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Zinc phosphate and glass-ionomer cements are not recommended for use with milled
ceramic restorations</td></tr><tr valign=top height="15" style="background-
color:White;"><td width="30" style="border:solid 1px black; border-
collapse:collapse ">8</td><td width="250" style="border:solid 1px black; border-
collapse:collapse ">thermal shock : cracking and crazing of the fired dental
porcelain results due to</td><td width="200" style="border:solid 1px black;
border-collapse:collapse ">A: too rapid cooling after glazing<br><br>B: Very slow
cooling<br><br>C: both of the above<br><br>D: none of the above<br><br></td><td
width="50" style="border:solid 1px black; border-collapse:collapse ">C</td><td
width="250" style="border:solid 1px black; border-collapse:collapse ">reference:
Craig dental materials page no: 556
Explanation:
Too-rapid cooling of the outer layers may result in surface crazing or cracking;
this is also called thermal shock.
Very slow cooling (e.g., in a furnace) as well as multiple firings, might induce
the formation of additional leucite and increase the overall coefficient of thermal
expansion of the ceramic, and may also result in surface cracking and crazing.
General visceral Efferents (GVEor VE) viscera that receive parasympathetic supply
III (Edinger westphial nucleus
VII (Superior salivatory nucleus)
IX (Inferior salivatory nucleus)
X (Dorsal motor nucleus)</td></tr><tr valign=top height="15" style="background-
color:#EFF3FB;"><td width="30" style="border:solid 1px black; border-
collapse:collapse ">53</td><td width="250" style="border:solid 1px black; border-
collapse:collapse ">Which of this part of vertebral canal will show secondary
curves with concavity backwards?</td><td width="200" style="border:solid 1px
black; border-collapse:collapse ">A: Cervical<br><br>B: Thoracic<br><br>C:
Sacral<br><br>D: Coccyx<br><br></td><td width="50" style="border:solid 1px black;
border-collapse:collapse ">A</td><td width="250" style="border:solid 1px black;
border-collapse:collapse ">Primary Curves: Embryonic and Fetal Curvatures
• Embryonic body appears flexed
• Curves are convex dorsally (concave anteriorly)
• Primarily in the thoracic and pelvic region.
Erythropoetin acts on Erythroid precursors in the bone marrow and stimulates the
rate of red cell production
This results in:
Increased Reticulocyte count
Increased Haematocrit
Increased oxygen carrying capacity of blood (Increased Haemoglobin)</td></tr><tr
valign=top height="15" style="background-color:White;"><td width="30"
style="border:solid 1px black; border-collapse:collapse ">58</td><td width="250"
style="border:solid 1px black; border-collapse:collapse ">A 30 year old female
patient presents with non progressive dysphagia, for both solids and liquids. The
characteristic finding on baruim swallow that will confirm the probable diagnosis
is:</td><td width="200" style="border:solid 1px black; border-collapse:collapse
">A: Dilated esophagus with narrow lower end (Rat tail esophagus)<br><br>B:
Multiple sacculations and pseudodiverticulae (Corkscrew esophagus)<br><br>C: Narrow
and irregular esophageal lumen<br><br>D: Stricture ulcer in the
esophagus<br><br></td><td width="50" style="border:solid 1px black; border-
collapse:collapse ">B</td><td width="250" style="border:solid 1px black; border-
collapse:collapse ">
• The presence of non progressive dysphagia to both solids and liquids suggests the
diagnosis of diffuse esophageal
spasm. The charachteristic barium swallow findings in diffuse esophageal spasm is
the corkscrew esophagus
• Rat tail esophagus with a dilated proximal esophagus and narrow lower end
suggests a diagnosis of Achlasia
cardia. Achlasia is associated with Progressive dysphagia to both solids and
liquids</td></tr><tr valign=top height="15" style="background-color:#EFF3FB;"><td
width="30" style="border:solid 1px black; border-collapse:collapse ">59</td><td
width="250" style="border:solid 1px black; border-collapse:collapse ">A 28 year old
patient has a FPD in 11, 12, 21 and 22 region following severe trauma. He is quite
happy with the prosthesis except for the black triangles which are present in the
area of gingival embrasures and is of esthetic concern to the patient? How can you
modify the prosthesis to enhance the patients esthetics?</td><td width="200"
style="border:solid 1px black; border-collapse:collapse ">A: use pink porcelain on
the old FPD, hence covering the gingival embrasures this enhancing the
look.<br><br>B: fabricate a new prosthesis<br><br>C: Andrews bridge<br><br>D:
Either A or C<br><br></td><td width="50" style="border:solid 1px black; border-
collapse:collapse ">D</td><td width="250" style="border:solid 1px black; border-
collapse:collapse ">Reference: Nallaswamy, Textbook of prosthodontics, pg no: 515
Explanation:pontic modifications:
if edentulous space is resorbed, large gingival embrasures (black triangles appear)
these can be treated by:
a.pink porcelain
b. or by fabricating Andrews bridge
Andrews bridge: Fixed removable modified fpd. It has afixed bar connected to the
lingual surface of the retainer whereas the removale prosthesis is attached onto
the bar.
</td></tr><tr valign=top height="15" style="background-color:White;"><td width="30"
style="border:solid 1px black; border-collapse:collapse ">60</td><td width="250"
style="border:solid 1px black; border-collapse:collapse ">Degeneration of the
basement membrane is mediated by:</td><td width="200" style="border:solid 1px
black; border-collapse:collapse ">A: Oxidases<br><br>B: Elastases<br><br>C:
Hydroxylases<br><br>D: Metallo proteinase<br><br></td><td width="50"
style="border:solid 1px black; border-collapse:collapse ">D</td><td width="250"
style="border:solid 1px black; border-collapse:collapse ">Basement membrane is a
component of ECM and largely consists of collagen (Type IV).
Matrix Mettaloproteins MMP9 and MMP2 are collagenases that cleave type IV collagen
of epithelial and vascular
basement membranes
ProvocativeTests:
1) (ScaleneTest)Adson test: Patient is instructed to: take breath and hold it,
extend the neck fully, turn the face towards the side---Obliteration or the
diminution of the radila pulse suggest the diagnosis.
2) MilitaryPosition)CostoclavicularTest: pateint instructed to Draw shoulders
downwards---Obliteration of radial pulse or reproduction of symptoms indicates
compression
OTHER FACTS:
• Small (Narrow) cuff leads to falsely high blood pressure recordings
(overestimation)
• Large (wide) cuff leads to falsely lower blood pressure recordings (under
estimation)
Note
• More accurate pressure can be recorded in obese individuals by using a wider cuff
size
• More accurate pressure can be recorded in the thigh by using a wider cuff
size</td></tr><tr valign=top height="15" style="background-color:#EFF3FB;"><td
width="30" style="border:solid 1px black; border-collapse:collapse ">69</td><td
width="250" style="border:solid 1px black; border-collapse:collapse ">Which of the
following hormone is not secret ed by the kidney:</td><td width="200"
style="border:solid 1px black; border-collapse:collapse ">A: Renin<br><br>B:
Angiotensin I<br><br>C: Erythropoietin<br><br>D: 1,25DHCC<br><br></td><td
width="50" style="border:solid 1px black; border-collapse:collapse ">B</td><td
width="250" style="border:solid 1px black; border-collapse:collapse ">The kidneys
produce three hormones, 1, 25 Dihydrochole calciferol, Renin and Erythropoetin.
Angiotensin I is not secreted by any organ. It is formed by the action of Renin
(secreted by kidney) on Angiotensinogen (secreted by Liver).</td></tr><tr
valign=top height="15" style="background-color:White;"><td width="30"
style="border:solid 1px black; border-collapse:collapse ">70</td><td width="250"
style="border:solid 1px black; border-collapse:collapse ">The latest WHO protocol
for classification and measurement of disability is:</td><td width="200"
style="border:solid 1px black; border-collapse:collapse ">A: ICIDH<br><br>B:
ICF<br><br>C: WHODAS<br><br>D: DALY<br><br></td><td width="50" style="border:solid
1px black; border-collapse:collapse ">B</td><td width="250" style="border:solid
1px black; border-collapse:collapse ">The latest WHO initiative for classification
of disability is the International Classification of Functioning, Disability and
Health which is termed as ICF</td></tr><tr valign=top height="15"
style="background-color:#EFF3FB;"><td width="30" style="border:solid 1px black;
border-collapse:collapse ">71</td><td width="250" style="border:solid 1px black;
border-collapse:collapse ">Psychrometer is used to measure:</td><td width="200"
style="border:solid 1px black; border-collapse:collapse ">A: Humidity<br><br>B: Air
velocity<br><br>C: Room temperature<br><br>D: Radiant temperature<br><br></td><td
width="50" style="border:solid 1px black; border-collapse:collapse ">A</td><td
width="250" style="border:solid 1px black; border-collapse:collapse ">Psychrometer
is a device to measure humidity.
Psychrometer (Synonym: Wet and Dry bulb thermometer / Hygrometer)</td></tr><tr
valign=top height="15" style="background-color:White;"><td width="30"
style="border:solid 1px black; border-collapse:collapse ">72</td><td width="250"
style="border:solid 1px black; border-collapse:collapse ">Which of the following is
the most radiosensitive phase of the cell cycle?</td><td width="200"
style="border:solid 1px black; border-collapse:collapse ">A: G2M<br><br>B:
G2<br><br>C: S<br><br>D: GI<br><br></td><td width="50" style="border:solid 1px
black; border-collapse:collapse ">A</td><td width="250" style="border:solid 1px
black; border-collapse:collapse ">Most sensitive phase is the junction of G2M phase
Susceptibility orvarious phases orcell cycle to radiation: G2M> G2 >= M > G1 >
Early S > Late S
Facts to remember:
• Dividing part of cells are most sensitive to RT
• Non dividing cells are relatively resistant
• Hypoxic cells are relatively resistant
• Phase of cell cycle that is most sensitive to radiation: G2M > M
• Phase of cell cycle that is most resistant to radiation: End of Sphase
• Phase of cell cyle in which radiation exposure leads to chromosomal aberration:
G1
• Phase of cell cycle in which radiation exposure leads to chromatid aberration:
G2</td></tr><tr valign=top height="15" style="background-color:#EFF3FB;"><td
width="30" style="border:solid 1px black; border-collapse:collapse ">73</td><td
width="250" style="border:solid 1px black; border-collapse:collapse ">The cell
bodies of proprioceptive nerves carrying information from the periodontal ligaments
are located in the</td><td width="200" style="border:solid 1px black; border-
collapse:collapse ">A: nucleus ambiguus.<br><br>B: spinal nucleus of V.<br><br>C:
main sensory nucleus of V.<br><br>D: mesencephalic nucleus of V.<br><br></td><td
width="50" style="border:solid 1px black; border-collapse:collapse ">D</td><td
width="250" style="border:solid 1px black; border-collapse:collapse ">Various
structures and functions of cranial nerve (CN) V (trigeminal) include the
trigeminal ganglion, near the cavernous sinus, which sends out the maxillary,
mandibular, and ophthalmic nerves. The mesencephalic nucleus receives information
about proprioception from the muscles of mastication, TMJ joint, and periodontal
ligament. The main sensory nucleus of CN V (choice C) lies near the motor nucleus
and receives tactile sensation. The spinal nucleus of CN V (choice B) receives pain
and thermal sensation. Nucleus ambiguus (choice A) is located in the pons
and sends fibers to CN IX, X, and XI.</td></tr><tr valign=top height="15"
style="background-color:White;"><td width="30" style="border:solid 1px black;
border-collapse:collapse ">74</td><td width="250" style="border:solid 1px black;
border-collapse:collapse ">In the posterior mediastinum, the thoracic duct usually
lies</td><td width="200" style="border:solid 1px black; border-collapse:collapse
">A: anterior to the phrenic nerves.<br><br>B: posterior to the
esophagus.<br><br>C: on the anterolateral surface of trachea.<br><br>D: does not
reach<br><br></td><td width="50" style="border:solid 1px black; border-
collapse:collapse ">B</td><td width="250" style="border:solid 1px black; border-
collapse:collapse ">The thoracic duct runs from the cisterna chyli (between the
aorta and azygos vein on the bodies of the first two lumbar vertebrae) through the
aortic opening of the diaphragm to the posterior mediastinum, along the vertebral
column, behind the esophagus, and through the superior mediastinum. It arches over
the apex of the pleura, descends across the left subclavian artery, and enters the
junction of the left internal jugular and subclavian veins. It is found posterior
and medial to the phrenic nerve and posterior to the trachea.</td></tr><tr
valign=top height="15" style="background-color:#EFF3FB;"><td width="30"
style="border:solid 1px black; border-collapse:collapse ">75</td><td width="250"
style="border:solid 1px black; border-collapse:collapse ">If an axonal membrane
transiently becomes very permeable to Na+ ions, then the membrane potential of the
cell will approach</td><td width="200" style="border:solid 1px black; border-
collapse:collapse ">A: -70 mV.<br><br>B: -60 mV.<br><br>C: -50 mV.<br><br>D: +60
mV.<br><br></td><td width="50" style="border:solid 1px black; border-
collapse:collapse ">D</td><td width="250" style="border:solid 1px black; border-
collapse:collapse ">► The diffusion of an ion through a membrane, without the
concomitant diffusion of its counter-ion, creates a membrane potential. Na+ ions
are present in higher concentration in the extracellular fluid than in axoplasm. If
the axonal membrane becomes permeable to Na+, the inward diffusion of this positive
ion, without similar diffusion of its negative counter-ion, will make the inside of
the neuron electrically positive in relation to the extracellular fluid. Thus, the
membrane potential will have a positive value.
