connects into the trachea - most common
D EA with double TEF Both upper and lower ends of the esophagus open
into the trachea by a fistula
E Isolated TEF Esophageal atresia but with fistula between
normal esophagus and trachea
Clinical Manifestation:
Diagnosis:
Management:
1. Prevention of aspiration pneumonia
2. Surgery to repair anomaly
- Several stages- indicated for premature, multiple anomalies, in poor condition
LIGATION OF TEF
Gastrostomy - for gastric decompression and jejunostomy feeding
Provision of constant drainage of the esophageal pouch - tube in the mouth to
upper pouch
Complications of Surgery:
Anastomostic leak
Stricture due to tension
Esophageal motility disorders
Nursing Care:
* Pre-operative
1. Maintain open airway
2. NPO
3. Administer IVF as ordered
4. Oxygen to relieve respiratory distress
5. Positioning
* Post-operative
1. Promote respiratory function
2. Provide optimum nutrition
A. Gastrostomy tube feedings
* elevated and secured t a point above level of the stomach (4‘’)
B. Oral feeding when anastomosis site is healed
C. Maintain IVF
D. Use of Pacifier
E. Diet - Liquid - small frequent feedings
4. GASTROESOPHAGEAL REFLUX
- return of gastric contents into the esophagus
Risk Factors:
1. Prematurity
2. Neurologic disorders
3. Asthma
4. TEF/EA repair
5. Gastrostomy placement
6. Neurologic disorders
Causes:
1. Related to the dysfunction of the lower esophageal sphincter (LES)
2. Delayed gastric emptying
3. Increased abdominal pressure
Clinical Manifestations:
1. Passive regurgitation, spitting up/vomiting
2. Poor weight gain
3. Hematemesis and melena
4. Irritability or excessive crying
5. Heartburn in older clients
6. Anemia
7. Respiratory problems - pneumonia, chronic cough, wheezes, stridor, gagging, choking at end of
feeding
8. Dysphagia
Diagnosis:
1. History and clinical manifestations
2. Barium swallow (esophagography)
3. 24 hour pH probe study
Complications of GERD
1. Persistent esophagi is with scarring
2. Recurrent aspiration pneumonia
* Barret’s syndrome disorder of the lower esophagus marked by benign ulcer like lesions resulting
most often from chronic irritation of the esophagus by gastric reflux of acidic digestive juices
Management:
1. Diet
2. Pharmacologic
A. H2 antagonist
- Cimetidine (Tagamet), Famotidine (Pepcid), Ranidine (Zantac)
B. Proton Pump Inhibitor
- Omeprazol (Prilosec), Lansoprazole (Prevacid)
C. Prokinetic Agents
- Metochlopromide Hcl (Placil)
5. INTUSSUSCEPTION
- An invagination or telescoping of one portion of intestines into another
Incidence:
- onset: abrupt
- one of the most frequent cause of intestinal obstruction between 3 months to five years of age; 50
% below 1 year (3-12 mos.) of age
- 2X greater in males than in females
Cause: unknown , likely a result of hypertrophy of intestinal lymphoid tissue secondary to viral infection
Clinical Manifestations:
1. Pain (acute), severe, colicky abdominal pain
2. Vomiting
3. Passage of red currant jelly like stools
4. Palpable sausage-shaped in RUQ
5. Empty RLQ (Dance sign)
6. Tender, distended abdomen
7. Lethargy
Complications:
1. Perforation
2. Peritonitis
3. Sepsis
Diagnosis:
1. History and physical exam
2. Abdominal radiograph to detect intraperitoneal air from a bowel perforation
3. Barium enema.
4. Ultrasound
5. Rectal Exam
Management:
1. Non-surgical
A. HYDROSTATIC REDUCTION
B. PNEUMATIC INSUUFFLATION
2. Surgical-manual reduction of invagination and resection of nonviable intestines when
necessary
Nursing Care:
1. Monitoring for S/s of complications
2. Maintain or establishing fluid and electrolyte balance
3. Preparing for hydrostatic reduction and/or surgery
Incidence:
- 1:5000
- Accounts for ¼ of all cases of neonatal obstruction although diagnosed in later infancy/childhood
- 4X greater in males than in females
- More common in children with Down’s syndrome
- can present as an acute or chronic disorder
Complications:
1. Obstruction
2. Bowel perforation
A. Peritonitis - s/s: relief of pain, increased RR and PR, fever, vomiting, absence of
bowel sounds and increased abdominal distention
B. Bleeding
3. Enterocolitis - s/s: fever, explosive watery diarrhea, dehydration, severe prostration
4. Dehydration
Diagnosis:
1. History and clinical manifestations
2. Barium enema
3. Rectal Biopsy
Nursing Care:
1. Monitoring vs. including abdominal circumference and ominous sign (enterocolitis)
2. Maintain fluid and electrolyte balance
3. Maintain adequate nutrition
4. Help parent adjust to the congenital defect in the child
5. Prepare client for surgery
6. Post-OP care
7. Discharge - assisting parents in proper colostomy care