Beruflich Dokumente
Kultur Dokumente
Basic EKG
Electrical Physiology
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PR interval
QT interval
RR interval
If Irregular rate
Arrhythmia
Theory of Tachyarrhythmia
-Abnormal impulse formation
-Enhance normal automaticity: auto phase 4 depolarization
SA,AV node,His –Purkinje
** resting membrane potential,threshold potential,slope of phase 4 depol
-Abnormal automaticity
Myocardial [Atrial &Ventricle] irritability generate impulse
-Trigger activity
-Early after depolarization
ex. Torsade de point[Polymorphic ventricular tachycardia]
Prolong QT syndrome,brady or phase dependent antiarrhythemic drug
-Delay after depolarization
ex. digitalis toxicity
-Abnormal impulse conduction
Reentry
-Classical reentry
slow conduction zone[unidirectional block]
-Random reentry:AF
Bradyarrhythmia
- SA block ,Sinus pause ,sinus arrest and Sickness Sinus Syndrome
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-AV block
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3.Axis
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4.Chamber enlagement
Atrium
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Ventricle
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Rt Ventricular :
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Lt Ventricular :
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Strain pattern
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5.Ischemic pattern
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-Injury
ST segment change
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Reciprocal change=……………………………………………………………………….
-Infarction
Q wave pathological Q wave
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Non-standard leads
Right ventricle—Right sided chest leads V1R to V6R
Posterior wall—Leads V7 to V9
The ECG does not have a lead that faces directly to the posterior wall of the heart. However, abnormalities of
depolarisation will cause reciprocal or mirror changes in the anterior leads. The important leads are V1,2,3 of which
V2 is the most important. The 3 classical changes to be sought are: