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DISASTER PREDICTABILITY
Note: Time, Impact
a) SHORT ONSET
o Typhoon Haiyan – strongest storm b) SUDDENT ONSET
o July 16, 1990 – M7.8 Earthquake – Baguio, o Predictable – cyclones, volcanic eruptions
Cabanatuan, Dagupan o Unpredictable – Earthquake, Landslide
o 1991 – Pinatubo – Aetas
Disaster according to NUMBER OF VICTIMS
Defined as an ecologic disruption of a severity and INVOLVED
magnitude that results in deaths, injuries, illness and
property damage that cannot be effectively managed a) MULTIPLE PATIENT – 10 casualties
using routine procedures or resources and that b) MULTIPLE CASUALTY – 100 casualties
require outside assistance (Landesman, et. al, 2001). c) MASS CASUALTY - >100 casualties
o Serious, unexpected, and often dangerous a) Pre- Impact – begins prior to onset of a disaster
situation requiring immediate action. b) Impact – (Rescue Phase) – continues to the
initiation of post-impact
Disaster according to OCCURRENCE c) Post Impact – (Recovery Phase) – disaster has
a) MAN-MADE been evaluated, Development of independence
o Disasters in which the principal direct causes are o Measure vulnerability – a)
identifiable human actions, deliberate or Preparedness, b) Finances (relocation
otherwise (war, bombing) site), c) man involved
o E.g. Fire in Ozone, Transportation accidents (Oil Disaster according to different FACTORS/
Spills), 9-11 VULNERABILITIES
b) NATURAL:
o Results of an ecological disruption or threat that a) Personal Factors – a) Uncontrolled (age –
exceeds the adjustment capacity of the young and old, disabilities), b) Controlled
community (Preparation, Physical Fitness)
NATURAL DISASTER CATEGORIES, TYPES, AND SUBTYPES
HYDROMETEOROLOGICAL
BIOLOGICAL GEOPHYSICAL
HYDROLOGICAL METEOROLOGICAL
~Epidemic ~Earthquake ~Flood ~Storm
Viral, Bacterial, ~Volcano General Flood Tropical Cyclone
Parasitic, Fungal, Prion ~Mass Movement (dry) Storm Surge/ coastal flood Extra-tropical cyclone
Infectious Disease Rockfall Local Storm
Landslide Mass Movement (Wet)
~Insect Infestation Avalanche Rockfall CLIMATOLOGICAL
Subsidence Landslide
~Animal Stampede Avalanche ~Extreme Temperature
Subsidence Heat wave
Cold wave
Extreme winter condition
~Drought/ Wildfire
Forest Fire
Land Fire
** Cross train nursing staff to perform other tasks to 4) RECOVERY: (short and long term) – includes
maintain services during staffing crisis actions of responders, government, victims that
help return an affected community to normal by
Fire Safety Measures: stimulating community cohesiveness and
government movement
1. Provide adequately wide entrances and exits in
all buildings to facilitate quick dispersal of
a) Short term:
children and staff in the event of emergency
o Restoring necessary lifeline systems
2. Ensure that wall openings in classrooms are not
(communication, water, electricity,
<20% of floor area
transportation)
3. Remove/Replace all inflammable material in
o Providing for basic human needs
school buildings
o Provide crisis interventions
4. Construct kitchen shed for mid-day meals in a
o Needs assessment
manner so as to minimize fire hazards
b) Long Term:
5. Provide Fire-Fighting arrangements in all
o Recovery may continue for a number
schools
of years after a disaster whose
6. Regular training of teachers and children in fire
purpose is to return life to a normal or
safety and evacuation drills
improved levels.
E) ABDOMEN A) OLDCART
o 1st intervention: identify painful area – assess o Onset of symptoms
last pain may affect assessment findings o Location of problem
o Note: symmetry, color o Duration of symptoms
o Cullen’s Sign – ecchymosis and pruritus o Characteristics of symptoms
around umbilicus o Aggravating factors
o Grey Turner’s Sign – ecchymosis and o Relieving factors
pruritus on the flank area o Treatment administered
o Ker’s Sign – pain on ths tip of the shoulder to B) PAIN (PQRST)
the tip of the scapula indicate splenic o Pain
rupture o Quality
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o Radiation o Individuals with highest risk/ highest
o Severity possibility of suicide
o Timing
Special considerations
C) HISTORY (SAMPLE) o Pediatric Trauma
o Signs and Symptoms Address not only the child’s behavior
o Allergies but also the accompanying adult
o Medications Fear of separation
o Past Medical History Disorganized behavior
o Last oral Intake; Last day of menses
o Events preceding the injury o Geriatric Trauma
All trauma in the elderly are
Pediatric Triage History potentially life-threatening
Limited physiologic reserves severely
A) C.I.A.M.P.E.D.S affect the heart and lung dynamics
o Chief complaint oxygenation and hemodynamics are
o Immunization, isolation prone to failures
o Allergies Decreased fat store, CHON reserves
o Medication sources of glucose; muscles (stores
o Past medical history of glycogen)
o Events surrounding problem Fight/ Flight – increase epi and nor epi
o Diet / Diaper increased HR, RR, sugar, BP
o Symptoms No compensation increase Demise
Atherosclerosis – decrease vasomotor
B) Head to Toe (SAVE A CHILD) response persistent hypertension
o Skin (complication)
o Activity Limited Ejection fraction – decreased
o Ventilation cardiac output compromise
o Eye-contact ischemia infarct MODS
o Abuse – look into diagnostic tests &
P.E (different levels of healing DEBRIEFING - Psychological Debriefing:
process & bone regeneration) o Encourage to verbalize
o Crying
o Heat/ Temperature COMPLICATIONS OF TRAUMA
o Immune status
o Level of consciousness 1. Compartment Syndrome:
o Dehydration o Result from bleeding or edema due to:
i. Fractures
NURSING ROLES IN THE ii. Crash injuries
PSYCHOLOGICAL MANAGEMENT OF iii. Arterial disruptions
PATIENTS AND FAMILIES IN EMERGENCY iv. Burns
AND CRISIS SITUATIONS v. Prolonged compressions
** CASTS: assess for presence of tingling sensation,
A) POST-TRAUMATIC STRESS DISORDER pulse and circulation
(PTSD)
o The development of characteristic symptoms 5 P’s – pain, pallor, pulselesness, paresthesia,
after a psychologically stressful event that is paralysis + poikilothermia
generally outside the range of human
experience Management:
o Common in soldiers o Remove cast – cause prolonged
compression
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o Maintain level of the limb at the level of
the heart TRAUMA – COMMON EMERGENCIES
o Apply Cold compress
o Remove constrictive clothing 1. Traumatic Brain Injury:
o Fasciotomy – surgical decompression o Alter cognitive activity and perception
o 2/3 of the affected are below 30 y/o
2. Fat Embolism o More males are affected than females
o Due to mobilization of fat globules in o 50% have positive alcohol blood levels
association with injury 2* to fracture of long o Main causes are vehicular accidents, falls,
bone and assaults
o Pulmonary embolism
i. DOB, chest pain 2. Focal Brain Injuries:
ii. Ecchymosis of chest o Confined in one area of brain
iii. **Acute respiratory failure o Result from severe blow to the head
o * open injury occurs when something
Management pierces the skill and enters brain tissue
o Stabilize fracture ASAP splints, o * closed injury occurs when parts of the
immobilize skull presses into the brain
o Minimize handling and moving patient o C1 & C2 – innervation of heart and lungs
decrease escape of fat globules o Optic chiasm – below PG
o Remove fat emboli o Parietal lobe – cranial nerves
o Pulmonary angiogram
3. Penetrating Injury:
3. Disseminated Intravascular Coagulopathy o Emergency management
(DIC) i. Stabilization of ABC including
o Systemic activation of blood coagulation external hge should be achieved
which results in deposition of fibrin leading ii. Inspect superficial wound
to thrombi in various organs iii. ID entrance and exit wounds (*bullets)
o May result to bleeding release of plasmin iv. Note for CSR leaks, bleeding, or brain
(lyse clot) as body’s way to cope parenchyma
Management: 4. Contusion:
o Prepare to administer clotting factors and o Bruising on the surface of the brain
blood or blood products o Cause: severe trauma to the head
o Administer heparin prevent o Common: Frontal and temporal
coagulation o Patient becomes unconscious immediately
(5 minutes)
4. Adult Respiratory Distress Syndrome (ARDS) o Loss of reflexes
o Cause: infections, chest-trauma damage o Transient cessation of respiration
alveoli (type I – epithelial cells, type II – o Brief period of bradycardia
Surfactant, type III – macrophage); o HPoN
destruction of elastin lose ability to
expand 5. Coup-Countrecoup
o Injury (inhalation, trauma) o injuries where the patient sustains a
o Causes lung swelling and fluid buildup in the combined injury
alveoli inhibits oxygen passing in to the o at the point of impact & at the opposite
bloodstream o ‘E’ Management:
Management: i. Dexamethasone/ steroid therapy
o PEEP ii. Surgery: craniotomy, burr-holing
o Corticosteroids iii. Decrease swelling by diuresis:
o Antibiotic mannitol, acetazolamide
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4. Subdural Hemorrhage:
6. Concussion o Bldg happens between dura and arachnoid
o Momentary interruption of brain function space
o No structural damage quick recovery o Cause: MVA
o With or without loss of consciousness o Types
o Management: a) ACUTE:
i. Observe for: H/A. irritability, Symptoms seen within 24* of the
insomnia, poor concentration and injury
memory (post-concussion syndrome) Start with H/A, drowsiness,
ii. Gradual resumption of activities restlessness, agitation, slowed
cognition, confusion
OTHER HEAD INJURIES Worsens over time & progress to
loss of consciousness, respiratory
1. Scalp Injuries: challenges (Biots breathing), and
o Damage underneath may be greater even pupils dilate
small injuries are sutured b) SUB-ACUTE: 48* - 2 weeks
c) CHRONIC: weeks- months
2. Skull Fracture
o Break in the continuity of the skull caused 5 CATEGORIES OF NEUROLOGIC
by a force FUNCTION THAT ARE CRITICAL TO THE
o Depressed Skull Fracture risk for EVALUATION PROCESS
piercing the brain
o Linear Skull Fracture 1. LOC
o Basilar Fracture – over the base of the skull, 2. Pattern of breathing
base of frontal, temporal 3. Pupillary changes
a) Leak CSF to nose, ear 4. Occulomotor responses
b) Raccoon’s eye 5. Motor responses
c) Battle’s sign
d) Hemotympanism SPINAL CORD INJURIES
e) Otorrhea, Rhinorrhea Compression along length of spinal cord, average
f) Meningeal Irritation (BONK): (18 inches)
Brudzinski, Opisthotonus, Nuchal
Rigidity, Kernig’s Sign
3. Epidural Hematoma
o Corrects between skull and dura
o Cannot be seen outside
o ** Brain atrophies with age bldg in geria
is not evident because of the space
o A momentary loss of consciousness
followed by a lucid period where
consciousness is regained, followed by
rapid progression of unconsciousness
Tension Pneumothorax
Closed
Primary due to ruptured bled
Tachypnea
Mild dyspnea
Mediastinal shift - due to excess air pushing
heart (note for tracheal deviation)
** signs and symptoms depend on the size of
pneumothorax
B. Hemothorax
Cause:
Chest trauma
MVA
Crushing injuries
Management: cover with wet cloth Check for fluctuations (+)
to prevent air entrance close system
Rib Fractures – 2* hitting steering wheel Bubbles leak in system
**First 3 ribs – High mortality No fluctuations
Puncture lung detachment, kinks,
pneumothorax; puncture obstruction, re-expansion of
trachea, aorta, SVC lungs (**)
5 - 10th rib – cardiopulmonary
th
** DO NOT MILK CORD
compromise, liver and spleen injuries increase pressure in the lung
Floating rib – renal injuries parenchyma tension
Assessment pneumothorax
Pain on inspiration and Transport bottle below
coughing patient’s body
Bruising over the fractured ** pressure – 2cm H2O – submerge
site retroperitoneal bldg. tube 2cm ONLY deeper
Crepitus (subQ emphysema) submersion = higher pressure
Management:
No binders and strappings -
*risk of punctures
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C. Flail Chest Primary Assessment
Initiate immobilization of the spine
Fractures of 2 or more ribs at multiple places Airway obstruction can occur due to:
leading to free floating ribs Tongue swelling
Bleeding or
Signs and symptoms Broken teeth
Dyspnea Breathing may be impaired
Paradoxical breathing Cricoidotomy / Cricothyrotomy
Rapid RR Tracheostomy
Crepitus
Diminished breath sounds Subsequent Interventions
A-B-B: Early (R. Alkalosis), Late (R. Ice pack – control bldg
Acidosis) Elevate HoB – control bldg.