► It is just such a transient increase in membrane permeability to Na+ that is the
mechanism responsible for the neuronal action potential. During the impulse, the
membrane voltage goes from a resting potential of about –75 mV to a peak of about
+30
mV, approaching the equilibrium potential for sodium of about +60 mV.</td></tr><tr
valign=top height="15" style="background-color:White;"><td width="30"
style="border:solid 1px black; border-collapse:collapse ">76</td><td width="250"
style="border:solid 1px black; border-collapse:collapse ">The most potent propionic
acid derivative is</td><td width="200" style="border:solid 1px black; border-
collapse:collapse ">A: ibuprofen<br><br>B: naproxen<br><br>C:
flurbiprofen<br><br>D: ketoprofen<br><br></td><td width="50" style="border:solid
1px black; border-collapse:collapse ">B</td><td width="250" style="border:solid
1px black; border-collapse:collapse ">Reference: Tripathi, Essentials of
Pharmacology,6th edition, pg no: 193
Explanation: Naproxen is the most potent propionic acid derivative.
Ibuprofen is the most used over the counter drugs.It also has a high theraputic
index.</td></tr><tr valign=top height="15" style="background-color:#EFF3FB;"><td
width="30" style="border:solid 1px black; border-collapse:collapse ">77</td><td
width="250" style="border:solid 1px black; border-collapse:collapse ">Most
endogenous cholesterol in the liver is usually converted into which of the
following?</td><td width="200" style="border:solid 1px black; border-
collapse:collapse ">A: Glucose<br><br>B: Steroids<br><br>C: Cholic acid<br><br>D:
Oxaloacetate<br><br></td><td width="50" style="border:solid 1px black; border-
collapse:collapse ">C</td><td width="250" style="border:solid 1px black; border-
collapse:collapse ">►Cholic acid, one of the bile acids, is synthesized by the
liver from cholesterol. In the conversion to cholic acid, hydroxyl groups and a
carboxyl group are added to the steroid nucleus of cholesterol. Bile acids are
important in the intestinal absorption of lipids.
►Neither glucose (choice A), oxaloacetate (choice D), nor ketone bodies (choice E)
are
derived from cholesterol. Steroids (choice B) are synthesized from cholesterol but
are not
produced in the liver. They are produced in the gonads and adrenal
glands.</td></tr><tr valign=top height="15" style="background-color:White;"><td
width="30" style="border:solid 1px black; border-collapse:collapse ">78</td><td
width="250" style="border:solid 1px black; border-collapse:collapse ">Which of the
following substances is LEAST polar?</td><td width="200" style="border:solid 1px
black; border-collapse:collapse ">A: Ethanol<br><br>B: Cholesterol<br><br>C:
Palmitic acid<br><br>D: Glycocholic acid<br><br></td><td width="50"
style="border:solid 1px black; border-collapse:collapse ">B</td><td width="250"
style="border:solid 1px black; border-collapse:collapse ">► Cholesterol is
considered a nonpolar and very hydrophobic lipid.The other three choices are less
hydrophobic and more polar than cholesterol, and are therefore incorrect.
► Ethanol (choice A) is a simple alcohol molecule and the most polar of the group.
Palmitic acid (choice C) is a saturated, straight-chain fatty acid that is nonpolar
but less so than cholesterol. Glycocholic acid (choice D) is a conjugated bile
acid.</td></tr><tr valign=top height="15" style="background-color:#EFF3FB;"><td
width="30" style="border:solid 1px black; border-collapse:collapse ">79</td><td
width="250" style="border:solid 1px black; border-collapse:collapse ">Syneresis and
imbibition are inherent properties that affect the dimensions of which of the
following impression materials (a) Silicones (b) Polysulfide rubbers (c) Reversible
hydrocolloids (d) Irreversible hydrocolloids (e) Impression modeling
compounds</td><td width="200" style="border:solid 1px black; border-
collapse:collapse ">A: (a) and (b) only<br><br>B: (a). (b) and (e)<br><br>C: (c)
and (d)<br><br>D: (c) and (e)<br><br></td><td width="50" style="border:solid 1px
black; border-collapse:collapse ">C</td><td width="250" style="border:solid 1px
black; border-collapse:collapse ">Ref: Craig, Ed: 11th,Pg no: 344, phillips, 10th
ed, pg no 161 and 147, Expl: As with alginate impressions, agar hydrocolloid
impressions are best stored in 100% relative humidity if the gypsum models cannot
be prepared immediately. Even in 100% humidity they can be stored for only limited
times, such as 1 hour, without shrinkage of the impression material caused by
syneresis. The best procedure is to pour up the impression immediately after
removing, rinsing, disinfecting, and superficial drying.Synerisis and imbibition
deos not occur in Impression compound. But rubber materials like polysulphide
aremuch more stable dimentionally when they are stored in air than the hydrocooliod
imp. materials. Addition silicones are the most dimentional stable imp. material of
all existing imp.materials.</td></tr><tr valign=top height="15" style="background-
color:White;"><td width="30" style="border:solid 1px black; border-
collapse:collapse ">80</td><td width="250" style="border:solid 1px black; border-
collapse:collapse ">In cobalt-chromium alloys, the constituent responsible for
corrosion resistance is</td><td width="200" style="border:solid 1px black; border-
collapse:collapse ">A: silver<br><br>B: nickel.<br><br>C: chromium.<br><br>D:
cobalt.<br><br></td><td width="50" style="border:solid 1px black; border-
collapse:collapse ">C</td><td width="250" style="border:solid 1px black; border-
collapse:collapse ">Ref: Craig, Ed: 11th, Pg no: 481, Expl: Chromium is
responsible for the tarnish and corrosion resistance of these alloys. When the
chromium content of an alloy is higher than 30%, the alloy is more difficult to
cast. With this percentage of chromium, the alloy also forms a brittle phase, known
as the sigma (0) phase. Therefore cast base-metal dental alloys should not contain
more than 28% or 29% chromium. In general, cobalt and nickel, up to a certain
percentage, are interchangeable elements. Cobalt increases the elastic modulus,
strength, and hardness of the alloy more than does nickel.</td></tr><tr valign=top
height="15" style="background-color:#EFF3FB;"><td width="30" style="border:solid
1px black; border-collapse:collapse ">81</td><td width="250" style="border:solid
1px black; border-collapse:collapse ">Viruses may cause disease by</td><td
width="200" style="border:solid 1px black; border-collapse:collapse ">A: lysing
many cells of the host<br><br>B: transforming cells to malignant cells<br><br>C:
disrupting the normal defence mechanism<br><br>D: all of the above<br><br></td><td
width="50" style="border:solid 1px black; border-collapse:collapse ">D</td><td
width="250" style="border:solid 1px black; border-collapse:collapse ">Ref:
ananthanarayan, Ed: 8th ,Pg no:444, Expl: At the cellular level, the virus causes
cytocidal effect or cytolytisisEx: poliovirus. Other may produce cellular
