Mata

table of content
Welcome Messages
Chairman of Organizing Committee 3

President of IOA 5
IOA Council Members 6
Committees 8
Plenary Lectures 10
Ophthalmologists Graduate 2015-2016 18
Welcome Reception 21
Congress information 22
Venue Map 24
Speaker Information 29
Scientific Program Overview 31
Scientific Program Day 1 37
Free Paper 85
E Poster 105
Abstract 116
Exhibitors 154
PERDAMI 14th NATIONAL CONGRESS & 41st ANNUAL SCIENTIFIC MEETING 2016 1
Welcome Messages
Ari Djatikusumo, MD
Congress Chairman
It is my honor and pleasure to welcome you all to our 14th National Indonesian Ophthalmology Association
Congress and 41st Annual Meeting 2016. The conference is held in Jakarta, the capital city of Indonesia.
This conference is a mixture of all sub specialty fields aims to improve quality of eye care. It offers a unique
blend of Science, Clinical Knowledge, and Cutting Edge Technology in the field of Ophthalmology and beyond.
All of us, organizers, speakers, and sponsors spare no time or effort to put bring to you the most up to date
developments in various fields of Ophthalmology.
This Congress is also featuring a high profile session with ORBIS. In this session, the ORBIS volunteer faculties
will share with us their vast knowledge and experience as well as the latest in the field of Pediatric Ophthal-
mology
I am sure that this conference will be of the greatest help to develop our knowledge and sharpen our skills in
pursuing the goal that we all share, to provide our patients with the best possible solutions for their eye care
needs.
I hope you all have a good time.
4 PERDAMI 14th NATIONAL CONGRESS & 41st ANNUAL SCIENTIFIC MEETING 2016
Welcome Messages
Prof. Nila F. Moeloek, MD

IOA President
Dear Friends and Colleagues,

It is a great pleasure for me, on behalf of the Indonesian Ophthalmologist Association to extent all of you, the
warmest welcome to the 14th National Assembly and the 41st Annual Scientific Meeting.
The main aims of our National Assembly are to seek and to vote the new leaders in Indonesian Ophthalmologist
Association (Perdami), the Indonesian College of Ophthalmology (KOI), and it’s Internal Affair Bodies; Ethics
Committee (DKEDK) and Financial Auditor (BPK). The global pace of science and technology in our profession
is one of fastest amongst all medical disciplines, there are a new local regulations and regional de-regulations
which demanding ourselves to keep adapted to the global pace of professional’s best practice; however, we also
have to have rot to our local situations. We do believe that all Perdami member will use your “organization right”
to vote the right person to be our new “captain of the ships”.
In order to keep our acquaintance to the global pace of ophthalmological best practice, we dedicated to
enhance the continuing professional development (CPD) scheme; and yet strengthening our brotherhood in
ophthalmology amongst the members.
We do hope that those aims could be achieved through a wide varieties of simultaneous exciting scientific
information which will arousing in all of our sub-specialties’ symposia and courses, wet labs, free papers, posters,
and memorial’s lecture, and last, the exhibition hall.
We urge that Perdami members will behave as active participants in the coming event, in the spirit of sharing
experience, knowledge and skills in order to provide a better serving performance to our patients.
I would like to express my gratitude to all speakers, audience, and the organizing committee, and wish to
acknowledge their valuable contributions throughout the meeting. It is the combined efforts of them all that
makes such a meeting fruitful and enjoyable, and trust that these efforts will help in bringing benefits to the
Indonesian Vitreo-retina Society and to the people of Indonesia. We also welcome all ophthalmic-related
pharmacies, industries, and distributors to contribute and participate in this meeting.
Last, we do hope that all of you will enjoy the scientific quality as well as the friendly atmosphere. Jakarta is a
capital city that provide a lot of leisure and culinary. You will enjoy Jakarta.
IOA Council Members
President
Vise-Presidents
Iwan Sovani, MD
Johan Hutauruk, MD
Soemartono Samadikoen, MD
Secretary
M. Sidik, MD
Astrianda Nadia Suryono, MD
Treasurer
Indriani Pudjiastuti, MD
Mutmainah, MD
Public Relation
Gitalisa Andayani, MD
Ferdiriva Hamzah, MD
Yulia Azizah, MD
Website
Achmad Juandy, MD
Mario Hutapea, MD
Organization
Hikmat Wangsaatmadja, MD. (Chairman)
Riki Tsan, MD
Andrew M. Knoch, MD
DP3 (Departemen Pembinaan dan Pengembangan Profesi)

Ari Djatikusumo, MD (Chairman)
Ni Retno Setyoningrum, MD (Secretary)

CME : Julie Barliana, MD
Eko Firdianto Karim, MD

Training Program : Amir Shidik, MD

MOI : Dyah Winarti, MD

Annual Meeting : Ari Djatikusumo, MD

IOA Council Members
Special Interest Groups
Coordinator : Habibah Setyawati, M. MD
Cataract & Refractive Surgery : Setiyo Budi Riyanto, MD
Vitreo-retina : Iwan Sovani, MD
Glaucoma : Andhika Prahasta, MD
Infection & Immunology : Fatma Asyari, MD
Neuro-ophthalmology : Bambang Setioadji, MD
PO & Strabismus : Feti Karfiati Memed, MD
Oncology & Reconstruction : Ratna Dumilah, MD
Refraction & Contact Lens : Karmelita, MD
Neuro-ophthalmology : Bambang Setioadji, MD
Ophthalmology Community : Nina Ratnaningsih, MD
DP3M (Departemen Pengembangan dan Peningkatan Pelayanan Masyarakat)

Aldiana Halim, MD (Chairman)
Mayang Rini, MD
Darmayanti, MD
Sindy Boru, MD
SPBK
Yeni Dwi Lestari, MD (Chairman)
Indra Wiryawan, MD
Anna P. Bani, MD
M. Iqbal Sofyan, MD
Umar Mardianto, MD
D.A.N. Candra Sari, MD
Pembinaan Dokter Umum, Profesi Para-oftalmologi

Amyta Miranty, MD
Nylvia Lamsari, MD
DPKA (Departemen Permberdayaan Kesejahteraan Anggota)

Rina La Distia Nora, MD
Virna Dwi Oktarina, MD
DP4 (Departemen Pertimbangan Pemberian Penghargaan Perdami)

Prof. Khailul Rahman, MD
Prof. Wasisdi Gunawan, MD
P2KB (Pengembangan Pendidikan Keprofesian Berkelanjutan)

Tri Rahayu, MD
Virna Dwi Oktarina, MD
M. Iqbal Sofyan, MD
Devina Nur Annisa, MD
Sri Sutarsih, MD
Organizing Committee
Advisors Prof. Nila F. Moeloek, MD

Prof. Rita S. Sitorus, MD
Tjahjono D. Gondhowiardjo, MD
Widya Artini, MD
Johan Hutauruk, MD
Chairman Ari Djatikusumo,MD
Co-Chairman Ni Retno Setyoningrum, MD
Secretary Syska Widyawati, MD

Devina Nur Annisa,MD
Nylvia Lamsari,MD
Treasurer Julie D. Barliana, MD

Pradnya Pramitha, MD
Rien Widyasari, MD
Scientific Chair Tri Rahayu, MD
Scientific Secretary Syntia Nusanti, MD

Referano Agustiawan, MD
Yeni Dwi Lestari, MD
Virna D. Oktariana, MD
Sindy Boru Sembiring, MD
Yulia Aziza, MD
Dialika, MD
Mario Hutapea, MD
Sita Paramita Ayuningtyas, MD
Suci Mustafa, MD
Azrina Noor, MD
Rini Sulastiwati, MD
Video Session Amir Shidik, MD)

M. Iqbal, MD
National Assembly Ni Retno Setyoningrum, MD

Astrianda Suryono, MD
Indra Wiryawan, MD
Nasrudin, MD
Umar Mardianto, MD
Program Ceremony Gitalisa Andayani, MD
Anna P. Bani, MD
Grace Setyohardjo, MD
Florence M. Manurung, MD
Ferdiriva Hamzah, MD
Utami Noor Syahbaniah, MD
Congress Banquet Yunia Irawati, MD

Neni Anggraini, MD
Bangkit Damayanti, MD

Logistic & Transportation Umar Mardianto,MD

Nasrudin, MD
Indra Wiryawan, MD
Publication & Emil Fahrinandi Sjahreza, MD

Documentation Andito Adisasmita, MD

Registration Retno S. Wulandari, MD

Dian Estu Yulia, MD
Muthmainah, MD
D. A. N Candra Sari, MD
Audiovisual Lukman Edwar, MD

Achmad Juandy, MD
Zeiras Eka Djamal, MD
Sponsorship & Exhibition Yudisianil E. Kamal, MD

Eko F. Karim, MD
Anggun Rama Yudantha, MD
Ucok Parlindungan, MD
Security & Permit Robby Hilman Maulana, MD

M. Sulaiman, MD
Puranto Budi, MD
PLENARY LECTURE
MENGENAL & MEMILIH INVESTASI YANG TEPAT

UNTUK PROFESI DOKTER
Safir Senduk
Thursday, 29 September 2016, 16.30-18.00

• Opening Ceremony • Ball Room
Di masyarakat, profesi Dokter saat ini identik dengan profesi yang memiliki penghasilan besar. Itulah kenapa
di Indonesia profesi ini banyak diburu sehingga peminat Sekolah Kedokteran selalu membanjir. Dan setiap
tahunnya selalu ada saja Dokter-Dokter baru ·yang muncul. Dari situ tidak sedikit dari para Dokter tersebut yang
berhasil dalam karirnya dan memiliki penghasilan yang sangat-sangat besar.
Sayangnya penghasilan besar ini seringkali tidak diimbangi dengan penqetahuan yang baik tentang investasi.
Padahal dengan berinvestasi mereka bisa memutarkan uangnya untuk kelak bisa memberikan penghasilan juga
dari situ.
Ada dua pertanyaan yang muncul kalau bicara tentang investasi. Pertanyaan pertarna adalah altematif inveslasi
apa saja yang ada diluar sana. lni menarik karena di Indonesia kebanyakan orang masih hanya rnengenal produk-
produk investasi yang umum (sepertt Deposilo & Properti) dan sering tidak tahu bahwa masih banyak produk
investasi lain yang bisa jadi lebih menarik.
Pertanyaan kedua yang biasa muncul adalah bagaimana cara memilih Produk lnvestasi yang tepat diantara
banyaknya alternatif yang tersedia. Ini penting karena harus diakui bahwa tidak ada satupun investasi yang
cocok untuk semua orang mengingat adanya perbedaan pengetahuan, jumlah dana yang dimiliki, karakter,
pengaruh lingkungan, dan tingkat kebranian dalam mengambil risiko.
Itulah kenapa, penting sekali bagi para Dokter untuk rnendapatkan materi tentang bagaimana mengenal dan
memilih investasi yang tepat. Materi yang dibahas diantaranya:
• Apa itu Investasi?
• Bagaimana menetapkan tujuan dalam investasi?
• Mengenali Produk-produk lnvestasi apa saja yang ada diluar sana serta apa saja kelebihan dan
kekurangannya. Seperti Deposito, Obligasi, Saham, Reksa Dana, Emas, Properti, Valuta Asing, dan
sebagainya.
• Mengetahui Tips & Triks dalam memilih Produk investasi yang tepat
• Sekilas ekonomi dan hubungannya dengan pemilihan Produk investasi
• Risiko,risiko lnvestasi dan bagaimana menghadapinya
• Adakah perbedaan Strategi lnvestasi antara Profesi Dokter yang memiliki Penghasilan tidak tetap dan
• Profesi Karyawan yang memiliki Penghasilan Tetap?
• Menyusun Portofolio investast ala Manajer lnvestasi
• Mengetahui Strategi investasi yang tepat untuk Jangka Panjang maupun Jangka Pendek.
Disampaikan dengan bahasa yang sederhana, penuh humor dan diselingi contoh kasus. Seminar ini akan
membuat para Dokter menjadi lebih cerdas tentang investasi dan termotivasi untuk langsung melakukannya.
PLENARY LECTURE
ASPEK ETIK DAN MEDIKOLEGAL PATIENT SAFETY DALAM
PRAKTEK DOKTER MATA
dr. Tjetjep DS, SpF, SH.
Departemen Forensik dan Medikolegal FKUI - RSCM
Thursday, 29 September 2016, 16.30—18.00

• Closing Ceremony • Ball Room
Setiap tindakan medik yang dilakukan oleh dokter mata selalu mengandung risiko buruk, mulai dari gangguan
penglihatan sampai mengakibatkan kebutaan, bahkan dapat mendatangkan bahaya maut bagi pasien, sehingga
harus dilakukan upaya pencegahan ataupun tindakan mereduksi risiko dengan tingkat kehati-hatian dan
kepedulian yang tinggi.
Di dalam profesi kedokteran, paling sedikit terdapat tiga komponen penting yang harus dimiliki oleh Dokter
Mata agar pelayanan kesehatan dapat memberikan rasa nyaman, aman baik bagi dokter, pasien maupun
tenaga kesehatan lainnya. Ketiga komponen tersebut adalah komponen ilmu dan technologi kedokteran,
komponen moral dan etik kedokteran, serta komponen hubungan interpersonal antara dokter dan pasien.
Standar hubungan dokter pasien merupakan suatu seni di bidang kedokteran (art of medicine) yang mengatur
bagaimana berkomunikasi efektif, berempati, simpati, sopan santun dan penuh perhatian terhadap pasien
dengan masalah kesehatannya. Komunikasi dokter pasien yang kurang baik, sering menimbulkan kekecewaan
pasien atau keluarganya yang akhirnya menimbulkan konflik antara dokter dengan pasien dan bahkan dapat
berakibat timbulnya masalah hukum.
International Faculty of Speakers
John Jarstad, M.D., F.A.A.O., F.R.S.M. UK (oph)
John Jarstad is an Associate Professor of Clinical Ophthalmology and Director of

Cataract and Refractive Surgery at Mason Eye Institute - University of Missouri School
of Medicine. He is the founding partner of Evergreen Eye Centers 4 clinics near Seattle,
Washington, USA.
He received degrees from Brigham Young University (Utah), the M.D. from University
of Washington, Seattle. His internship and residency in ophthalmology at Mayo Clinic.
Dr. Jarstad was a Research Fellow at the National Institutes of Health (Bethesda, Maryland) where he studied
choroidal melanomas with radio labeled monoclonal antibodies, movement disorders in neuroophthalmology
with Dr. David Cogan at the and ophthalmic pathology of melanomas at Armed Forces Institute of Pathology
with Dr. Lorenz Zimmerman and Dr. Ian McLean.
He is the inventor of many commonly used devices in ophthalmology including the Jarstad Refractive Cataract
Surgery Marker, the Jarstad Cataract Surgery simulator and teaching head, the first injectable IOL inserter (Chiron
Passport) and helped develop many drugs and devices used in ophthalmology including the first topical mast
cell stabilizer, first multifocal IOL, and the first teledioptric IOL for macular degeneration. His current research
involves collagen cross-linking for keratoconus using inexpensive dietary riboflavin and natural sunlight. Also
the effects of immediate adjustment of post op IOP following anterior segment surgery.
Professor Jarstad is a guest host of Radio and TV medical talk shows in Seattle and Columbia, Missouri.
He has lectured at scientific meetings throughout the USA and taught eye surgery as a visiting professor in
Indonesia, Austria, the Philippines, North Korea, Vietnam, Cambodia, Angola, Madagascar, Zimbabwe, Egypt,
Nigeria and England, where he was elected into the Royal Society of Medicine in 2006. Along with Professor
Istiantoro, he introduced no-stitch phaco and foldable IOL’s in Indonesia in 1994.
Dr. Jarstad has been named by Consumer Research Council one of “America’s Top Ophthalmologists” and by
Newsweek magazine as one of the top 15 Laser Eye Surgeons in the USA in 2011. He is the author of over 60
publications or presentations and one book.
Prof. Nathan Congdon
Professional Objective: To transform local eye health challenges into innovative and
sustainable global solutions, by joining world-class program design and execution with
groundbreaking research and far-reaching advocacy.
MD, Johns Hopkins University School of Medicine, Alpha Omega Alpha, 1993
MPH, Johns Hopkins University School of Hygiene and Public Health, Delta Omega,
1993
MPhil, Cambridge University, Oriental Studies, 1987
AB, Princeton University, Phi Beta Kappa, Summa Cum Laude, Oriental Studies, 1985
Professional Training & Current Work Experience

2015 – present Ulverscroft Chair of Global Eye Health and Director of the Translational Research for Equitable
Eyecare (TREE) Center at Queen’s University Belfast
2009 - present Senior Advisor, ORBIS International North Asia

Helped to steer ORBIS North Asia’s 5-year strategic planning process for China and Mongolia,
including drafting the main documents
Held primary responsibility for the design of ORBIS programs in North Asia totaling USD10
million, including two of the organization’s largest and most innovative projects, Guangdong
CREST and Shanxi CHEER
Wrote a successful USD400,000 grant application to World Diabetes Foundation, and developed
a cutting edge, electronically-networked rural model of comprehensive eye care linking 10
county hospitals in the Guangdong CREST project
Took primary responsibility for crafting the clinical and research elements of ORBIS’ 5-year
strategy, which I have presented to the Board of Directrors on several occasions
2009-present Specially-appointed Professor, Division of Preventive Ophthalmology, Zhongshan Ophthalmic

Center, Sun Yat-sen University, Guangzhou, PRC
Reporting directly to the Hospital Director, I have built and manage a full-time team of over 20
program managers, clinical trainers, statisticians, IT personnel, an image grading center and
graduate students
Secured a highly-competitive Thousand Man Plan grant from the Chinese government to create
novel programmatic solutions in the prevention and treatment of children’s eye disease in rural
China
Designed and managed the PRECOG study, serving as principal coordinator for a team of over
400 persons at 40 hospitals in 11 countries in Asia, Latin America and Africa, which established
novel tools for evaluation of cataract surgery outcomes where patient follow-up is lacking, now
forming the basis for developing a freely-available app for gobal use
Assisted in the design and execution of a major trial of 20,000 children in rural western China,
which has provided the first evidence that improving children’s vision with glasses improves
academic performance significantly, and has led to government-supported demonstration
projects in two provinces
Noemi Lois, MD, PhD, FRCS(Ed), FRCOphth
Noemi Lois, MD, PhD, FRCS(Ed), FRCOphth

Noemi Lois is a Clinical Professor of Ophthalmology at Queens University, Belfast, and
an Honorary Consultant Ophthalmic Vitreoretinal Surgeon at the Belfast Health and
Social Care Trust, Northern Ireland, UK. Prof Lois leads a programme of preclinical
and clinical research into diabetic retinopathy and its complications (diabetic macular
oedema and proliferative diabetic retinopathy). This programme of research extends
from pathogenic mechanisms of disease and risk stratification to new therapies, including determining the most
cost-effective therapeutic alternatives for this disease. The value of Prof. Lois current active grants (competitive
public or charity funding) as principal applicant or co-applicant is over £3 million. Prof. Lois has published over
100 articles in peer review journals (Scopus h-index 31), 13 book chapters and is editor of 3 textbooks, one on
its second edition. Prof Lois is ad hoc referee for 22 national and international peer-review journals and six
grant funding bodies, has given over 40 lectures as guest speaker at national and international meetings and is
editorial board member of three international journals. Prof Lois has participated, as member of the Evidence
Review Group, in 10 Single Technology Appraisals for the UK National Institute of Health and Care Excellence
(NICE) on new therapies for eye diseases, three of these on diabetic macular oedema.
Prof. Aniki Rothova

was born in Prague, former Czechoslovakia but lives and works in the Netherlands.
She received her M.D. and also her Ph.D. cum laude for a thesis entitled “Uveitis and
Systemic Disease”. She worked as an ophthalmologist and a senior researcher in the
department of Ophthalmo-immunology at the Netherlands Ophthalmic Research
Institute in Amsterdam. Later, she worked in University Medical Centre in Utrecht,
where she set up a referral centre for diagnostics of uveitis, focusing on analysis of
intraocular fluids and supervised many PhD theses on uveitis. She was appointed as full
professor in ophthalmology in 2004.
She has participated in many international projects assessing the problems of uveitis in Africa and
has initiated the formation of a diagnostic laboratory for intraocular infections in Chiang Mai, Thai-
land, South-East Asia. Her work received multiple prizes and distinctions and she was repeatedly an
invited speaker in the international meetings including American Academy of Ophthalmology.
Since four years, she works in Erasmus Medical Center in Rotterdam and her assignment is to set up
a center for patients with uveitis and to build up a creative research program in ophthalmo-immunol-
ogy. She has written more than 250 papers on uveitis, specifically focusing on the clinically relevant
research projects.

Dr. Subhadra Jalali
Dr. Subhadra Jalali, did her MBBS from govt. Medical College Jammu in 1986 and
MS from PGIMER, in 1989. She completed two year fellowship from LVPEI in 1993,
and further fellowships in USA in Ocular genetic diseases, Electrophysiology of vision
and Posterior Uveitis (1995) and in management of Retinopathy of Prematurity and
Paediatric retinal disorders (1998).
Presently she is working as a Consultant at the LVPEI since 1993 and running a very
successful exclusive Paediatric retina service. She was amongst the first group of
pioneering women in India to go for exclusive Retinal surgery practice that was an exclusive male domain
at that time. She has over 450 presentations including orations and named lectures and 135 publications in
National and International journals and many book chapters. She is a Co-investigator in various multicentric
international studies.
She is the recipient of State, National and International awards including the American Academy Achievement
award, ISCEV travel grant, P. Siva Reddy award to name a few. Her crowning glory is however the more than
350 fellows trained by her in ROP from Mexico to Azerbaijan to Indonesia and all over India through one of
the first dedicated one month hands-on ROP training program. The IJO platinum award is for her pioneering
work published on outcomes of setting up a city-wide ROP program, the first one in India and in most of the
countries. She is now working for setting up similar programs in cities and towns of India and also in neighbouring
countries. She loves dancing and enjoying various cultures around the world.
Karla Chaloupka, MD., PhD.
Education/professional training:
Subspecialty training in Neurophthalmology (AB Safran, Geneva/Switzerland)
and Oculoplastics, Lacrimal and Orbital surgery (A. McNab, Melbourne/Australia)
PhD in Nanotechnology, UCL, London/UK
Professional career:
Head and Founder of Oculoplastics, Orbital and Lacrimal Surgery since 2003
at the University Hospital Zurich, Switzerland
Regular teaching sessions at the university in Zurich (UZH/USZ) and London (RFH/UCL)
Worldwide invited lecturer and teacher for oculoplastic, orbital and lacrimal on hands surgery
Regular humanitarian commitments in Mexico to reduce avoidable blindness (project Heuberger)
Patent on a novel synthetic lacrimal tube
Prof. Clement Tan
Prof. Clement Tan, Associate Professor(A/Prof) Clement Tan took over Headship of
Ophthalmology Department, National University Hospital and National University
Singapore effective 1 June 2014. He obtained his MBBS from the National University of
Singapore in 1993. After completing his basic and advanced Ophthalmology training in
Singapore, A/Prof Clement completed a fellowship in Neuro-ophthalmology at King’s
College Hospital and the National Hospital for Neurology and Neurosurgery in London.
He received his Masters in Health Professions Education from University of Maastricht
in June 2014.
He has special interests in eye movement and pupil disorders and heads the Neuro-ophthalmology service at
the National University Hospital.
He also plays a significant role as NUHS Associate Designated Institutional Official in Residency Program and
is passionate about teaching and has been recipient of National University Hospital(NUH) Teaching Excellence
Award for 5 consecutive years (2011-2015) as such he has been awarded the NUH Eminent Teacher Award in
2016, this award is given to educators who have won the Teaching Excellence Award 5 times in a row.

Rajiv Khandekar, MD
Ophthalmologist (India), epidemiologist (UK) and low vision rehabilitation (Hong Kong)
Current work:
Guide and assist postgraduate fellows and consultant ophthalmologists of King Khaled
Eye Specialist Hospital, Riyadh Saudi Arabia – it is WHO collaborative center for
prevention of blindness in the middle east.
Subeditor of Middle East Africa Journal of Ophthalmology (MEAJO)
Editorial board member of three international journals of ophthalmology and reviewer of 15 ophthalmology
journals.
More than 140 indexed publications as first or lead author.
Work in the Past:

Clinical and surgical ophthalmology: From 1981 till 1993
Prevention of blindness and deafness: public health in ophthalmology, National health program management
from 1993 till 2012.
Post graduate diploma in epidemiology in 2003
Trained in low vision rehabilitation by WHO and Hong Kong society for the blind – 2005.
Worked as WHO consultant to government and non-government organizations for eye and ear health care
Hon attaché physician to University of British Columbia, Vancouver, Canada from 1999 till 2005
Research guide to post graduate ophthalmology students five eye institutes ofIndia and Nepal
Sirisha Senthil, MD
Dr. Sirisha Senthil completed her medical education from Rajah Muthiah Medical
College, Annamalai University, followed by postgraduate training in ophthalmology
from Aravind Eye Hospital, Madurai, Tamil Nadu, securing a first rank in ophthalmology
and otorhinolaryngology from the university (1993-1994). She did her FRCS in
Ophthalmology from Edinburgh in 2000, followed by a fellowship in glaucoma at LVPEI
and training in contact lens, phacoemulsification and small incision cataract surgery at
Chennai and Madurai. She worked at several hospitals as registrar, assistant professor and consultant before
joining LVPEI in 2007. She is an investigator for several studies, has published scientific papers in peer-reviewed
journals and co-authored a book ‘Clinical Ophthalmology Made Easy’. She has presented and published over
60 papers at national and international fora. She received the “Best scientific paper” award at the National
and International meetings. Glaucoma Society meetings in 2007, APOC 2012, APAO and AIOS 2013, and “Best
video” award at GSI in 2007, 2009 and 2011, 2012, Fusion 2012 and WGC 2015. She specialises in managing
refractory adult and paediatric glaucoma. Her other areas of interest are glaucoma drainage implants and
managing combined cataract and glaucoma.
OPHTHALMOLOGISTS GRADUATE 2015-2016
Universitas Airlangga Universitas Brawijaya

1 dr. Aquirina Caesari Putri, Sp.M 1 dr. Afriani Jacob Musu, Sp.M
2 dr. Erwanda Fredy P, Sp.M 2 dr. Ahriani Achmad, Sp.M
3 dr. Fheryanto, Sp.M 3 dr. Harir Rahmaniah, Sp.M
4 dr. Fitria Kusumastuti, Sp.M 4 dr. Helen Kusumaningsih, Sp.M
5 dr. Gredy Editha Aryani, Sp.M 5 dr. Irma Suryani, Sp.M
6 dr. Indra Ihsan Utama, Sp.M 6 dr. Mutia Hapsari R., Sp.M
7 dr. Irfan Reza Primadi, Sp.M 7 dr. Niken Nuringsih R., Sp.M
8 dr. Joko Triyono, Sp.M 8 dr. Nuke Erlina Mayasari, Sp.M
9 dr. Kholid Maharri, Sp.M 9 dr. Ria Indriyanie, Sp.M
10 dr. Marthasari Rosalina, Sp.M 10 dr. Saiful Rijal, Sp.M
11 dr. Masmeryani, Sp.M 11 dr. Shanti Widya Haryati, Sp.M
12 dr. Neni Daniati, Sp.M 12 dr. Teddy Kristian, Sp.M
13 dr. Nevita Yonnia Ayu Soraya, Sp.M 13 dr. Yasmin Sani Aprilliantina, Sp.M
14 dr. Rezekian, Sp.M
15 dr. Riska Andriani, Sp.M Universitas Diponegoro
16 dr. Rochmatul Hidayati, Sp.M 1 dr. Agus Setyawan, Sp.M
17 dr. Sigit Arihandoko, Sp.M 2 dr. Amy Aurelian, Sp.M
18 dr. Siswi Hapsari Wahyuningtyas, Sp.M 3 dr. Andhika Guna Dharma, Sp.M
19 dr. Sukoto, Sp.M 4 dr. Bolia, Sp.M
20 dr. Tri Muliasih, Sp.M 5 dr. Denti Puspasari, Sp.M
21 dr. Vinca Desyandri, Sp.M 6 dr. Dera Tresna Utami, Sp.M
22 dr. Windi Indria Rini, Sp.M 7 dr. Hadijah, Sp.M
23 dr. Maitri Anindita, Sp.M 8 dr. Indrianingrum, Sp.M
9 dr. Leidina Rachmadian, Sp.M
UNIVERSITAS ANDALAS 10 dr. Raras Suksmaprasasta, Sp.M
1 dr. Afida Yanti, Sp.M 11 dr. Riani Nilam Sari P., Sp.M
2 dr. Fitriliza Hamdy, Sp.M 12 dr. Yuda Saputra, Sp.M
3 dr. Haves Ashan, Sp.M
4 dr. Ike Rahayu, Sp.M
5 dr. Indah Niani, Sp.M
6 dr. Linda Wira Putri, Sp.M
7 dr. Mayasari Nasrul, Sp.M
8 dr. Obdes Maharni Emputri, Sp.M
9 dr. Riko Kurniawan, Sp.M
Universitas Gajah Mada Universitas Indonesia

1 dr. Ahmad Ikliluddin, Sp.M 1 dr. Alia Arianti, Sp.M
2 dr. Ahmad Prasetianto Utomo, Sp.M 2 dr. Asri Setiawati, Sp.M
3 dr. Atik Rahmawati, Sp.M 3 dr. Astriviani Widyakusuma, Sp.M
4 dr. Cut Elvi Dewi, Sp.M 4 dr. Azrina Noor, Sp.M
5 dr. Daruwati, Sp.M 5 dr. Cut Putri Samira, Sp.M
6 dr. Hasiholan Tigor Adityo Hasibuan, Sp.M 6 dr. Damara Andalia, Sp.M
7 dr. Nindyan Prawasari, Sp.M 7 dr. Dian Farikha, Sp.M
8 dr. Reinne Natali Christine, Sp.M 8 dr. Faresa Hilda, Sp.M
9 dr. Sofia Risti Widhyarti, Sp.M 9 dr. Monika Ratnasari, Sp.M
10 dr. Vita Ayu Paramita, Sp.M 10 dr. Nashrul Ihsan, Sp.M
11 dr. Seruni Era Lestari, Sp.M
UNIVERSITAS HASANUDIN 12 dr. Tri Rejeki Herdiana, Sp.M
1 dr. A. Amna Rahmi, Sp.M
2 dr. Afriani Djibran, Sp.M Universitas Diponegoro
3 dr. Aisyah Muhlisah, Sp.M 1 dr. Anggitya Rullantika, Sp.M
4 dr. Aminah, Sp.M 2 dr. Anita Oktaputri, Sp.M
5 dr. Andi Hasyim Asy'ari, Sp.M 3 dr. Ariawan Priguna, Sp.M
6 dr. Ariyanie Nurtania, Sp.M 4 dr. Arief Akhdestira Mustaram, Sp.M
7 dr. Fahmiyanti Arsi, Sp.M 5 dr. Astri Avianty, Sp.M
8 dr. Faradillah Nasaruddin, Sp.M 6 dr. Fitri Wisnuwardani, Sp.M
9 dr. Ferdiana Sarunggallo, Sp.M 7 dr. Heru Syahputra, Sp.M
10 dr. Hendra Kusuma, Sp.M 8 dr. Nina Manggiasih, Sp.M
11 dr. Junelly Vimala Jaury, Sp.M 9 dr. Putri Hartini, Sp.M
12 dr. Miftahul AKhyar Latief, Sp.M 10 dr. R. Sophia Marviani, Sp.M
13 dr. Muhammad Misbah, Sp.M 11 dr. Riani Kartika Asrindi, Sp.M
14 dr. Ratih Natasha M., Sp.M 12 dr. Sandraningrum Tri Putranti, Sp.M
15 dr. Ririn Nislawati, Sp.M 13 dr. Sri Hardwiyani, Sp.M
16 dr. Rosmiaty Z. A., Sp.M 14 dr. Syafarudin Fadli, Sp.M
17 dr. Soraya, Sp.M 15 dr. Verna Kusuma, Sp.M
18 dr. Sulyanti Rachman B., Sp.M 16 dr. Wirawan Adikusuma, Sp.M
Universitas Sam Ratulangi Universitas Sumatra Utara