Pain medications
Management ** Generally No Blowing of Nose,
High fowlers Coughing, Sneezing due to possibility of
Prepare for intubation CSF leaks cough out brain particles and
Mechanical ventilation (PEEP) IICP
O2 by BVM DBE – for pain and anxiety
Stabilize chest wall
Turn towards affected side improve ABDOMINAL TRAUMA
oxygenation pool blood towards affected - Common area; vague in assessment due to # of
side improve perfusion organs
D. Pulmonary Contusion
E. Maxillofacial Trauma
- Fractured mandible, maxilla, orbitals,
zygomatic bone
Management Management
Local wound exploration Resuscitation prior to definitive repair
Apply stabilizing dressing Laparotomy is mandatory if shock,
Prophylactic antibiotics – broad spectrum evisceration, or peritonitis
** IN GENERAL: leave the foreign bodies in ** Pancreas rupture amylase,
and remove in the OR lipase, trypsin, chymotrypsin auto
digestion
Management of Peritoneal Abdominal Trauma Diagnostic studies used to determine need for
In the thoracoabdomen laparotomy
Consider diaphragmatic injury WoF abdominal compartment syndrome
Note breathing pattern Abdominal distention
Paradoxical breathing – Decrease peristalsis
sinking of abdomen Muffled bowel sound
Diagnostic evaluation
LIVER TRAUMA
Signs and Symptoms of Diaphragmatic Injury Most common cause of death due to
Decrease breath sounds abdominal trauma
Paradoxic breathing
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Perforated liver rupture GB bile into direct bilirubin) indirect bilirubin
peritoneum peritonitis & breakdown of blood skin
interstitial organ (cirrhosis)
Signs and Symptoms of Large Intestine
Signs and Symptoms of Liver injuries Obstruction
Persistent HpON Abdominal pain
Guarding over RUQ/LUQ Peritoneal irritation
(+) murphy’s sign Blood on rectal examination
Rebound abdominal tenderness – Peritoneal Fever
involvement
Direct – indicates organ involvement POISONING
Dullness to percussion 90% of events occurring at home
Abdominal distention and peritoneal Cause: loose paint, (preservatives)
irritation
Persistent thoracic bleeding – 2* Poison
emulsification of diaphragm Substance that causes : Injury, Illness, Death
Enters through – Ingestion, Inhalation,
Absorption
SPLEEN INJURIES
Lymphatic A) Ingested Poison
Sequestration of old RBC, WBC,
Platelet 2 Categories of Poisoning
Use raw materials: Fe,
albumin Bacterial Food Poisoning
Produce lymphocytes Hives, N/V
** 3-5 months for body to recuperate **most common: Salmonella typhi
from injury ** most dangerous: Botulism
**Any injury to the spleen can cause Chemical Poisoning
severe internal hge & shock
Corrosive Poisons
Signs and Symptoms of Spleen Injuries Agents that cause tissue destruction
HPoN, tachycardia, SOB late signs
Peritoneal irritation Alkaline
LUQ pain Encourage vomiting if within 1 hour of
ingestion
BOWEL INJURIES Toilet bowl cleaners
Associated as complications of trauma Bleach
Infection, abscess, fistula Non-phosphate detergents
Oven cleaners
Signs and Symptoms of Small Intestine Injuries Button batteries
Abdominal pain – radiating to the testicles
Referred pain to shoulders, chest, and back Acid
Peritoneal irritation Hcl
Fever, jaundice, intestinal obstruction Toilet bowl cleaners
Jaundice : 2* obstruction to sphincter Swimming pool cleaners
of oddi backflow of pancreatic Metal cleaners
juice (autodigestion pancreatitis Rust removers
bldg); backflow of bile to GB & liver
(decrease conjugation of indirect to
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Signs and Symptoms
Immediate – burning sensation ( mouth, ** DO NOT IDUCE VOMITING WITH
throat, esophagus, stomach) CORROSIVE AGENTS
Intense thirst