proliferation or malignancy Ex: oncogenic viruses. In some instances it causes
cytopathic effects.</td></tr><tr valign=top height="15" style="background-
color:White;"><td width="30" style="border:solid 1px black; border-
collapse:collapse ">82</td><td width="250" style="border:solid 1px black; border-
collapse:collapse ">An antibacterial substance found in saliva, tears, and eggs
white is</td><td width="200" style="border:solid 1px black; border-
collapse:collapse ">A: albumin<br><br>B: isozyme<br><br>C: amylase<br><br>D:
lysozyme<br><br></td><td width="50" style="border:solid 1px black; border-
collapse:collapse ">D</td><td width="250" style="border:solid 1px black; border-
collapse:collapse ">Ref: Guyton, Ed: 11th, Pg no: 794, Expl: Saliva contains
several factors that destroy bacteria. One of these is thiocyanate ions and another
is several proteolytic enzymes—most important, lysozyme—that (a) attack the
bacteria, (b) aid the thiocyanate ions in entering the bacteria where these ions in
turn become bactericidal, and (c) digest food particles, thus helping further to
remove the bacterial metabolic support. Saliva contains two major types of protein
secretion: (1) a serous secretion that contains ptyalin (ana-amylase), which is an
enzyme for digesting starches, and (2) mucus secretion that contains mucin for
lubricating and for surface protective purposes.
</td></tr><tr valign=top height="15" style="background-color:#EFF3FB;"><td
width="30" style="border:solid 1px black; border-collapse:collapse ">83</td><td
width="250" style="border:solid 1px black; border-collapse:collapse ">If the pH of
an area is lower than normal body pH, the membrane theory of local anesthetic
action predicts that the local anesthetic activity would be</td><td width="200"
style="border:solid 1px black; border-collapse:collapse ">A: greater, owing to an
incre.1se in the free-base form of the drug.<br><br>B: greater, owing to an
increase in the cationic form of the drug.<br><br>C: less, owing to an increase in
the free-base form of the drug .<br><br>D: less, owing to a decrease in the free-
base form of the drug.<br><br></td><td width="50" style="border:solid 1px black;
border-collapse:collapse ">D</td><td width="250" style="border:solid 1px black;
border-collapse:collapse ">Ref: Monheims, Ed: 7th, Pg no: 131, Expl: The local
aneasthetic solution is water soluble. The salts of local aneasthetic compounds
exist as both uncharged molecules , also called free base, and positiively charged
molecules, called the cation, in equlibrium with each other. This relative
proportion between the free base and the cation depends on the pH of the solution
and the pKa (dissociation constant). Since pKa is constant for any specific
compound, the relative proportion of free base and the cation depends on the pH of
the solution.If the pH is low, more cation will be present than free base( fat
soluble) form. The free base forms is responsible for the optimal diffusion through
the nerve sheath.Thus deprotonation is prevented in acidic pH and the LA solution
fails to penetrate the nerve.</td></tr><tr valign=top height="15"
style="background-color:White;"><td width="30" style="border:solid 1px black;
border-collapse:collapse ">84</td><td width="250" style="border:solid 1px black;
border-collapse:collapse ">The most controllable route for administration of a
general anesthetic is</td><td width="200" style="border:solid 1px black; border-
collapse:collapse ">A: rectal.<br><br>B: inhalation.<br><br>C:
intramuscular.<br><br>D: intravenous.<br><br></td><td width="50"
style="border:solid 1px black; border-collapse:collapse ">B</td><td width="250"
style="border:solid 1px black; border-collapse:collapse ">Ref: Tripathi, Ed: 5th,
Pg no: 9, Expl: Inhalational GA is absorbed from the alveoli and the action is
very rapid. When discontinued the drug diffues back and is rapidly eliminated in
expired air. Thus, controlled administration is possible with moment to moment
adjustment.</td></tr><tr valign=top height="15" style="background-
color:#EFF3FB;"><td width="30" style="border:solid 1px black; border-
collapse:collapse ">85</td><td width="250" style="border:solid 1px black; border-
collapse:collapse ">The fluid that leaks out of vessels in noninflammatory
conditions, such as cardiac failure, is</td><td width="200" style="border:solid
1px black; border-collapse:collapse ">A: exudate<br><br>B: ecchymoses<br><br>C:
metachysis<br><br>D: transudate<br><br></td><td width="50" style="border:solid 1px
black; border-collapse:collapse ">D</td><td width="250" style="border:solid 1px
black; border-collapse:collapse ">Ref: Harsh Mohan, Ed: 3rd, Pg no: 189, 201. Expl:
Transudate is the filtrateof blood plasma without changes in endothelial
permeability. It is a noninflammatory oedema Ex: Oedema in congestive cardiac
failure. Where as Exudate is oedema of inflamed tissue associated with increased
vascular permeability. Ex: Purulent exudate such as pus. Ecchymoses is Large
extravasations of blood into the skin and mucous membranes seen in haemorrhagic
disorders.</td></tr><tr valign=top height="15" style="background-color:White;"><td
width="30" style="border:solid 1px black; border-collapse:collapse ">86</td><td
width="250" style="border:solid 1px black; border-collapse:collapse ">the hormones
responsible for blood calcium level are</td><td width="200" style="border:solid
1px black; border-collapse:collapse ">A: parathormone<br><br>B:
calcitonin<br><br>C: 1,25 dihydroxycalciferol<br><br>D: all of the
above<br><br></td><td width="50" style="border:solid 1px black; border-
collapse:collapse ">D</td><td width="250" style="border:solid 1px black; border-
collapse:collapse ">Ref: Sembulingam, 2nd ed, pg no: 301
Exp: all the hormones are responsible for regulating blood calcium level:
CALCITONIN: By decreasing bone resorption
PARATHORMONE: By increasing bone resorption
1,25 DIHYDROXYCALCIFEROL: By increasing calcium absorption from
intestine</td></tr><tr valign=top height="15" style="background-color:#EFF3FB;"><td
width="30" style="border:solid 1px black; border-collapse:collapse ">87</td><td
width="250" style="border:solid 1px black; border-collapse:collapse ">On the crown
of the maxillary canine, the height of contour is normally located in the cervical
third of which of the following surfaces?</td><td width="200" style="border:solid
1px black; border-collapse:collapse ">A: Labial<br><br>B: Lingual<br><br>C: Both
labial and lingual<br><br>D: Neither labial nor lingual<br><br></td><td width="50"
style="border:solid 1px black; border-collapse:collapse ">C</td><td width="250"
style="border:solid 1px black; border-collapse:collapse ">► The height of contour,
or crest of curvature, is the greatest elevation
of the tooth, either incisocervically or occlusocervically, on a specific surface
of the crown. The labial and lingual surfaces of a tooth also have a height of
contour that is easily seen when viewing the tooth’s profile from the proximal.
►On the crown of the maxillary canine, the height of contour is normally located in
the cervical third of both labial and lingual surfaces, similar to all anterior
teeth. Choice D, neither labial or lingual surface, is incorrect.
► For posterior teeth, the height of contour for the crown’s labial (choice A)
surface is in the cervical third.</td></tr><tr valign=top height="15"
style="background-color:White;"><td width="30" style="border:solid 1px black;
border-collapse:collapse ">88</td><td width="250" style="border:solid 1px black;
border-collapse:collapse ">The non-working pathway of the maxillary cusps on the
mandibular posterior teeth is toward the</td><td width="200" style="border:solid
1px black; border-collapse:collapse ">A: distofacial.<br><br>B:
distolingual.<br><br>C: mesiofacial.<br><br>D: mesiolingual.<br><br></td><td
width="50" style="border:solid 1px black; border-collapse:collapse ">A</td><td
width="250" style="border:solid 1px black; border-collapse:collapse ">►When the
mandible moves into lateral excursive movement, the
nonworking-side condyle moves forward and down, and the working side rotates. This
causes the maxillary teeth to follow a pathway that is facial and distal to the
mandibular molars on the nonworking side.
► ( refer: diagrams in the Aim MDS Keynotes/ Videos Dental anatomy) for more
path</td></tr><tr valign=top height="15" style="background-color:#EFF3FB;"><td
width="30" style="border:solid 1px black; border-collapse:collapse ">89</td><td
width="250" style="border:solid 1px black; border-collapse:collapse ">The principal
fibrous elements of the periodontal ligament in adults consist chiefly of</td><td
width="200" style="border:solid 1px black; border-collapse:collapse ">A: elastic
fibers<br><br>B: collagen fibers<br><br>C: reticular fibers<br><br>D: a mixture of
elastic and collagen fibers<br><br></td><td width="50" style="border:solid 1px
black; border-collapse:collapse ">B</td><td width="250" style="border:solid 1px
black; border-collapse:collapse ">Ref: Orbans, Ed: 10th, Pg no: 216, Expl: The
fibers in periodontal ligament are the collagen and the oxytalan fibers. The
elastic fibers are restricted to the walls of the blood vessels.The majority of the
fibers are collagen ( predominently Type I collagen , but type III collagen is also
present).</td></tr><tr valign=top height="15" style="background-color:White;"><td
width="30" style="border:solid 1px black; border-collapse:collapse ">90</td><td
width="250" style="border:solid 1px black; border-collapse:collapse ">The principal
types of nerves in the dental pulp are</td><td width="200" style="border:solid 1px
black; border-collapse:collapse ">A: parsympathetic and afferent fibers<br><br>B:
sympathetic and afferent fibers<br><br>C: parasympathetic fibers only<br><br>D:
sympathetic fibers only<br><br></td><td width="50" style="border:solid 1px black;
border-collapse:collapse ">B</td><td width="250" style="border:solid 1px black;
border-collapse:collapse ">Ref: Orbans, Ed: 10th, Pg no: 156, Expl: The majority of
the nerves that enter the pulp are nonmyelinated and are sympathetic in nature. The
bundles of nerve fibers enter the tooth through apical foramen and proceed to the
coronal area. The peripheral axons form a network of nerves located adjacent to the
cell-rich zone. This is termed as the plexus of Rashkow or the parietal layer of
nerves. The large myelinated fibers carry the sensation of pain that may be caused
by external stimuli.</td></tr><tr valign=top height="15" style="background-
color:#EFF3FB;"><td width="30" style="border:solid 1px black; border-
collapse:collapse ">91</td><td width="250" style="border:solid 1px black; border-
collapse:collapse ">In the healing of an ulcer, the epithelium that eventually will
cover the defects is derived from</td><td width="200" style="border:solid 1px
black; border-collapse:collapse ">A: intact epithelium at the ulcer
margin<br><br>B: metaplasia of fibroblasts to epithelial cells<br><br>C:
transformation of fibrinous exudate to epithelium<br><br>D: transformation of
endothelial cells to epithelial cells<br><br></td><td width="50"
style="border:solid 1px black; border-collapse:collapse ">A</td><td width="250"
style="border:solid 1px black; border-collapse:collapse ">Ref: Shafers, Ed: 5th, Pg
no: 816 , Expl:The replacement of the lost tissue in ulcer is by granulation
tissue filling the defect followed by epithelialization. The epidermal growth
factor , produced by the epithelium around the damaged area helps in regeneration
of the epithelial tissue. However, the fibroblast growth factor released by
macrophages mediates the fibroblast activity and the endothelial growth factor
triggers the formation of the blood vessels.</td></tr><tr valign=top height="15"
style="background-color:White;"><td width="30" style="border:solid 1px black;
border-collapse:collapse ">92</td><td width="250" style="border:solid 1px black;
border-collapse:collapse ">In erythema multiforme, typical oral involvement
consists of</td><td width="200" style="border:solid 1px black; border-
collapse:collapse ">A: fissured tongue<br><br>B: acute gingivitis<br><br>C: diffuse
mucosal ulcerations<br><br>D: white straitions on the buccal mucosa<br><br></td><td
width="50" style="border:solid 1px black; border-collapse:collapse ">C</td><td
width="250" style="border:solid 1px black; border-collapse:collapse ">Ref: Shafers,
Ed: 5th, Pg no: 1112, 124 . Expl: Oral mucous membrane lesions are macules ,
papules, or vesicles which erode or ulcerate and bleed freely. Tongue, B. mucosa
and gingiva are the most common places in o. cavity.Bulls eye shaped lesions on
skin is a charcteristic feature of E. multiforme.
