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  • Acute Coronary Syndrome

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    Renz Castañaday
    512 Ansichten2 Seiten
    The document summarizes the pathophysiology of atherosclerotic heart disease. It lists age and genetics as predisposing factors and diabetes mellitus type 2 and hypertension as precipitating factors that can lead to endothelial injury and desquamation of the endothelial lining. This increases permeability and adhesion molecules, allowing lipids and platelets to accumulate and form plaque. Over time this can lead to reduced coronary tissue perfusion, diminished myocardial oxygenation, chest pain, and potentially a ST elevation myocardial infarction if the blockage is complete. Cardiac enzymes may be released indicating cell death.

    Originalbeschreibung:

    Pathophysiology of acute coronary syndrome

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    The document summarizes the pathophysiology of atherosclerotic heart disease. It lists age and genetics as predisposing factors and diabetes mellitus type 2 and hypertension as precipitating factors that can lead to endothelial injury and desquamation of the endothelial lining. This increases permeability and adhesion molecules, allowing lipids and platelets to accumulate and form plaque. Over time this can lead to reduced coronary tissue perfusion, diminished myocardial oxygenation, chest pain, and potentially a ST elevation myocardial infarction if the blockage is complete. Cardiac enzymes may be released indicating cell death.

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    © All Rights Reserved
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    Markieren Sie unangemessene Inhalte
    512 Ansichten2 Seiten

    Acute Coronary Syndrome

    Hochgeladen von

    Renz Castañaday
    The document summarizes the pathophysiology of atherosclerotic heart disease. It lists age and genetics as predisposing factors and diabetes mellitus type 2 and hypertension as precipitating factors that can lead to endothelial injury and desquamation of the endothelial lining. This increases permeability and adhesion molecules, allowing lipids and platelets to accumulate and form plaque. Over time this can lead to reduced coronary tissue perfusion, diminished myocardial oxygenation, chest pain, and potentially a ST elevation myocardial infarction if the blockage is complete. Cardiac enzymes may be released indicating cell death.

    Copyright:

    © All Rights Reserved
    Als DOCX, PDF, TXT herunterladen oder online auf Scribd lesen
    Markieren Sie unangemessene Inhalte
    von 2

    V.

    Pathophysiology
    Predisposing Factors Precipitating factors
    -Age (70 Years Old) -Diabetes Mellitus Type 2
    -Genetics (FBS= 10.98mmol/L)
    -Hypertension (130/70)

    Endothelial injury

    Desquamation of endothelial
    lining

    Increase Permeability/adhesion
    molecules

    Lipids and Platelets accumulate in the area

    LDL attracts monocytes and


    macrophages
    (Inflammatory Response)

    Formation of Plaque

    Atherosclerotic Heart Disease

    Reduced Coronary tissue perfusion

    Renz Darwin C. Castañaday


    BS Nursing
    St Jude College Cavite
    Diminished Myocardial
    Oxygenation Anaerobic metabolism

    Lactic Acid production

    (Lactic Acidosis)
    Decreases
    Cardiac Output Myocardial Ischaemia

    Decrease O2 Chest Pain


    supply in the body

    -Fatigue
    -Difficulty in ST elevation
    Breathing Complete Blockage

    NO YES
    Partial Blockage
    Cardiac Tissue Begin
    To Die
    (Necrosis)
    Unstable Angina NSTEMI

    STEMI

    No Cardiac
    Enzymes
    Released

    Release of Cardiac enzymes


    -CK-MB
    -Troponin I

    Renz Darwin C. Castañaday


    BS Nursing
    St Jude College Cavite

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