1 dr. Franciscus Paparang, Sp.M 1 dr. M. Syukri Hamonangan, Sp.M
2 dr. Franky Richard Kasih, Sp.M 2 dr. Mila Karmila, Sp.M
3 dr. Linda Vonica, Sp.M 3 dr. Musda Hidayati, Sp.M
4 dr. Roy Umboh, Sp.M 4 dr. Azrina Noor, Sp.M
UNIVERSITAS SRIWIDJAYA UNIVERSITAS UDAYANA

1 dr. Irnawati Nuna, Sp.M 1 dr. Dewa Gede Benny Raharja Prabawa, Sp.M
2 dr. Agus Nugroho, Sp.M 2 dr. Etika Widhiastuti, Sp.M
3 dr. Chair Jauhari, Sp.M 3 dr. Harumi Purwa Prahesty, Sp.M
4 dr. Edwin Bayu Saputra, Sp.M 4 dr. I.G.A. Ratna Suryaningrum, Sp.M
5 dr. Frida Lestari, Sp.M 5 dr. Ni Made Oka Handayani, Sp.M
6 dr. Junaidi, Sp.M 6 dr. Ni Made Widya Mahayani, Sp.M
7 dr. Mahar Jaya Purba, Sp.M 7 dr. Novianti Primasari, Sp.M
8 dr. Mira Retna Tetiana, Sp.M 8 dr. Nyoman Novita Rismawati, Sp.M
9 dr. Novia Nasir, Sp.M 9 dr. Nyoman Yenny Khristiawati, Sp.M
10 dr. Nurul Widiati, Sp.M 10 dr. Yulia Dewi Suandari, Sp.M
11 dr. Nyimas Farida, Sp.M
12 dr. R.M. Irsan, Sp.M
13 dr. Rafrida Amnovebrianthy, Sp.M
14 dr. Ratna Juwita, Sp.M
15 dr. Wisudawan Abdullah, Sp.M
Congress Information
Congress Venue
Shangrila Hotel
Jakarta, Indonesia.
Secretariat
During The Congress
VIP Room
Shangri-La Hotel
After The Congress

Baile Bld. Room 101
Jl. Kimia
Jakarta Pusat
Language
English is the official language of the congress. However, in some presentations Indonesian language
may be used. There will be no simultaneous translation.
Registration and Badge Pickup

Venue: Main Lobby, Shangri-La Hotel
Please bring along your registration confirmation letter.
The Registration Desk will operate during the following hours:
Day Date Time
Day 0 Wednesday, 28 September 2016 16.00—20.00
Day 1 Thursday, 29 September 2016 07.30—20.00
Day 2 Friday, 30 September 2016 07.30—18.00
Day 3 Saturday, 1 October 2016 07:30—17.00
Nametag
Please wear your nametag during all sessions.
Certificate of Attendance
Your certificate of attendance will be sent via email after the conference
Internet and E-mail

Free internet facilities are available in certain booth exhibition during congress hours.
Coffee Breaks
Coffee, tea, and snack will be served in the exhibition area during the coffee breaks between
session, as specified in the Timetable.
Lunch
Box lunch will be served for participants who attend the lunch symposium on Friday 30 September
and Saturday 1 October from 12.:00-13:30. Please see the scientific program detail for lunch
symposium’s topics
Continuing Medical Education Accreditation

This meeting is recognized by the Indonesian Medical Association for a minimum of 18 CME Credits,
Speakers are eligible for an additional 14 credits.
CME Examination
CME Examination will be applied in the following subjects:
• Cataract
• Infection and Immunology
• Pediatric Ophthalmology
• Glaucoma
Prayer Room
Prayer room located at the shopping arcade area. Friday praying will be held at the Ceria Room
Acknowledgements
We wish to thank the following companies who,
through their generosity, have helped make this congress possible:
DIAMOND
PLATINUM
GOLD
SILVER
Venue Map

Information for Speakers
Speakers Preview Room
The Speakers Preview Room is located at Board Room 1, level 3 Shangri-la Hotel. Speakers are requested to
upload their presentations at the Speakers Preview Room at least 3 hours before the start of their sessions. If
your presentation is scheduled in the early morning session, please upload your presentation slides the day
before.
Important Note: It is compulsory for all free paper presentations to be uploaded in the Speakers Preview Room
at least 3 hours before the start of their sessions. Free Paper Presenters will not be allowed to operate their own
computers at the podium. The committee will provide the computer and operate the presentation from the
multi media counter in the meeting room.
For Speakers who wish to use Keynote for Mac OS computers will need to use their own computers at the
podium.
The Speakers Preview Room will operate during the following hours:
Day Date Time

Day 0 Wednesday, 28 September 2016 16.00—20.00
Day 1 Thursday, 29 September 2016 07.30—20.00
Day 2 Friday, 30 September 2016 07.30—18.00
Day 3 Saturday, 1 October 2016 07:30—17.00
The data you provide will be temporarily stored on the server in the Speakers Preview Room. The organizer will
take responsibility for erasing all data after the conference. Overhead projector slides, video tapes, and sound
data cannot be used for presentations.
Presentation format
Only presentations using PowerPoint are acceptable. Overhead projector slides, video tapes, and sound data
cannot be used for presentations.
Data Format
MS Office: PowerPoint 2000, 2003, 2007, 2010, 2013, and 2016
Mac OS X: Office PowerPoint 2004, 2008, 2011, and 2015
Video Format
Windows: Media Player Classic
Mac OS X: QuickTime Player and VLC Player
Video presentations must be in high resolution to ensure clarity on digital HD screens.
For Speakers Using Own Personal Computer (Not applicable for free paper presentations)
AV staff will check the output of your computer using an LCD Monitor. Please bring your own power cable for
you computer. If you are using a Mac computer, please bring your cable connection to VGA projector.
The Time Keeping
Speakers should take note, session chair will notify the presenter 1 (one) RING 1 minute before presentation
time ends and 2 (two) RINGS when presentation time is finished.
PROGRAM OVERVIEW
PROGRAM OVERVIEW Day1, THURSDAY, 29 September 2016
Time Ballroom (1) Ballroom (2) Ballroom (3) Satoo Garden (4) Ceria Room (5)
08.30 - 10.00 RAP 1 IKPAMI
10.00 - 10.30 Coffee Break 1
The Commission The Commission The Commission The Commission

10.30 - 12.00 IKPAMI
Meeting Meeting Meeting Meeting
12.00 - 13.00 Lunch
13.00 - 15.10 RAP 2
15.10 - 16.15 Coffee Break 2
16.15 - 18.00 Opening Ceremony IKPAMI
PROGRAM OVERVIEW Day1, FRIDAY, 30 September 2016
Glaukoma INASCRS Refraction

07.30 - 08.30 INAVRS Meeting
Meeting Meeting Meeting
Update in
PO
Retina Glaucoma Indonesia Eye Oculoplasty
08.30 - 10.00 Strabismus
(RET S 2.1) (GLAU S 2.1) Health Care (ROO DC 2)
(POS S2)
System
10.00—10.30 Coffee Break 1
ORBIS Diabetic Misc. Problems Video Session Medical

10.30 - 11.30 Retinopathy of Retina Cataract Education
(ODR 2) (RET-MP 2) (ZEISS) (MEW 2)
11.30 - 12.30 Friday Prayer
Abbott Lunch Pfizer Lunch

12.30 - 14.00
Symposium Symposium
PO
Retina Glaucoma Oculoplasty
14.00 - 15.30 Strabismus
(SRET S 2.2) (GLAU S 2.2) Ophthalmology (ROO S 2)
(POS S2)
Community
(OPHCOM 2)
PO Oculoplasty
Retina Glaucoma
15.30 - 17.00 Strabismus (OS 2)
(SRET S 2.2) (GLAU S 2.3
(POS VS 2)
Coffee Break 2
PROGRAM OVERVIEW, Day 2, FRIDAY 30 September 2016
Surabaya Padang Room Medan Room Denpasar Yogyakarta Bandung

Time
Room (6) (7) (8) Room (9) Room (10) Room (11)
INOIIS
07.30 - 08.30 ROO Meeting NO Meeting POS Meeting
Meeting
08.30 - 10.00 FP FP FP FP FP FP
10.00 - 10.30 Coffee Break 1
10.30 - 11.30 FP FP FP FP FP FP
11.30 - 12.30
12.30 - 14.00
14.00 - 15.30 FP FP FP FP
CATARACT FP
15.30 - 17.00
(CAT FP 3)
Coffee Break 1
PROGRAM OVERVIEW Day 3, SATURDAY 1 October 2016
Infection Neuro
Cataract Video Contest Refraction
08.30 - 10.00 Immunology Ophthalmology
(CAT S 3.1) (VC 3) (REF S 3.1)
(II S 3.1) (NO 3.1)
10.00 - 10.30 Coffee Break 1
Video Session
Cataract Retina Infeksi Neuro
Refraction
10.30 - 12.00 (CAT S 3.2) (OPTIK Immunology Ophthalmology
(LV W 3)
TUNGGAL) (II S 3.2) (NO 3.2)
SMEC Lunch Kalbe Lunch Lunch Social

12.00 - 13.30
Symposium Symposium Program
Infection Neuro Ophthalmology

Cataract Refraction
13.30 - 15.00 Immunology Ophthalmology Community
(COR S 3) (REF S 3.2)
(II S 3.3) (NO 3.3) (OPHCOM 3)
15.00 - 15.30 Coffee Break 1
15.30 - 16.00 Plenary lecture
16.00 - 16.30 Closing
PROGRAM OVERVIEW Day 3, SATURDAY 1 October 2016
Surabaya Padang Room Medan Room Denpasar Yogyakarta Bandung

Time
Room (6) (7) (8) Room (9) Room (10) Room (11)
08.30 - 10.00 ARSAMI IAPB
10.00 - 10.30
RSCM
10.30 - 12.00 ORBIS PO IAPB KIRANA COE
(COE 3)
12.00 - 13.30
13.30 - 15.00 IAPB

15.00 - 15.30

15.30 - 16.00
16.00 - 16.30
Day 1, THURSDAY, 29 September 2016
OPENING CEREMONY
Ball Room
16.30 – 18.30
Time Description
16.30 - 16.32 Opening session begin
16.32 - 16.38 National Anthem & PERDAMI Hymne
16.38 - 16.41 Video Presentation
16.41 - 16.45 Betawi Traditional Dance
Remark by Chairman
16.45 - 16.53
Ari Djatikusumo, MD
Remark by Ministry of Health Republic Indonesia

16.58 - 17.08
17.08 - 17.13 Remark by the newly elected IOA President

17.13 - 17.40 Commencement Ophthalmology Residency 2015 -2016
Mardiono Marsetio Memorial Lecture

17.40 - 17.55 Introduction: Fatma Asyari, MD
Lecturer: Tjahjono D. Gondhowiardjo, MD. PhD.
Plenary Lecture: Financial Planning

17.55 - 18.25
Safir Senduk
18.25 - 18.30 Closing
DAY
2
FRIDAY 30 September 2016
Day 2, FRIDAY, 30 September 2016
RETINA SYMPOSIUM (RET S 2.1)
Ballroom 1
08.30 - 10.00 hrs.
Chair Prof. Khalilul Rahman, MD Padang
Co-Chair Sauli Ari Widjaja, MD Surabaya
Time Topic Speaker
RET S 2.1.1 08.30-08.42 Current update in AMD Prof. Nathan Congdon, MD Hongkong
Systematic approach to
RET S 2.1.2 08.42-08.54 Prof. Noemi Lois, MD Northern Ireland
pediatric retinal disease
RVO : Laser, steroid and

RET S 2.1.3 08.54-09.06 Gilbert W. Simanjuntak, MD Jakarta
Anti-VEGF
RET S 2.1.4 09.06-09.18 CMV Retinitis Erwin Iskandar, MD Bandung
Anti-VEGF therapy and

RET S 2.1.5 09.18-09.30 progression of geographic Elvioza, MD Jakarta
atrophy
RET S 2.1.6 09.30-09.42 Approach to Chronic CSR Angela Nurini Agni, MD Yogyakarta
09.42-10.00 Discussion
CME
ORBIS - PERDAMI DIABETIC RETINOPATHY (ODR 2)
Ballroom 1
10.30 - 11.30 hrs
Chair : Gitalisa Andayani, MD Jakarta
Co Chair : Mario Hutapea, MD Jakarta
Time Topic Speaker
Lessons from Orbis DR

ODR 2.1 10.30 – 10.45 Programs in China and Other Prof. Nathan Congdon, MD Hongkong
Settings
Diabetic Retinopathy in
ODR 2.2 10.45 – 11.00 Indonesia: Prevalence, Gitalisa Andayani, MD Jakarta
Changes, and Challenges
New Approaches to Clinical

ODR 2.3 11.00 – 11.15 DR Treatment Which May be Prof. Noemi Lois, MD Northern Ireland
Relevant to Indonesia
Discussion: How can we (Orbis, QUB, Indonesian colleagues) best collaborate

11.15 – 11.30
around the issue of DR in Indonesia?
SURGICAL RETINA SYMPOSIUM (SRET S 2.2)
Ballroom 1
14.00 - 15.30 hrs.
Chair Iwan Sovani, MD Bandung
Co-Chair Afrisal Hari Kurniawan, MD Semarang
Time Topic Speaker
Macular hole: posture and

SRET S 2.2.1 14.00-14.12 Sjakon G. Tahija, MD Jakarta
peeling
Scleral Buckling: Pearls and

SRET S 2.2.2 14.12-14.24 Djonggi P. Pangabean, MD Bandung
Pitfall for beginner
The Role of Anti-VEGF in VR

SRET S 2.2.3 14.24-14.36 Andi Arus Victor, MD Jakarta
Surgery
SRET S 2.2.4 14.36-14.48 How to prevent PVR Waldensius. Girsang, MD Jakarta
VR surgery post operative

SRET S 2.2.5 14.48-15.00 Wimbo Sasono, MD Surabaya
complication
Simple VR procedure for

SRET S 2.2.6 15.00-15.12 Arif Wildan, MD Semarang
general ophthalmologist
MISCELLANEOUS PROBLEMS OF RETINA (RET-MP 2)
Ball Room 2
10.30 - 11.30 hrs.
Chair Prof. Noemi Lois, MD Northern Ireland
Co-Chair Referano Agustiawan, MD Jakarta
Time Topic Speaker
Antithrombotic agents in
RET-MP 2.1 10.30 - 10.50 Susilo Chandra, MD Jakarta
retinal surgery – friend or foe
Vitrectomy with Wide Field

Viewing System in Managing
RET-MP 2.2 10.50 - 11.00 Retina Detachment Ari Djatikusumo, MD Jakarta
Associated with Anterior
Retina Break
Update Inherited Retinal

RET-MP 2.3 11.00 - 11.10 Prof. Noemi Lois, MD Northern Ireland
Dystrophy
Unspecified Clinical Sign of

RET-MP 2.4 11.10 - 11.20 Anggun Rama Yudantha, MD Jakarta
Retinitis Pigmentosa
Neovascular Glaucoma in
RET-MP 2.5 11.20 - 11.30 Prof. Nathan Congdon, MD Hongkong
Diabetic Retinopathy
Discussion
RETINA SYMPOSIUM (RET S 2.3)
RECENT INNOVATION IN VITREO-RETINA
Ballroom 1
15.30 - 17.00 hrs.
Chair Rumita Kadarusman, MD Jakarta
Co-Chair Anggun Rama Yudantha, MD Jakarta
Time Topic Speaker
OCT angiography: principles

RET S 2.3.1 15.30-15.42 Gitalisa Andayani, MD Jakarta
and applications
A new technology for

RET S 2.3.2 15.42-15.54 Prof. Noemi Lois, MD Northern Ireland
vitrectomy
Prospects and limitations of

RET S 2.3.3 15.54-16.06 artificial visions, stem cell Arief S Kartasasmita, MD Bandung
therapy and gene therapy
The New Generation of anti

RET S 2.3.4 16.06-16.18 Heksan, MD Padang
VEGF
Future Pharmacotherapies
RET S 2.3.5 16.18-16.30 Habibah Muhiddin, MD Makassar
for Diabetic Retinopathy
RET S 2.3.6 16.30-16.42 Curent trend of Laser in VR Eko F. Karim, MD Jakarta
GLAUCOMA SYMPOSIUM (GLAU S 2.1)
Ballroom 2
08.30 - 10.00 hrs.
Chair : Maksum Effendi, MD Malang
Co Chair : Andrini Ariesti, MD Padang
Time Topic Speaker
GLAU S 2.1.1 08.30-08.40 Target Intraocular Pressure Fitratul Ilahi, MD Padang
Update in pathogenesis of
GLAU S 2.1.2 08.40-08.50 Fifin L Rahmi, MD Semarang
glaucoma
Is this glaucomatous optic

GLAU S 2.1.3 08.50-09.00 Sirisha Senthyl, MD India
neuropathy?
GLAU S 2.1.4 09.00-09.10 When to do trabeculectomy Astrianda Suryono, MD Jakarta
GLAU S 2.1.5 09.10-09.20 Basic trabeculectomy Maula Rivada, MD Bandung
GLAU S 2.1.6 09.20-09.30 Releasable suture Agus Kusumadjaja, MD Bali
Techniques to enhance
GLAU S 2.1.7 09.30-09.40 Yulia Primitasari, MD Surabaya
successful trabeculectomy
Intraoperative
GLAU S 2.1.8 09.40-09.50 trabeculectomy complication Elsa Gustianty, MD Bandung
management
Postoperative
GLAU S 2.1.9 09.50-10.00 trabeculectomy complication Evelyn Komaratih, MD Surabaya
management
CME
Ballroom 2
14.00 - 15.30 hrs.
Chair : Fidalia, MD Palembang
Co Chair : Aulia Rahman, MD Malang
Time Topic Speaker
Problem arising due to

GLAU S 2.2.1 14.00-14.15 Virna D Oktarina, MD Jakarta
glaucoma mismanagement
Congenital glaucoma and its

GLAU S 2.2.2 14.15-14.30 Retno Ekantini, MD Yogyakarta
management
Management of co-exsisting
GLAU S 2.2.3 14.30-14.45 Nurwasis, MD Surabaya
cataract and glaucoma
Ocular hypertension and its

GLAU S 2.2.4 14.45-15.00 Masitha Dewi Sari, MD Medan
management
GLAU S 2.2.5 15.00-15.15 Glaucoma due to trauma Novanita Satolom, MD Manado
How to deal with uveitic

GLAU S 2.2.6 15.15-15.30 Andhika Prahasta, MD Bandung
glaucoma
Ballroom 2
15.30 - 17.00 hrs.
Chair : Syukri Mustafa, MD Jakarta
Co Chair : Maharani Cahyono, MD Semarang
Time Topic Speaker
Update in glaucoma
GLAU S 2.3.1 15.30-15.45 Noro Waspodo, MD Makassar
medications
Current update of Angle

GLAU S 2.3.2 15.45-16.00 Widya Artini, MD Jakarta
Closure Glaucoma
Management of Malignant
GLAU S 2.3.3 16.00-16.15 Donny Istiantoro, MD Jakarta
Glaucoma
What we have to do with

GLAU S 2.3.4 16.15-16.30 Tatang Talka Gani, MD Yogyakarta
normal tension glaucoma?
Tips and tricks of Glaucoma

Drainage Devices
Complicated cases in
Glaucoma Drainage Devices
PEDIATRIC OPHTHALMOLOGY & STRABISMUS SYMPOSIUM (POS S2)
Ballroom 3
08.30 - 10.00 hrs.
Chair : Luki Indriaswati, MD Surabaya
Co Chair : Mayasari Wahyu, MD
Time Topic Speaker
Underdiagnosis in Retinal
POS S2.1 08.30-08.45 Prof. Rita Sitorus, MD, PhD Jakarta
Dystrophy
What Do They Really See

POS S2.2 08.45-09.00 Subhadra Jalali, MD India
After ROP Management?
Management of sixth nerve

POS S2.3 09.00-09.15 Luki Indriaswati, MD Surabaya
palsy
Familial Exudative
Vitreoretinopathy
Neonatal Eye Problems

with Special Emphasis on
Non-ROP Neonatal Retinal
Problems
POS S2.6 09.45-10.00 Blurred or double vision? Anna Bani, MD Jakarta
CME
PEDIATRIC OPHTHALMOLOGY & STRABISMUS PANEL DISCUSSION (POS PD 2)
Ballroom 3
14.00 - 15.30 hrs.
Chair : Prof. Rita Sitorus, MD, PhD Jakarta
Panelists : Feti K. Memed, MD, PhD Bandung
Gusti G. Swardana, MD Jakarta
Time Topic Speaker
Congenital Cystic Eye, How

POS PD 2.1 14.00-14.15 Julie D. Barliana, MD Jakarta
To Manage?
Secondary Glaucoma in
POS PD 2.2 14.15-14.30 Kemala Sayuti, MD Padang
Anterior Segment Dysgenesis
Is It PHPIFAP (Ichtyosis
POS PD 2.3 14.30-14.45 Follicularis, Alopecia, and Primawita Oktarima, MD Bandung
Photophobia) Syndrome
Optic disc mass in a patient

with acute lymphoblastic
POS PD 2.4 14.45-15.00 Eka Sutyawan, MD Bali
leukemia: leukemic
infiltration or primary tumor?
POS PD 2.5 15.00-15.15 Macular edema in children Yulinda Simiatno, MD Jakarta
Case report : Lens luxation

POS PD 2.6 15.15-15.30 Liana Ekowati, MD Semarang
in Homocystinuria
PEDIATRIC OPHTHALMOLOGY & STRABISMUS VIDEO SESSION (POS VS 2)
Ballroom 3
15.30-17.00 hrs.
Chair : Feti K. Memed, MD, PhD Bandung
Co Chair : Gusti G. Suardana, MD Jakarta
Time Topic Speaker
How to manage anterior

POS VS 2.1 15.30-15.40 Balgis Desy, MD Medan
Persistent Fetal Vasculature
Primary Posterior
Capsulotomy & Anterior
POS VS 2.2 15.40-15.50 Prof. Rita Sitorus, MD, PhD Jakarta
Vitrectomy: Pars Plana
Approach
POS VS 2.3 15.50-16.00 Intermitten XT Gusti G. Suardana, MD Jakarta
POS VS 2.4 16.00-16.10 Learning strabismus surgery Lely Wulandari, MD Malang
POS VS 2.5 16.10-16.20 Traumatic cataract Dian Estu Y, MD Jakarta
POS VS 2.6 16.20-16.30 Marfan syndrome Florence Manurung, MD Jakarta
POS VS 2.7 16.30-16.40 Secondary implant Adhi Wicaksono, MD Jakarta
Improvement in patient's
quality of life following small
POS VS 2.8 16.40-16.50 angle esotropia surgery: Rozalina Loebis, MD Surabaya
focusing on psychological
impact
POS VS 2.9 16.50-17.00 Myotomi inferior oblique Feti K. Memed, MD, PhD Bandung
OPHTHALMOLOGY COMMUNITY (OPHCOM 2)
SOSIALISASI KOMITE MATA NASIONAL
Satoo Garden Room
14.00 - 17.00 hrs.
Chair : Syumarti, MD Bandung
Co Chair : Hera Dwi Novita, MD Malang
Time Topic Speaker
OPHCOM Introduksi Komite Mata

14.00-14.20 Andy Flores Noya Jakarta
2.1 Nasional
OPHCOM Program Kerja Departemen

14.20-14.40 Nina Ratnaningsih, MD Bandung
2.2 Pendataan
OPHCOM Program kerja Departemen

14.40-15.00 Tri laksana Nugraha, MD Semarang
2.3 Pengembangan SDM

15.00-15.20 Syumarti, MD Bandung
2.4 Operasional Op. Katarak
Program Kerja Departemen

OPHCOM
15.20-15.40 Gangguan penglihatan pada Mayang Rini, MD Bandung
2.5
anak
Strategi Bisnis on Hospital Prof. Mardjo Soebiandono,

OPHCOM 2.6 15.40-16.00 Jakarta
Enterpreneurship MD
Sosialisasi Program
OPHCOM
16.00-16.20 Pengembangan koordinasi M. Sidik, MD Jakarta
2.7
dengan Organisasi Profesi
16.20-17.00 Pertemuan Seminat Oftalmologi Komunitas
RRO DIDACTIC COURSE (RRO DC 2)
ORBITAL INFLAMMATION
Ceria Room
08.30 - 10.00 hrs.
Chair : Kentar Arimadyo MD Semarang
Co Chair : Elfina G. Sadono MD
Time Topic Speaker
Pathophisiology of Orbital
ROO DC 2.1 08.30-08.50 Karla Chaloupka, MD Switzerland
inflammation
Management of orbital
ROO DC 2.2 09.00-09.20 Rossalyn Sandra, MD Jakarta
inflammation
Pseudo tumor and it’s

ROO DC 2.3 09.30- 10.50 Karla Chaloupka, MD Switzerland
management
ROO SYMPOSIUM (ROO S 2)
SIMPLE OCULOPLASTY PROCEDURE
Ceria Room
14.00 - 15.30 hrs.
Chair : Rinaldi Dahlan, MD
Co Chair : Iskandar, MD
Time Topic Speaker
Primary Clossure of Eyelid

ROO S 2.1 14.00-14.15 Rahmawati Lubis, MD Medan
Laceration
ROO S 2.2 14.15-14.30 When and How To Probing Riani Erna, MD Palembang
Diagnostic and Management

ROO S 2.3 14.30-14.45 Laksmi Utari, MD Bali
of Tearing Eye
Management of Phtysis
ROO S 2.4 14.45-15.00 Sutjipto, MD Surabaya
Bulbi/Mikroftalmi
Management of Involutional
ROO S 2.5 15.00-15.15 Hernawita Suharko, MD Jakarta
Entropion
ONKOLOGY SIMPOSIUM (OS 2)
SIMPLE OCULAR ONCOLOGY PROCEDURE
Ceria Room
15.30 - 17.00 hrs.
Chair : Hendrian Dwi Koloso S . MD Surabaya
Co Chair : Ardizal Rahman MD Padang
Time Topic Speaker
Small Lid Margin Tumor

OS 2.1 15.30-15.45 Nina Mumainah, MD Jakarta
Removal
OS 2.2 15.45-16.00 Conjuctival Tumor Removal Tri laksana, MD Semarang
OS 2.3 16.00-16.15 Dermoid Cyst Removal Ibrahim Sani, MD Surabaya
Haemangioma Capillary
OS 2.4 16.15-16.30 Neni Anggraeni, MD Jakarta
Management Make It Easy
OS 2.5 16.30-16.45 Chalazion VS Meibomian CA Purjanto Tepo Utomo, MD Yogyakarta
UPDATE IN CURRENT INDONESIA EYE HEALTH CARE SYSTEM (JKN 2)
Satoo Garden Room
08.30 - 10.00 hrs.
Chair : dr. Anang Tribowo, SpM(K)
Co Chair : dr. Fatimah Dyah, SpM
Time Topic Speaker
Latar Belakang & Proses Re- Pusat Pembiayaan Jaminan

JKN 2.1 08.30 - 08.45 Hongkong
klasifikasi INACBG Kesehatan KEMENKES RI
Prof. Dr. dr. Armen

Dasar Pemilihan Obat dalam
Muchtar, Sp.FK
JKN 2.2 08.45 - 09.00 Formularium Nasional & Jakarta
(Tim Ahli Formularium
Rasionalisasi Penggunaannya
Nasional)
Re-klasifikasi INACBG dalam
dr. Virna D.
Layanan Mata dan Obat-obat
JKN 2.3 09.00 - 09.20 Oktariana, Sp.M(K) Northern Ireland
Mata dalam Formularium
(DPKA PP Perdami)
Nasional
Sistem BPJS dalam Pelayanan

DR. dr. Bayu Wahyudi, SpOG,
JKN 2.4 09.20 - 09.35 Mata di Tingkat Primer,
MPH, MHKes, MM
Sekunder, & Tersier
09.35 - 10.00 Discussion
MEDICAL EDUCATION WORKSHOP (MEW 2)
FACILITATING REFLECTIVE HEALTHCARE PRACTITIONERS THROUGH PORTFOLIO ASSESSMENT: THE
ROLE OF CLINICAL TEACHERS IN GUIDING AND ASSESSING REFLECTION
Ballroom 2
10.30 - 11.30 hrs.
Chairs : Diantha Soemantri, MD, PhD Jakarta
Syntia Nusanti, MD Jakarta
Natalia Widiasih, MD Jakarta
Time Topic Speaker
Introduction and group

MEW 2.1 10.30 – 10.35 Syntia Nusanti, MD Jakarta
formation
Introduction: Principles of
MEW 2.2 10.35 – 10.50 reflection on learning and Diantha Soemantri, MD, PhD Jakarta
portfolio development
Group work: Developing and

MEW 2.3 10.50 – 11.10 Syntia Nusanti, MD Jakarta
assessing students’ reflection
Debriefing: Reflection upon

the process of guiding and
MEW 2.4 11.10 – 11.25 Natalia Widiasih, MD Jakarta
assessing reflection and
portfolio
Summary & take home

MEW 2.5 11.25 - 11.30 Diantha Soemantri, MD, PhD Jakarta
message (DS)
VIDEO SESSION - CARL ZEISS
Ball Room 3
10.30 - 11.00 hrs.
Chair: Johan Hutauruk, MD
Time Topic Speaker
State of Art Fundus Viewing System for

Optimal Optical Quality, Efficiency and Emil Sjahreza, MD
Patient Safety
Toric Implants make EASY – Zeiss Cataract

Setiyo Budi Riyanto, MD
Suite
The Beauty of (ReLEx) SMILE Johan Hutauruk, MD
SOSIALISASI KOMITE MATA NASIONAL
Satoo Garden Room
14.00 - 17.00 hrs.
Chair : Syumarti, MD Bandung
Co Chair : Hera Dwi Novita, MD Malang
Time Topic Speaker
OPHCOM Introduksi Komite Mata

14.00-14.20 Andy Flores Noya Jakarta
2.1 Nasional

14.20-14.40 Nina Ratnaningsih, MD Bandung
2.2 Pendataan
OPHCOM Program kerja Departemen

14.40-15.00 Tri laksana Nugraha, MD Semarang
2.3 Pengembangan SDM

15.00-15.20 Syumarti, MD Bandung
2.4 Operasional Op. Katarak
Program Kerja Departemen

OPHCOM
15.20-15.40 Gangguan penglihatan pada Mayang Rini, MD Bandung
2.5
anak
OPHCOM Strategi Bisnis on Hospital Prof. Mardjo Soebiandono,

15.40-16.00 Jakarta
2.6 Enterpreneurship MD
Sosialisasi Program
OPHCOM
16.00-16.20 Pengembangan koordinasi M. Sidik, MD Jakarta
2.7
dengan Organisasi Profesi
LUNCH SYMPOSIUM—ABOOTT
PEACE OF MIND - PREDICTABLE SUCCESSFUL IOLs AND PHACO SURGERY
Ball Room 1
12.30 - 14.00 hrs.
Co Chair : Mayang Rini, MD
Time Topic Speaker
PhacoDynamics (Mastering The Basic for Successful

13: 05 – 13:20 Johan A Hutauruk, MD
Phaco Surgery)
13:20 – 13:35 Expanding Options for Pump & Power Technology Setiyobudi Riyanto, MD
Expanding Options for Astigmatism & Presbyopia

13:35 - 13:50 Iwan Soebijantoro, MD
Correction

LUNCH SYMPOSIUM - PFIZER
Ball Room 2
12.30 - 14.00 hrs.
Chair Andika Prahasta, MD
Time Topic Speaker
Guideline in Glaucoma medication Virna D Oktarina, MD
Management of Chronic angle closure Glaucoma Ikke Sumantri, MD
CATARACT FREE PAPER (CAT FP 3)
Surabaya Room
08.30 - 10.00 hrs.
Chair : Johan Hutauruk, MD Jakarta
Co Chair : Fitria Romadiana, MD Gresik
Time Topic Speaker
How to modify
operating theater by
CAT FP 3.1 08.30 - 08.39 Dharmawan M. Sophian, MD Pandeglang
Phacoemulsification in mass
Cataract Surgery at rural area
Fluids in Phaco... mastering

CAT FP 3.2 08.39 - 08.48 Ikhsan Revino, MD Jakarta
OVD & hydroprocedure
CAT FP 3.3 08.48 - 08.57 CCC Make it easy Uyik Unari, MD Gresik
CAT FP 3.4 08.57 - 09.06 Safe IA Fitria Romadiana, MD Gresik
Hard Cataract for Young

CAT FP 3.5 09.06 - 09.15 Novita Sitompul, MD Bandung
Phaco Suegeon
CAT FP 3.6 09.15 - 09.24 Scafold technique in PCR Imam Tiharyo, MD Serang
Patient selection in multifocal Vidyapati Mangunkusumo,

CAT FP 3.7 09.24 - 09.33 Jakarta
IOLs MD
The learning curve in Toric Vidyapati Mangunkusumo,

CAT FP 3.8 09.33 - 09.42 Jakarta
IOL implantation MD
Pressure induced stromal

CAT FP 3.9 09.51 - 10.00 Johan Hutauruk, MD Jakarta
keratitis (PISK)
DAY
3
SATURDAY 1 October 2016
Day 3, SATURDAY, 1 October 2016
CATARACT SYMPOSIUM (CAT S 3.1)
RECENT ADVANCES IN CATARACT SURGERY
Ballroom 1
08.30 - 10.00 hrs.
Chair : John Jastad, MD USA
Tjahjono D. Gondhowiardjo,
Co Chair : Jakarta
MD, PhD
Time Topic Speaker
How to select your suitable

Vidyapati Mangunkusumo,
CAT S 3.1.1 08.30-08.45 candidates for Multifocal IOL Jakarta
MD
implants
A comparison of two
methods in measuring and
adjusting immediate post-
CAT S 3.1.2 08.45-08.58 John Jarstad, MD USA
operative IOP following
cataract surgery. What are
the 3 benefits?
CAT S 3.1.3 08.58-09.11 Retro Pupillary iris claw Imam Tiharyo, MD Serang
CAT S 3.1.4 09.11-09.24 Markerless Toric IOL surgery Johan Hutauruk, MD Jakarta
Femtosecond LASER Cataract

CAT S 3.1.5 09.24-09.37 Setiyo Budi Riyanto, MD Jakarta
Surgery with Premium IOL
CAT S 3.1.6 09.37-09.50 Nano Laser Cataract Surgery Gangolf Sauder, MD Germany
My first cases with Nano

CAT S 3.1.7 09.50-10.03 Hadi Prakoso, MD Jakarta
Laser Cataract Surgery
CATARACT SYMPOSIUM (CAT S 3.2)
SOLVING THE PROBLEM IN YOUR DAY TO DAY CATARACT SURGERY
Ballroom 1
10.30 - 12.00 hrs.
Chair : Darwan M. Purba, MD Jakarta
Co Chair : Novita Sitompul, MD Bandung
Time Topic Speaker
Effective Efficient Technique

CAT S 3.2.1 10.30-10.40 And Cost On Complicated R. Prabowo, MD Yogyakarta
Case Cataract Surgery
Iris prolaps: small problem

CAT S 3.2.2 10.40-10.50 Ikhsan Revino, MD Jakarta
but disaster
Managing Small Pupils
CAT S 3.2.3 10.50-11.00 Without Pupil-expanding Novita Sitompul, MD Bandung
Devices
Vitreous Prolapsduring
Phaco. How to handle the
CAT S 3.2.4 11.00-11.10 Amir Shidik, MD Jakarta
Vitreous, lens mass & IOL
Implantation
Management of post cataract
CAT S 3.2.5 11.10-11.20 surgery complication in top Syska Widyawati, MD Jakarta
refferal hospital
Scleral fixation with foldable
CAT S 3.2.6 11.20-11.30 Ucok P. Pasaribu, MD Jakarta
IOL
Phacoemulsification in
CAT S 3.2.7 11.30-11.40 Budiman, MD Bandung
Pseudoexfoliations Syndrome
Aphakic IOL as an alternative

CAT S 3.2.8 11.40.11.50 for secondary IOL Budiman, MD Bandung
Implantation
Is it possible to implant
CAT S 3.2.9 11.50-12.00 premium IOL in challenging Hadi Prakoso, MD Jakarta
situation?
CME
CORNEA SYMPOSIUM (COR S 3)
CORNEA & REFRACTIVE SURGERY
Ballroom 1
13.30 - 15.00 hrs.
Chair : Prof. Suhardjo, SU, MD Yogyakarta
Co Chair : Soeharnila, MD Jakarta
Time Topic Speaker
Tjahjono D. Gondhowiardjo,
COR S 3.1 13.30-13.43 Indonesian Eye Bank Jakarta
MD, PhD
Patching the impending

COR S 3.2 13.43-14.56 Johan Hutauruk, MD Jakarta
corneal perforation
COR S 3.3 13.56-14.09 No Touch DMEK Soeharnila, MD Jakarta
The effects of dietary

riboflavin and natural UV
light exposure on corneal
COR S 3.4 14.09-14.21 John Jarstad, MD USA
cross-linking in keratoconus
and kerectasia - a multi-
center collaborative study
CXL combined with PRK for

COR S 3.5 14.21-14.34 treating mild and moderate Prof. Suhardjo, SU, MD Yogyakarta
Keratoconus
Outcomes of Presby Lasik

Vidyapati Mangunkusumo,
COR S 3.6 14.34-14.47 . A two years follow up at Jakarta
MD
Jakarta Eye Center
COR S 3.7 14.47-14.49 ReLex Smile: Why we start it? Bambang Triwiyono, MD Jakarta
Central discoid cornea: How

COR S 3.8 14.49-15.00 Sharita Siregar, MD Jakarta
to make a baby smile
VIDEO CONTEST (VC 3)
Ball Room 2
08.30 - 10.00 hrs.
Time Topic Producer
External DCR with silicone

VC 3.1 08.30 - 09.00 Ratna Doemilah, MD Surabaya
tube intubation
Surgical approach of long

VC 3.2 09.00 - 09.30 standing traumatic cataract Devina, MD
in children
One haptic scleral fixation of

secondary posterior chamber
VC 3.3 09.30 - 10.00 Andhika Prahasta, MD Bandung
intraocular lens in aphakia
patient with iridodialysis :
Day 3, SATURDAY, 1 Oktober 2016

VIDEO SESSION — OPTIK TUNGGAL
EVA SYSTEM : ULTRA HIGH SPEED CUTTER WITH SMART FLUIDICS
Ball Room 2
10.30 - 11.00 hrs.
Chair: Ari Djatikusumo, MD

Mario Hutapea, MD
Anggun Rama Yudanta, MD
Referano Agustiawan, MD
Elvioza, MD
Ari Djatikusumo, MD
INFECTION AND IMMUNOLOGY SYMPOSIUM (II S 3.1)
TIPS AND TRICKS IN MANAGING EXTERNAL DISEASES
Ballroom 3
08.30 - 10.00 hrs.
Chair : Anang Triwibowo, MD Palembang
Co Chair : Sammy Malingkas, MD Manado
Time Topic Speaker
Allergy as an interfering
II S 3.1.1 08.30 – 08.42 factor in Ocular Surface Ratna Sitompul, MD Jakarta
problem
II S 3.1.2 08.42 – 08.54 Fungal Corneal Infection Made Susiyanti, MD Jakarta
II S 3.1.3 08.54 - 09.06 Bacterial Corneal Ulcer Prof. Suhardjo, SU, MD Yogyakarta
II S 3.1.4 09.06 – 09.18 Viral corneal infection Prof. Winarto, MD Semarang
II S 3.1.5 09.18 – 09.30 Scleritis Susi Heryati, MD Bandung
What is new in the

II S 3.1.6 09.30 – 09.42 Rosi Aldina, MD Malang
management of Dry eye
II S 3.1.7 09.42 – 09.54 Infectious conjunctivitis I Gusti Ayu Made Juliari, MD Bali
09.54 – 10.00 Discussion
WHAT IS NEW IN MANAGING UVEITIS : ARE YOU UP TO DATE ?
Ballroom 3
10.30 - 12.00 hrs.
Chair : Fatma Asyari, MD Jakarta
Co Chair : Dina Novita, MD Semarang
Time Topic Speaker
Step by step in reaching

II S 3.2.1 10.30 – 10.42 the etiological diagnosis of Prof. Aniki Rothova, MD Netherlands
uveitis
II S 3.2.2 10.42 – 10.54 Tuberculous Uveitis Rina La Distia Nora, MD Jakarta
II S 3.2.3 10.54 – 11.06 Syphilitic Uveitis Randi Montana, MD Surabaya
II S 3.2.4 11.06 – 11.18 Viral Uveitis Prof. Aniki Rothova, MD Netherlands
Retinal vasculitis: what to

II S 3.2.5 11.18 – 11.30 Soedarman Sjamsoe, MD Jakarta
do?
Endophthalmitis: prevention
II S 3.2.6 11.30 – 11.42 Lukman Edwar, MD Jakarta
and treatment
Therapeutic strategy in the

II S 3.2.7 11.42 – 11.54 Prof. Aniki Rothova, MD Netherlands
treatment of uveitis
CME
CHALLENGING CASES
Ballroom 3
13.30 - 15.00 hrs.
Chair : Endang M. Johani, MD Jakarta
Co Chair : Kemala Dewi, MD Jakarta
Panelists : Prof. Winarto, MD Semarang
Prof. N. K. Niti Susila, MD Bali
Fatma Asyari, MD Jakarta
Getri Sukmawati, MD
Time Topic Speaker
II S 3.2.1 13.30 - 13.50 Corneal ulcer Havriza Vitresia, MD Padang
II S 3.2.2 13.50 - 14.10 Non infectious keratitis Prof. Niti Susila, MD Bali
II S 3.2.3 14.10 - 14.30 Uveitis Ovie Sofia, MD Malang
II S 3.2.4 14.30 - 14.50 Scleritis Rifna Lutfiamida, MD Jakarta
NEURO-OPHTHALMOLOGY (NO 3.1)
OPTIC NEUROPATHY
Satoo Garden (4)
08.30 - 10.00 hrs.
This case oriented session will discuss optic neuropathy as well as chiasmal lesion. The objective of this
session is to increase knowledge and skill in diagnosing optic neuropathy cases. Therefore the management
of optic neuropathy will be better.
Chair : Muhammad Sidik, MD Jakarta
Co Chair : Antonia Kartika, MD Bandung
Time Topic Speaker
Current management of
NO 3.1.1 08.30-08.45 Prof. Clement Tan, MD Singapore
Optic Neuritis in Children
Multiple Sclerosis and Optic

NO 3.1.2 08.45-09.00 Bambang Setiohadji, MD Bandung
Neuritis
Controversy of steroid in
NO 3.1.3 09.00-09.15 A.A. Mas Putrawati, MD Bali
Traumatic Optic Neuropathy
Hereditary Optic Neuropathy,
NO 3.1.4 09.15-09.30 M. Hidayat, MD Padang
Is it really Exsist?
NO 3.1.5 09.30-09.45 Work Up for Chiasmal Lesion Devy Azri, MD Palembang
Ethambutol Toksik Optik

NO 3.1.6 09.45-10.00 Riski Prihatningtias, MD Semarang
Neuropathy: What’s new?
OPHTHALMOPLEGIA
Satoo Garden (4)
10.30 - 12.00 hrs.
Double vision caused by ophthalmoplegia is a challenging case for ophthalmologist. Comprehensive review
will help ophthalmologist to manage the cases.
Chair : Prof. Diany Yogiantoro, MD Surabaya
Co Chair : Devy Azri, MD Palembang
Time Topic Speaker
Simple Evaluation in
NO 3.2.1 10.30-10.45 Prof. Clement Tan, MD Singapore
Nystagmus
Myasthenia Gravis is
NO 3.2.2 10.45-11.00 the Great Mimicker of Syntia Nusanti, MD Jakarta
Ophthalmoplegia
Multiple Sclerosis and

NO 3.2.3 11.00-11.15 Antonia Kartika, MD Bandung
Ophthalmoplegia
Ophthalmoplegia and
NO 3.2.4 11.15-11.30 Yunita Mansyur, MD Makassar
Chronic Red Eye
NO 3.2.5 11.30-11.45 Frozen Eye Muhammad Sidik, MD Jakarta
Neuroimaging in
NO 3.2.6 11.45-12.00 Batari T. Umar, MD Makassar
Ophthalmoplegia
CME
CHALLENGING CASES
Satoo Garden (4)
13.30 - 15.00 hrs.
Systemic disease can affect the eye including the optic nerve and ocular motor nerve. By discussing neuro-
ophthalmic manifestation of systemic diseases, we could manage these problems comprehensively.
Chair : Bambang Setiohadji, MD Bandung
Co Chair : Syntia Nusanti, MD Jakarta
Time Topic Speaker
Neuro-Ophthalmic
NO 3.3.1 13.30-13.45 Manifestation of Tumor Prof. Clement Tan, MD Singapore
Intracranial
NO 3.3.2 13.45-14.00 Visual Field Defect in Stroke Hartono, MD Yogyakarta
Neuro-Ophthalmic Problems
NO 3.3.3 14.00-14.15 Bobby Sitepu, MD Medan
in Meningitis
NO 3.3.4 14.15-14.30 Management of Adie’s Pupil Wino Vrieda Vierlia, MD Malang
Sleep Apneu Disease and

NO 3.3.5 14.30-14.45 Seskoati, MD Malang
Ischemic Optic Neuropathy
Migrain in Neuro-Ophthalmic
NO 3.3.6 14.45-15.00 Lukisiari, MD Surabaya
Perspective
REFRACTION SYMPOSIUM (REF S 3.1)
Ceria Room
08.30 - 10.00 hrs.
Chair : Irayanti Rafki, MD Bandung
Co Chair : Alie Solahuddin, MD Palembang
Time Topic Speaker

Accommodation and it’s
REF S 3.1.1 08.30-08.42 Ani Ismail, MD Palembang
problem
How to determine refraction
REF S 3.1.2 08.42-08.54 Yudisianil E. Kamal, MD Jakarta
target in choosing IOL power
How to manage high myopia
REF S 3.1.3 08.54-09.06 Rinda Wati, MD Padang
in children
Contact lens in dry eye
REF S 3.1.4 09.06-09.12 Ariesanti TH, MD Bali
patients
Vision improvement of high
REF S 3.1.5 09.12-09.24 Lucia Sutedja, MD Jakarta
myopic wearing RGP
Contact Lens in Children
REF S 3.1.6 09.24-09.36 Christina, MD Surabaya
(problems & solve)
LOW VISION WOORKSHOP (LV W 3)
Ceria Room (5)
10.30 - 12.00 hrs.
Chair : Susanti Natalya, MD Bandung
Co Chair : Elly Silalahi, MD Medan
Time Topic Speaker

Visual acuity assessment in
LV W 3.1 10.30-10.45 Erna Niza, MD Palembang
low vision patient
How to determine
LV W 3.2 10.45-11.00 magnification power for Rajiv Khandekar, MD Saudi Arabia
distance near acuity
How to choose appropriate

LV W 3.3 11.00-11.15 Rajiv Khandekar, MD Saudi Arabia
low vision devices
The importance of visual

LV W 3.4 11.15-11..30 function assessment in low Umar Mardianto, MD Jakarta
vision patient
How to train low vision

LV W 3.5 11.30-11.45 patient to use their low Karmelita S, MD Bandung
vision devices
REFRACTION SYMPOSIUM (REF S 3.2)
Ballroom 2
13.30 - 15.00 hrs.
Chair : Prillia TS , MD Surabaya
Co Chair : Yudowati, MD Jakarta
Time Topic Speaker

Prescribing bifocal or
REF S 3.2.1 13.30-13.42 Fatimah Dyah, MD Semarang
multifocal spectacle?
Visual skill in infant and

REF S 3.2.2 13.42-13.54 preverbal children , how to Nanda Anindita, MD Malang
train
How to create a comfort

REF S 3.2.3 13.54-14.06 Ria Sandy Deneska, MD Surabaya
vision in astigmatism patients
Fitting and chose appropiate

REF S 3.2.4 14.06-14.12 contact lens type in baby and Tri Rahayu, MD Jakarta
toddler
REF S 3.2.5 14.12-14.24 Soft contact lens or RGP ? Widjajanti U, MD Bandung
Visual acuity assessment in

REF S 3.2.6 14.24-14.36 Elly Silalahi, MD Medan
severe visual impairmant
Role of ophthalmologists and
REF S 3.2.7 14.36-14.48 optometrist's in low vision Rajiv Khandekar, MD Saudi Arabia
rehabilitation services
PROGRAM KESEHATAN MATA BERBASIS KOMUNITAS
Ceria Room
13.30 - 15.00 hrs.
Chair : Aldiana Halim, MD Bandung
Co Chair : Mayang Rini, MD Bandung
Time Topic Speaker

Sight First project &
OPHCOM 3.1 13.30 -13.50 Ahmad Ashraf, MD Makassar
Childhood Blindness Program
Diabetic Retinopathy
Screening Program : from
OPHCOM 3.2 13.50-14.10 Yeni Dwi Lestari, MD Jakarta
hospital based to community
based
Program Oftalmologi
OPHCOM 3.3 14.10-14.30 Komunitas PERDAMI Cabang Hera Dwi Novita, MD Malang
Malang
Program Oftalmologi
OPHCOM 3.4 14.30 -14.50 Komunitas PERDAMI Cabang Sriana Wulansari, MD Mataram
NTB
Program Oftalmologi
OPHCOM 3.5 14.50-15.00 Komunitas PERDAMI Cabang Ni Made Ari Suryathi, MD Bali
Bali
PERDAMI - IAPB Vision 2020 Meeting
SESSION 1 BLINDNESS STAKE HOLDER PERSPECTIVE AND PROGRAMMING
Medan Room
08.30 - 15.00
Community Oph.
Chair: Dr. Nina Ratnaningsih
Chairman
Time Topic Speaker
Co-Chair IAPB/Ministry of
08.30 – 08.34 Opening Remarks: Health/Member of Parliement/
Chairman PERDAMI
Refreshing on 2014-2019 WHO Universal Eye

08.35 – 08.40 Health - Global Action Plan with situational Dr.Gilbert Simanjuntak (IAPB cc Indonesia)
analysis of Indonesia 2016
RAAB Result, what was done and to be done
08.41 – 09.15 BALITBANGKES KEMENKES
(perspective)
MoH strategy on WHO Global Action Plan after Dit.PPTM
09.16 – 09.40 dr.Lily Setyowati, MM
RAAB done KEMENKES
National Eye Committee (NEC) role and
09.41 – 09.50 NEC
strategy to fill the gap
09.51 – 10.00 Perdami/KOI role and strategy to fill the gap Dr. Tjahjono D. Gondhowiardjo KOI
10.00 – 10.30 Coffee and tea time (served)
Dr. Syumarti,SpM(K), MSc RSM Cicendo

DR. dr.H. Noor Syamsu,
Presentation from different provinces on BKMM Makasar
10.01 – 11.00 SpM(K), M.Kes
existing service delivery
dr. Eko Budi Priyanto, MARS BKMM Cikampek
Wandi S Brata MATAHATI
Presentation from existing eye program in Mr.Matthew Hanning CBM

11.01 – 11.30 Indonesia (CBM, HKI, Fred Hollow) to support
WHO UEH-GAP in Indonesia Mr. Prakteek Gupta HKI
Discussion and build final draft of recommendation of role description of blindness stake holder in
11.31 – 12.00
Indonesia
Conclusion, Vote of thanks and Group photographs
PERDAMI - IAPB Vision 2020 Meeting
SESSION 2 PROBLEM FINDINGS AND PROBLEM SOLVING IN LOCAL AREA BLINDNESS
Medan Room
13.30 - 15.00
Chair: Dr. Syumarti,SpM(K), MSc
Time Topic Speaker

How to find blindness magnitude/
13.30 – 13.45 BALITBANGKES KEMENKES
situational analysis
13.46 – 14.00 Programming and step forward Mr.Prakteek Gupta (HKI)
Working with district government:

14.00 – 14.15 Dr.dr.Habibah, SpM
advocacy and cooperation
14.16 – 14.30 Building cooperation with NGO CBM
14.31 – 14.45 Report and analysis Dr. Phuc Huynh Tan FHF
ORBIS - PERDAMI CHILDHOOD BLINDNESS SYMPOSIUM (OCB 3)
Padang Room (7)
10.30 - 12.00 hrs.
Chair : Prof. Rita S. Sitorus, MD, PhD Jakarta
Co Chair : Feti K. Memed, MD, PhD Bandung
Time Topic Speaker
Solving the Problem of

OCB 3.1 10.30 - 10.50 Uncorrected Refractive Error Nathan Congdon, MD, MPH Hongkong
Among Children in Asia
Establishment of the First

Child-Friendly Pediatric
OCB 3.2 10.50 - 11.10 Habibah Muhiddin, MD Makassar
Eye Care Center in South
Sulawesi in Indonesia
Implementing Eye Health

OCB 3.3 11.10 - 11.30 Screening of Newborns in Subhadra Jalali, MS India
Middle Income Countries
ROP Pearls and Pitfall: South

OCB 3.4 11.30 - 11.50 Marliyanti N. Akib, MD Makassar
Sulawesi Experience
COE RSCM KIRANA (COE 3)
Denpasar Room
10.30 - 12.00 hrs.
Time Topic Speaker
Epidemiologic Clinical
COE 3.1 10.30-10.45 Manifestation of Sri Linuwih Menaldi, MD Jakarta
Lepromatous Infection
Neurological Involvement in
COE 3.2 10.45-11.00 Manfaluthy Hakim, MD Jakarta
Lepra
COE 3.3 11.00-11.15 Ocular Inflammation in Lepra Made Susiyanti, MD Jakarta
COE 3.4 11.15-11.30 Uveitis Indicator in Leprosy Rosdeni, MD Jakarta
Management of
COE 3.5 11.30-11.45 Yunia Irawati, MD Jakarta
Lagophthalmos
ASOSIASI RUMAH SAKIT MATA INDONESIA (ARSAMI)
Padang Room
08.30 - 10.00 hrs
dr. Enny Cahyani Permatasari,

Chair :
Sp.M.,M.Kes
Co Chair : dr. A.K. Ansyori, SpM
Time Topic Speaker
08.30 - 08.45 Pelayanan Mata dr. Anang Tribowo, SpM(K)
08.45 - 09.00 Usulan Pentarifan JKN untuk Pelayanan Mata Prof DR Dr. Kadir SpTHT-KL(K)
09.00 - 09.20 Penilaian Alat Kesehatan drg. Arianti Anaya, MKM
09.20 - 09.35 Tinjauan Tarif JKN di RS Mata Pemerintah dr. Irayanti, SpM(K), MARS
09.35 - 10.00 Tinjauan Tarif JKN di RS Mata Swasta dr. Imsya Satari, SpM(K)
Lunch Symposium : ENHANCING YOUR OPHTHALMOLOGY PRACTICE TOGETHER WITH SMEC -
IN SCIENCE AND BUSINESS
Ball Room 1
12.00 - 13.30 hrs.
Moderator : Ikhsan Revino, MD
Co-Moderator : Balgis Desy, MD
Time Topic Speaker
12.00 - 12.05 Opening by Moderator
12.05 - 12.10 Company profile presentation
The challenges that will be faced by Ophthalmologists

12.10 - 12.25 and Eye Hospitals in the era of ASEAN Free Trade Area Imsyah Satari, MD
(AFTA) and National Health Insurance Program
Preparing the future of your ophthalmology practice: a

12.25 - 12.35 Heri Purwoko, MD
real-world tips for young ophthalmologists
12.35 - 12.45 Unraveling the story behind SMEC Manado Devy Christofel Mandagi, MD
FREE PAPER
Lunch Symposium by KALBE VISION
EYE NEUROPROTECTOR, SMALL ACTION BIG IMPACT
Ball Room 1
12.00 - 13.30 hrs.
Chair Elvioza, MD
Topic Speaker
Eye Neuroprotector, Empowerment for Child Vision Feti K. Memed, MD, PhD
Citicoline in Amblyopic Children, Does it Really Help? Florence Manurung, MD
RG-Choline, Eye Neuroprotection for All Generations Shendy Isyanto, MD
FREE PAPER
Surabaya Room
08.30 - 10.00 Chair: Retno Sasanti, MD

The Effect of Phacoemulsification and Opposite
FP-R-I- Clear Corneal Incision (OCCI) Modification to
08.30 - 08.39 NIKEN NURINGSIH Univ. Brawijaya
KBR-01 the Changes of Corneal Astigmatism in Cataract
Patients with Pre-existing Corneal Astigmatism
Visual Outcome Comparison Between Spectacle
FP-R-I- Univ. Gadjah
08.39 - 08.48 Corrected Visual Acuity and Phakic Intraocular ARYA PRADIPTA
KBR-02 Mada
Lens Implantation for High Myopia
Comparison of Intrastromal and Subconjunctival
FP-R-I- RIANI KARTIKA
08.48 - 08.57 Bevacizumab Injection Effectiveness for Corneal Univ. Padjajaran
KBR-03 ASRINDI
Neovascularization
FP-R-II- Endothelial Density and Central Corneal KARINA SATYANI Univ. Gadjah
08.57 - 09.06
KBR-04 Thickness in Diabetes Mellitus PRATIWI Mada
FP-R-II- Phacoemulsification in Post Vitrectomy Eyes;
09.06 - 09.15 WIDYA ANANDITA Univ. Indonesia
KBR-05 Result and Complications
FP-R-II- P53 Expression in Primary and Recurrent RATIH NATHASHA Univ.
09.15 - 09.24
KBR-06 Pterygium M. Hasanuddin
Retinal Acuity Meter Result and Visual Acuity
FP-R-II- Univ.
09.24 - 09.33 Post Cataract Surgery on the Patient with Age FAHMIYANTI ARSI
KBR-07 Hasanuddin
Related Cataract
FP-R-II- Dry Eeye after Phacoemulsification Cataract FERDIANA Univ.
09.33 - 09.42
KBR-08 Surgery SARUNGGALLO Hasanuddin
Visual Outcome after Cataract Surgery in
FP-O-II- ARMANTO
09.42 - 09.51 Corporate Social Responsibility Program at Tritya Univ. Airlangga
KBR-09 SIDOHUTOMO
Eye Clinic Surabaya, East Java
FP-O-II-
09.51 - 10.00 My First 1-Year Experience in Cataract Surgery RIZKI ADELIA Tritya Eye Clinic
KBR-10
10.00 - 10.30 COFFEE BREAK

FREE PAPER
Surabaya Room
10.30 - 11.30 Chair: Lukman Edwar, MD
The Effect of Topical DLBS 1425 in Various

FP-R-I- Univ.
10.30 - 10.39 Concentration on Epithelial Proliferation and BOLIA
IIM-01 Diponegoro
Hyperplasia in Rabbit Meibomian Gland
Comparison of Intravitreal Antifungal 100

FP-R-I- µg Voriconazole and 5 µg Amphotericin B in
10.39 - 10.48 ALIA ARIANTI Univ. Indonesia
IIM-02 Experimental Aspergillus flavus Endophthalmitis
Model in Rabbits
The Effect of Toxic to the Various Topical
FP-R-I- Concentration DLBS1425 towards the Univ.
10.48 - 10.57 AMY AURELIAN
IIM-03 Proliferation Degree of Goblet Cell and the Diponegoro
Secretion Mucin of Rabbit's Eyes
Outcome of fresh amniotic membrane

FP-R-II- Univ. Gadjah
10.57 - 11.06 transplantation using suturing technique in RENY SETYOWATI
IIM-04 Mada
patients with corneal ulcers
Demographic Profile, Clinical Characteristics, and

FP-R-II-
11.06 - 11.15 Treatment Evaluation of Contact Lens-Related DESRINA Univ. Indonesia
IIM-05
Corneal Ulcer in RSCM Kirana 2013-2014
Cytomegalovirus Serology Test Accuracy towards

FP-R-II- NI NYOMAN
11.15 - 11.24 CMV Retinitis in HIV Patients with CD4 Counts Univ. Airlangga
IIM-06 GERIPUTRI
Less than 100 cells/mm3
FREE PAPER
Surabaya Room
14.00 - 15.30 Chair: Umar Mardiono, MD

Correlation Between Best Corrected Visual
FP-R-II- Acuity Acquired by Snellen Chart with Potential DERA TRESNA Univ.
14.00 - 14.09
REF-11 Visual Acuity of Retinometri in Ametropic UTAMI Diponegoro
Patients
Comparison of Intelligence Quotient and Body
FP-R-II-
14.09 - 14.18 Mass Index in Myopia and Non Myopia Students INDAH NIANI Univ. Andalas
REF-12
of Senior High Schools in Padang
The Difference in Delta Spherical Equivalent
FP-R-II- According to the Level of Axial Length Standard Univ.
14.18 - 14.27 INDRIANINGRUM
REF-13 Deviation of Post Phacoemulsification in Cataract Diponegoro
Surgery Patient at Kariadi Hospital Semarang
FP-R-II- Characteristic of Myasthenia Gravis in National SRI REZEKI
14.27 - 14.36 Univ. Padjajaran
NO-11 Eye Centre Cicendo Eye Hospital HANDAYANI
FP-R-II- Visual Field Deffect and Ocular Motor AISYAH Univ.
14.36 - 14.45
NO-12 Dysfunction in Stroke Patient MUHLISAH Hasanuddin
FP-R-II- Visual Field Defect in Pulmonary Tuberculosis
14.45 - 14.54 AGUS NUGROHO Univ. Sriwijaya
NO-13 Patient with Ethambutol Therapy
Karakteristik Klinis Pasien Melanoma Maligna
FP-R-II-
14.54 - 15.03 Okular di Pusat Mata Nasional Rumah Sakit Mata PUTRI HARTINI Univ. Padjajaran
ROO-11
Cicendo
Karakteristik Penderita Grave’s Ophthalmopathy
FP-R-II-
15.03 - 15.12 di Pusat Mata Nasional Rumah Sakit Mata IVA YULIA Univ. Padjajaran
ROO-12
Cicendo Bandung
Profile of Primary Malignant Eye Tumors Patients
FP-R-II-
15.12 - 15.21 in Reconstruction, Oculoplasty and Oncology SHANTI WIDYA H. Univ. Brawijaya
ROO-13
Subdivision dr. Saiful Anwar Hospital Malang

FREE PAPER
Padang Room
08.30 - 10..00 Chair: AA. Mas Putrawati T, MD
The Effect of Mirtogenol Towards The Changes
FP-R-I- in Retinal Nerve Fiber Layer Thickness and ASTRIVIANI
08.30 - 08.39 Univ. Indonesia
GLA-01 Visual Field in Primary Open Angle Glaucoma WIDYAKUSUMA
The Change of Intraocular Pressure Between

FP-R-I- Hyperbaric Normoxia Conditions in Naval IRFAN REZA
08.39 - 08.48 Univ. Airlangga
GLA-02 Personnel PRIMADI
Visual Field Defect Based on Cup Disc Ratio of

FP-R-II- the Optic Nerve Head and Peripapillary Retinal KRISNA DWI Univ. Gadjah
08.48 - 08.57
GLA-03 Nerve Fiber Layer PURNOMO JATI Mada
Corneal Endothelial Cell Density and Central

FP-R-II- Corneal Thickness in Glaucoma at Dr. Sardjito Univ. Gadjah
08.57 - 09.06 NISITA SURYANTO
GLA-04 General Hospital Yogyakarta Mada
Role of Peripapillary Retinal Nerve Fiber

FP-R-II- Layer and Ganglion Cell-Inner Plexiform Layer DAMARA
09.06 - 09.15 Thickness as Diagnostic Factors in Primary Angle Univ. Indonesia
GLA-05 ANDALIA
Closure and Primary Angle Closure Glaucoma
The Difference of Axial Length, Anterior
Chamber Depth, Lens Thickness and Central
FP-R-II- Corneal Thickness between the Eye with History YURIKE TIURMA Univ.
09.15 - 09.24
GLA-06 of Glaucoma Attack and the Contralateral Eye in PARSAULIN Diponegoro
Primary Angle Closure Glaucoma
Effect of Intravitreal Bevacizumab in Conjunction
FP-R-II- with Glaucoma Drainage Device in Treatment of NINA ASRINI
GLA-07 Neovascular Glaucoma NOOR
Perbandingan Ocular Pulse Amplitude (OPA)

FP-R-II- Penderita Glaukoma Primer dengan dan Tanpa
09.33 - 09.42 VERNA KUSUMA Univ. Padjajaran
GLA-08 Diabetes Melitus
FP-O-II- Serum Antibody Responses of Glaucoma Group FIFIN LUTHFIA Univ.

09.42 - 09.51 Compare to Non-Glaucoma Group
GLA-09 RAHMI Diponegoro
The Correlation of Body-Mass Index and Waist
Circumference towards Intraocular Presssure of Univ.
FP-O-II- MUHAMMAD
09.51 - 10.00 the Academic Community in Medical Faculty of Tanjungpura
GLA-10 ASRORUDDIN
Tanjungpura University Pontianak
FREE PAPER
Padang Room
08.30 - 10.00 Chair: Made Susiyanti, MD
FP-R-II- Diagnostic Study Using PCR to Detect Hepatitis C

10.30 - 10.39 Virus in Tears Fluid of Hemodialysis Patients NOVIA NASIR Univ. Sriwijaya
IIM-07
FP-R-II- Ocular Surface Disease Profile in Saiful Anwar MUTIA HAPSARI

10.39 - 10.48 General Hospital Malang, East Java, Indonesia Univ. Brawijaya
IIM-08 R.
Comparison Between Luteinizing Hormone

FP-R-II- and Degree of Dry Eye Syndrome in Polycystic TESHA
10.48 - 10.57 Ovary Syndrome Patient in Mohammad Hoesin Univ. Sriwijaya
IIM-09 RATNAFURY
Hospital Palembang.
Karakteristik Penderita Keratitis Akantamuba di

FP-R-II- Pusat Mata Nasional RS Mata Cicendo Bandung
10.57 - 11.06 IVA YULIA Univ. Padjajaran
IIM-10 Periode Januari 2012 - Desember 2014
FP-R-II- Karakteristik Klinis Pasien Endoftalmitis Pasca NELLY CHRISTINA

11.06 - 11.15 Bedah Katarak Univ. Padjajaran
IIM-11 SIAHAAN
Bacterial Mapping and Predisposing Factors of

FP-R-II- Corneal Ulcers in Dr. Soetomo Hospital Surabaya JOKO TRIYONO
IIM-12 and Undaan Eye Hospital Surabaya
11.30 - 14.00 BREAK

FREE PAPER
Padang Room
14.00 - 15.30 Chair: Astrianda S., MD

Effect of Methanol Extract of Moringa oleifera
FP-R-I- to Decrease in Malondialdehyde (MDA) Levels
14.00 - 14.09 in Trabecular Meshwork Primary Congenital IRMA SURYANI Univ. Brawijaya
GLA-11
Glaucoma Cell Line
FP-R-II- Distribution of Glaucoma at Ophthalmology
14.09 - 14.18 Department RSMH Palembang in 2015 TIARA MAYA SARI Univ. Sriwijaya
GLA-12
FP-R-II- Clinical Features of Primary Open angle

14.18 - 14.27 Glaucoma in Dr.M.Djamil Hospital VERA Univ. Andalas
GLA-13
FP-R-II- Clinical Characteristics of Juvenile Glaucoma in ELFI RISALMA

14.27 - 14.36 Dr. M. Djamil General Hospital Padang Univ. Andalas
GLA-14 PUTERI
Acute Primary Angle-Closure: Profile and

FP-R-II- Characteristic Patient in Saiful Anwar Hospital
14.36 - 14.45 AHRIANI ACHMAD Univ. Brawijaya
GLA-15 Malang East Java
FP-R-II- The Difference on Axial Length between Primary AMANDA NUR Univ. Gadjah
14.45 - 14.54 Open Angle Glaucoma and Normal Population
GLA-16 SHINTA PERTIWI Mada
Severity of Diabetic Retinopathy in Patients

FP-R-II- Treated with Laser Photocoagulation in Dr. SABRINA Univ. Gadjah
14.54 - 15.03
RET-11 Sardjito General Hospital HANIFAH Mada
Prevalence and Risk factor of Diabetic

FP-R-II- Retinopathy among Senile Patients with Type 2 Univ.
15.03 - 15.12 PRABOWO
RET-12 Diabetes in Halmahera Primary Health Care Diponegoro
FP-R-II- Vitreous Hemorrhage in Dr.Sardjito General IRENE TITIN Univ. Gadjah

15.12 - 15.21 Hospital
RET-13 DARAJATI Mada

FREE PAPER
Medan Room
08.30 - 10.00 Chair: Referano Agustiawan, MD

Comparison of 532 nm Panretinal Laser
Photocoagulation of 20 ms Duration Single
Session and 100 ms Duration Single Session
FP-R-I- and Multiple Session toward Central Macular
08.30 - 08.39 FARESA HILDA Univ. Indonesia
RET-01 Thickness (in Severe Non-Proliferative Diabetic
Retinopathy and Non-High Risk Proliferative
Diabetic Retinopathy)
FP-R-I- Electroretinography Result of Pycnogenol Effect
08.39 - 08.48 on Non Proliferative Diabetic Retinopathy DIAN FARIKHA Univ. Indonesia
RET-02
Prevalence and Unmodified Risk Factor of KASIHANA
FP-R-II- Diabetic Retinopathy Among Elder Patients with Univ.
08.48 - 08.57 HISMANITA
RET-03 Type 2 Diabetes in Primary Health Care Diponegoro
SOPHA
Evaluation on the Management of Open Globe
Injury Through an Analysis of the Match between
FP-R-II- Final and Predictive Visual Acuity Based on Univ.
08.57 - 09.06 A. AMNA RAHMI
RET-04 Ocular Trauma Score (OTS) at Dr. Wahidin Hasanuddin
Sudirohusodo Hospital in Makassar, 2014
Correlation of Apolipoprotein Level with
FP-R-II- Diabetic Retinopathy Grading and Diabetic Univ.
09.06 - 09.15 RIRIN NISLAWATI
RET-05 Macular Edema Hasanuddin
The Comparison of Handheld Portable Retinal
FP-R-II- Camera with Non-portable Fundus Photography HENDRA KUSUMA Univ.
09.15 - 09.24
RET-06 for Grading Diabetics Retinopathy Hasanuddin
Evaluation of Primary Pars Plana Vitrectomy and
FP-R-II- Scleral Buckling Surgery in Rhegmatogenous
09.24 - 09.33 Retinal Detachment at Cipto Mangunkusumo ANNA NUR UTAMI Univ. Indonesia
RET-07
Hospital: 2013-2014
The Clinical Feature and Treatment of
FP-R-II- Neovascular Age Macular Degeneration at Cipto ASTI
RET-08 Mangunkusumo Hospital in 2014 AYUDIANINGRUM
Retinal Reattachment and Visual Acuity
after Primary Vitrectomy, Scleral Buckle, and
FP-R-II- Pneumatic Retinopexy in Rhegmatogenous HELDA
RET-09 Retinal Detachment’s Patients in National Eye PUSPITASARI
Centre Cicendo Eye Hospital
Coagulation Parameters and Thrombocyte MUHAMMAD
FP-R-II- Index in Patient with Proliferative Diabetic Univ.
09.51 - 10.00 MU'AMAR
RET-10 Retinopathy Diponegoro
HABIBIE

FREE PAPER
Medan Room
10.30 - 11.30 Chair: Anna P. Bani, MD
Inflammation after Congenital Cataract Surgery
FP-R-I- with or without Intraoperative Intracameral MUHAMMAD Univ.
10.30 - 10.39
POS-01 Triamcinolone Injection: A Comparative Study MISBAH Hasanuddin
Comparison of Monocular Recession-Resection

FP-R-II- to Bilateral Recession for Intermitent Exotropia MAULIA FITRA
POS-02 in Cipto Mangunkusumo Hospital 2007-2014 PURNAMA
Comparison of Visual Acuity Result Using LEA

FP-R-II- Symbols, HOTV, E CHART and LANDOLT C in
10.48 - 10.57 SUKOTO Univ. Airlangga
POS-03 Children Ages 4-6 Years
The Prediction of Visual Acuity After Open Globe

FP-R-II- Injury in Children Using Pediatric Ocular Trauma ALBAAZA NUADY Univ. Gadjah
10.57 - 11.06
POS-04 Score (POTS) Mada
FP-R-II- Pediatric Cataract Surgery: Comparison between AQUIRINA

11.06 - 11.15 Ages at Surgery Univ. Airlangga
POS-05 CAESARI PUTRI
Ocular Manifestation in Seropositive Toxoplasma

FP-R-II- and or Rubella Age 0-36 months in Dr. Soetomo MAITRI ANINDITA Univ. Airlangga
11.15 - 11.24
POS-06 General Hospital
11.30 - 14.00 BREAK

FREE PAPER
Medan Room
14.00 - 15.30 Chair: Syska Widyawati, MD

The Relationship between Anti Double
FP-R-II- Stranded-DNA (Anti ds-DNA) and Retinal
14.00 - 14.09 Vasculitis in Patients Systemic Lupus CHAIR JAUHARI Univ. Sriwijaya
IIM-15
Erythematosus
The Comparison of the Profiles of Conjunctival
FP-R-II- Bacteria between the Vaginal Delivery and the Univ.
14.09 - 14.18 AMINAH
IIM-16 Cesarean Section Section Newborns Hasanuddin
FP-R-II- Risk Factors of Dry Eye in Sritex's Worker Univ.

14.18 - 14.27 Sukoharjo RIANI NILAM
IIM-17 Diponegoro
Prevalence of Bacterial Corneal Ulcer at dr.
FP-R-II- Kariadi Hospital January 2011 to December MONIKA YOKE Univ.
14.27 - 14.36
IIM-18 2015 LUSIANI Diponegoro
Effect of Autologous Serum on Corneal Ulcer

FP-R-II- Patient Underwent Fresh Amnion Membrane KRISTIAN Univ. Gadjah
14.36 - 14.45
IIM-19 Transplant GOENAWAN Mada
FP-R-II- The Change in Bacterial Corneal Ulcer Size after

14.45 - 14.54 Definitive Therapy MASMERYANI Univ. Airlangga
IIM-20
FP-R-II- Clinical Profile of Anterior Uveitis at Saiful HARIR

14.54 - 15.03 Anwar Hospital Malang Univ. Brawijaya
IIM-21 RAHMANIAH
FP-R-II- Clinical Profile of Vernal Keratoconjunctivitis YASMIN SANI

15.03 - 15.12 (VKC) patients at Saiful Anwar Hospital Malang Univ. Brawijaya
IIM-22 APRILLIANTINA
The Correlation between Corneal Sensitivity
FP-R-II- and the Degree of Dry Eye in Type 2 Diabetes ANDI HASYIM Univ.
15.12 - 15.21
IIM-23 Mellitus Patients ASY'ARI Hasanuddin

FREE PAPER
Denpasar Room
08.30 - 10.00 Chair: Syntia Nusanti, MD
Effect of Mitomycin C on Human Tenon

FP-R-I-
08.30 - 08.39 Fibroblast Proliferation in Contracted Socket MIRZA METITA Univ. Brawijaya
ROO-01
Tissue
FP-R-II- Outcome of Involutional Entropion Surgical

08.39 - 08.48 SHERLY MUCHLIS Univ. Andalas
ROO-02 Reconstruction in M Djamil Hospital Padang
FP-R-II- Hormonal Contraception and Risk of REINNE NATALI Univ. Gadjah

08.48 - 08.57
ROO-03 Orbitocranial Meningioma Among Women CHRISTINE Mada
FP-R-II- Meningioma Characteristic In National Eye ANTONIUS DWI
ROO-04 Center Cicendo Eye Hospital Bandung 2011-2015 JUNIARTO
FP-R-II- Characteristics Of Nasolacrimal Duct Obstruction

09.06 - 09.15 RANI HIMAYANI Univ. Padjajaran
ROO-05 Patients at Cicendo Eye Hospital in 2014
Outcome of Eyelid Defect Management Post-

FP-R-II- wide-excision Due to Malignant Eyelid Tumor in ROMMEL
ROO-06 Plastic and Reconstruction Division RSCM Kirana: ALEDDIN DR.
January 2012-December 2015
Karakteristik dan Hasil Penatalakasanaan

FP-O-II- YULIANA Jakarta Eye
09.24 - 09.33 Tindakan Bedah Epiblefaron di Jakarta Eye
ROO-07 HARTONO Center
Center (Periode 2011-2015)
FP-R-II- The Pattern of Eyelid Tumors Cases at Dr. Sardjito EUNIKE AMELIA Univ. Gadjah
09.33 - 09.42
ROO-08 Hospital, Yogyakarta LAHAGU Mada
Karakteristik Pasien Ocular Surface Squamous SANDRANIN-

FP-R-II-
09.42 - 09.51 Neoplasia di Pusat Mata Nasional RS Mata GRUM TRI PU- Univ. Padjajaran
ROO-09
Cicendo Periode Januari 2012 – Juni 2014 TRANTI
FP-R-II-
09.51 - 10.00 Carcinoma Metastatic to the Orbit ROZY ONETA Univ. Andalas
ROO-10
FREE PAPER
Denpasar Room
10.30 - 11.30 Chair: Neni Anggraini, MD

Characteristics and Management of Anterior
FP-O-II-
10.30 - 10.39 Segment Dysgenesis in Pediatric Ophthalmology IRAWATI IRFANI Univ. Padjajaran
POS-07
Cicendo Eye Hospital Bandung, Indonesia.
FP-R-II- Characteristics Strabismus Patient in Sanglah HARUMI PURWA

10.39 - 10.48 Univ. Udayana
POS-08 General Hospital Denpasar PRAHESTHY
FP-R-II- DYAH TJINTYA

10.48 - 10.57 Evaluation of Refractive Amblyopia Therapy Univ. Indonesia
POS-09 SARIKA
FP-R-II- Esotropia Profile in Saiful Anwar General Hospital NOVI FIRMAN

10.57 - 11.06 Univ. Brawijaya
POS-10 Malang January 2010 – December 2014 SYAH
FP-R-II- Profil Bilateral Retinoblastoma in Dr. M. Djamil FICKY FERLIANDO

11.06 - 11.15 Univ. Andalas
POS-11 Hospital Padang between 2003 - 2015 IRWAN
FP-R-II- Interventional Management of Retinopathy of

11.15 - 11.24 KUKUH PRASETYO Univ. Padjajaran
POS-12 Prematurity (ROP); A Case Series
11.30 - 14.00 BREAK

FREE PAPER
Denpasar Room
14.00 - 15.30 Chair: Amir Shidik, MD

TIME CODE TOPIC SPEAKER INSTITUTION
The Pattern of Visual Improvement after PRK and

14.00 - 14.09 Phakic IOL Implantation on High Myopic Patients Widyandana
KBR-13 Mada
at YAP Eye Hospital, Yogyakarta, Indonesia
Comparison of Serum Nitric Oxide Concentration

FP-R-II- MAHAR JAYA
14.09 - 14.18 in Diabetes Mellitus Patient with and without Univ. Sriwijaya
KBR-14 PURBA
Cataract
Comparison of Visual Outcomes Between
FP-R-II- MSICS and Phacoemulsification Performed by OKTARIA WIDYA Univ. Gadjah
14.18 - 14.27
KBR-15 Ophthalmology Residents during Community PUTRI Mada
Health Service
FP-R-II- Intraocular Pressure and Endothelial Cell Counts SYAM Univ. Gadjah
14.27 - 14.36
KBR-16 after Iris Fixated Phakic IOL in High Myopia SUHARYONO Mada
FP-R-II- Timing of Suture Removal in Surgically Induced Univ.
14.36 - 14.45 LARAS WIDAYANTI
KBR-17 Astigmatism Diponegoro
Factors Related to The Incident of Pterygium

FP-R-II-
14.45 - 14.54 on PT. Pal Employees Surabaya with Radiation FHERYANTO Univ. Airlangga
KBR-18
Exposure
FP-R-II- Pencapaian Target Refraksi Operasi Katarak di ANGGITYA

KBR-19 Pusat Mata Nasional Rumah Sakit Mata Cicendo RULLANTIKA
FP-R-II- Karakteristik Pasien Ectopia Lentis di Pusat Mata

15.03 - 15.12 GRACE PURNOMO Univ. Padjajaran
KBR-20 Nasional RS Mata Cicendo
Clinical Experience with the First 40 Cases of

FP-O-II- LISTYA DYAH Tritya Eye Clinic,
15.12 - 15.21 Phacoemulsification Cataract Surgery: Safety of
KBR-21 RIHARDINI Surabaya
the Learning Curve

FREE PAPER
Yogyakarta Room
08.30 - 10.00 Chair: Tri Rahayu, MD
Pengaruh Pemberian Citicolin Terhadap Fenotip

FP-R-I- Univ.
08.30 - 08.39 Penderita Leber Hereditary Optic Neuropathy AGUS SETYAWAN
NO-01 Diponegoro
(LHON) Di Kecamatan Wanadadi Banjarnegara
The Effect of Zinc Distribution for New TB

FP-R-I-
08.39 - 08.48 Patients Who Have Ethambutol Therapy toward NENI DANIATI Univ. Airlangga
NO-02
Visual Field Defect
Visual Finding in Patient with Pituitary

08.48 - 08.57 Macroadenoma Underwent Transphenoid RENY SETYOWATI
NO-03 Mada
Surgery at Dr Sardjito General Hospital
Clinical Characteristics and Therapeutic Outcome
FP-R-II-
08.57 - 09.06 of Carotid-Cavernous Fistula at Ophthalmology RESSA YUNETA Univ. Indonesia
NO-04
Department of Cipto Mangunkusumo Hospital
The Effect of Ethambutol Duration Treatment

FP-R-II- MARTHASARI
09.06 - 09.15 to Color Perception Based on Ishihara VS Univ. Airlangga
NO-05 ROSALINA
Fransworth D15 Test
FP-R-II- Etiologies Of Nonglaucomatous Optic Atrophy at SEKAR HARRY Univ. Gadjah
09.15 - 09.24
NO-06 Dr.Yap Eye Hospital in Yogyakarta, Indonesia SATYAWATI Mada
FP-R-II- Profile of Cortical Visual Impairment at Dr. M. SUCI PERMATA
09.24 - 09.33 Univ. Andalas
NO-07 Djamil Hospital Padang West Sumatera SARI
The Alteration of Visual Field Defect in Patient

FP-R-II-
09.33 - 09.42 with Stroke Ischemic After Three Months FRIDA LESTARI Univ. Sriwijaya
NO-08
Therapy
Correlation between Ethambutol Theraphy

FP-R-II- MIRA RETNA
09.42 - 09.51 Duration and Zinc Serum Level with Colour Univ. Sriwijaya
NO-09 TETIANA
Vision Deficiency in Tuberculosis Patients
FP-R-II- Incidence, Etiology and Risk Factor of Third Univ. Gadjah

09.51 - 10.00 IKA NURAITA
NO-10 Nerve Palsy at Dr. Yap Eye Hospital, Yogyakarta Mada

FREE PAPER
Yogyakarta Room
10.30 - 11.30 Chair: Nina Ratnaningsih, MD
Differences between Health Promotion with The

Counseling Pocketbook Prevention of Childhood
FP-R-I- Univ.
10.30 - 10.39 Blindness by The Cadres and The Use of A HADIJAH
OFK-01 Diponegoro
Pocketbook Independently of The Mother’s Level
of Knowledge
The Prevalence of Uncorrected Refractive Errors

FP-R-II- INDRA TRI Univ. Gadjah
10.39 - 10.48 in Urban, Exurban, Suburban and Rural Primary
OFK-02 MAHAYANA Mada
School Children
FP-O-II- Situational Analysis of Eye Care Services in South Univ.
10.48 - 10.57 AHMAD ASHRAF
OFK-03 Sulawesi Hasannudin
Pengaruh Pelatihan Buku Saku Pencegahan

FP-R-I- LEIDINA Univ.
10.57 - 11.06 Kebutaan pada Anak terhadap Tingkat
OFK-04 RACHMADIAN Diponegoro
Pengetahuan Kader Kesehatan
The Use of Educative Flipchart as A Simple

FP-R-II- DHIMAS HARI Univ. Gadjah
11.06 - 11.15 Method for Improving Pediatric Ophthalmology
OFK-05 SAKTI Mada
Knowledge in The Community.
Questionnaire-Based Survey on Public’s

FP-R-II- Univ.
11.15 - 11.24 Knowledge and Attitudes towards Glaucoma in IFFAH ZULFA
OFK-06 Diponegoro
Semarang, Indonesia
11.30 - 14.00 BREAK

FREE PAPER
Yogyakarta Room
14,00 - 15.30 Chair: Anggun R, MD
Gambaran Klinis Penderita Retinopati Diabetika

FP-R-II-
14.00 - 14.09 yang Terjaring Program Skrining Retinopati MEGA PRAYOGA Univ. Padjajaran
RET-14
Diabetika di Kota Bandung
Gambaran Tajam Penglihatan dan Keadaan

FP-R-II- Anatomis Retina Setelah Tiga Bulan Pemasangan
14.09 - 14.18 SRI HARDWIYANI Univ. Padjajaran
RET-15 Bakel Sklera pada Penderita Ablasio Retina
Regmatogen
FP-R-II- The Toxic Effect of Ethambutol Treatment to

14.18 - 14.27 IRNAWATI NUNA Univ. Sriwijaya
RET-16 Retinal Nerve Fiber Layer Thickness Changes
The levels of TGFΒ1 Serum and Vitreous in The
FP-R-II- Patients of Proliferative Diabetic Retinopathy Univ.
14.27 - 14.36 ROSMIATY Z.A.
RET-17 With or Without Panretinal Photocoagulation Hasanuddin
Laser.
Comparison of Increasing Intraocular Pressure
FP-R-II-
14.36 - 14.45 between Pars Plana Vitrectomy With C3F8 and TRI MULIASIH Univ. Airlangga
RET-18
Silicon Oil Tamponade
The Effect of Visual Function to Quality of Life
FP-R-II- in Diabetic Retinopathy Patient According HELEN
RET-19 RETDQOL (Retinopathy Dependent Quality Of KUSUMANINGSIH
Life) Questioneer
FP-R-II- Corelations Retinal Nerve Fiber Layer Thickness
14.54 - 15.03 KHOLID MAHHARI Univ. Airlangga
RET-20 with Long Axis Axial Ball Eyes and Myopia Degree

FREE PAPER
Bandung Room
08.30 - 10.00 Chair: Yeni Dwi Lestari, MD

Difference of Accomodative Asthenopia
FP-R-I- Univ.
08.30 - 08.39 Symptoms in Sewing Operators Who Were Given DENTI PUSPASARI
REF-01 Diponegoro
and Not Given Addition Lens.
Comparison of Corneal Thickness and
Endothelial Cell Morphology in Myopic Patients
FP-R-I- CUT PUTRI
08.39 - 08.48 with Daily Wear Hydrogel (Nelfilcon A) and Univ. Indonesia
REF-02 SAMIRA
Silicon Hydrogel (Lotrafilcon B) Contact Lenses: A
Pilot Study
Differences of Refractive Errors in Patient after
Cataract Surgery Based on Biometric Ocular
FP-R-II- Univ.
08.48 - 08.57 Measurements : Applanation Ultrasound, YUDA SAPUTRA
REF-03 Diponegoro
Immersion Ultrasound, and Partial Coherence
Interferometry
Comparison of The Visual Outcome Between
FP-R-II- Spectacles and Rigid Gas Permeable Contact Lens BANU AJI Univ. Gadjah
08.57 - 09.06
REF-04 in Myopia & Compound Myopic Astigmatism DIBYASAKTI Mada
Patient
The Prediction Accuracy of Cataract Surgery
FP-R-II- Refractive Results Using SRK/T and Haigis DEWI MEGASARI
REF-05 Formula (with and without Personalized JUHRIE
Constants)
FP-R-II- The Role Of ZNF644 Gene Mutation in the
09.15 - 09.24 R.M. IRSAN Univ. Sriwijaya
REF-06 Degree of Myopia
Biometry Predictability of Refractive Outcome THERESIA
FP-R-II-
09.24 - 09.33 Evaluation in Cataract Surgery in Klinik Kasih YINSKI PISTARI Univ. Indonesia
REF-07
Sayang (January-December 2014) GONDOSARI
Correlation Between Axial Length with Central
FP-R-II-
09.33 - 09.42 Corneal Thickness and Degrees of Myopia (Cross RISKA ANDRIANI Univ. Airlangga
REF-08
Sectional Study)
Comparison Quality of Life in Low Vision Patients
MARHAENI
FP-R-II- Before and After Rehabilitation in Low Vision
09.42 - 09.51 PUSPOSETYANIN- Univ. Padjajaran
REF-09 Clinic National Eye Center Cicendo Eye Hospital
GRUM
Bandung
FP-R-II- Correlation between Ocular Biometric RARAS SUKS- Univ.
09.51 - 10.00
REF-10 Components with Correction of Myopia MAPRASASTA Diponegoro

FREE PAPER
Bandung Room
10.30 -11.00 Chair: Yeni Dwi Lestari, MD
FP-R-II- Corneal Ulcer Profile in Saiful Anwar Hospital NUKE ERLIANA

IIM-13 Malang 2013-2014 MAYASARI
The Outcome of Vitrectomy in Acute

FP-O-II- Postoperative Endophthalmitis in Cipto SINDY BORU
IIM-14 Mangunkusumo Hospital: What an Eight Years SEMBIRING
Experience (2007-2015) had Taught Us?
Kesesuaian Hasil Penapisan Status Refraksi

FP-R-II- NINA
10.48 - 10.57 Antara Dokter Umum dan Dokter Spesialis Mata Univ. Padjajaran
OFK-07 MANGGIASIH
dengan Menggunakan Uji Brückner
Conjunctivitis Prevalence and Risk Factors

FP-O-II- MARIE YUNI
10.57 - 11.06 Among Boarding School Students in Kediri, West Univ. Mataram
OFK-08 ANDARI
Lombok
Effect Of Anti-Vascular Endothelial Growth Factor
FP-R-I- OBDES MAHARNI
11.06 - 11.15 (Anti - VEGF) Injection on Translucency Grade Univ. Andalas
KBR-11 EMPUTRI
and Length of Primary Pterygium
Tear fluid MMP-8 Concentration Predicts

FP-R-II- AGUNG Univ. Gadjah
11.15 - 11.24 Inflammation after Photorefractive Keratectomy
KBR-12 NUGROHO Mada
(PRK)
11.30 - 14.00 BREAK

FREE PAPER
Bandung Room
14.00 - 15.30 Chair: Mayasari Wahyu, MD
Risk Factors of Infants that Undergone Screening

FP-R-II-
14.00 - 14.09 of Retinopathy o f Prematurity in Dr. M. Djamil RIRI HANDAYANI Univ. Andalas
POS-13
Hospital Padang
Correlation and Comparison Contrast Sensitivity

FP-R-II- Test Using Lea Symbols Low Contrast and Mars SISWI HAPSARI
POS-14 Number Contrast Sensitiviy Chart in A Class Al- WAHYUNINGTYAS
Iman Kindergarten Surabaya
Comparison of Intraocular Pressure after

FP-R-II- JUNELLY VIRMALA Univ.
14.18 - 14.27 Congenital Cataract Surgery With or Without
POS-15 JAURY Hasanuddin
Intraocular Lens Implantation
FP-R-II- Congenital Cataract due to Congenital Rubella

14.27 - 14.36 DIAN ARIANI Univ. Sriwijaya
POS-16 Syndrome
The Correlation between Near Point

FP-R-II- SULYANTI Univ.
14.36 - 14.45 Convergence and The Convergence Insufficiency
POS-17 RACHMAN B. Hasanuddin
in Computer Users
E POSTER
NO CODE CATARACT TOPIC PRODUCER INSTITUTION
Ocular Trauma With Good Visual Acuity After GREDY EDHITA Universitas
1 POS-KBR-001
Surgery ARYANI Airlangga
Iris Suture Fixated Posterior Chamber Intraocular YENNI Universitas
2 POS-KBR-002
Lens Without Adequate Capsular Support POERNAMA SARI Udayana
Cataract Extraction In Patient With Aniridia AA. RATIH Universitas
3 POS-KBR-003
Syndrome KEMALA DEWI Udayana
Direct Placement Of Foldable Iol Into The Capsular Eye Community
4 POS-KBR-004 JAMALUDDIN
Bag Hospital Java
Necrotizing Or Corneal Calcification In Alkaline
Universitas
5 POS-KBR-005 Chemical Injury (Slake Lime, Ca(Oh)2) , Could It Be GUNTUR FAZWAT
Andalas
Disappear ?
Secondary Iris Cyst With Complications Following
KING HANS Universitas
6 POS-KBR-006 Cataract Surgery: How To Commence The
KURNIA Indonesia
Treatment?
Corneal Cryotheraphy For Descemetocele : A Way I.G. AYU MADE Universitas
7 POS-KBR-007
When Corneal Donor Unavailable JULIARI Udayana
Phacoemulsification In Mature Cataract With DYAH PURWITA Universitas
8 POS-KBR-008
Stromal Corneal Dystrophy TRIANGGADEWI Airlangga
Management Of Intraocular Foreign Body (Iofb) In Universitas
9 POS-KBR-009 EKO WIDAYANTO
Anterior Chamber Airlangga
Refractive Surprise Following Uncomplicated Universitas
10 POS-KBR-010 EUNIKE
Refractive Lens Extraction On High Myopic Eyes Hasanuddin
The Efficacy Of Corneal Incision Approach In Post Universitas
11 POS-KBR-011 TRI SUBEKTI
Keratoplasty Complicated Cataract Airlangga
Urrets-Zavalia Syndrome (Fixed And Dilated Pupil) MUH. FADLY Universitas
12 POS-KBR-012
Following Uneventful Cataract Surgery HIDAYAT Hasanuddin
Phacoemulsification Approach To Posterior Polar Universitas
13 POS-KBR-013 FEBRINA ART
Cataract Sriwijaya
Corneal Collagen Cross-Linking (Cxl) Followed By R.A. KANIRARAS
Universitas
14 POS-KBR-014 Photorefractive Keratectomy (Prk) In Keratoconus LINTANG
Gadjah Mada
Patients : A Case Series Study PRAMESWARI
RINI Rsu Dr. Wahidin
15 POS-KBR-015 Wood Penetrating Ocular Injury KUSUMAWAR Sudiro Husodo
DHANY Mojokerto
NO CODE INFECTION IMMUNOLOGY TOPIC PRODUCER INSTITUTION
Atypical Presentation Of Acanthamoeba Keratitis

Universitas
1 POS-IIM-001 : A Challenge In Diagnosis And Treatment :A Case FITRI MUSLIM
Indonesia
Report
IDA AYU ARY Universitas
2 POS-IIM-002 Episcleritis As A Sign Of Carotid Cavernous Fistula
PRAMITA Udayana
Universitas
3 POS-IIM-003 Endophthalmitis After Pars Plana Vitrectomy SARI MARINA
Udayana
Challanges In Managing Steven Johnson Syndrome
JOSHUA P.F. Universitas
4 POS-IIM-004 With Chronic Ocular And Extraocular Sequele : A
LUMBANTOBING Indonesia
Case Report
NI PUTU
Permanent Visual Loss In Severe Dengue Infection Universitas
5 POS-IIM-005 NGURAH SRI
: A Case Report Udayana
YULIASTINI SJ.
A Successful Management Of Sympathetic VINCENT WANG Universitas
6 POS-IIM-006
Ophtalmia: A Case Report TAHIJA Indonesia
Treatment Of Acute Endophthalmitis After
Universitas
7 POS-IIM-007 Cataract Surgery At Undaan Eye Hospital Surabaya RITA TJANDRA
Airlangga
On February 2016
YENITA
Anterior Uveitis Exacerbate During Pregnancy : Is Universitas
8 POS-IIM-008 KHATANIA
There Any Correlation? Udayana
ARDJAJA
HIMAWATI Universitas
9 POS-IIM-009 Ocular Behcet Disease
NIRMALASARI Airlangga
Universitas
10 POS-IIM-010 Endophthalmitis On One Seeing Eye PARAMITA PUTRI
Airlangga
Rsup Prof. Dr.
Chorioretinitis Et Causa Toxoplasma Gondii With ANNE MERRY
11 POS-IIM-011 R. D. Kandou
Pathology Myopia SEVANI UMBOH
Manado
Severe Inflammation After Cataract Surgery In I.G. AYU MADE
12 POS-IIM-012 Rsud Singaraja
Diabetic Patient JULIARI
Shield Ulcer As Complication Of Vernal Universitas
13 POS-IIM-013 TRI LESTARI
Keratoconjungtivitis Samratulangi
I.G. AYU MADE Universitas
14 POS-IIM-014 Scleritis In Patient With Antiphospholipid Syndrome
JULIARI Udayana
Scleritis After Vitreoretinal Surgery : Is It Any I.G. AYU MADE Universitas
15 POS-IIM-015
Correlation ? JULIARI Udayana
I.G. AYU MADE Universitas
16 POS-IIM-016 Corneal Cryotheraphy For "Hot Eye" Corneal Ulcer
JULIARI Udayana
Universitas
17 POS-IIM-017 Panuveitis In Cutaneuos T-Cell Lymphoma NAFITRI AULIA
Brawijaya
Post Traumatic Endophthalmitis And Intraocular
TESTIANA GALUH Universitas
18 POS-IIM-018 Foreign Body With Poor Posterior Segment
RESCAHYANTI Airlangga
Visualization
MARHAENI
Investigation Of Retinal Vasculitis With Vitreous Universitas
19 POS-IIM-019 PUSPOSETYA-
Hemorrhage Caused By Behchet’ Disease Padjajaran
NINGRUM
MOHAMAD
Management Of Corneal Perforation With Fascia Universitas
20 POS-IIM-020 ARIEF
Lata Graft Padjajaran
HERDIAWAN
Visual Improvement After Oral Valacyclovir In The