Fissured tongue is associated with Downs syndrome and Melkerson-Rosenthal syndrome.
White straitions are usually seen in leukoplakia, lichen planus, chronic cheek
bite,etc.</td></tr><tr valign=top height="15" style="background-color:#EFF3FB;"><td
width="30" style="border:solid 1px black; border-collapse:collapse ">93</td><td
width="250" style="border:solid 1px black; border-collapse:collapse ">Which of the
following dental findings is frequently observed in cases of Pagets disease of
bone</td><td width="200" style="border:solid 1px black; border-collapse:collapse
">A: Hypercementosis<br><br>B: Apical root resoption<br><br>C: Internal resorption
of the pulp<br><br>D: Widening of the periodontal ligament<br><br></td><td
width="50" style="border:solid 1px black; border-collapse:collapse ">A</td><td
width="250" style="border:solid 1px black; border-collapse:collapse ">Ref: White &
Pharoah, Ed: 5th, Pg no: 367, Ingle, 5thed, pgno 139 ,Expl:Hypercementosis occurs
in patients with Pagets disease of bone and with hyperpituitarism (gigantism and
acromegaly). The effects of inflammation on surrounding cancellous bone include
stimulation of bone formation, resulting in a sclerotic pattern, or bone
resorption, resulting in radiolucency. The periodontal ligament space involved in
the lesion will be widened; this widening is greatest at the source of the
inflammation. For example, with periapical lesions the widening is greatest around
the apical region of the root; in periodontal disease the widening is greatest at
the alveolar crest. With chronic infections, root resorption may occur and cortical
boundaries may be resorbed. Internal resorption of pulp is seen in chronic pulpal
inflammation, it also occurs as an idiopathic dystrophic change. Trauma in the form
of an accidental blow, or traumatic cavity preparation, has often been indicted as
a triggering mechanism for internal resorption.</td></tr><tr valign=top height="15"
style="background-color:White;"><td width="30" style="border:solid 1px black;
border-collapse:collapse ">94</td><td width="250" style="border:solid 1px black;
border-collapse:collapse ">Multiple periapical radiolucencies are common in
patients with which of the following conditions?</td><td width="200"
style="border:solid 1px black; border-collapse:collapse ">A: Taurodontism<br><br>B:
Dentinal dysplasia<br><br>C: Amelogenesis imperfecta<br><br>D: Dentinogenesis
imperfecta<br><br></td><td width="50" style="border:solid 1px black; border-
collapse:collapse ">B</td><td width="250" style="border:solid 1px black; border-
collapse:collapse ">Ref: White & Pharoah, Ed: 5th, Pg no: 348 -49, Expl:
Association of these periapical radiolucencies with noncarious teeth is an
important feature for recognition of this particular entity.The differential
diagnosis for dentin dysplasia may include only one other entity, dentinogenesis
imperfecta. Because these two conditions seem to fortn a continuum, their
differentiation may be difficult at first. Both entities can produce altered color
and occluded pulp chambersfinding a thistle-tube-shaped pulp chamber ina
singlerooted tooth strengthens the probability of dentin dysplasia If the roots are
short and narrow, the condition is likely to be dentinogenesis imperfecta. On the
other hand, normal-appearing roots or practically no roots at all should suggest
dentin dysplasia. Periapical rarefying osteitis in association with noncarious
teeth are more commonly seen in dentin dysplasia.</td></tr><tr valign=top
height="15" style="background-color:#EFF3FB;"><td width="30" style="border:solid
1px black; border-collapse:collapse ">95</td><td width="250" style="border:solid
1px black; border-collapse:collapse ">Which of the following is an advantage of IV
administration of a drug?</td><td width="200" style="border:solid 1px black;
border-collapse:collapse ">A: it eliminates side effects<br><br>B: minimal skill is
necessary<br><br>C: it allows titration of the drug<br><br>D: sedatives drugs are
compatible with IV<br><br></td><td width="50" style="border:solid 1px black;
border-collapse:collapse ">C</td><td width="250" style="border:solid 1px black;
border-collapse:collapse ">Ref:Tripathi, Ed: 5th, Pg no: 10, Expl: one big
advantage of IV route is titration of the dose with the response is possible. One
of the main disadvantage is that it is the most risky route - vital organs like
Heart, brain, etc get exposed to high concentration of the drug.</td></tr><tr
valign=top height="15" style="background-color:White;"><td width="30"
style="border:solid 1px black; border-collapse:collapse ">96</td><td width="250"
style="border:solid 1px black; border-collapse:collapse ">The most common cause of
postextraction bleeding is</td><td width="200" style="border:solid 1px black;
border-collapse:collapse ">A: failure of the patient to follow postextraction
instructions<br><br>B: congenital coagulation factor deficiency ( eg:
hemophilia)<br><br>C: acquired coagulation factor deficiency (eg: anticoagulants
like Coumadin)<br><br>D: inhibition of ADP release by platelets because of
ingestion of analgesics containig aspirin<br><br></td><td width="50"
style="border:solid 1px black; border-collapse:collapse ">A</td><td width="250"
style="border:solid 1px black; border-collapse:collapse ">Ref:, Ed:, Pg no:, Expl:
With proper medical and drug history B, C and D are ruled out. Where as option A is
common because the patient is anxious and may not follow the post operative
instructions.</td></tr><tr valign=top height="15" style="background-
color:#EFF3FB;"><td width="30" style="border:solid 1px black; border-
collapse:collapse ">97</td><td width="250" style="border:solid 1px black; border-
collapse:collapse ">A group of antibiotics related both structurally and by mode of
action to the penicillins is</td><td width="200" style="border:solid 1px black;
border-collapse:collapse ">A: cycloserines.<br><br>B: cephalosporins.<br><br>C:
chloramphenicols.<br><br>D: polymyxins<br><br></td><td width="50"
style="border:solid 1px black; border-collapse:collapse ">B</td><td width="250"
style="border:solid 1px black; border-collapse:collapse ">
Reference:Elander, R. P. (2003). "Industrial production of beta-lactam
antibiotics". Applied microbiology and biotechnology 61 (5–6): 385–392.
Explanation: Both penicillins and cephalosporins belong to B lactam antibiotics.
