Chronic Bronchitis

 Is an inflammation of the lower airways

characterized by excessive secretion of mucus,
hypertrophy of mucous glands, and recurring
infection, progressing to narrowing and
obstruction of airflow.

Assessment:

1. Signs and symptoms of chronic bronchitis

(insidious onset):

 Productive cough lasting at least 3 months

during a year for 2 successive years.
 Thick, gelatinous sputum (greater amounts produced during superimposed infections).
 Dyspnea and wheezing as disease progresses.

Diagnostic Evaluation:

1. Pulmonary function tests, to demonstrate airflow obstruction-reduced forced

expiratory volume in 1 second (FEV1), FEV1 to forced vital capacity ratio; increased
residual volume to total lung capacity (TLC) ration, possibly increased TLC.
2. Chest X-rays to detect hyperinflation, flattened diaphragm, increased retrosternal space,
decreased vascular markings, possible bullae (all in late stages).
3. Arterial blood gases, to detect decreased arterial oxygen pressure (PaO2), pH, and
increased arterial carbon dioxide pressure (Paco2).
4. Sputum smears and cultures to identify pathogens.

Therapeutic and Surgical Interventions:

1. Smoking cessation to stop the progression and preserve lung capacity.

2. Low-flow oxygen to correct severe hypoxemia in a controlled manner and minimize
carbon dioxide retention.
3. Home oxygen therapy, especially at night to prevent turnal oxygen desaturation.
4. Pulmonary rehabilitation to reduce symptoms that limit activity.
5. Chest physical therapy, including postural drainage and breathing retraining.
6. Lung transplant in severe cases of alpha1-antitrypsin deficiency.

Pharmacologic Intervention:

1. Bronchodilators to reduce dyspnea and control bronchospasm delivered by metered-dose

inhaler, other handheld devices, or nebulization.
2. Inhaled corticosteriods may be useful for some with severe airflow limitation and
frequent exacerbations.
3. Corticosteroids by mouth or I.V. in acute exacerbations.
 4. Antimicrobials to control secondary bacterial infections in the bronchial tree, thus
clearing the airways.
5. Alpha1-antitrypsin replacement delivered by I.V. infusion.

Nursing Interventions:

1. Monitor for adverse effects of bronchodilators-tremulousness, tachycardia, cardiac

arrhythmias, central nervous system stimulation, hypertension.
2. Monitor oxygen saturation at rest and with activity.
3. Eliminate all pulmonary irritants, particularly cigarette smoke. Smoking cessation usually
reduces pulmonary irritation, sputum production, and cough. Keep the patient’s room as
dust-free as possible.
4. Use postural drainage positions to help clear secretions responsible for airway
obstruction.
5. Teach controlled coughing.
6. Encourage high level of fluid intake (8 to 10 glasses; 2 to 2.5 L daily) within level of
cardiac reserve.
7. Give inhalations of nebulized saline to humidify bronchial tree and liquefy sputum. Add
moisture (humidifier, vaporizer) to indoor air.
8. Avoid dairy products if these increase sputum production.
9. Encourage the patient to assume comfortable position to decrease dyspnea.
10. Use pursed lip breathing at intervals and during periods of dyspnea to control rate and
depth of respiration and improve respiratory muscle coordination.
11. Discuss and demonstrates relaxation exercises to reduce stress, tension, and anxiety.
12. Encourage frequent small meals if the patient is dyspneic; en a small increase in
abdominal contents may press on diaphragm and impede breathing.
13. Offer liquid nutritional supplements to improve caloric intake and counteract weight loss.
14. Avoid foods producing abdominal discomfort.
15. Encourage use of portable oxygen system for ambulation for patients with hypoxemia
and marked disability.
16. Encourage the patient in energy conservation techniques.