FAIKA Universitas
21 POS-IIM-021 Treatment Of Acute Retinal Necrosis Caused By
NOVADIANAZ Sriwijaya
Herpetic Viruses
Successful Cryotherapy For Corneal Ulcer With Universitas

22 POS-IIM-022 MUTIA
Hypopion After Failed Bridging-Flap Airlangga
GRACIA
Universitas
23 POS-IIM-023 Symphathetic Opthalmia : A Case Report MARGARET
Samratulangi
SIWU
DIAN EKA Universitas
24 POS-IIM-024 Early Manifestation Of Herpes Zoster Ophtalmicus
SAPUTRA Andalas
NO CODE GLAUCOMA TOPIC PRODUCER INSTITUTION
Diagnosis And Management Of Iridocorneal

YULINDA ARTY Universitas
1 POS-GLA-001 Endothelial Syndrome In Combination With
LAKSMITA Indonesia
Primary Angle Closure
Posner-Scholssman Syndrome : Mimic Variety Of AA AYU DIAH Universitas
2 POS-GLA-002
Ocular Disorders CITRADEWI Udayana
Combination Trabeculectomy And Bevacizumab I GUSTI PUTU
Universitas
3 POS-GLA-003 Intravitreal Injection : A Succesful Management EKA SURYAWAN
Udayana
Of Neovascular Glaucoma In Sanglah Hospital WIDNYANA
Paracentesis And Trabeculectomy As Surgical
HENRY SANTOSA Universitas
4 POS-GLA-004 Interventions In Secondary Glaucoma Due To
SUNGKONO Udayana
Traumatic Hyphema
Universitas
5 POS-GLA-005 Juvenile Glaucoma M. FAISAL
Sumatera Utara
Universitas
6 POS-GLA-006 Cyclocryotherapy In End Stage Glaucoma EKA SAFIATI
Sumatera Utara
RANNY Universitas
7 POS-GLA-007 Bilateral Iridocorneal Endothelial Syndrome
LAIDASURI Andalas
The Outcome Of Modified Trabeculectomy In DESTY RIA
8 POS-GLA-008
Glaucoma Secondary To Sturge-Weber Syndrome TIFFANI
Glaucoma In Axenfeld-Rieger Syndrome: A Case RIANI Universitas
9 POS-GLA-009
Report WITJAKSANA Samratulangi
Antiglaucoma Therapy In Patient With Steroid Universitas
10 POS-GLA-010 FANEISHA
Induced Glaucoma Sriwijaya
SUCI PERMATA Universitas
11 POS-GLA-011 A Case Of Lens Luxation In Exfoliative Glaucoma
SARI Andalas
Management Of Secondary Glaucoma Associated NI PUTU DIAN Universitas
12 POS-GLA-012
With Lens Subluxation After Trauma AYU Brawijaya
Management Of Patient With Open Angle
INTAN DWI Universitas
13 POS-GLA-013 Glaucoma With Glaucoma Drainage Devices (Gdd)
RAHAYU Padjajaran
Implant
Glaucoma Secondary To Subluxated Cataract In AMELIA SAFITRI Universitas
14 POS-GLA-014
The Last-Eye RAMADHANI Airlangga
Secondary Glaucoma After Penetrating Universitas
15 POS-GLA-015 MARIA RIANDIKA
Keratoplasty Airlangga
Secondary Angle Closure Glaucoma Due To Rsud Dr. Saiful
16 POS-GLA-016 AYU PUSPITASARI
Iridocorneal Endothelial Syndrome A Case Report Anwar Malang
Outcome Of Trabeculectomy Surgery In Chronic Universitas
17 POS-GLA-017 ROLAN IQBAL
Angle Closure Glaucoma Sriwijaya
RECONSTRUCTION OCULOPLASTY
NO CODE PRODUCER INSTITUTION
TOPIC
Ectropion And Lagophthalmos After Eyelid RATNA
1 POS-ROO-001 Unair
Trauma. How Best To Manage The Eyelid Trauma DOEMILAH
A Rare Case Of Retrobulbar Kimura Disease: ANISSA N. Universitas
2 POS-ROO-002
Challenge In Diagnostic And Management WITJAKSONO Indonesia
Systemic Propanolol Reduces Progressive Capillary RINA Universitas
3 POS-ROO-003
Haemangioma WULANDARI Airlangga
Orbital Primitive Neuroectodermal Tumor: A Case Universitas
4 POS-ROO-004 IKA KARTIKA
Report Gadjah Mada
Frontal Region And Cheek Advancement Flap AMMAR Universitas
5 POS-ROO-005
Reconstruction After Orbital Exenteration FARDHANA Sriwijaya
Satisfactory Result Of Superior And Inferior
Palpebral Defect Reconstruction In Severe Universitas
6 POS-ROO-006 PUTRI A IDHAM
Superior And Inferior Palpebral Defect Due To Indonesia
Trauma
Ocular Dystopia And Lacrimal Pathway
Universitas
7 POS-ROO-007 Obstruction In Delayed Management Of Multiple VEGA CASALITA
Indonesia
Midfacial Fractures
Frontalis Suspension For Bilateral Congenital RATNA SARI Universitas
8 POS-ROO-008
Ptosis : A Case Report DEWI Udayana
Outcome Of Hughes Procedure On Basal Cell KRISTIAN Universitas
9 POS-ROO-009
Carcinoma GOENAWAN Gajah Mada
Buccal Mucosa Graft In Re-Reconstruction NI NYOMAN
Universitas
10 POS-ROO-010 Congenital Eyelid Coloboma With Symblepharon : RINA
Udayana
A Case Report KURNIASARI
Skin Graft Procedure After Debridement Of Facial Universitas
11 POS-ROO-011 SISCA MAYASARI
Ulceration In Diabetic Patient Padjajaran
MADE OKA Universitas
12 POS-ROO-012 Orbital Extrapleural Solitary Fibrous Tumor
HANDAYANI Udayana
Conjunctival Granuloma Clinically Masquerading NI MADE Universitas
13 POS-ROO-013
As Recurrent Squamous Cell Carcinoma DWIPAYANI Udayana
An Unusual Case Of Massive Proptosis Bulbi As A DWI MAYSAROH Universitas
14 POS-ROO-014
Manifestation Of Neuroblastoma ARSA Sumatera Utara
Successful Management Of Orbital
Universitas
15 POS-ROO-015 Rhabdomyosarcoma In Adult With Treatment On RENY SETYOWATI
Gadjah Mada
Multimodality Protocols
Wide Excition Eyelid Tumor Cystic Cell Fibrolipoma SRI ULINA Universitas
16 POS-ROO-016
With Pre-Auricular Graft GINTING Sumatera Utara
Isolated Congenital Bilateral Lacrimal Gland
DEVINA NUR Jakarta Eye
17 POS-ROO-017 Agenesis: Clinical Radiologic Finding And
ANNISA Center
Management
Chomprehensive Management Of Iris Melanoma PRIMA SUGESTY Universitas
18 POS-ROO-018
At Dr. Sardjito General Hospital Yogyakarta NURLAILA Gadjah Mada
The Clinical Outcome Of Cutler-Beard Procedure
ANDREAS SURYA
19 POS-ROO-019 For Upper Eyelid Reconstruction Following Wide Rsup Dr. Sardjito
ANUGRAH
Tumor Excision : A Case Series Study
Long-Term Gold Weight Implant Survival And

NILUH ARCHI SRI Universitas
20 POS-ROO-020 Success In Managing Lagophthalmos Due To Facial
RAMANDANI Indonesia
Paralysis: A Case Report
Local Recurrence Of Malignant Melanoma INDHA DWI Universitas
21 POS-ROO-021
Following Orbital Excenteration KARTIKASARI Brawijaya
Eyelid’s Defect Reconstruction In Various HENDRIATI
22 POS-ROO-022
Predilection Of Basal Cell Carcinoma JEFRIANTO
Case Series Of Two Patients With Optic Pathway MARTIN Universitas
23 POS-ROO-023
Glioma, To Cut Or To Wait HERTANTO Indonesia
RSAL
ARMANTO
24 POS-ROO-024 Eyelid Reconstruction Surgery In Cryptophthalmos Dr. Ramelan/
SIDOHUTOMO
Tritya Eye Clinic
Universitas
25 POS-ROO-025 Cavernosa Hemangioma Mimicking Cyst PRIMERY PUTRI
Andalas
Universitas
26 POS-ROO-026 Suprasellar Meningioma Mimicking Optic Neuritis RANI HIMAYANI
Padjajaran
IDHAM Universitas
27 POS-ROO-027 Ankyloblepharon Filiforme Adnatum MUSTAHID
ARIFIN Airlangga
Glabellar Flap For Medial Canthal Reconstruction MARETA GUSTIA Universitas

28 POS-ROO-028
After Skin Cancer Excision NINGSIH Padjajaran
GEORGE A.N. Universitas
29 POS-ROO-029 Traumatic Globe Subluxation
SITAYA Hasanuddin
Management Of Patient With Lagofthalmus Ec INTAN DWI Universitas
30 POS-ROO-030
Tension Graft RAHAYU Padjajaran
Papiledema Due To Tumor Infratentorial
31 POS-ROO-031 ADE IRAWAN
Meningioma With Nistagmus
IFAN Universitas
32 POS-ROO-032 Rare Case Of Neuroblastoma In Adolescent ROMADHON
LUKMANA Airlangga
Oral Mucosal Graft And Amniotic Membrane NI MADE AYU Universitas
33 POS-ROO-033 Transplantation For Conjunctival Malignant TRISNADEWI
Melanoma SUYASA Udayana
Ductal Adenocarcinoma Of The Nasolacrimal Duct KI AJENG WINDA Universitas
34 POS-ROO-034
: The First Reported Case NINGRUM P. Airlangga
The Characteristic And Clinical Outcome Post Universitas

35 POS-ROO-035 DISTY ANDRYANI
Lateral Orbitotomy Surgery Sriwijaya
NO CODE RETINA TOPIC PRODUCER INSTITUTION
Diagnosis And Management Of Central Retina AYU THEA Universitas

1 POS-RET-001 Vein Occlusion Caused By Carotid-Cavernous PRIMANITA
Fistula MAWAN Udayana
KOMANG DIAN Universitas

2 POS-RET-002 Systemic Therapy Of Lupus Retinopathy
LESTARI Udayana
Universitas
3 POS-RET-003 Diagnosis In Stargardt Disease: A Case Report LISA MAULIDA
Indonesia
Extraction Intraocular Foreign Body With C-Arm Universitas
4 POS-RET-004 ATHI PURNASARI
Guiding Airlangga
ELBETTY Universitas
Management Neuroretinitis In Acquired Syphilis :
5 POS-RET-005 AGUSTINA BR
A Case Report Udayana
SIMANJUNTAK
Spontaneous Reattachment Of Large Retinal Universitas
6 POS-RET-006 RANI HIMAYANI
Break Rhegmatogen Retinal Detachment Padjajaran
Significant Improvement Of Leukemic Retinopathy Universitas
7 POS-RET-007 After Leukapheresis In Chronic Myelogenous RUCHYTA RANTI
Leukemia With Leukostasis Airlangga
Secondary Glaucoma E.C Coats’ Disease & Universitas

8 POS-RET-008 AMELIA RIZAR
Cryotherapy Oculi Sinistra Sumatera Utara
Intra Ocular Foreign Body In The Posterior DANANG Universitas
9 POS-RET-009
Segment Of Eye PRASETYA K. Airlangga
Argon Laser Hyaloidotomy In Premacular DYAH AYU Universitas
10 POS-RET-010
Subhyaloid Hemorrhage Management WINDY AP. Hasanuddin
Can Mild Enhancement Of Low-Density Universitas
11 POS-RET-011 Lipoprotein Cause Central Retinal Vein Occlusion RISKA
In Young Patient ? Hasanuddin
Unilateral Or Asymmetric Retinitis Pigmentosa: ELYAS ADYTYA Universitas

12 POS-RET-012
How To Diagnose? PRADANA Indonesia
Intravitreal Bevacizumab In The Management Of SRI REZEKI Universitas

13 POS-RET-013
Coats Disease HANDAYANI Padjajaran
14 POS-RET-014 Macular Star, A Great Mimicker A Case Report IRMA CHOLIDAH
Retinal Capillary Hemangioma With Secondary ASTRID CHAIRINI Universitas
15 POS-RET-015
Macular Hole CHAIRI Padjajaran
Cytomegalovirus Retinitis In Systemic Lupus IDA AYU PUTRI Universitas

16 POS-RET-016
Erythematosus WIDHIASTUTI Udayana
Spontaneous Resolution Of Rhegmatogenous Universitas
17 POS-RET-017 Retinal Detachment In Clinically Schwartz-Matsuo VALENCHIA
Syndrome : A Case Report. Indonesia
Intravitreal Injection Of Ranibizumab And Universitas

18 POS-RET-018 Trabeculectomy In A Patient With Neovascular NURUL AINI
Glaucoma Due To Advanced Pdr Brawijaya
Vasculitis Mimicking Central Retinal Vein HERU Universitas

19 POS-RET-019
Occlusion Caused By Chronic Myeloid Leukemia SYAHPUTRA Padjajaran
Comparing The Visual Outcome Of Bevacizumab

FEBRIA RESTISSA
20 POS-RET-020 And Ranibizumab In Patients With Central Retinal
VALENTINA
Vein Occlusion
NO CODE PEDIATRIC OPHTHALMOLOGY TOPIC PRODUCER INSTITUTION

A Case Of Congenital Cytomegalovirus Retinitis In AMELYA Universitas
1 POS-PO-001
Infant : How To Manage ? PERMATA SARI Indonesia
ERLI NUR Universitas
2 POS-PO-002 Unilateral Of Intraocular Retinoblastoma
MAGDALENA Sumatera Utara
Bilateral Persistent Fetal Vasculature In Twin CYNTHIA DEWI Universitas
3 POS-PO-003
Balinese Girls M Udayana
Modified Jensen Procedure In Traumatic ASTRID CHAIRINI Universitas
4 POS-PO-004
Abducens Nerve Palsy CHAIRI Padjajaran
IDAYANI Universitas
5 POS-PO-005 Crocodile Tears (Congenital Aberrant Tearing)
PANGGALO Hasanuddin
Transposition Of Lateral Rectus And Superior Universitas
SORAYA
6 POS-PO-006 Rectus Muscle With Medial Rectus Muscle
RACHIMA Diponegoro
Recession On Heavy Eye Syndrome; A Case Report
Universitas
7 POS-PO-007 The Risk Factors Of Strabismus In Children AFRIANI DJIBRAN
Hasanuddin
Laser Photocoagulation As Management Of GRACE Universitas
8 POS-PO-008
Retinoblastoma PURNOMO Padjajaran
NO CODE NEURO-OPHTHALMOLOGY TOPIC PRODUCER INSTITUTION
Isolated Bilateral Retrobulbar Optic Neuritis In BUDIMAN Universitas

1 POS-NO-001 Childhood Challenging In Management And BINTANG
Prognosis, A Case Report PRAKOSO Indonesia
Third Nerve Palsy As First Clinical Manifestation In IKA CITRA Universitas

2 POS-NO-002
Carotid Cavernous Fistula : A Case Report SUSANTI Indonesia
Successful Management Of Bilateral Traumatic NURIADARA Universitas
3 POS-NO-003
Optic Neuropathy In A Young Girl SAMIRA Indonesia
Bilateral Optic Neuritis : A Very Rare Complication MADE PARAMITA Universitas

4 POS-NO-004
In Dengue Hemorrhagic Fever Case WIJAYANTI Udayana
Challenge In Diagnosing And Managing Carotid MARIA Universitas

5 POS-NO-005 Cavernous Fistula In Patient With Intermittent MAGDALENA
PURBA Indonesia
Angle Closure Glaucoma
Universitas
6 POS-NO-006 Bilateral Idiopathic Neuroretinitis RINCE LIYANTI
Andalas
Successful Management Of Toxic Optic RANI INDIRA Universitas
7 POS-NO-007
Neuropathy After Snake Bite : A Case Report SARI Indonesia
Cytomegalovirus Papillitis In Acquired DINDA ARKEN Universitas
8 POS-NO-008
Immunodeficiency Syndrome DEVONA, Indonesia
Cavernosus Sinus Thrombosis Infection With Universitas
9 POS-NO-009 RENI ANGRAINI
Cranial Nerve’s Paralysis Andalas
Bilateral Visual Loss As The First Manifestation Of GANESA Universitas
10 POS-NO-010
Chronic Kidney Disease WARDANA Hasanuddin
The Prognosis Of Binocular Visual Loss In Cortical KHAIRIAH
11 POS-NO-011
Blindness NEVRIANTY
WASISDI Universitas
12 POS-NO-012 Ophthalmophlegic Migrain: A Case Report
GUNAWAN Gadjah Mada
Management Of Atypical Optic Neuritis Caused By Universitas
13 POS-NO-013 MEGA PRAYOGA
Tuberculous Meningitis Padjajaran
Fast Visual Acuity Recovery After Hyperbaric YENITA Universitas
14 POS-NO-014 Oxygen Therapy In Bilateral Visual Loss Due To KHATANIA
ARDJAJA Udayana
Decompression Sickness
Wall-Eyed Bilateral Internuclear Ophthalmoplegia
WANTY Universitas
15 POS-NO-015 In Human Immunodeficiency Virus (Hiv) Infected
BURHANUDDIN Hasanuddin
Patient
Universitas
16 POS-NO-016 Neuroophthalmic Manifestation Of Snake Bite ARYA RADITYA
Padjajaran
Study Of 7 Cases Of Methyl Alcohol Poisoning INDRA TRI Universitas
17 POS-NO-017
Outbreak: The Clinical Courses And Outcomes MAHAYANA Gadjah Mada
Development Of Horner Syndrome In Association ASTRID CHAIRINI Universitas
18 POS-NO-018
With Lung Cancer CHAIRI Padjajaran
Universitas
19 POS-NO-019 Bilateral Retrobulbar Optic Neuritis Caused By Sle WIBAWANINDYA
Airlangga
Embolization Of Carotid Cavernous Fistule In ANDI NUR Siloam Hospital

20 POS-NO-020
Caused By Domestic Violence UMMAH Surabaya
Bitemporal Hemianopsia With Third And Fourth ANI NUR Universitas

21 POS-NO-021
Nerve Paralysis In Pregnancy WALUYO Airlangga
Successful Risk Factors Management Combined

With Corticosteroid Resulting Good Outcome In Universitas
22 POS-NO-022 HISAR DANIEL
Non-Arteritic Ischemic Optic Neuropathy Patients: Indonesia
A Case Report
Management Of Indirect Traumatic Optic Universitas

23 POS-NO-023 FAISAL RADHI
Neuropathy Sriwijaya
Universitas
24 POS-NO-024 Miller Fisher Syndrome : A Case Report GRIMALDI IHSAN
Padjajaran
NO CODE REFRACTION TOPIC PRODUCER INSTITUTION
Management Of Irregular Astigmatism In Young DANANG YOGA Universitas

1 POS-REF-001
Keratoconic Patient WIGUNA Airlangga
Management Of High Unilateral Corneal
DESTY RIA Universitas
2 POS-REF-002 Astigmatism With Rigid Gas Permeable Contact
TIFFANI Andalas
Lenses
Early Diagnosis, Prompt Occlusion Therapy
IRMA ANDRIANI
3 POS-REF-003 Are Keys To Successful Teraphy For Refractive
PASARIBU
Amblyopia
FARIZ Universitas
4 POS-REF-004 Low Vision Management In Aniridia Patient
NUFIARWAN Brawijaya
The Role Of Low Vision Aid In High Myopia Patient Universitas
5 POS-REF-005 PERLITA KAMILIA
With Chorioretinal Atrophy: A Case Report Indonesia
Helping High Myopia With Refractive Lens Universitas
6 POS-REF-006 SISKA
Exchange (Rle) In East Indonesia Udayana
MEIDINA
7 POS-REF-007 Correction Of High Hyperopia In Young Children
RAHMAH
ABSTRACT
CAT S 3.2.1
Effective Efficient Technique and Cost on Complicated Case Cataract Surgery
R. Prabowo, MD
Katarak brunescent, komplikata (post uveitis, post vitrektomi, post trabekulektomi), traumatika, sindrom
pseudoexfoliasi, IFIS, pupil kecil, subluxasi merupakan salah satu tantangan bagi operator katarak. Operasi katarak
pada kasus-kasus tersebut banyak menimbulkan komplikasi yang lebih tinggi jika tidak dipersiapkan dengan baik.
Karena alasan inilah pada kasus-kasus tersebut membutuhkan pembelajaran, persiapan pre-operasi yang hati-
hati, strategi dan follow-up pasien dengan cermat untuk memperoleh hasil operasi yang optimal. Kami melakukan
strategi operasi dengan teknik yang efektif pada tiap kasus dengan kualitas teknik chop yang tepat, dan efisien
pada bahan yang dipakai.
Kata kunci : Katarak brunescent, katarak komplikata, sindrom pseudoexfoliasi, IFIS, pupil kecil, subluxasi, efektif,
efisien, teknik operasi
CAT S 3.2.4
VITREOUS PROLAPS DURING PHACO.
HOW TO HANDLE THE VITREOUS, LENS MASS & IOL IMPLANTATION.
Amir Shidik, MF
Vitreous loss during phacoemulsification surgery is the most common complication in cataract surgery. Even
though visual outcome post phacoemusification with vitreous loss can be quite good. Still for every surgeon having
a posterior capture rupture and vitreous prolaps during phacoemulsification is stressful and energy consuming.
Early recognision and proper management to vitreous prolaps, lens cortex remnant followed by IOL implantation
would influence the surgical outcome. Vitreous prolaps could occurs in almost any step the phacoemulsification
surgery. Recognising vitreous prolaps is also challanging, for it would influence the following surgical step
management. From simple anterior vitrectomy, enlarging the wound to evacuate the remaining lens mass or in
the late stage, dealing with nucleus drop.
The following presentation would share surgeon expiriences on combining teoritical reasoning and reality fact
on the operating table, of how to recognise postrior capture rupture, dealing with vitreous prolaps, cleaning the
remaining lens mass followed by IOL implantation. An open discussion would enrich the presentation for a better
patient management.
CAT S 3.2.5
Management post cataract surgery complication
Syska Widyawati
Cipto Mangunkusumo Hospital as top referral hospital in Indonesia received 268 Patient with post cataract
surgery complication over 2014-2015. Data from registry in outpatient clinic revealed that posterior capsular
opacity is the most frequent problem referred to cataract and refractive surgery clinic. Surgery registration
showed that the most procedure performed due to post cataract surgery complication was secondary IOL
implantation . frequently intra operative cataract surgery complication end up with aphakia and many times
the anterior segment structure also damaged which would increase the challenge of management in order to
improve patient vision. In this topic we will give some tips and pitfall that would improve management during
the cataract surgery so then secondary procedure would be simpler.
COR S 3.3
No-Touch DMEK
Soeharnila, MD
Abstract :
The first clinically successful endothelial keratoplasty was performed in 1998. DSAEK may have become the
gold standard in the management of endothelial disorder in recent years, but the current trend may be shifting
towards DMEK. DMEK relatively safe and feasible procedure that provide instant visual rehabilitation in a majority
of patients., and creates a near perfect restoration of the corneal anatomy to reach its maximal visual potential.
Rejection after DMEK may occur in 1% of eyes, a 10 times lower risk than in DSAEK,and 20 times lower than
in PK. No- touch DMEK technique was developed by Gerrit Melles. DMEK–graft preparation technique started
with loosing peripheral descemet membrane from outside trabecular meshwork 360° with hockey stick. Then
use peripheral ring of TM to completely strip off DM from posterior stroma . After trephine DM , DM forms a
roll with endothelium on the outside. After staining, the DMEK –roll can be sucked into the ‘Melles injector’ and
carefully inject the DMEK-roll into the anterior chamber. I present two cases of ‘no-touched’ DMEK in patient
with Fuchs endothelial dystrophy and corneal decompensation after phakic IOL. Although endothelial cell
count decreased significantly after surgery, the cornea became clear and visual acuity improved.
COR S 3.5
Implementation of Corneal Collagen Cross Linking Before Photorefractive Kera-
tectomy
Suhardjo, MD., Dede N. Herani, MD., Reny Setyowati, MD., Widyandana, MD.
Yogyakarta Eye Study, Ophthalmology Dept., RSUP Dr. Sardjito, Universitas Gadjah Mada, Yogyakarta
Combining Corneal Collagen Cross Linking (CXL) with refractive surgery such as Photorefractive Keratectomy
(PRK) targeting for visual function improvement. This combination might improve corneal stabilization and
reshaping the corneal tissue. The debating issue is the appropriate timing between CXL and PRK on the safety
and efficacy of combined surgery, it should be performed before or after surgery?
The mechanism of CXL is believed that riboflavin as a photosensitizer which saturated cornea is exposed to
ultraviolet irradiation and is excited into an activate state generating some reactive oxygen species, which lead
to the formation of covalent bond between collagen molecules that increase the stiffnes of the cornea. Previous
studies CXL followed by PRK removed the cross-linked stiffer anterior cornea.
This presentation aimed to share recent study, and experiences from Corneal Laboratory Universitas Gadjah
Mada about combining PRK followed by CXL, that hope used as base for further study.
GLAU S 2.1.1
Target Intraocular Pressure
Fitratul Ilahi , MD
Ophthalmology Departmen Faculty of Medicine
Andalas University /Dr. M Djamil Hospital Padang West Sumatera
Intraocular pressure (IOP) is essential in the management of glaucoma patients, and the achievement of
target IOP is the main goal of glaucoma therapy. Generally, monitoring of normal variations and spontaneous
fluctuations of IOP implies better protection for the loss of vision and visual field impairment in glaucoma.
Lowering intraocular pressure is still the only available point to treat glaucoma pasient, the risk of further
damage is related to the degree of ganglion cell loss. Intraocular pressure plays a significant role in pathogenesis
of glaucoma and several studies have shown that the rate of glaucoma progression can stop with intraocular
pressure controlling even in advanced glaucoma and aggressively lowering of IOP decrese glaucoma progession.
Level of intraocular pressure is correlated with severity of the glaucoma patient and visual impairment. Target
pressure is different for each patient and it was influenced due to intra ocular and systemic factors. The value of
target IOP depends on the pretreatment level of intraocular pressure, optic nerve damage, glaucoma state, rate
of glaucoma progression, age and other factors.
Rate of desease progression is very important, the fast progressors should be treat more aggressively than slow
one for preventing visual loss and maintaining or enhancing quality of life for the patient.
GLAU S 2.1.2
UP DATE IN PATHOGENESIS OF PRIMARY OPEN- ANGLE GLAUCOMA
Fifin Luthfia Rahmi, MD
Ophthalmology Department Medical Faculty, Universitas Diponegoro
Glaukoma sudut terbuka primer (GSTaP) merupakan kelainan neurodegeneratif dengan etiopatogenesis
multifaktorial Tekanan intra okuler (TIO) yang tinggi bukan lagi merupakan satu-satunya faktor yang berperan
pada patogenesis glaukoma. Faktor-faktor lain yang terbukti berkaitan dengan glaukoma diantaranya adalah
proses apoposis, peningkatan kadar nitrit oksida, peningkatan kadar glutamat (Casson, 2006) dan keterlibatan
sistem imun (Grus, 2004). Beberapa diantaranya masih belum jelas apakah bertindak sebagai penyebab atau
akibat.
Proses di tingkat seluler bersifat sangat kompleks, terjadi secara simultan dan saling mempengaruhi. Berbagai
faktor yang diduga mendasari patogenesis glaukoma dapat saling berinteraksi yang pada akhirnya dapat
menyebabkan kematian sel ganglion retina. Pada fase awal/ primer kematian akson sel-sel ganglion retina
pada mata glaukomatous dari manusia maupun binatang percobaan disebabkan oleh apoptosis. Sedang pada
tahap lanjut terjadi proses nekrosis yang akan memperberat destruksi jaringan (Agarwal dkk., 2009; Caprioli,
2007).
Penelitian bidang genetika menemukan GLC1A sebagai gen glaukoma yang pertama kali diketahui. Gen tersebut
banyak ditemukan pada keluarga dengan glaukoma juvenile, terdapat di kromosom 1. Mutasi gen tersebut
akan menyebabkan pembentukan protein myocilin yang akan menyebabkan perubahan struktur trabecular
meshwork seperti pada individu yang mendapat terapi dexametason (Skuta dkk., 2010).
Tekanan intra okuler yang tinggi masih disepakati sebagai faktor risiko utama GSTaP. Kegagalan autoregulasi
akibat tekanan intra okuler tinggi memicu penurunan faktor pertumbuhan, peningkatan ekspresi methyl
metallo proteinase (MMP), kerusakan mekanis, peningkatan glutamat, peningkatan produksi nitrit oksida dan
peningkatan TNF-α (Agarwal dkk., 2009). Keadaan ini akan memicu apoptosis. Bila TIO tinggi berlangsung lama
maka akan berlanjut menjadi proses nekrosis jaringan yang bersifat patologis.
Patogenesis GSTaP yang tidak dipengaruhi TIO dimulai oleh adanya disregulasi vaskuler. Disregulasi vaskuler
dapat memicu pembentukan MMP, peningkatan sekresi endotelin, peningkatan TNF-α, peningkatan glutamat
serta peningkatan produksi nitrit oksida. Sebagaimana pada TIO tinggi, keadaan tersebut di atas akan memicu
apoptosis.
Glutamat secara khusus disebutkan sebagai salah satu substrat yang berperan penting pada patogenensis
glaukoma melalui perannya sebagai senyawa yang dapat menimbulkan eksitotoksisitas pada sel. Sel-sel glia yang
terdapat di retina diduga juga berperan dalam patogenesis glaukoma. Mekanisme lain yang diduga berperan
adalah adanya keterlibatan sistem imun.
GLAU S 2.1.3
IS THIS GLAUCOMATOUS OPTIC NEUROPATHY?
Sirisha Senthil, MD
GLAUCOMA is the leading cause of irreversible blindness worldwide. To be able to treat glaucoma appropriately,
diagnosis has to be early and accurate. Despite the recent advances in the structural assessment of optic nerve
head by imaging techniques, clinical evaluation of the optic disc is still considered the cornerstone in the
diagnosis and management of glaucoma. While early diagnosis of glaucoma is essential for early treatment
of the blinding disease, improper diagnosis or overdiagnosis poses a bigger challenge to this chronic disease,
with life long treatment and their side effects. Understanding and differentiating glaucomatous from non-
glaucomatous optic neuropathy is hence of utmost importance.
GLAU S 2.1.5
Basic Trabeculectomy
Maula Rifada, MD
Department of Ophthalmology Faculty of Medicine Universitas Padjadjaran Cicendo Eye Hospital
Incisional surgery in glaucoma is indicated when the intraocular pressure (IOP) of a patient does not achieve
the target pressure on maximum medical therapy and/or laser treatment. The goal of glaucoma surgery is to
lower IOP to the level that could maintain the patient’s vision. Trabeculectomy is still the standard of filtering
surgery to achieve the target pressure in glaucoma patient. The primary goals of glaucoma filtering surgery are
to bypass the conventional pathways and to maintain the integrity of conjunctival bleb. In this presentation, it
will be discussed about step by step of trabeculectomy.
GLAU S 2.1.6
TRABECULECTOMY WITH RELEASABLE SUTURES
Made Agus Kusumadjaja, MD., IGA Ratna Suryaningrum, MD.
Glaucoma Division, Opthalmology Department Faculty of Medicine Udayana University / Sanglah Hospital
Releasable scleral flap sutures have been effectively used with trabeculectomy. It permit control over the
intraocular pressure and anterior chamber depth after trabeculectomy. The advantage of placing releasable
sutures is it need no laser to release, can be removed even if the overlying tissue clarity is poor, or if the patient
has difficulty keeping the eye still.
During the early post operative period, this sutures prevent complications related to shallow or flat anterior
chambers. Shallow chambers lead to the formation of peripheral anterior and posterior synechia, corneal
decompensation, and cataract formation. Releasable sutures technique also prevent associated complication in
the posterior segment, including choroidal detachment, macular edema, and suprachoroidal hemorrage. There
are four releasable suture techniques such as Migdal, Kochler, Rootman, Cohen and Osher. The most widely
used technique for these sutures was developed by Cohen and Osher.
Keywords : releasable sutures, trabeculectomy, anterior chamber depth
GLAU S 2.1.7
Techniques to Enhance Successful Trabeculectomy
Yulia Primitasari, MD
Airlangga University, Surabaya
Trabeculectomy or glaucoma filtration surgery is performed to provide an alternative route for aqueous
humor efflux from anterior chamber and past the diseased trabecular meshwork to a space external to the
eye. One criteria for successful trabeculectomy is were graded as 1. Complete success: IOP below 21 mmHg
without medication, 2. Qualified success: IOP blow 21 mmHg with medication and 3. Compelete failure: IOP
at or above 21 mmHg despite maximum medications.
Many factors contribute to the final outcome of filtration surgery. Some risk factors for failure can
be modified or reduced by taking specific course of action before and during filtration surgery.
Preoperative assessment or patient selection is the most important factor that can influence the successful
outcome of trabeculectomy. Step by step surgical technique must be folowed, and each step in the
process of performingthis surgery must be carefully considered. Using anti fibrotic agents can improve the
post operative surgical success in both primary open angle and high risk glaucoma cases.
The process of glaucoma filtration surgery is a challenge to the ophthalmic surgeon. Meticulous technique
combined with careful and close observation are required throughout the perioperative period.
GLAU S 2.1.9
POST TRABECULECTOMY COMPLICATION MANAGEMENT
Evelyn Komaratih, MD
Departement of Ophthalmology Faculty of Medicine, Airlangga University
Dr. Soetomo General Hospital Surabaya
Glaucoma is an increasingly one of the leading cause of blindness as the world›s population life expentancy
increases . Surgical option for glaucoma is considered when other modalities are not sufficient in maintaining
intraocular pressure on target. As with most procedures, a little planning and foresight are the best ways to make
sure complications doesn›t impair the outcome. Successful trabeculectomy is a stable surgical fistula. The one
thing required to keep a fistula patent (open) is flow. The principal and most challenging complication is scarring
of the fistula; however, other complications may occur such as hypotony, chroridal detachment, conjunctiva
leak, and infection. To minimize the morbidity, it is very important that one should know how to prevent them,
recognize them and treat them. The goal of trabeculectomy is to achieve controlled intraocular pressure (IOP).
However this effort does not end after the surgical procedure is complete. Postoperative management of the
trabeculectomy requires the understanding of possible factors that influence IOP and suffiecient knowledge
to manage the IOP within target. Recent changes in interventions and surgical technique, such as the use
of antimetabolites, have provided new challenges in the postoperative management of trabeculectomy
patients. Complication management is directed to restore the flow of aqueous to the filtering bleb in the early
postoperative period, manage flat anterior chambers with both high and low intraocular pressures, manage a
failing filtering bleb, and to manage hypotony.
Keywords : trabeculectomy, trabeculectomy complication.
GLAU S 2.2.1
PROBLEM ARISING DUE TO GLAUCOMA MISMANAGEMENT
Virna Dwi Oktariana A.
CiptoMangunkusumo hospital/ Faculty of medicine universitas Indonesia
Glaucoma is a chronic progressive disease and it has classified as irreversible blindness. Thus diagnosing it as early
as possible will give benefits for the patients. Early diagnosis and proper management will halt or at least slower
the glaucoma progression. Glaucoma had a wide range variety from primary to secondary. Neverthelessmost
glaucoma cases are primary and the most of it are open angle glaucoma. Primary open angle glaucoma is mostly
asymptomatic and presented in a severe condition as the end stage of the glaucomatous optic neuropathy. In
most cases, glaucoma managed with medication but some of them especially angle closure glaucoma must have
iridectomy to prevent further damage or to prevent them from progression to another stage of the disease.
Secondary glaucoma should be managed depend on the cause. It will recover nicely if the management is to
relieve the causes, e.gphacolytic will be overcome by doing cataract extraction if the trabecular meshwork is
still functioning well.
GLAU S 2.2.3
Management Of Co-Exsisting Cataract And Glaucoma
Nurwasis, MD
Both glaucoma and cataract are diseases with an increasing prevalence with age, and thus one often
finds that they are coexistent in the elderly patient population. The association of glaucoma with cataract has
becoming more frequent as a result of increase in life expectancy. The use of anti glaucoma medication has
only strengthened their association. Furthermore, glaucoma surgery significantly increases the risk cataracts
development. The most important step before operating on a patient with cataract and glaucoma is the
preoperative evaluation as well as the decision regarding the type of surgery to be performed.
The goal of treatment in a glaucoma patient with cataract, is to achieve an adequate long term control
of intraocular pressure (IOP), avoid postoperative IOP spikes which are deleterious to the health of the optic
nerve head, obtain an optimal visual rehabilitation thus improving the quality of life of the patient.
In the management of a patient with a significant visual impairment due to cataract and coexisting
glaucoma, there are three basic surgical approaches: (a) cataract extraction alone, which may need to be
followed by a trabeculectomy later; (b) glaucoma filtering surgery alone, followed by cataract removal later
(two-stage approach); and (c) combined cataract and glaucoma surgery. One surgical experience maybe the best
for a patient.
Cataract extraction alone is suitable for patient with easily controlled glaucoma (for example with a
single medication) and with minimal optic nerve damage. Cataract surgery alone has significant effects on the
intraocular pressure. However, the effect is small, averaging around 2-4 mmHg and one cannot depend on this
as a means of lowering the IOP.
Glaucoma filtration surgery is used alone, in patients with very advanced optic nerve damage.
Trabeculectomy remains the gold standart technique for the management of adult hood glaucoma and by far is
the most preferred surgical technique.
The combined surgical technique of phacotrabeculectomy has become the standard technique for management
of eyes with co-existent cataract and glaucoma. Many surgeons perform trabeculectomy with phacoemulsification
surgery in patients with stable IOP, yet still on 2-3 anti-glaucoma medications.
GLAU S 2.2.4
Ocular Hypertension
Masitha Dewi Sari, MD
Ocular hypertension is a clinical term describing the presence of elevated intraocular pressure in the absence
of optic nerve damage and visual field loss with anatomically normal, open angles on gonioscopy. For most
individuals, the normal range of intraocular pressure is between 10-21 mmHg. Elevated intraocular pressure is
an important risk factor for glaucoma. The Ocular Hypertension Treatment Study determined that topical ocular
hypotensive medication delays or prevents the onset of Primary Open Angle Glaucoma.
GLAU S 2.2.6
How to deal with uveitic glaucoma ?
Andika P Gandasubrata, MD
Department of Ophthalmology Padjadjaran University / Cicendo Eye Hospital Bandung
The management of uveitic glaucoma depends on the underlying disease and on the individual patient. The
treatment rationale consists of 1) treating any underlying systemic disease, 2) treating the ocular inflammation,
and 3) treating the glaucoma. The ocular inflammation and glaucoma usually can be controlled with eye drops.
Often, treatment of the inflammation will control the IOP.
It is a general rule that surgery should be avoided, when possible, in the inflamed eye. However, if surgery is
required, the eye should receive maximal preoperative anti-inflammatory therapy to decrease the inflammation
as much as possible. In eyes with active uveitis, preparation for intraocular surgery should include perioperative
topical and, occasionally, systemic corticosteroid therapy to avoid exacerbation of uveitis and failure of filtering
surgery. If an elective surgical case is to be performed, the uveitis should be as quiet as possible for 3 months
prior to surgery.
GLAU S 2.3.3
Malignant Glaucoma
Vira Wardhana Istiantoro, MD
The term malignant glaucoma refers to a sustained ongoing process that is difficult to treat and characteristically
progresses to blindness. It is sometimes unresponsive and occasionally worsened with conventional management.
Modern vitreoretinal surgical techniques, as well as a better understanding of the disease, have improved the
prognosis to some extent.
GLAU S 2.3.4
WHAT WE HAVE TO DO WITH NTG ?
Tatang Talka Gani, MD
Fakultas Kedokteran Universitas Gadjah Mada RSUP Dr. Sardjito, Yogyakarta
Glaucoma is one of the leading causes of irreversible visual impairment in the world. One type of glaucoma
that is still becomes a challenge for an ophthalmologist is Normal Tension Glaucoma (NTG). There are still many
controversies on management and evaluation on NTG. Is not uncommon that disease progression is still found
despite standard therapy has been given. NTG does have distinctive features compared with primary open-
angle glaucoma: intraocular pressure–independent risk. Patient systemic conditions such as autoimmunity,
obstructive sleep apnea, the stability of the intracranial pressure and systemic blood pressure control gives an
important role in the progression of NTG. This article provides an overview and update on the current issues
surrounding the pathogenesis, diagnosis, management, evaluation and monitoring of NTG.
Keywords: Normal tension glaucoma, Glaucoma Management, Glaucoma Evaluation
GLAU S 2.3.5
TIPS AND TRICKS OF GLAUCOMA DRAINAGE DEVICES.
Sirisha Senthil, MD
Glaucoma drainage devices and implants are now being increasingly used in the management of refractory
glaucoma. With the rising number of secondary glaucomas after corneal and retinal surgeries, or multiple failed
trabeculectomies and challenging secondary glaucomas like uveitic, neovascular glaucoma etc, every glaucoma
specialist may need to offer glaucoma implant procedure in certain situations to help their patients better. In this
regard, understanding the surgical principles of GDD implantation, their techniques, associated complications
and their management is important.
GLAU S 2.3.6
GLAUCOMA DRAINAGE DEVICES IN THE MANAGEMENT OF
REFRACTORY CHILDHOOD GLAUCOMAS.
SirishaSenthil
Congenital glaucoma is a potentially blinding disease that is refractory to medical treatment. In pediatric
glaucoma, the mainstay of treatment is surgical and antiglaucoma medications are used as an adjunct to control
the IOP when the child is waiting for surgery. The surgical procedures for congenital glaucoma are Trabeculotomy,
goniotomy or combined Trabeculotomy-Trabeculectomy.
Combined Trabeculotomy-Trabeculectomy is our preferred surgical choice to manage primary congenital