The penicillins and cephalosporins which have so far proved clinically useful have
the general structures I and H respectively. The general similarity in structure of
the two fused ring systems is clearly due to common features in their biogenesis.
MODE OF ACTION: β-Lactam antibiotics are bacteriocidal, and act by inhibiting the
synthesis of the peptidoglycan layer of bacterial cell walls. The peptidoglycan
layer is important for cell wall structural integrity, especially in Gram-positive
organisms, being the outermost and primary component of the wall</td></tr><tr
valign=top height="15" style="background-color:White;"><td width="30"
style="border:solid 1px black; border-collapse:collapse ">98</td><td width="250"
style="border:solid 1px black; border-collapse:collapse ">A two-days old developing
plaque would consist primarily of</td><td width="200" style="border:solid 1px
black; border-collapse:collapse ">A: filamentous organism<br><br>B: gram-positive
cocci and rod-like organisms<br><br>C: a structureless, non-mineralized
pellicle<br><br>D: spirochetal organisms<br><br></td><td width="50"
style="border:solid 1px black; border-collapse:collapse ">B</td><td width="250"
style="border:solid 1px black; border-collapse:collapse ">Ref: Carranza, Ed: 11th,
Pg no: 143, Expl : Streptococci and actinomyces are the primary colonizers of
plaque. These organisma prepare a favourable envinronment for later( secondary
colonizers), which have a fastidius growth requirements.Ex: P. intermedia, P.
gingivalis.</td></tr><tr valign=top height="15" style="background-
color:#EFF3FB;"><td width="30" style="border:solid 1px black; border-
collapse:collapse ">99</td><td width="250" style="border:solid 1px black; border-
collapse:collapse ">The strength of dental investments for gold alloys is dependent
upon the amount of</td><td width="200" style="border:solid 1px black; border-
collapse:collapse ">A: silica<br><br>B: carbon<br><br>C: gypsum<br><br>D:
alumina<br><br></td><td width="50" style="border:solid 1px black; border-
collapse:collapse ">C</td><td width="250" style="border:solid 1px black; border-
collapse:collapse ">Ref: Phillips, Ed: 10th, Pg no: 483, Expl: The compressive
strength of the investment mold is the primary factor to be considered, in
addition to the expansion . The comp. strength is increased according to the
amount and type of gypsum binder present,Eg : the use of alpha hemihydrate instead
of plaster definitely increases the compressive strength of the
investment.</td></tr><tr valign=top height="15" style="background-color:White;"><td
width="30" style="border:solid 1px black; border-collapse:collapse ">100</td><td
width="250" style="border:solid 1px black; border-collapse:collapse ">When a liquid
wets a solid completely, the contact angle between the liquid and the solid
is</td><td width="200" style="border:solid 1px black; border-collapse:collapse
">A: 0degrees<br><br>B: 45degrees<br><br>C: 90degrees<br><br>D: 135
degrees<br><br></td><td width="50" style="border:solid 1px black; border-
collapse:collapse ">A</td><td width="250" style="border:solid 1px black; border-
collapse:collapse ">Ref:, Ed:, Pg no:, Expl: the spreading of liquids on solids, or
the tendency for wetting surfaces, by measuring the angle of contact between the
liquid and the solid surface.The greater the tendency to wet the surface, the lower
the contact angle, until complete wetting occurs at an angle equal to
zero.</td></tr><tr valign=top height="15" style="background-color:#EFF3FB;"><td
width="30" style="border:solid 1px black; border-collapse:collapse ">101</td><td
width="250" style="border:solid 1px black; border-collapse:collapse ">A patient
recently received a blow to the mouth. Radiographs show a horizontal mid root
fracture of a maxillary central incisor. The tooth is not mobile and is
asymptomatic; however, it does not respond to pulp testing. No radiographic lesion
is present. Proper treatment is to</td><td width="200" style="border:solid 1px
black; border-collapse:collapse ">A: institute root canal treatment to include both
segments ofthe tooth.<br><br>B: extract the coronal segment of the tooth and remove
surgically the apical segment<br><br>C: remove surgically the apical segment of the
tooth and retrofill the coronal segment<br><br>D: render no treatment at this time.
The tooth should be periodically rechecked clinically a:ld
radiographically.<br><br></td><td width="50" style="border:solid 1px black;
border-collapse:collapse ">D</td><td width="250" style="border:solid 1px black;
border-collapse:collapse ">Ref: Ingle, Ed: 5th, Pg no: 812, Expl: Root fractures
are not always horizontal; in fact, probably more often than not, the angulation of
fractures is diagonal. Treatment: If there is no mobility and the tooth is
symptomless, the fracture is likely to be in the apical one-third of the root, and
no treatment is necessary. If the coronal fragment is mobile, treatment is
indicated. The initial treatment consists of repositioning the coronal segment (if
it is displaced) and then stabilizing the tooth to allow healing of the periodontal
ligament supporting the coronal segment.Repositioning can be as simple as pushing
the tooth into place with finger pressure, or orthodontic intervention may be
required to move the displaced segment into properalignment and Splinting. The
splint should allow for functional movement of the tooth to promote healing, and
the length of stabilization time is 4 to 6 weeks. Following initial treatment by
reduction and stabilization, repair by calcific and/or fibrous deposition is very
likely. About 80% of properly treated root fractures heal
successfully.</td></tr><tr valign=top height="15" style="background-
color:White;"><td width="30" style="border:solid 1px black; border-
collapse:collapse ">102</td><td width="250" style="border:solid 1px black; border-
collapse:collapse ">Which of the following is the best space maintainer?</td><td
width="200" style="border:solid 1px black; border-collapse:collapse ">A: Nance
holding arch<br><br>B: A band and loop appliance<br><br>C: Removable accrylic
appliance<br><br>D: A pulpectomized primary molar<br><br></td><td width="50"
style="border:solid 1px black; border-collapse:collapse ">D</td><td width="250"
style="border:solid 1px black; border-collapse:collapse ">Ref: Ingle, Ed: 5th, Pg
no: 889, Expl: The treatment objectives in nonvital pulp therapy for primary teeth
are to (1) maintain the tooth free of infection, (2) biomechanically cleanse and
obturate the root canals, (3) promote physiologic root resorption, and (4) hold the
space for the erupting permanent tooth. The treatment of choice to achieve these
objectives is pulpectomy, which involves the removal of necrotic pulp tissue
followed by filling the root canals with a resorbable cement. Indications for this
procedure include teeth with poor chance of vital pulp treatment success, strategic
importance with respect to space maintenance, absence of severe root resorption,
absence of surrounding bone loss from infection, and expectation of
restorability.</td></tr><tr valign=top height="15" style="background-
color:#EFF3FB;"><td width="30" style="border:solid 1px black; border-
collapse:collapse ">103</td><td width="250" style="border:solid 1px black; border-
collapse:collapse ">Which of the following is least likely to resolve from
persistent long term thumb sucking?</td><td width="200" style="border:solid 1px
black; border-collapse:collapse ">A: A deep overbite<br><br>B: Protrusion of
maxillary incisors<br><br>C: Construction of maxilary arch<br><br>D: Rotation of
maxialry laetral incisors<br><br></td><td width="50" style="border:solid 1px
black; border-collapse:collapse ">A</td><td width="250" style="border:solid 1px
black; border-collapse:collapse ">Ref: Balaji, Ed: 2nd, Pg no: 107, 412, Expl: The
effects of thumb sucking are 1) Labial tipping of maxillary anteriors, 2) the
overjet increases due to proclination of maxillary anteriors, 3) anterior open
bite, the cheek muscles contract during thumb sucking resulting in a narrow
maxillary arch which predisposes to posterior cross bite. However, the etiology for
deep overbite is Skeletal which is genetic in origin or dental deep bite which due
to over eruption of anteriors or infra-occlusion of molars.</td></tr><tr valign=top
height="15" style="background-color:White;"><td width="30" style="border:solid 1px
black; border-collapse:collapse ">104</td><td width="250" style="border:solid 1px
black; border-collapse:collapse ">Which of the following conditions is usually
present in a Class-II, Divison-II, malocclusion?</td><td width="200"
style="border:solid 1px black; border-collapse:collapse ">A: Open bite<br><br>B:
Steep mandibular plane<br><br>C: Mesiocclusion of permanent first molars<br><br>D:
Lingual inclination of maxillary central incisors<br><br></td><td width="50"
style="border:solid 1px black; border-collapse:collapse ">D</td><td width="250"
style="border:solid 1px black; border-collapse:collapse ">Ref: Balaji, Ed: 2nd, Pg
no: 78, Expl: In Angels Class II Div 2 the upper central incisors are lingually
inclined and the lateral incisors are labially tipped overlapping the centrals. The
lingually inclined upper incisors gives the arch a squarish appearance and
mandibular gingival tissue is often traumatized . by excessive tipped upper
incisors. the patient exhibits normal perioral muscle activity.</td></tr><tr
valign=top height="15" style="background-color:#EFF3FB;"><td width="30"
style="border:solid 1px black; border-collapse:collapse ">105</td><td width="250"
style="border:solid 1px black; border-collapse:collapse ">Orthodontic correction of
which of the following is most easily retained?</td><td width="200"
style="border:solid 1px black; border-collapse:collapse ">A: Diastema<br><br>B:
Rotation<br><br>C: Expansion<br><br>D: Anterior crossbite<br><br></td><td
width="50" style="border:solid 1px black; border-collapse:collapse ">D</td><td
width="250" style="border:solid 1px black; border-collapse:collapse ">Ref: Balaji,
Ed: 2nd, Pg no: 445, Expl: Correction of anterior cross bites requires
no retention or a short trem retention. Where as , rotation, expansion and
diastema demands for prolonged or permanent retention</td></tr><tr valign=top
height="15" style="background-color:White;"><td width="30" style="border:solid 1px
black; border-collapse:collapse ">106</td><td width="250" style="border:solid 1px
black; border-collapse:collapse ">Which of the following results from addition of
more water initially to a mix of gypsum products(hemihydrate)?</td><td width="200"
style="border:solid 1px black; border-collapse:collapse ">A: more expansion, more
strength, more abrasion resistance<br><br>B: more expansion, less strength, less
abrasion resistance<br><br>C: less expansion, more strength, less abrasion
resistance<br><br>D: less expansion, less strength, less abrasion
resistance<br><br></td><td width="50" style="border:solid 1px black; border-
collapse:collapse ">D</td><td width="250" style="border:solid 1px black; border-
collapse:collapse ">Ref:Craig, Ed: 11th, Pg no:400,404,401. Expl: The compressive
strength is inversely related to the W/P ratio of the mix. The more water used to
make the mix, the lower the compressive strength. The W/P ratio of the mix also has
an effect, with an increase in the ratio reducing the setting expansion.The surface
hardness of unmodified gypsum materials
is related in a general way to their compressive strength. High compressive
strengths of the hardened mass correspond to high surface hardnesses. After the
final setting occurs, the surface hardness remains practically constant until most
excess water is evaporated from the surface, after which its increase is similar to
the increase in compressive strength..</td></tr><tr valign=top height="15"
style="background-color:#EFF3FB;"><td width="30" style="border:solid 1px black;
border-collapse:collapse ">107</td><td width="250" style="border:solid 1px black;
border-collapse:collapse ">The area of buccal frenum of a complete mandibular
denture must be thinned to allow for the contraction of which of the following
muscles</td><td width="200" style="border:solid 1px black; border-
collapse:collapse ">A: Caninus<br><br>B: masseter<br><br>C: zygomaticus<br><br>D:
triangularis<br><br></td><td width="50" style="border:solid 1px black; border-
collapse:collapse ">D</td><td width="250" style="border:solid 1px black; border-
collapse:collapse ">Ref: Bouchers, Ed: 9th, Pg no: 187, 128, Expl: The buccal frenm
connects as a continuos band through the modiolus at the corner of the mouth and on
up to the buccal frenum attachment of maxilla.Caninus muscle attaches beneath the
buccal frenum in maxillary arch and affects the position of buccal frenum in
maxillary arch . Masseter muscle decides the thickness of maxillary buccal flange
in denture.</td></tr><tr valign=top height="15" style="background-color:White;"><td
width="30" style="border:solid 1px black; border-collapse:collapse ">108</td><td
width="250" style="border:solid 1px black; border-collapse:collapse ">All of the
following are catagorised as secondary lymphoid organs except</td><td width="200"
style="border:solid 1px black; border-collapse:collapse ">A: Lymph nodes<br><br>B:
Spleen<br><br>C: Thymus<br><br>D: Subepithelial collections of
lymphocytes<br><br></td><td width="50" style="border:solid 1px black; border-
collapse:collapse ">C</td><td width="250" style="border:solid 1px black; border-
collapse:collapse ">Reference: Immunology: essential and fundamental By Sulabha
Pathak, Urmi Palan page no 90
Explanation : Primary lymphoid organs
The central or primary lymphoid organs generate lymphocytes from immature
progenitor cells.
The thymus and the bone marrow constitute the primary lymphoid tissues involved in
the production and early selection of lymphocytes.