glaucomas. Though the procedure is associated with good early success rates, about 20% of the surgeries
fail at the end of 1 year. The surgical success decreases over time due to rapid wound healing response and
excessive scarring in children. Secondary congenital or developmental glaucomas associated with anterior
segment dysgenesis, aniridia have poorer success rates. Glaucoma drainage devices are important alternatives
in the management of these refractory childhood glaucomas, specially when the filtering surgery has failed.
Good preoperative assessment, meticulous surgery, close follow up and visual rehabilitation help to achieve
substantial success in these eyes.
II S 3.1.3
Updates on Bacterial Corneal Ulcer :
Epidemiology, Antibiotic Sensitivity, and Recent Management
Suhardjo,MD., Reny Setyowati, MD., Widyandana, MD.
Yogyakarta Eye Study, Ophthalmology Department, RSUP Dr. Sardjito, Universitas Gadjah Mada, Yogyakarta
There are changing epidemiology of infectious bacterial keratitis cases. The incidence of community-acquired
infection is rising. During 2015 there were 79 cases of patients with corneal ulcer in RSUP Dr. Sardjito Yogyakarta.
There were 28 patients with severe corneal ulcer’s and 27 of them patients causative agent were bacterial.
Coagulase negative Staphylococcus being the most common organism cultured from corneal swab. The
pattern of antibiotic sensitivity is also changing. Amikacin and Trimetoprim-sulfametoxazole were agent with
high activity against the organism. Corneal collagen cross-linking (CXL) was initially developed to stabilize
keratoconus. Nowadays CXL has been shown to be an efficient treatment stabilizing corneal ulcers. Justified
use of newer antibiotics may help decrease the incidence of challenging infectious with more resistant strains
in future and CXL may also help reduce the burden of multi-drug-resistant pathogens and patient’s compliance
in those clinical situations.
II S 3.1.7
TIPS AND TRICK IN MANAGING INFECTIOUS CONJUNCTIVITIS
I Gusti Ayu Made Juliari, MD
Conjunctivitis is the most common cause red eye in the eyeclinic. Conjunctivitis is an inflammation on conjunctiva.
Conjunctivitis has a number of different causes, infectious or non infectious. Infectious conjunctivitis could be
divided into viral, bacterial, parasite, and fungal. Non infectious conjunctivitis could be caused by allergies,
dry eye, or toxic. Conjunctivitis caused by some bacteria and viruses can spread easily from person to person,
but it’s not a serious health risk if diagnosed promptly. Conjunctivitis in newborn babies, however, should be
reported to doctor immediately, as it could be a vision-threatening infection. Clinically judgment and sometime
laboratory examination will help in managing conjunctivitis.
II S 3.2.1
INFECTIOUS CORNEAL ULCER
Havriza Vitresia, MD
Ophthalmology Department, Andalas University, Dr M Djamil Hospital
Padang, West Sumatera
Purpose : To report cases of interesting of infectious corneal ulcer and it’s management
Method : A case report of five patients with infectious corneal ulcer with various caused of microorganism. Two
cases with fungal corneal ulcer on clinical presentation, but culture and sensitivity test result show the different
microorganism. One case found no microorganism and the other found MRSA with multi drug resistent. There is
a case ofgonococcal neonatal conjunctivitis, with thick membran on the tarsal conjunctiva, and the other cases
are viral corneal ulcer and bacterial corneal ulcer from contact lens wear. Culture test result found Seratia
Marcesscens, a gram negatif cocobacilli. All patient were treated with anti microbial agent and show clinical
improvement.
Results ; Some organism produce lesions of particular shapes, color or have some distinctive features, and a
mere clinical examination of such lesions may aid in establishing the etiological diagnosis, although clinical
observation should not replace the laboratory investigation of direct microscopy and culture of corneal scrapings.
Conclusion : Based on the presenting clinical history, risk factors, predisposing ocular and systemic factors and
distinctive clinical signs, infectious corneal ulcer may be easy to diagnose. However, there may be few factors
which may alter the typical clinical features.
II S 3.2.2
Thygeson Superficial Punctate Keratitis
N. K. Niti Susila, MD
Thygeson superficial punctate keratitis adalah kelainan di epitel kornea bagian superfisial idiopatik, bilateral dan
biasanya remisi dan eksaserbasi. Umumnya menyerang dewasa muda tetapi dapat juga menyerang segala umur.
Tujuan penulisan makalah ini adalah untuk refresing kembali tentang Thygeson superficial punctate keratitis.
Metode dari penulisan ini adalah dengan menelusuri kepustakaan yang ada, sehingga dapat diuraikan tentang
Thygeson superficial punctate keratitis, mulai dari anatomi kornea, patogesis, gejälá kliñis serta penangáñäfi
Thygesõñ superficial ÞUfiCtate kéfátitiS.
Ringkasan yang dapat disampaikan adalah keluhan utama yaitu terasa iritasi yang sering timbul berulangkali,
fotofobia, berair, visus menrun. Gambaran klinis yang tampak mata tenang tidak merah, pungtata erosi kornea
dengan tes fluorsein positif dan lesi terletak disentral kornea. Terapi dapat diberikan lubrikasi, steroids tetes
mata ataupun cyclosporine.
Kunci : keratitis superficial puntata dari Thygeson
LV W 3.1
Visual acuity assessment in low vision patient
Erna Niza, MD
Low vision is significantly reduced vision, that is visual acuity is less (worse) than 6/18 or visual fields are less than
20 degress in diameter. After treatment or with refractive correction, vision cannot be corrected to “normal”,
visual performance maybe affected. A person with low vision can use her reduced vision for learning about the
world and for planning and doing task that need vision.
The World Health Organization (WHO) classifies people with low vision as follows:
1. 6/18 (20/60) [0.5] to 6/60 (20/200) [1.0] : mild
2. 6/60 (20/200) [1.0] to 3/60 (20/400) [1.3] : moderate
3. 3/60 (20/400) [1.3] to 1/60 (20/1200) [1.8] : severe
Reading from left to right are in meters, feet and Log Mar values.
The factors that need to be considered when choosing and using object are: size, distance, contrast, colour,
position, light on and ground the object. Beforming low vision evaluation can be one of the fulfilling this business,
but remember that every situation will be unique.
LV W 3.2
How to determine the magnification power for near/distance viewing
for low vision devices
Rajiv Khandekar, MD
There are theoretical estimates for determining the required magnification of a low vision device. However, there
is significant variation in an individual’s needs and selection of magnification for a low vision device. Experience
and practical tips help the low vision practitioner in the selection of the initial device for trial/ training of patients
and determining the final prescription. Magnification primarily improves near visual function. However, there
must be an appropriate balance between magnification and the constriction of the field of vision. Determination
of the magnification of the device to be dispensed also depend on the patient’s occupation, whether it is a hands-
free device, duration of viewing, binocularity, and other factors. The selection of a device for magnification and
the power has become less important in the era of smartphones and apps.
LV W 3.3
How to choose appropriate low vision devices
Rajiv Khandekar, MD
Magnifiers are used for improving near vision and telescopes for the distance vision. Environmental changes and
use of non-optical devices complement the visual functions with optical devices. These devices are less expensive
and easy to adopt. Hence non-optical devices should be the initial recommendation or tried concurrently with
other optical aids. Filters that reduce glare are very useful. The selection of low vision device is mainly based on
the cost of the device, patient’s requirements, residual vision, binocularity, mobility and status of other sensory
functions. A disability scoring system allows the quantification of the change before and after selection of the
device. The scoring system provides effective monitoring of the progress of low vision care.
LV W 3.4
The Importance of Visual Function Assessment in Low Vision Patient
Umar Mardianto, MD
Consultant of Ophthalmology in Refractive Division FKUI-RSCM Kirana (National Referral hospital)
Visual function assessment played an importance role in low vision patient. In managing low vision cases
Ophthalmologist must have baseline data about the condition of each patients. These data were very important
not only to make a proper diagnose but also for formulating therapy and rehabilitation which is individual for
each patients.
LV W 3.5
How to train your Low Vision patient to use their low vision devices.
Karmelita S, MD
We often give Low Vision devices prescription after a long examination just by give simple instructions. But the
success of low vision rehabilitation requires a comprehensive teaching of the devices. Teaching how to use the
device is one package with the devices itself.
As simple as how much will the reading distance be for a certain device according to the power of the devices.
Many people will gain their confidence in using it, many patients will come for follow up because they can
get full benefit of their devices that work well. We have to give patients the state of the art of the Low Vision
rehabilitation services and those devices. We should give the written instruction along with the devices.
NO 3.1.3
Controversy of Steroid in Traumatic Optic Neuropathy
AA Mas Putrawati, MD
Traumatic optic neuropathy (TON) is a condition that refer to an acute injury of the optic nerve secondary to
trauma and potentially with blinding complication . Vision loss can be immediate or may take days, weeks, or
even months to develop. The optic nerve axons may be damaged either directly or indirectly and the visual loss
may be partial or complete. The commonest aetiology is optic nerve compression from orbital haemorrhage
(orbital compartement syndrome).The pathogenesis and treatment of TON are inconclusive. Following the
initial injury, optic nerve swelling within the optic canal can result in secondary retinal ganglion cell death.
There have been no randomized, placebo-controlled treatment trials till now and no convincing data to support
any treatment as being effective for TON. There are some evidence to treat TON including; systemic steroid,
intravitreal steroid, surgery or neuroprotectants agent.
Pulse steroid therapy has been frequently used as a treatment of choice, but the International Optic Nerve
Trauma Study (IONTS) was unable to demonstrate that high-dose of intravenous methylprednisolone (MP)
therapy or surgery was more beneficial than no treatment. Furthermore, it is important to consider the adverse
effects of steroid in Traumatic cases.
Our descriptive study in Sanglah Hospital during 2013-2015, we found 41 patient were included in these study.
13 patients received high dose corticosteroid intravenous and 28 patients received observation. We found no
statiscally significant difference in improvement of visual acuity ³ 2 line between patients treated with dose
corticosteroid and observation (p =0,755).
Individual decision-making treatment regarding to patients general condition, age and visual function is essential.
In the future, further preclinical trials with neuroprotectans and subsequent clinical trials may bring forth a new
horizon for TON.
Keyword: Traumatic Optic Neuropathy, Steroid.
NO 3.1.5
Chiasmal Lession Work Up
Devi Azri Wahyuni, MD
Ophthalmology Departement of Mohammad Hoesin Hospital
Medical Faculty of Sriwijaya University
Importance of thorough history-taking, meticulous examination and tailored investigations cannot be

underestimated in neuroophthalmology. Lesions producing a relative or absolute bitemporal hemianopia,
without affecting central vision, can be localized to the mid or posterior optic chiasm. The differential diagnosis
for this lesions includes pituitary adenoma, parasellar meningioma, craniopharyngioma, parasellar internal
carotid artery aneurysm, chiasmal glioma, abscess, and inflammatory, metastatic or demyelinating lesions. A
detailed history regarding onset, duration, and progress of the complaints must be taken.Visual field loss due to
chiasmal and retrochiasmal lession is characterized by temporal defect that align along the vertical meridian. In
chiasmal syndrome optic disc may appear normal initially, even with significant visual field loss. With progressive
damage, the optic discs show typical atrophy, often in temporal portion of the disc. CT scan of the brain with
sellar images may be sufficiently specific and can detect tumor calcifications. However, the detail is generally
inferior to that of MRI. Patient with nonsecreting tumor typically present with vission loss. Hormonally active
tumor, however, are often detected before vission loss because of systemic system.
NO 3.1.6
ETHAMBUTOL TOXIC OPTIC NEUROPATHY: WHAT’S NEW?
Riski Prihatningtias, MD
Ophthalmology Department, Faculty of Medicine, Diponegoro University, Semarang
Ethambutol toxic optic neuropathy is a well-recognized adverse ocular event in patients who receive this drug for
tuberculosis treatment. The World Health Organization estimates that there are about 9.2 million new cases of
tuberculosis each year. About 55% of these patients take ethambutol each year as a treatment for tuberculosis
or Mycobacterium avium. If we take the conservative estimate that 2% of these individuals will experience
significant and irreversible visual loss, then the annual incidence of this serious iatrogenic complication is
100,000. The patient develops slowly progressive loss of visual acuity in conjunction with central or cecocentral
scotomas, dyschromatopsia, and loss of high spatial frequency contrast sensitivity.
Ethambutol is a metal chelator, destroying bacteria by inhibiting arabinosyltransferase, an important enzyme
in mycobacterial cell wall synthesis. Due to the similarity between mammalian mitochondrial DNA (mtDNA)
and bacterial ribosomes, ethambutol also disrupts oxidative phosphorylation and mitochondrial function by
interfering with iron-containing complex I and copper-containing complex IV. Classically, the ocular toxicity is
described as dose and duration related, and is largely reversible on drug discontinuation. However, the issue of
reversibility is challenged by many recent studies.
How should we change our approach to the use of ethambutol to still effectively treat tuberculosis? Proper
management includes identification of patients at risk, adjusting the dose regimen for impaired renal function,
body weight, and age, regular monitoring for early signs of ocular toxicity, and patient education.
NO 3.2.2
Mysthenia Gravis is the great mimicker of Ophthalmoplegia
Syntia Nusanti, MD
Faculty of Medicine University of Indonesia Cipto Mangunkusumo Hospital
Mysthenia is one of the “great mimickers “and can almost simulate almost any ocular motility disorder. Its
ophthalmic clinical hallmark are ptosis and/or diplopia are usually fatiguable and variable. If ophthalmoplegia
limited to one or more extraocular muscle without ptosis, in can be very difficult to diagnose. Usually myasthenia
gravis is idiopathic but sometime can be induced by drugs. Sometime patients with myasthenia have systemic
symptoms such as dyspnea, dysphagia, fatigue and lack of energy. Some clinical test can be done to diagnose
myasthenia gravis such as sleep test, ice test, tensilon test and electromyography. Myasthenia is a potentially life
threatening disease, in general treatment is given to keep the patient is comfortable and safe as possible until
the disease stop it self.
NO 3.2.3
Multiple Sclerosis and Ophthalmoplegia
Antonia Kartika, MD
Neuro-Ophthalmology Unit, National Eye Center-Cicendo Eye Hospital
Multiple Sclerosis is a chronic inflammatory autoimmune disease of the central nervous system. It attack
myelinated axon in CNS destroying the myelin and the axon to varying degree. No evaluation of a patient
either suspected or diagnosed with MS is complete without a systematic examination of their vision and eye
movements (Leigh and Zee 2006) .
The most common eye movement abnormalities are saccadic dysmetria, internuclear ophthalmoplegia (INO),
disorders of the vestibulo-ocular reflex (VOR), and gaze-evoked nystagmus.
Clinical detection of INO and saccadic abnormalities in MS may be challenging (Frohman TC et al 2003), and
measurement of eye movements may help confirm the diagnosis during early stages of the disease.
The neuro-ophthalmologic examination including eye movement assessment also provides insights into the
nature of the disorder and even in estimating prognosis.
NO 3.2.4
Ophthalmoplegia and chronic red eye
Yunita Mansyur, MD
Neuro-ophthalmology Subdivision Ophthalmology Department Hasanuddin University, Makassar
Ophthalmoplegia with eye swelling, and redness may represent a diagnostic challenge to many ophthalmologists.
The differential diagnosis is broad and includes both no emergent and emergent diagnoses. While the emergent
diagnoses must be made and treatment initiated immediately, the no emergent differential also includes
life- and vision-threatening entities. We report a case-based discussion including distinguishing features,
diagnostic modalities, and treatment of similar entities involving external ophthalmoplegia with red eye on
neuroophthalmology point of view.
NO 3.2.5
Frozen Eyes
Mohamad Sidik, MD
Ophthalmology Department, Faculty of Medicine, Universitas Indonesia
– Cipto Mangunkusumo Hospital, Jakarta.
Frozen eye is a term referred to an abnormal condition of eye movements which is indicated by unability of
the globe to move to any direction. Abnormality of eye movement could affect to one or both eyes and the
direction affected could be to certain or all directions of movement.
The causes of this abnormality were varied and could be differentiated to one of the following : extra ocular
muscles diseases, toxic or metabolic disorders, central or peripheral nervous system abnormalities, genetic or
hereditary and neuromuscular junction disorder.
Careful ophthalmology examination, certain laboratory testing and imaging examination should be performed
to establish diagnosis and exact therapy.
NO 3.2.6
NEUROIMAGING IN OPHTHALMOPLEGIA
Batari Todja Umar, MD
Ophthalmoplegia refers to weakness or paralysis of one or more extra-ocular muscles, which responsible for eye
movements. An ophthalmoplegia results in double vision with many potential causes that can involve many different
structures. The cranial nerves responsible for ocular movements, the third, the fourth and the sixth nerve, can be
affected intrinsically or extrinsically along their nuclei, their course in the brainstem, in the cisterns, skull base,
cavernous sinuses and orbits. As a result, neuroimaging is mandatory to clarify the cause of ophthalmoplegia
and to guide the treatment but deciding to do the neuroimaging or not is important since some ophthalmoplegia
cases does not need imaging immediately depends on the mechanism of muscle weakness. Clinical
presentation should suggest lesion localization and prompt the most appropriate neuroimaging techniques.
MRI is the most accurate imaging modality in the setting of ophthalmoplegia while non- enhanced CT is
the most common initial imaging examination in the emergency setting. In selected cases, vascular imaging with
MR angiography, CT angiography or conventional catheter digital subtraction angiography (DSA) is also necessary.
NO 3.3.2
VISUAL FIELD DEFECT IN STROKE
Hartono, MD
To know the pattern of visual field defect in occipital stroke, the route of upper and lower part of lower radiation
of geniculocalcarine tract (optic radiation) through parietal and temporal lobe, end in the occipital stroke,
more exactly in the calcarine cortex, must be understood. Homonymous visual field defect without other
neurologic signs/symptoms mostly caused specifically by lesion of occipital lobe. Most of the occipital lesions
are caused by stroke, characterized by sudden characteristic of visual field defect. Visual field defects caused by
occipital stroke are as follow: 1) Right or left homonymous hemianopia with macular sparring. 2) Right or left
homonymous hemianopia with macular sparring and temporal crescent. 3) Right or left superior homonymous
quadrantanopia (pie in the sky). 4) Right or left inferior homonymous quadrantanopia (pie in the floor). 5) Cross
quadrantic (checkered board) hemianopias with or without temporal crescent. 6) Right or left homonymous
hemianopic scotoma. 7) Homonymous bihemianopia (central or cortical blindness).
NO 3.3.3
NEURO-OPHTHALMIC PROBLEMS IN MENINGITIS
Bobby R.E Sitepu, MD
Department Ophthalmology, Faculty of Medicine, University of North Sumatera
Meningitis is an acute inflammation of the protective membranes covering the brain and spinal cord known
collectively as the meningens. This may be caused by bacteria, viruses, or other microorganisms, but can be
a result of injury, cancer, or certain drugs. Meningitis can be life-threatening because of the inflammation’s
proximity o the brain and spinal cord.
The type of meningitis and its cause can only be determined by a physician using laboratory test results. A lumbal
puncture diagnoses or excludes meningitis. The clinical expression of meningitis depends on the underlying
medical condition and the immune status of the patient.
Manifestation neuro ophthalmic are diplopia. Pupillary disorder, lossof visual field, optic neuritis and papilledema
can be develop.
NO 3.3.4
MANAGEMENT OF PUPILLARY ABNORMALITIES
Vierlia, Wino Vrieda, MD
Department of Ophthalmology Saiful Anwar Hospital -Faculty of Medicine Brawijaya University, Malang
The value of pupillary size and motility observation in the evaluation of patients with neurologic disease is highly
important. In many patients with visual loss, an abnormal pupillary response is the only objective sign of organic
visual dysfunction. Pupillary testing serves two purposes which are to find disorders of pupillary function itself and
to detect disorders of the afferent visual system and the autonomic innervation of the eye. Prompt examination of
pupillary reaction to light and near stimuli, the difference in anisocoria in light and dark, and attention to distinctive
associated signs and symptoms facilitate differentiating the abnormalities in pupil size and response to stimuli.
Pupillary disorders may be classified into two major categories , the afferent and efferent. The afferent pupillary
defects interfere with the input of light to the pupillomotor system by light blockage or deficits in any of the retinal
layers, into the optic nerve, chiasm, optic tract, or midbrain pretectal area. All of these result in a symmetrical
decrease in the contraction of both pupils to light given to the damaged eye compared with light given to the
other less damaged or normal eye. While the efferent pupillary defects interfere with contraction or dilatation
of the pupil due to damage in the midbrain, in the peripheral nerve that supplies the iris muscles, or in the iris
muscles themselves that often leading to anisocoria. If the anisocoria is greater in dim light then the defect is
in the sympathetic innervation of the pupil while if the anisocoria is greater in bright light then the lesion is in
the parasympathetic innervation of the pupil. Some major abnormalities in pupillary function that are detected
through examination are physiologic anisocoria, Horner’s syndrome, Adie’s tonic pupil, third nerve palsy, and
Argyll-Robertson pupil. The treatment will depend on each individual case.
NO 3.3.5
SLEEP APNEU DISEASE AND NONARTERITIC ANTERIOR ISCHEMIC OPTIC NEUROPATHY
Seskoati P , MD
Faculty of Medicine, Brawijaya University Saiful Anwar Hospital
Nonarteritic anterior ischemic optic neuropathy (NAION) is a common cause of visual loss from optic nerve
dysfunction. It is characterized by sudden or rapidly progressive, painless visual loss, and altitudinal visual field
defect. Initially accompanied by segmental or diffuse optic disc edema, later optic atrophy and retinal arteriolar
narrowing. This condition is due to circulatory insufficiency within the optic nerve head, because of obstruction
or decreased perfusion through short posterior ciliary arteries, but the specific mechanism and location of
the vasculopathy remain unknown. However, some of systemic conditions accompanied with NAION consist of
systemic hypertension, ischemic heart disease, hypercholesterolemia, stroke, smoking, nocturnal hypotension
and atherosclerosis.
Obstructive sleep apnea syndrome (OSAS) is a temporary cessation or absence of breathing during sleep that
common yet underdiagnosed condition that may be associated with significant morbidity if left untreated. It is
characterized by recurrent interruption of normal breathing during sleep, owing to upper airway obstruction
(apneic spells). The apneic spells can cause a decrease in the arterial oxygen saturation and an increase in
the carbon dioxide saturation during sleep. OSAS has been associated with an increased risk of cardiovascular
disease, hypertension (HT), and stroke.
Therefore, we report the several cases of OSAS that underwent optic nerve function examination such as
perimetry, contrast sensitivity, and Ishihara test for the diagnosis of NAION. We also evaluated patients with
NAION underwent overnight polysomnography for the diagnosis of OSAS and calculation of apnea-hypopnea
index (AHI). We evaluated the possible association between OSAS and NAION.
OCB 3.2
Establishment of First Child-Friendly Pediatric Eye Care Center in South Sulawesi,
Indonesia
Habibah S Muhiddin, MD
Children blindness becomes a big issue in the future. Perdami Sulsel, Orbis International, HKI, and CBM have
made a proposal to develop children eye care system in South Sulawesi. One of the programs is to establish
a Child-Friendly Pediatric Eye Care Center in Hasanuddin University Hospital. The activities start from 2015,
consisted of renovation of the area, human resources capacity building, developing good quality assurance
system and developing links to other stakeholders and organization, which associated with blindness in children.
Grand opening was performed on July 23rd, 2016.
OPHCOM 2.2
PROGRAM DEPARTEMEN PENDATAAN KOMATNAS 2015-2019
Nina Ratnaningsih, MD
2 2 2 2 2
NO
SASARAN
INDIKATOR KEGIATAN POKOK 0 0 0 0 0 PENANGGUNG
STRATEGIS 1 1 1 1 1 JAWAB
5 6 7 8 9
Menyempurnakan
system informasi
V
Adanya informasi kesehatan indera Pusdatin
gangguan penglihatan Pemda Propinsi dan
penglihatan dan Mengintegrasikan Dinkes Kab/Kota
kebutaan yang kedalam system Dan Komda dan
V V V V
Penguatan sistem terintegrasi dengan informasi kesehatan Komatnas
informasi dan SIK NAS nasional
1
penyediaan Monitoring dan
V V V V
data gangguan evaluasi
penglihatan dan Mapping propinsi
kebutaan tempat pelaksanaan V V V V V
Adanya data hasil survey Komatnas-Komda
Rapid Assessment
Penyusunan jadwal, Litbangkes dan
of Avoidable
anggaran dan team V V Perdami
Blindness (RAAB)
pelaksana survey
Pelaksanaan survey
V V V V V
Monitoring, evaluasi
OPHCOM 2.3
Program kerja Departemen Pengembangan SDM KOMATNAS
Trilaksana Nugroho, MD
Department of Ophthalmology, Faculty of Medicine Diponegoro University
dr. Kariadi General Hospital Semarang
Prevalensi kebutaan di Indonesia merupakan tertinggi kedua setelah Ethiopia di antara seluruh negara di
dunia dan terus meningkat pada tahun 1993 sebesar 1,5% sampai dengan tahun 2014 – 2015 (Survey RAAB)
berkisar antara 2,7% - 4,5%. Untuk menurunkan prevalensi sebanyak 25% (dalam periode lima tahun) gangguan
penglihatan dan kebutaan di seluruh belahan dunia, WHO menyusun Global Action Plan yang salah satu
strateginya adalah peningkatan kapasitas dan kompetensi dokter mata, perawat, dan optometrist. Di
Indonesia pada tahun 2015 dibentuk Komite Mata Nasional (Komatnas) sebagai bagian dari Global Action
Plan. Departemen Pengembangan SDM Komatnas mempunyai tugas mengembangkan program koordinasi dan
peningkatan kualitas dan kuantitas dokter spesialis mata, perawat mahir mata, perawat mata masyarakat, dan
pembinaan kader kesehatan mata di masyarakat dengan berkoordinasi dengan institusi / pusat pendidikan dokter
spesialis mata, Kolegium Oftalmologi Indonesia, Puskesmas / Dinas Kesehatan, Rumah Sakit Umum Daerah, dan
instansi terkait untuk menjamin kesiapan dan ketersediaan sumber daya manusia di bidang kesehatan yang
berkualitas sesuai kebutuhan.
OPHCOM 2.5
Program Kerja Departemen Gangguan penglihatan pada anak
Mayang Rini, MD
Kehilangan penglihatan pada anak berdampak pada seluruh aspek perkembangan anak tersebut.
Dampak tersebut terjadi pada bidang pendidikan, pekerjaan dan sosial. Anak yang mengalami kehilangan
penglihatan berisiko mengalami kesulitan dalam tingkah laku, psikologis dan emosional, terganggunya rasa
percaya diri dan integrasi sosial.
Vision 2020 The Right to Sight dari Badan Kesehatan Dunia menempatkan kebutaan pada anak sebagai
salah satu prioritas programnya karena berbagai alasan. Pertama, jumlah “tahun buta” akibat kebutaan pada anak
terjadi lebih dari dua kali lipat “tahun buta” akibat katarak pada orang dewasa. Kedua, penyebab kebutaan pada
anak sangat berbeda dengan penyebab kebutaan pada orang dewasa, dan strategi untuk menangani kebutaan
pada orang dewasa tidak akan bisa diterapkan untuk mengangani kebutaan pada anak. Ketiga, keterlambatan
penanganan terapi untuk beberapa penyebab kebutaan dapat berakibat amblyopia yang tidak akan terjadi pada
orang dewasa. Keempat, mata anak-anak bukan seperti mata orang dewasa yang kecil, akan berespon berbeda
terhadap terapi, sehingga membutuhkan keahlian, peralatan dan pelatihan khusus.
Komite Mata Nasional di Indonesia telah dibentuk kembali sejak tahun 2015, yang berfungsi kembali
sebagai badan koordinator nasional untuk kesehatan mata. Salah satu program yang ada adalah program kerja
departemen gangguan penglihatan pada anak. Merujuk pada universal eye health a global action plan 2014
-2019 yang bertujuan untuk menurunkan prevalensi gangguan penglihatan yang dapat dihindari sebanyak
25% pada tahun 2019, maka prioritas program kerja gangguan penglihatan pada anak adalah menanggulangi
gangguan penglihatan pada anak akibat kelainan refraksi.
OPHCOM 3.1
SIGHTFIRST PROJECT AND CHILDHOOD BLINDNESS PROGRAM IN SOUTH SULAWESI
Ahmad Ashraf Amalius, MD
Departemen Ilmu Kesehatan Mata FK UNHAS / RS Universitas Hasanuddin
Purpose : To report the sightfirst project by Lions Club International Foundation (LCIF) and Childhood Blindness
Program by Standard Chartered Bank and consortium of NGO (Orbis International, Helen Keller Indonesia and
Christoffel Blindenmission Indonesia).
Method : The sightfirst project focused in 3 districts that are Bantaeng, Palopo and Bone. The activities
were training for staffs, donated equipment for PHC and hospitals. After the training, there were screening
for elementary school students and general public. Program for Childhood blindness were hospital based
hands on training program for ophthalmologist, establishment of children eye care center, training abroad for
ophthalmologists, anesthesiologists, nurses, optometrist, ortoptist, counselor.
Results : During the activities, there were 9 ophthalmologists, 1 anesthesiologist, 30 GPs, 5 mid-level opthalmic
personnel, 1 counselor, 1 optometrist, 1 ortoptist, 30 nurses, 100 teachers, 100 cadres trained by LCIF and Orbis.
In all 3 districts area, there were screening in elementary school for 6.135 students and the result showed 425
students suffered from refractive error. Screening in public area showed 735 participants and the result showed
378 people were refractive error and 71 were cataract. Children eye center were established in Hasanuddin
University Hospital. The other program by HKI still on progress.
Conclusion : The sightfirst project and childhood Blindness program in our area gives big impact in eye care
services. The program also need to implemented to other districts in our province.
OPHCOM 3.3
BLINDNESS, WHAT SHOULD WE DO?
Hera Dwi Novita, MD
Medical Faculty of Brawijaya University, Malang, East Java
Blindness persistently becomes a big health problem in our country despite a finding from RISKESDAS that shows
a decreased rate of blindness. However, the data was even strengthened by RAAB blindness survey, (Rapid
Assesment Avoidable Blindness) which is still in progress, that the average range of blindness accounts for  2,5%.
From WHO : Universal eye Health : a global action plan 2014-2019 focuses on 3 goals which constitute : Evidence
based advocacy, Health system approach for strengthening comprehensive eye care, multisectoral engagement
and effective partnerships. Component health system consists of health information system, leadership and
governance, service delivery, essential medicines, human resource for health, health financing, slightly different
from Global action plan in the previous year which was disease control, infrastructure development and human
resource development but it is still a persistently continuous program. A question deserves to be a self question is
whether we have been sufficiently active to eliminate blindness through government program in accordance with
vision 2020? Where is our current position?. We can still also participate actively despite our small scope in our
local area. Then it comes to a question where our position is. There are still some hindrances encountered.
Some ophthalmological-related activities in Malang city comprise several fields: a) Eye Health Promotion; This
health promotion have been conducted in varying activities such as holding seminars of eye health for public
community, cooperating with radio stations to broadcast eye health, ,b)Human Resource development ; training
cataract screening addressed to Medical faculty students, basic ophthalmological health examination trainings
for Public Health Centre general practitioners and nurses, c)Screening ; cataract screening jointly collaborative
with SPBK Malang branch, refraction screening in elementary schools of Malang regency, screening diabetic
Retinopathy, d)Eye Health Education, ; eye health seminar for ophthalmologist, general practitioners and nurses as well
as refractionists, eye health seminar for public health center cadres, e) Referral system ; shortening referral system paths
for cataract medical case which health personnel already found in which they have got medical trainings and
the patients are directly registered to SPBK, waiting for surgery in voluntary activities for those who are in need,
f) multi sectoral coordination : collaborating with city and regency health Board routinely by holding medical
training for GP and nurses, cooperating with NGO in voluntary/social medical activities, which is, in turn, followed
by the next collaboration with education board to conduct screening of blindness in school-aged-children. And
g) Evidence base; participating in health survey such as RISKESDAS, RAAB health survey, reporting the number of
cataract surgery social activities held by SPBK and analyzing cataract outcomes, database of refraction cataract of
elementary school children.
Contribution is surely needed to find out our program. Furthermore, evaluation of each program needs to be
conducted to find out whether our programs run well or not. SWOT analysis (Strengths, Weakness, Opportunities,
and Threats) is used for assessment. It is said that if you know your enemies and know yourself, you can win a
hundred battles without a single loss. If you only know yourself, but not your opponent, you may win or lose. If you
know neither yourself nor your enemy, you will always endanger yourself. Together we can, eliminate Blindness.
OPHCOM 3.4
Cataract Training For General Doctor & Nurse At Public Health Center In Central
Lombok & Sumbawa Besar NTB
Sriana Wulansari, MD
Balai Kesehatan Mata Masyarakat NTB
Background :
Rapid Assesment Of Avoidable Blindness (RAAB) NTB in 2014 result is 4% . The majority cause of blindness is
cataract, about 78%. In order to decrease cataract blindness, we need to increase Cataract Surgical Rate (CSR).
CRS can increase if referal System from public health need a good knowledge of general doctors and nursse
about cataract at public health centers.
Purpose :
To improve knowledge general doctors & nurses about cataract.
Methode :
cataract training for 50 general doctosr 50 nurses in Central Lombok district and Sumbawa Besar district has
been done at September 2014. We compare proper diagnose at screening cataract in charity before and after
training.
Result :
In Sumbawa Besar accuracy proper diagnosis level increase from 40,3% to 80,4%.
In Central Lombok accuracy proper diagnosis level increase from 30% to 85,4%.
OPHCOM 3.5
BLINDNESS ERADICATION PROGRAM BY PERDAMI BALI
Ni Made Ari Suryathi, MD., Ariesanti T Handayani, MD
Cataract is still being the main cause of blindness in Bali. RAAB survey in Bali on 2015 showed the number of
blindness in Bali was 2,7% while 77% of blindness were caused by cataract. PERDAMI Bali has a responsibility of
blindness eradication in Bali and east part of Indonesia.Recently, collaboration between PERDAMI Bali and Bali
Government via Bali Mandara Eye Hospital and Non-Government Organization via The John Fawcett Foundation
through a program called “Pemberantasan Gangguan Penglihatan dan Kebutaan” or PGPK can increase the
Cataract Surgical Rate (CSR) in Bali from 915 in 2014 to 1485 in 2015. The Cataract surgery were performed at
entire Bali with Mobile Cataract Unit.
PERDAMI Bali also responsible for Blindness Eradication in other area of Bali, such as Nusa Tenggara Timur
(NTT). Since 2015, SeksiPemberantasanButaKatarak (SPBK) PERDAMI Bali and Non-Government Organization in
NTT called Besipae have already held a routine Cataract operation at Soe, NTT every three months.
The future goal of Blindness Eradication Program by PERDAMI BALI will emphasized of cataract case finding in
the level of Public Health Service, and other eye disease screening such as refractive error, diabetic retinopathy,
glaucoma and infection disease.
Key word: PERDAMI Bali, cataract, blindness
OS 2.2
CONJUNCTIVAL TUMORS REMOVAL
Trilaksana Nugroho, MD
Department of Ophthalmology, Faculty of Medicine Diponegoro University – dr. Kariadi General Hospital
Semarang
Conjunctival tumors have a wide clinical spectrum of conjunctival lesions, ranging of benign inflammatory
lesions to malignant-aggressive and life threatening diseases. Clinical differentiations among tumors were
distinguished by history and the typical clinical appearance of illnesses. Conjunctival tumors are generally
classified into congenital lesions, and acquired lesions obtained, where the acquired lesions were subdivided by
origin tissues. The conjunctiva is a thin-visible membrane structure, so that conjunctival tumors / lesions can be
detected easily. An accurate diagnosis can be confirmed by an external examination by slit lamp biomicroscopy.
Many conjunctival tumors can be removed by simple technique, but sometimes complicated. Understanding
of the anatomy of conjunctiva and related structures were required to establish the definitive diagnosis and
management of conjunctival tumors. This paper will discuss the clinical and histopathological appearance, and
management of the conjunctival tumors, especially surgical technique.
OS 2.3
ORBITAL DERMOID CYSTS
Ibrahim Sani, MD
Introduction
→ Orbital dermoid is a choristoma à histologically normal tissue at abnormal location
→ It is derived from displacement of ectoderm to a subcutaneous location along embryonic lines of closure
→ These dermoid cysts are usually isolated and unassociated with systemic diseases
→ Solid epibulbar dermoids are less common but are more important in visual stand point
→ They occur at corneal limbus, or lateral canthus, or extending subconjunctivally
→ Dermoid cysts are most common epithelial cysts accounting for 33% of cysts and nearly 50% of orbital
leisons of childhood
→ Usually occur at superotemporal orbit in relation to suture lines of orbital bones often with a bony defect
→ The cyst contents consists of keratin, sebaceous secretions & hairs that are grossly recognisable
→ Previous rupture of cyst lining leads to replacement of wall by giant cell foreign body granulomatous reaction
OS 2.4
HEMANGIOMA KAPILER ORBITA DAN KELOPAK
Neni Anggraini, MD
Departemen Medik Ilmu Kesehatan Mata Fakultas Kedokteran Universitas Indonesia
Hemangioma kapiler merupakan neoplasma jinak yang berasal dari pembuluh darah. Hemangiomakapiler
merupakan tumor orbita dan kelopak mata tersering pada anak-anak dan meningkat pada saat usia bayi.
Hemangioma kapiler mempunyai banyak nama lain seperti infantile hemangioma, juvenile hemangioma,
hemangioblastoma, benign hemangioendothelioma, dan hypertrophic hemangioma. Kejadian hemangioma
kapiler sekitar 10% pada bayi baru lahir dan anak-anak usia kurang dari 1 tahun. Pada tahun pertama kehidupan,
hemangioma kapiler berproliferasi cepat dan akan proliferasi lambat pada 5-7 tahun kemudian. Sekitar 30
persen dari seluruh kasus hemangioma kapiler pada orbita maupun kelopak mata, didiagnosis pada saat baru
lahir dan hampir semua kasus dapat didiagnosis pada enam bulan pertama kelahiran.
Gambaran klinis hemangioma kapiler berupa superfisial strawberry nevus, hemangioma subkutan dan orbital
dalam. Sebagian besar hemangioma kapiler dapat didiagnosis berdasarkan pemeriksaan fisik, namun untuk
diagnosis yang akurat dibutuhkan pemeriksaan ultrasonografi (USG), computed- tomography scanning (CT scan)
maupun magnetic resonance imaging (MRI). Komplikasi yang dapat terjadi pada hemangioma kapiler adalah
strabismus, ambliopia, dan atrofi optik.
Indikasi tatalaksana hemangioma kapiler meliputi beberapa indikasi, diantaranya indikasi visual meliputi oklusi
dari aksis visual, kompresi nervus optik, dan anisometropia ambliogenik. Sedangkan indikasi kulit meliputi
maserasi dan erosi pada epidermis yang disebabkan oleh hipertrofi berat pada epidermis dan jaringan subkutan.
Indikasi sistemik meliputi gejala obstruktif (nasofaring, oral, atau subglotis), hematologi (trombositopenia atau
anemia hemolitik), dan kardiovaskular (gagal jantung kongestif). Pilihan tatalaksana pada hemangioma kapiler
adalah observasi, oklusi vaskular, eksisi, radiasi, interferon, laser, kortikosteroid sistemik ataupun intralesi, dan
terapi propanolol.
OS 2.5
SEBACEOUS GLAND CARCINOMA VS CHALAZION
Purjanto Tepo Utomo, MD
Departemen Ophthalmologi FK UGM/ KSM Mata RSUP Dr. Sardjito Yogyakarta
Sebaceous Gland Carcinoma (SGC) adalah keganasan pada kelopak mata yang sering ditemukan pada orang Asia.
Insidensinya kira kira 27-‐40% dari seluruh keganasan pada kelopak mata, lebih sering ditemukan pada wanita
decade 6 -‐7 terutama di kelopak mata atas. SGC ini secara klinis kadang sulit dibedakan dengan chalazion,
mengingat asalnya yaitu dari glandula meibom. Akan tetapi terdapat beberapa perbedaan klinis diantara
keduanya yang bisa menjadi penanda perbedaan tersebut. Hal ini penting diketahui karena manajemen dari
kedua jenis kondisi ini berbeda.
POS PD 2.1
Congenital Cystic Eye
Julie Dewi Barliana, MD
Pediatric Ophthalmology Division, Department of Ophthalmology,
Faculty of Medicine Universitas Indonesia, Cipto Mangunkusumo Hospial Jakarta
A newborn baby girl, presented with left orbital mass. The orbital mass was seen since in uterine from 3D
ultrasound examination. The baby was born fullterm through sectio cesarean. There was no history of
consanguinity and same condition in the family. On examination, a large erythematous cystic mass was seen
in the left orbit, stretching the upper eyelid and bulging out of the palpebral fissure. No globe was identified.
The right eye was normal. Orbital CT scan was performed and revealed the large eyeball, extended to the
superior orbital fissure and involved the optic nerve. The diagnosis was congenital cystic eye. Brain MRI was
also planned to perform to see whether there is brain malformation and/or intracranial communication of the
cystic eye. But until now, the brain MRI was not already performed. The type of surgey is still questionable
because the technique to enucleate the eyeball is not simple and easy.
Keywords: congenital cystic eye
POS PD 2.3
IFAP (ICHTHYOSIS FOLLICULARIS, ALOPECIA, AND PHOTOPHOBIA) SYNDROME
Primawita Oktarima, MD
Introduction:
The IFAP (ichthyosis follicularis, alopecia, dan photophobia) syndrome is a rare congenital ectodermal disorder
characterized by the triad of follicular ichthyosis, alopecia, and photophobia.
Purpose:
To report a rare case of 13 years old boy with IFAP syndrome suspect with congenital cataract as an
additional manifestation and its management.
Case report:
A 13 years old boy patient came to Pediatric Ophthalmology and Strabismus Unit Cicendo Eye Hospital
with the chief complaint of blurred vision in both eyes. Clinical findings revealed alopecia, ichthyosis
follicularis, and photophobia. From ophthalmologic examination, his visual acuity was close face
counting finger (CFFC), nystagmus, corneal scar with neovascularization, and bilateral congenital
cataract. Other dermatologic finding was onychodystrophy. Based on the clinical findings, a diagnosis of
IFAP syndrome suspect, bilateral congenital catract, and deprivation amblyopia was made. We
performed cataract extraction with intraocular lens implantation in the right eye followed by the left eye.
Conclusion:
Congenital cataract could be an additional feature of ocular manifestation in the IFAP syndrome. The
pathogenesis of this IFAP syndrome still remains obscure and awaits further clarification as more cases
are accumulated with the understanding of the pathogenesis of this disorder, to look for other treatment
possibilities.
Keyword: alopecia, ichthyosis follicularis, photophobia, corneal scar, cataract, nystagmus,

onycodystrophy
POS PD 2.4
Optic disc mass in a patient with acute lymphoblastic leukemia: leukemic infiltra-
tion or primary tumor?
Sutyawan, Eka I W, MD
Opthalmology Department of Udayana University, Denpasar, Bali
Introduction and Objective: About 90% of cases with optic nerve involvement occur in patients with acute
forms of leukemia. Leukemic cells may also infiltrate the optic disc to form circumscribed edematous mass,
white elevated lesion associated with yellow deposits and peripapillary hemorrhage. The differential diagnosis
of optic disc edema in patients with leukemia includes leukemic infiltration of the CNS with secondary increased
intracranial pressure, tumor infiltration resulting in ischemic papilitis and perivascular tumor infiltration leading
to venous engorgement. Early, aggressive intratechal chemotheraphy and radiotheraphy is the most effective
treatment of incipient optic nerve head infiltration by leukemia.
Case Description: Five years old boy with acute lymphoblastic leukemia (ALL) standar risk, was on the 58th week
therapy phase maintenance of ALL (6-Mercaptopurine) and observation cephalgia. Pediatric department want to
know there was any possibility of an intracranial process. Right eye visual acuity was light perception, restricted
movement to temporal, superotemporal and inferotemporal. Right eye optic nerve head was blurry edge, with
the cup disc ratio difficult to be evaluated; artery and vein was irregular and turtous; there were blots, dots and
flame shape bleeding on retina. Ultrasonography (USG ) examination of right eye found an intraocular mass on
optic disc, with no calcification and shadowing effect. There was a solid mass posterior intraocular on Magnetic
resonance imaging (MRI), no invasion and suspect metastase process with differential diagnosis retinoblastoma.
After patient have done chemotherapy intrathecal three cycles, patient didn’t come to control.
Keywords: acute lymphoblastic leukemia, leukemia infiltration, optic disc mass
POS PD 2.5
Macular Edema in Children
Yulinda I Soemiatno, MD
Klinik Mata Nusantara, Jakarta
To report a case of an 11 year- old boy who had complained of deterioration in vision since 5 years ago. He was seen
by an ophthalmologist elsewhere and referred for having macular edema. Vision on the right and left eyes were
0.3 and 0.1, respectively. Complete ophthalmologic examination and optical coherence tomography were done.
Funduscopy showed bilateral cyst-like structures at the macula developing a cartwheel pattern. Average central
macular thickness were 516 micrometer and 576 micrometer, for right and left eyes, respectively.
Cystic changes in the macula may be due to a variety of causes. Macular edema is often owing to inflammation,
retinal vein occlusion, diabetic retinopathy, and even dominantly inherited cystoid macular edema. With the
characteristic sign of cartwheel pattern, x-linked retinoschisis is most likely to be the cause of the macular
change in this patient, however, further examination such as electroretinography and genetic testing will help
confirm the diagnosis.
Keywords: macula, edema, cyst, x-linked retinoschisis
POS PD 2.6
Anterior lens luxation in homocystinuria. A case report
Liana Ekowati,MD
A 6 year-old girl had anterior lens luxated in left eye and subluxated lens in right eye. She had developmental delay
and mental retardation. Biochemical study detected a profile of increase plasma homocysteine levels consistent
with suspected homocystinuria. Episodes of pupillary block glaucoma prompted lens extraction in left eye. The
luxated lens was extracted and scleral fixation IOL implanted under general anaesthesia. A good result without
serious complications was obtained. Visual acuity improved from 1/60 to 6/20 with Lea symbol’s. She was
started on treatment with vitamin B6, vitamin B12 and folic acid. A good response was noted after treatment.
Homocystinuria is a metabolic disease usually presenting at an early age as vascular, skeletal and neuropsychiatric
abnormalities, as well as ectopia lentis. Our case is atypical because of the absence of thromboembolism. It is
necessary to rule out homocystinuria in patients with ectopia lentis, even the absence of systemic symptoms.
POS S2.3
Management of sixth nerve palsy
luki Indriaswati, MD
Department of Ophthalmology Faculty of Medicine Airlangga University/ Dr. Soetomo General Hospital Sura-
baya
Sixth nerve palsy is one of the most common oculomotor nerve palsy, manifested by limited abduction of the
affected eye and diplopia. Management of sixth nerve palsy and paresis remains challenging. Therapeutic
options are varied depend on the etiology and degree of neuromuscular dysfunction.. The most crucial step is
to identify the causative factor which is different according to the age groups, and treat the underlying disease.
The predominant etiology in children either post viral or tumor and trauma. In young adults the causative factor
is most difficult to assess. Elderly patients have microvascular diseases frequently. Excluding increased intra
cranial pressure and papilledema is essential. Treatment of sixth nerve palsy may be conservative or surgical.
It is generally considered a waiting period of at least 6 months after the onset of paralysis for a spontaneous
recovery. If this condition is not achieved, surgery is indicated, depending on the residual function of the
neuromuscular complex. Recession and resection technique is procedure of choice for sixth nerve paresis.
Transposition techniques is recommended if sixth nerve paralysis is found. The rate of reoperation is not high
when treatment is appropriately selected according to clinical condition. Conservative or surgical treatment
of paralysis itself is not designed to recover lost function but to improving symptoms, eliminating diplopia in
primary position and recovery of binocular visual field. It’s important to highlight the role of a careful imaging
and a good interdisciplinary collaboration in management of this paralysis. Key words: sixth nerve, parese,
paralysis, conservative , surgical
POS VS 2.4
LEARNING STRABISMUS SURGERY
Lely Retno Wulandari, MD
Department of Ophthalmology Medical Facultyy Brawijayya University
It is said that the only surgeons who do not have complications are those who do not operate and those that
lie about having no complications. The surgical experience must feature a progressive development of surgical
practice but the individual learning abilities vary a lot. All surgeries carry risks of complications, and there is no
way to avoid ever having one. Strabismus surgery is no different in this regard. Strabismus surgery is typically
recommended when a patient’s eye alignment can no longer be treated with conservative measures such as
eyeglasses, eye patching, prisms, and orthoptic exercises. Like many other ophthalmic procedures, strabismus
surgery is very safe and effective, but difficulties and complications can occur and need to be prevented to
optimize post-operative outcome. There are methods to reduce the risk of a complication during or after
surgery, and these steps should always be taken. When a complication occurs, it is important to first recognize
it and then manage it appropriately to allow for the best outcome possible. This video will discuss some of the
more common and/or most devastating complications that can occur during or after strabismus surgery as well
as thoughts on how to avoid them and manage them should they happen.
POS VS 2.6
Management of Ectopia Lentis in Marfan Syndrome
Florence M. Manurung, MD., Tjahjono D. Gondhowiardjo, MD
Marfan syndrome (MFS) is an autosomal dominant connective tissue disorder involving the cardiovascular,
skeletal, and ocular systems. Ophthalmic problems are often the presenting symptom in childhood and can
seriously affect vision. The characteristic opthalmic problems are bilateral lens subluxation, cataract, myopia,
retinal detachments, and early onset glaucoma. MFS is the most frequent cause of heritable ectopia lentis.
Classically, the direction of lens dislocation occurs in a bilateral, symmetrical, and superior temporal pattern.
Severe dislocation requires surgical intervention in order to avoid amblyopia.
POS VS 2.8
Improvement in patient›s quality of life following small angle
esotropia surgery: focusing on psychological impact
Rozalina Loebis, MD
Pediatric Ophthalmology and Strabismus Division Ophthalmology Department Airlangga University
Dr Soetomo General Hospital Surabaya
Purpose: To report a case of small angle sixth nerve palsy caused by embolization after carotid cavernous fistula,
focusing on the psychosocial impact of the patient.
Methods: A case report
Results:
A man came to our strabismus clinic with the complaining of diplopia and crossed eye that made him jobless.
This complaints began since he had traffic accident 1 year ago and made him suffered from carotid cavernous
fistula. Embolization was done immediately but diplopia and crossed eye after embolization remains.
Examination reveals limitation on the right gaze (around -1) Hirschberg test esotropia < 15 degrees and prism
cover test 10 prism diopter. Saccadic movement was floating and Force generation was positive suggesting sixth
nerve paretic on right eye. Prism glasses was tried but failed because patient still diplopia at work.
Recession of right medial rectus 4 mm was performed on the right eye in adjustable suture. After underwent this
surgical procedure, the patient achieve satisfying results. There is no diplopia and the eye become orthophoric.
The important result is the good impact of his psychosocial life because he gained his job back
Conclusion:
Recession medial rectus surgery seems to be a suitable procedure for the treatment of abduscen nerve
paresis. Since the force of the medial rectus muscle is still good, this surgical technique is preferred compare to
transposition procedure. Even the deviation is small (only 10 prism diopter), surgical approach was done to give
patient ability to gain his job as surveyor in civil engineering field.
Keyword: Sixth Nerve Palsy , Embolization , Carotid Cavernous Fistula, Psychosocial Impact
REF S 3.1.1
Accommodation and it’s problem
Ani Ismail, MD
Accommodation is the process by which the eye changes optical power to maintain a clear image or focus on an
object as its distance varies. Distances vary for individuals from the far point - the maximum distance from the
eye for which a clear image of an object can be seen, to the near point -the minimum distance from the eye for
which a clear image of an object can be seen. Accommodation occurs when the ciliary muscle contracts, causing
tension of the choroid and posterior zonula resulting into a convex lens.
There are several theories mechanisms of accommodation: Helmholtz theory, Ticherig theory and Thsemig
theory. Duke-Elder classified a number of accommodative dysfunctions which are Accommodative insufficiency,
Accommodative Excess and Accommodative infacility. There are also disorder associated with age accommodation
is presbyopia, in which symptoms appear upon entering the age of 40 years in which the patient could not see
clearly at close distances. To help people with presbyopia accommodation power, it can be used the positive
lens power to increase the strength of lens in accordance with age.
REF S 3.1.5
Vision Improvement of High Myopic Patients wearing RGP Contact Lenses
Lucia Sutedja, MD
Purpose: to report the Visual Acuity (VA) and the improvement long term VA of wearing RGP on high myopic
patients
Methods: Case report of 4 cases of high myopic patients wearing RGP Cls. The Patients were performed
comprehensive eye examination on each visit; they were free from any other ocular diseases at the time.
Result:
Case 1: a 34 year-man has visited the clinic at 6th may 2000, VOD wearing RGP (-12.00 D) was 6/12, VOS wearing
RGP (-13.00) was 6/12. After refitting the VA could be maintained 6/6. At present VOD wearing RGP (-22.00 D) is
6/6; VOS wearing RGP (-23.50 D) is 6/6. The RGP powers changes were OD: -13.00 D, -14.00 D, -17.00 D, -19.00
D, -22.00 D respectively, and OS: -15.00 D, -17.00 D, -18.50 D, - 19.50 D, -23.50 D.
Case 2: a 45 year-woman has visited the clinic at 26th February 2011, for the first time fitting RGP. VOD wearing
RGP (-23.00 D) was 6/15; VOS wearing RGP (-19.00D) was 6/12. The next month follow up, VOD wearing RGP
was 6/9, VOS wearing RGP was 6/9, then VOD gained 6/7.5; 6/6 respectively. VOS wearing RGP was 6/9, 6/6
respectively.
Case 3: a 24 year-woman has visited the clinic at 15th January 2002, VOD wearing
RGP (-16.00D) was 6/6, VOS wearing RGP (-15.50D) was 6/6. On the follow up RGP OD changed to -17.00 D,
-18.00D, -18.50 D at present respectively, RGP OS is still -15.50 till present.
Case 4: a 29 year-woman has visited the clinic at 4th September 2004; VOD wearing RGP (-12.00 D) gained 6/6,
VOS wearing RGP (-13.00) gained 6/6. VODS could be maintained 6/6, but OD RGP were changed to -13.25 D,
-16.50 D -17.00 D, -18.00, 19.00 D at present respectively. OS RGP were changed to -15 D, - 17.75D, - 18.00, -
19.50 at present respectively.
Conclusion: High Myopic Patients will have VA improvement if it is manage properly, also to slow myopia
progression.
Key words: high myopia, RGP contact lenses, Visual acuity
REF S 3.1.6
Contact Lens in Children (problems & solve)
Chirstina, MD
Contact lens in children has modalities for visual acuity or clearance of vision, because spectacles maybe
cosmetically unappealing, uncomfortable to wear, visually disturbing and too easy to remove. It is important
things that influence parents decision to fit child in contact lens. Many factors that can make doctor giving
prescribed contact lenses for children ≥ 7 years old maybe capable of handling the responsibility of contact lens
wear and can be fitted using techniques similar to what would be performed on adults. Disposible contact lens
for children ≤ 12 years, meanwhile reusable contact lens for children 13–17 years old.
It is important to examine the children with contact lens routinely every week in the first 1-2 months of contact
lens wear, and then every 1-3 months until the child reaches school age. The power should be reduced to
correct emmetropia as the child begins to walk and requires greater viewing distances. Smaller diameter contact
lens for infants. The size could be similar like adult when children 2 years old and continue until 8 years old. Soft
contact lens use daily wear to reduce neovascular or infection when become a toddler.
Rigid Gas Permeable (RGP) contact lens for aphakia with lenticular design are use for reducing contact lens
thickness, reducing center of gravity, and improve lid attachment. RGP to reduce the progression of myopia
have many tips for fitting such are use topical anesthetic placed in the contact lens prior to insertion, dispensing
visits, having fixate on a target, firmly holding the eyelids open, and quick insertion contact lens. Most of the
children can be fitted with power 9.2 Dioptre and optic zone diameter 7.8 mm. Girls tipically more successfull in
RGP wear than boys because they are more mature in similar age and they can insert, remove and care without
assistance from the parents.
Keywords contact lens, soft contact lens, Rigid Gas Permeable contact lens
REF S 3.2.1
Prescribing Bifocal or Multifocal?
Fatimah Dyah, MD
Refraction, Contact Lens and Low Vision Sub Division.
Kariadi General Hospital, Diponegoro University.
Bifocal lens defined as a lens that consist of two power zone. The primary lens provide the distance power
zone and the segment provide near power zone. All bifocal lens provide a large distance viewing zone with
homogenous power and most also provide large near-viewing zone with homogenous power. For good visual
outcome, bifocal lens must be properly fitted within the frame and the frame must be properly fitted to the face.
Multifocal lenses defines as a lens that provide distance power zone, intermediate power zone and near power
zone. One type multifocal that used mostly now a days is Progressive addition lenses (PALs). PALs provide
continuous change from distance through intermediate to near, provide the wearer with seamless visual space
and eliminates unusual view of visual area caused by top line of bifocal segment. PALs also have visual and
cosmetic advantages compared to segmented multifocal. In another study showed a strong preference for PALs
; 265 habitual bifocal wearer were fitted with PALs, 92% of these patients preferred to PALs.
REF S 3.2.2
Simple Visual Acuity Assessment in Infant and Children
Nanda W Anandita, MD
Department of Ophthalmology Faculty of Medicine, Brawijaya University Malang
Visual acuity measurement in Infant and Children are important for early detection of ocular disorder in infant
and children to avoid lifelong visual impairment. Measurement visual acuity in infant and children is an art,
requiring some techniques that is different from adult’s and interpretation of the child’s visual acuity within the
global evaluation of the child. There are many guidelines for measurement the visual acuity in them which was
obtained by surveys of practitioners and are based upon experience and acquired many years. In age of 3-12
moths this is phase of fast emmetropisation. The most important before examining the visual acuity is knowing
about the history of ocular problems and past illness that maybe cause the visual problem. At the 3-6 years
old there is less changes. Children who starting to walk typically has working distance acuity 1-2 m. Children in
kindergarten do most things at school at desk. Children in the school years emmetropisation is complete at 6
years old. The visual demand is look like adult. Children are not simply little adult. There is a unique needs based
upon their visual demands and their developing visual system.
REF S 3.2.3
How to create comfort vision with astigmatism correction
Ria Sandy Deneska, MD
Ophthalmology Dept, Medical Faculty, Universitas Airlangga
Some patients not tolerate the full astigmatic correction. Some ophthalmologists stated that full correction of
a high astigmatic error may initially result in considerable blurring of vision, while others said that with the full
astigmatic correction the image is too sharp and the patient is not used to seeing so clearly. Statements such as
these are not only misleading; they are incorrect.
The cause of intolerance of astigmatic spectacle corrections is distortion caused by meridional magnification.
Unequal magnification of the retinal image in the various meridians produces monocular distortion manifested
by tilting lines or altered shapes of objects. But monocular distortion by itself is rarely a problem; the effect is
too small. The clinically significant problem occurs only under binocular conditions.
There are a lot of things we have to understand how the distortion occurred. And there are some procedure we
can perform to minimize these distortion and create more comfort visions for our patients.
REF S 3.2.4
Fitting and choose appropriate contact lens type in baby and toddler
Tri Rahayu, MD
Department of Ophthalmology FMUI-RSCM Kirana, and Jakarta Eye Center
Infant and toddler visual development requires constant and appropriate correction of refractive error so as to
maximize their visual potential and binocularity. Contact lenses play an important role in correcting infant’s and
toddler’s vision, as contact lenses give less cosmetically unappealing, more comfortable to wear, less visually
disturbing, and hard for a young child to remove, compare to spectacles.
Infants and toddlers who consistently remove their glasses and need constant visual correction can benefit from
contact lens wear and can be fitted with that. Contact lenses give benefit for infants and toddlers for a variety of
reasons ranging from correction of refractive error to vision therapy.
Fitting contact lenses on baby is easily performed in the operating theatre under general anaesthesia. However,
it also could be performed in clinical setting with patient, carefull, and quick approache. Fitting contact lenses
on toddler is the most challenging procedure for us. The doctor who fit a contact lens for a toddler must be very
adept at several methods since there are no universal procedures appropriates for it. If fitting procedures in
outpatient clinic room is unsuccessful, a fitting under general anaesthesia should be considered.
There are two main category of contact lenses available for infant and toddler. Soft contact lenses are initially
more comfortable, require less time and effort to fit, and can be dispenses at the fitting visit. They are available
in standard daily wear, frequent replacement, daily, weekly, 2 week, and monthly disposable. Despite its
advantages soft contact lens have a potential risks that should be anticipated.
Rigid gas permeable (RGP) contact lenses may provide clearer vision than soft contact lenses, especially for
correcting astigmatism caused by corneal toricity.There are many tips in RGP contact lense fitting to make the
process easier.
REF S 3.2.5
Soft Contact Lenses Or Rigid Gas Permeable Contact Lenses - How Do We Choose ?
Widjajanti U, MD
Below is a brief comparison of soft and rigid gas permeable (RGP) contact lenses. A thorough eye examination
and a better understanding of your specific vision requirements will help your doctor of optometry determine
the best options for you. Soft contact lenses are composed of malleable plastic polymers. They are very flexible
and, when fit properly, will form to the cornea (the front surface of the eye). They are the most common type of
contact lenses worn. Rigid Gas Permeable (RGP) contact lenses have been around since the 1960s. Newer RGP
contact lenses offer the advantage of allowing more oxygen to pass through to the eye. They are often referred
to as oxygen permeable contact lenses and are available in daily wear and extended wear options.
Both types of contact lenses are very easy to use and handle. The two things to avoid are: wearing contact
lenses whilst sleeping and wearing the lenses more than the recommended schedule. If you follow the proper
procedure and care for the contact lenses, it is unlikely that you will have any problems with infection.
REF S 3.2.7
Role of Ophthalmologists & Optometrists in low vision services.
Rajiv Khandekar, MD
Eye care comprises preventive, curative and rehabilitative services. Unfortunately, less attention is paid to visual
rehabilitation while training eye care professionals. This presentation, highlights the role and possible contribution
of ophthalmologists and optometrists in rehabilitating vision. Some pearls for rehabilitation include; defective
vision and low vision disability are different; distance visual acuity is just one component of visual function and
other components are required for daily living activities. The phrase ‘Nothing further can be done to improve
vision’ should never be used. A disabled individual can use residual vision with low vision devices and alterations
in environment to enhance his/her working capabilities. Low vision rehabilitation requires a team effort. Both
patients and care providers have crucial and specific roles. Simple low cost interventions such as head gear (like a
hat) and adjusting illumination can be effective. Eye care professionals can counsel the disabled and their relatives
to actively seek out low vision practitioners to enhance their quality of life. This presentation will encourage
ophthalmologists and optometrist to work as team members in offering low vision services.
RET S 2.1.6
Approach to Chronic Central Serous Chorioretinopathy
Angela Nurini Agni, MD
Central Serous Chorioretinopathy (CSCR) is a disorder in the posterior pole retina with a characteristic sign of
neurosensory retinal detachment (SDR) due to serous fluid accumulation between the photoreceptor and the
retinal pigment epithelium (RPE). The distinction between acute and chronic CSCR is considered by its temporal
course ( 3-6 months duration from the onset of the disease) and by the characteristic widespread abnormality
of the RPE. Chronic CSCR comprises about 5 % of CSCR. Unlike acute CSCR which occurs predominantly in
young healthy males, the chronic variant of CSCR occurs in older people, and affecting more women than the
acute one, with a worse visual prognosis. While in the acute CSCR most of the serous neurosensory detachment
can be resolved spontaneously, the chronic CSCR needs more active management to resolve the SDR. The
management of chronic CSCR includes Conventional laser photocoagulation, Subtreshold diode micropulse
Laser, Photodynamic therapy, and medicamentous treatment such as intra vitreal injection of anti VEGF agent,
antagonist of mineralocorticoid reseptor, etc. However, only PDT is supported by the good quality of evidences
( with reduced dose and reduced laser fluency to resolve SDR with minimal side effects) at the moment. It can
be concluded that PDT is the best available treatment for Chronic CSCR nowadays.
Keywords: Chronic CSCR, photodynamic therapy, Micropluse subthreshold diode laser, Anti VEGF, anti
mineralocorticoid receptor.
RET S 2.3.5
Future Pharmacotherapies for Diabetic Retinopathy
Habibah S. Muhiddin, MD
Laser photocoagulation is still a gold standard in diabetic retinopathy treatment, however it is often associated with
visual field reduction and other ocular side effect. Currently, many researchers have been performed to introduce
some adjunctive treatments to achieve better result and visual acuity improvement. Based on pathogenesis of
diabetic retinopathy, some medical treatments were applied as adjunctive treatment such as anti VEGF intra-
vitreal, fenofibrate and ACE inhibitors. These kinds of treatments showed some benefits in diabetic retinopathy
treatment, especially in the early phase.
ROO DC 1.1
PATHOPHYSIOLOGY OF ORBITAL INFLAMMATIONS
Karla Chaloupka, MD
The orbit presents with a big variety of different pathomechanisms in inflammatory disease ranging from
the endocrine orbitopathy to idiopathic inflammations and from localized disease to systemic response.
The presentation discusses also the systemic connections which are very distinct compared to the ocular
inflammatory disease.
ROO DC 1.2
Penatalaksanaan Non Specific Orbital Inflammation (Pseudotumor)
Rossalyn Sandra Andrisa,,MD
Divisi Tumor, Departemen Ilmu Kesehatan Mata, Fakultas Kedokteran Universitas Indonesia- RSCM Kirana
Non Specific Orbital Inflammation (NSOI) merupakan kondisi inflamasi pada orbit yang disebut juga sebagai
kondisi pseudotumor orbita. NSOI dapat digambarkan sebagai kondisi inflamasi non malignant pada orbit yang
dikarakteristikan dengan adanya infiltrat limfoid polimorf dengan variasi derajat fibrosis, tanpa diketahui penyebab
lokal maupun sistemik. Kondisi ini terjadi paling sering pada dewasa dengan puncak insiden terjadi antara usia
40 dan 60 tahun. Tanda dan gejala klinis NSOI meliputi nyeri, protopsis, kemosis, dan hambatan gerakan otot
ekstraokular. NSOI biasanya terjadi secara unilateral dengan insidensi terbesar bilateral pada anak-anak.
Diagnosis dapat ditegakkan melalui kombinasi antara anamnesis, pemeriksaan fisik, dan gambaran pencitraan
orbita. Hampir semua kondisi NSOI dapat diterapi dengan kortikosteroid, tetapi terapi tambahan imunosupresi,
radiasi lokal dan bedah dapat dipertimbangkan. Tujuan didactic course ini adalah agar peserta course memiliki
pemahaman yang baik tentang NSOI, baik dari segi penegakan diagnosis hingga penatalaksanaannya.
ROO DC 1.3
Pseudotumor and it’s management
Karla Chaloupka, MD
The unspecific orbital inflammation covers a big pool of still undiagnosed diseases. Newer entities such as IgG4-
related inflammations emerge thanks to better imaging, safe surgical approach and advanced histopathology,
allowing targeted therapy. The presentation gives an approach to diagnostics and an overview on treatment
options.
ROO S 2.1
PRIMARY CLOSURE OF EYELID LACERATION
Rodiah Rahmawaty Lubis, MD
Recostructive, Oculoplasty and Oncology Division Ophthalmology Departement, Madical Faculty
University of Sumatera utara, Medan
Laceration to the eyelid requires a thoughtful, well-planned approach in order to provide the best outcome and
reduce the chances of postoperative complications. The surgical repair of eyelid lacerations differs somewhat
from that of most skin lacerations. The reasons for this include the need to maintain a functional eyelid and
ensure a cosmetic outcome as well as the fact that the eyelid is more vascularized than many other skin regions.
A functional upper eyelid is essential to maintain the health of the globe itself. If the eyelid is incomplete or
immobile, it is unable to physically protect the eye and is unable to spread the tear film over the ocular surface.
Proper management of eyelid lacerations requires thorough knowledge of the anatomy of the eyelids and
periorbital structures, careful examination for associated ocular injury, and prompt referral to a subspecialist
when complicated lacerations are encountered.
Keywords: Primary Closure, eyelid laceration, functional eyelid
ROO S 2.3
DIAGNOSIS AND MANAGEMENT OF TEARING EYE
Ni Made Laksmi Utari, MD
Bagian Ilmu Kesehatan Mata, Fakultas Kedokteran Universitas Udayana/RSUP Sanglah Denpasar Bali
Tearing eye or epiphora is the most common reported symptoms of any ocular pathology and sometimes requires
referral to specialist opthalmic units. A stable tear film is vital for maintaining optical quality and function of
the eye. Epiphora can develop at any age. As life expectancy increases, according to demographic trends in
many countries, the epiphora prevalence will continue to increase and significantly affect quality of life. This
condition is due to a disruption in the balance between tear production and tear loss. Numerous etiologies lead
to an excess of tears, and there are a number of ways to diagnose and manage it. It is important to distinguish
between terms epiphora and pseudoepiphora or hyperlacrimation. Epiphora can be due to functional or
anatomical obstruction in lacrimal pathway. The goal of evaluation of patient with epiphora is to differentiate it
and to find out the cause. The evaluation can be divided into detailed clinical history, local examination of the
adnexal structure, ancillary testing and nasal evaluation. Ancillary investigation like dacryocystography, lacrimal
scintigraphy and imaging are required to determine cause of epiphora. Management of epiphora depend on all
the causes. Successful management of tearing patient requires the clinician to determine the underlying cause
of the epiphora, type and level of blockage in cases of anatomical obstruction, but this can be difficult because
the causes are often multifactorial.
Keyword: tearing eye, epiphora, lacrimal obstruction
ROO S 2.5
Management of involutional entropion
Hernawita S., MD
Involutional entropion usually occurs in the lower eyelid and in old patient. The factors that can cause involutional
entropion are horizontal laxity, disinsertion of lower eyelid retractors and overriding of the preseptal orbicularis
muscle over the pretarsal orbicularis muscle.
Horizontal laxity can be detected by snap back test and distraction test. Disinsertion of lower eyelid retractors
allows inferior border of the tarsus to ride forward and superiorly with the eyelid margin rotating inward.
Surgical procedure is needed to repair involutional entropion. The surgery consist of tightening the lower eyelid
and reattach disinsertion of the lower eyelid retractors.
There are many surgical procedures available to repair involutional entropion. In this presentation i will share
my approach on how to repair involutional entropion.
SRET S 2.2.1
Macular Holes: Peeling and Positioning
Sjakon G Tahija, MD
Klinik Mata Nusantara Jakarta, Indonesia
There have been many different techniques used for macular hole surgery. Originally only vitreous detachment
was done without I LM peeling and strict positioning was carried out for at least 2 weeks using a long acting gas.
Macular hole surgery has now shifted to nearly everyone using I LM peeling or folding techniques, and short
acting gas and shorter positioning times.
I now fold I LM in all large, long standing, and myopic macular holes and use SF6 exclusively with positioning
for a maximum of 5 days with an emphasis on the first 24 hours. I will share these techniques and my personal
experience in managing macular holes.
SRET S 2.2.3
Scleral Buckling Pearls and Pitfalls
Djonggi Panggabean, MD
Scleral buckling is an operative procedure for the treatment of rhegmatogenous retinal detachment, a condition
where the neurosensoric layer becomes separated from the RPE (retinal pigment epithelium) layer as a result of
fluid accumulating behind the retina due to a retinal break.
The purpose of the operation is to seal the retinal break, remove the subretinal fluid, loosen any traction on the
retina, thus reattaching the retina back to the RPE layer.
This presentation discusses the steps of the scleral buckling procedure, what is best to do and to avoid in various
conditions.
SRET S 2.2.5
Role of Anti VEGF in Vitreoretinal Surgery
Andi Arus Victor, MD
The recent years we have seen development of anti VEGF in the management of retinal vascular disease. Anti
VEGF agents have been used in preventing retinal neovascularization also resolving macula edema by improving
retinal vascular permeability. Due to its effect, Anti VEGF is being used as an additive treatment in vitreoretinal
surgery.
Several studies have been reported that Anti VEGF agents’ adjunct to pars plana vitrectomy in Diabetic
Retinopathy can achieve a better result compared to those without anti VEGF. Preoperative treatment with
anti VEGF agents causes in rapid regression of active neovascularization and lower the risk of intraoperative
hemorrhage. Most studies frequently used between 3 and 20 days before surgery. Other study suggests that
combination between intravitreal anti VEGF and pars plana vitrectomy in diabetes retinopathy should be done
to restrict VEGF development in vitreous so the complication can be prevented.
The most feared complication is glaucoma neovascular. Delayed in diagnosis and treatment can result to
complete vision loss. The application anti VEGF agent to treat glaucoma neovascular has been observed to cause
regression of new vessels in anterior chamber within a few days. This will lead to reduce intraocular pressure.
As the second most common retinal vascular disease, retinal vein occlusion has a major impact to the patient.
Complication includes vitreous haemorrhage, macula edema, and vascular ischemic will lead to blindness. Anti
VEGF shown to resolve the macula edema complication.
Eales disease is defined by perivascular sheathing, peripheral retinal nonperfusion, and neovascularization
of disc and retina. Main therapy of this condition includes laser photocoagulation or cryotherapy to remove
ischemic retina. Anti VEGF has emerged the promising therapeutic strategy to regress the neovascularization
within 2 weeks of treatment. It will allow laser photocoagulation to remove the ischemic retina.
RET-MP 2.1
OPHTHALMIC SURGERY AND ANTI THROMBOTIC AGENT: FRIEND OR FOE
Susilo Chandra, MD, FRCA (Anesthesiologist)
Patient safety is the absence of preventable harm to a patient during the process of health care. The discipline
of patient safety is the coordinated efforts to prevent harm, caused by the process of health care itself, from
occurring to patients. In eye surgery patients, issue of patient safety become more complex, because of patients
often elderly with significant co-morbidity, may be on antithrombotic agents such as aspirin, anticoagulant,
antiplatelet, DOA (direct oral anticoagulant) and others. Antithrombotic withdrawal predisposes to risk of
thromboembolic events. Antithrombotic continuation predisposes to risk of bleeding during surgery. This
mandates an awareness of these agents, allowing optimal patient management. We review traditional and
newer agents in the context of cataract, vitreoretinal, glaucoma and oculoplastic surgery. Recommendations
are given for continuation, cessation and re-commencement of these agents in order to minimize the risk of
bleeding and thrombotic/ thromboembolic complications.
EXHIBITORS
LIST OF EXHIBITORS
Booth # Exhibitor Address
PT CENDO PHARMCEUTICAL INDUSTRIES Jl. Cicendo no 25,Bandung, Jawa Barat.Indonesia

Titan Center, 7th Floor
Jl. Boulevard Bintaro Jaya Sektor 7
Tangerang 15224, Indonesia
1 PT FERRON PAR PHARMACUITICALS
Ph: 021 7454 333, 021-30433 333
Fax: 021 7453 939
www.Ferron-pharma.com
Buaran Regency Blok C no 21-22
Jakarta 13450
1A PT ENAM WARNA INDONESIA
Ph: 021 8690 3921 Fax: 021 8690 3922
Email: enam@indo.net.id
Kompleks Fatmawati Mas
Blok III Kav. 316
2,3 PT MEGA MEDICAL ABADI Jl Tarogong Raya, Jakarta
Ph: 021 7591 1489
www.mega-medical-abadi.indonetwork.co.id
3A PERMATA BOOK
Jl. Kyai Caringin no. 29—31
3B PT GAUDIUM VITAE Jakarta Pusat.
Jl. Terusan Arjuna Utara no. 1

Kedoya, Jakarta Barat
4,9,10 JAKARTA EYE CENTER
Ph: 021 2922 1000,
Email: jec@jakarta-eye-center.com
Komplek Griya KemayoranJalan Industri 9-11 Blok
R/41
5,6,7,8 PT TRIASTRI MEDITAMA
Jakarta10720Ph: 021 626 0637, 021 626 0638Fax:
021 626 0638
11, 12, AXA Tower Lt 25Jl. Prof. dr. Satrio Kav 18, JakartaPh:
ALCON PHARMACEUTICALS, LTD
13, 14 021 3048 0600
Jl. Bukit Duri Utara no 31,Jakarta SelatanPh: 021 830
15,16 PT AFINA SINAR CEMERLANG
2119 Fax: 021 831 9274
MGK Kemayoran Kantor Blok A-7
Jl. Angkasa Kav B-6, kota baru,
17,18 PT OPHTHALINDO JAYA
Bandar Kemayoran, Jakarta Pusat
Ph: 021 266 45184 Fax: 021 266 45185
Mega Glodok Kemayoran lt. 1
Blok A3 no. 1 Gunung Sahari Selatan,
19,20 PT TERAMED INDONESIA
Kemayoran Jakarta 10610
Ph: 021 4971 4442 Fax: 021 4980 0208
Jl. Pulo Gadung No. 5
21 PT SOHO GLOBAL MEDIKA Kawasan Industri Pulo Gadung, Jakarta
Ph: 021 460 5550 Fax: 021 461 0474
Jl. Letjend. Suprapto no 60 unit 6-H,JakartaPh: 021
22,23 PT SURGIKA ALKESINDO
425 3634 Fax: 021 425 3635
Jl. Mayor Hasibuan
Ruko Sun City Square E27
Bekasi, JABAR.
24,25 PT SURYA TAMA MEDIKA
Ph: 021 8886 3658
Email: suryatamedika@yahoo.co.id
www.suryatamamedika.com
Jl. Senen Raya 135, Ruko AtriumBlok C no 15, Jakar-
26,27,28 PT BERJAYA MANDARIN OPTO-MEDIC
taPh: 021 351 5478
Maspion Plaza, Jalan Gunung Sahari Raya
Kav. 18, 3rd Floor Unit 3F,
Jakarta Utara , Indonesia 14420
29,30,31 PT META MATA MEDIKA
Ph: 021 6470 1108 Fax : 0 21 6470 1114
Email:info@metamedika.com
www.metamedika.com
Gedung 88, 38TH Floor Unit GH 04
Jl. Kasablanka Raya no. 88
32,33 TOPCON SINGAPORE MEDICAL Jakarta Selatan
Ph: 021 2963 8004
Email: marketing-e@topcon.co.id
Kompleks Fatmawati Mas
Blok II No. 229
34, 35, 36,
PT PANCARAYA KRISNAMANDIRI Jl. Tarogong Raya, Jakarta Selatan
37, 38
Ph: 021 769 6181 Fax: 021 769 6180
www.pancaraya-krisnamandiri.com
Perkantoran Hijau Arkadia,
Tower A Lt 6 Suite 601,
39, 40, 43,
CARL ZEISS INDONESIA Jl. TB. Simatupang, Jakarta
44
Ph: 021 7884 8028, Fax: 021 7884 8027
www.zeiss.com
Jl. Pangeran No 77 RT 04/02
Condet, Jakarta Timur
41 CV SOETAN NOERDIN
Ph: 021 800 0014 Fax: 021 808 77939
cv_soetan_noerdin@yahoo.com
Pulogadung Trade Center Blok C/22
42 PT GLOBAL SELARAS MEDIKA Jakarta
Ph: 021 46836025 Fax: 021 4689475
Jl. RA Kartini no. 99, Lebak Bulus
45, 46, 49, Jakarta Selatan 12440
KLINIK MATA NUSANTARA
50 Ph: 021 751 6688 Fax: 021 751 9344
www.klinikmatanusantara.com
Jl. Kedoya Kembangan Raya 10, Jakarta
47,48 PT MULYA HUSADA JAYA
Ph: 021 5835 5942 Fax: 021 5830 2888
51,52, 53, Jl. Cikini Raya no. 89Jakarta 10330Ph: 021 390 8005 Fax:
PT OPTIK TUNGGAL
54, 55, 56 021 3192 3992
57, 58, 61, Kawasan Industri Pulo GadungJl. Rawa Kepiting no 4,
PT TIMUR RAYA LESTARI
62 Jakarta TimurPh: 021 461 0154 Fax: 021 583 55640
Gandaria 8 Office Tower
59 PT GLOBAL JAYA MEDIKA 11th floor unit A & KJl. Iskandar Muda, Kebayoran
Lama,JakartaPh: 021 290 36364
Ruko Puri Niaga,. Puri Kencana,
60 PT OPMIC PERKASA MANDIRI
Jakarta, 11610Ph: 021 582 3582
Ruko Serenade Center Blok A No. 27
63, 64, 67, Gading Serpong, Tanggerang
PT UNIVERSAL STAR MULTILINK
68 Ph: 021 2900 1517–19 Fax: 021 2900 1419
Email: usm.jhalim@gmail.com
Kompleks Perkantoran Bintang Toejoeh
Jl. Jend. A. Yani no. 2 Pulomas
65 PT KALBE VISION
Ph: 021 4786 2288 Fax: 021 4786 2533
http://www.kalbe.co.id
Jl. Tebet Timur Dalam Raya 125 A,Jakarta Selatan
66 PT KHASANAH
12820Ph: 021 3390 2265
Delta Building Blok A20-23.
Jl. Suryopranoto No.1.
69 PT KATAMATA OPTO MEDIK
Jakarta 10160 – Indonesia.
Ph: 021 380 8308. Fax: 021 380 1655
Mid Plaza 1 lt. 12
70 PT BAYER INDONESIA Jl. Jend. Sudirman Kav. 10-11, Jakarta
Ph: 021 570 3661 Fax: 021 570 0591
Jl. Taman Sari 10Bandung 40116, JABARPh: 022 420 7725
71 PT SANBE FARMA (DIVISI VISION)
Fax 022 423 8476
Jl. Tanah Abang II / 4 Jakarta Pusat
72 PT MEIJI INDONESIA Ph: 021 384 5584
Email: handa@meiji.co.id
Jl. Utan Kayu Raya Gd. Utaka 87 lt 2
Rawamangun Jakarta Timur
A PT MEDEQ MANDIRI UTAMA
Ph: 021 7041 9999
Email: medeq@medeqmandiri.com
Gd. Bank Resona Perdania Lt. 7
Jl. Jend. Sudirman Kav. 40-41 Jakarta 10210
PT ROHTO LABORATORIES INDONESIA
B,C Ph: 021 570 7370 Fax. 021 570 7374
Email : customercare@rohtolab.com
http://www.rohto.co.id/
D ZABBYS INDIA
SPONSORS OF INDUSTRY SYMPOSIA
Jl. Bukit Duri Utara no 31,

1 Jakarta Selatan
PT AFINA SINAR CEMERLANG
Ph: 021 830 2119
Fax: 021 831 9274
Kompleks Perkantoran Bintang Toejoeh
Jl. Jend. A. Yani no. 2 Pulomas
2 PT KALBE VISION Ph: 021 4786 2288
Fax: 021 4786 2533
http://www.kalbe.co.id
Jl. Pemuda no. 36
Rawamangun, Jakarta Timur
Ph: 021 471 4848
3 SUMATRA EYE CENTER
Fax: 021 475 5703
Email: smec_jakarta@smec-group.com
www.smec-grouup.com
4 PT PFIZER INDONESIA

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table of content

Chairman of Organizing Committee 3

I hope you all have a good time.

Prof. Nila F. Moeloek, MD

Dear Friends and Colleagues,

DP3 (Departemen Pembinaan dan Pengembangan Profesi)

DP3M (Departemen Pengembangan dan Peningkatan Pelayanan Masyarakat)

Pembinaan Dokter Umum, Profesi Para-oftalmologi

DPKA (Departemen Permberdayaan Kesejahteraan Anggota)

DP4 (Departemen Pertimbangan Pemberian Penghargaan Perdami)

P2KB (Pengembangan Pendidikan Keprofesian Berkelanjutan)

Advisors Prof. Nila F. Moeloek, MD

Chairman Ari Djatikusumo,MD

Co-Chairman Ni Retno Setyoningrum, MD

Secretary Syska Widyawati, MD

Treasurer Julie D. Barliana, MD

Scientific Chair Tri Rahayu, MD

Scientific Secretary Syntia Nusanti, MD

Video Session Amir Shidik, MD)

National Assembly Ni Retno Setyoningrum, MD

Congress Banquet Yunia Irawati, MD

Logistic & Transportation Umar Mardianto,MD

Publication & Emil Fahrinandi Sjahreza, MD

Registration Retno S. Wulandari, MD

Audiovisual Lukman Edwar, MD

Sponsorship & Exhibition Yudisianil E. Kamal, MD

Security & Permit Robby Hilman Maulana, MD

MENGENAL & MEMILIH INVESTASI YANG TEPAT

Thursday, 29 September 2016, 16.30-18.00

Thursday, 29 September 2016, 16.30—18.00

John Jarstad is an Associate Professor of Clinical Ophthalmology and Director of

Professional Training & Current Work Experience

2009 - present Senior Advisor, ORBIS International North Asia

2009-present Specially-appointed Professor, Division of Preventive Ophthalmology, Zhongshan Ophthalmic

Noemi Lois, MD, PhD, FRCS(Ed), FRCOphth

Prof. Aniki Rothova

Karla Chaloupka, MD., PhD.

Work in the Past:

Universitas Airlangga Universitas Brawijaya

Universitas Gajah Mada Universitas Indonesia

Universitas Sam Ratulangi Universitas Sumatra Utara

UNIVERSITAS SRIWIDJAYA UNIVERSITAS UDAYANA

After The Congress

Registration and Badge Pickup

Day Date Time

Day 0 Wednesday, 28 September 2016 16.00—20.00

Day 1 Thursday, 29 September 2016 07.30—20.00

Day 2 Friday, 30 September 2016 07.30—18.00

Day 3 Saturday, 1 October 2016 07:30—17.00

Internet and E-mail

Continuing Medical Education Accreditation

Day Date Time

08.30 - 10.00 RAP 1 IKPAMI

10.00 - 10.30 Coffee Break 1

The Commission The Commission The Commission The Commission

12.00 - 13.00 Lunch

13.00 - 15.10 RAP 2

15.10 - 16.15 Coffee Break 2

16.15 - 18.00 Opening Ceremony IKPAMI

Glaukoma INASCRS Refraction

10.00—10.30 Coffee Break 1

ORBIS Diabetic Misc. Problems Video Session Medical

11.30 - 12.30 Friday Prayer

Abbott Lunch Pfizer Lunch