Dysphagia, retching Other Important Treatments:
Hematemesis Stomach tubes and emetics are
contraindicated
Nursing Assessment Levin tubes can be passed within 1*
Note for:
Unusual odors B) Inhaled Poison
Flames/ smoke
An open medicine cabinet lead to what Carbon Monoxide
Binds to circulating Hgb to decrease the
type of poison was ingested
oxygen carrying capacity (CarboxyHgb)
Open or spilled containers
** Hgb absorbs CO approximately 200x
Overturned or damaged plants more readily than it absorbs oxygen
Bring paraphernalia or empty container * Odorless
Important Assessment Signs of CO poisoning
Determine
H/A
What – type help determine proper Nausea
treatment
Dizziness
When – time to encourage vomiting
Breathlessness
How much substance was ingested
Collapse
Loss of consciousness
Signs and Symptoms of Poisoning and Tissue
** CO causes encephalopathy deficits
Damage
Decreased Level of consciousness
General Management
Decreased DTR
Expose patient to fresh air ASAP
Pinpoint pupils
Administer O2 at 100% - Face mask @
10Lpm
Interventions for those with Ingested Poisons
Remove the toxin or decrease its absorption
C) Absorbed Poisons
Use emetics, cathartics when
Comes into contact with the skin
appropriate
Absorbed poisons come from plants such as
Gastric lavage
poison ivy, poison oak, and poison sumac, as
Activated charcoal
well as from fertilizers and pesticides
Administration of specific antagonist
as early as possible
Care for Absorbed Poisons
Emergency Management Immediately rinse the area
Note for:
ACIDS Rashes or weeping lesions
Weak Alkalis e.g. Ca & Mg OH cold, running water for 5-10 minutes
Along with plenty of water ** EPINEPHRINE 0.3 mL – first line
Neutralizing agents like Milk or Egg
Albumin Management for Chemical Contact
ALKALIS If chemical is dry:
Neutralize poison by giving acids like Brush off the chemicals using gloves
Acetic acid, citric acid Flush with running tap H2O
Mix with large quantities of water If chemical is wet
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Flush with large amounts of cool, NEAR DROWNING
running tap water at least 20 minutes. Episode in which a person initially survives
immersion in liquid
D) Injected Poisons Fresh Water Drowning: destroys
Injected poisons enter the body through: surfactant atelectasis
Bites or stings of certain insects (bees, Salt Water Drowning increase Na
hornets, wasps) or + H2O osmosis pleural effusion
Drugs or
Misused Management
Maintain cerebral perfusion
Signs and Symptoms Oxygen by mask with reservoir –
Generalized urticarial 10Lpm
Anxiety, malaise Treat hypothermia if suspected
Anaphylaxis bronchospasms Insert NGT
Decompress the stomach and
Management: Prevent the patient from aspirating
Epinephrine injected SubQ gastric content ARDS RF
Assess patient for anaphylactic reactions
(dyspnea, urticarial, bronchospasm)
3. Contusion:
Damage of soft tissue
Ecchymosis and hematoma
Tears
Management
RICE
Hot – Cold Modalities
ICE injury within 24 - 48*
vasoconstriction
Warm – relief of pain
** If severe – cast immobilization or surgical
repair may be necessary
Assess neurovascular status frequently
Capillary refill
6 P’s
B) Lacerations
Disrupted continuity of the skin
Management
Clip or shave hair D) Dislocations
Clean wound with NSS Misalignment of the bone on the articulating
Debridement – remove necrotic surfaces
portions A temporary separation b/n articulating ends
Dressing and a joint
Signs and symptoms: athlete often reports
C) Fractures Snapping or popping sound
Disruptions in the continuity of the bone Followed by dysfunction deformity
(lengthening)
a. Simple – bone ends remain intact within
surrounding soft tissue E) Subluxation
b. Compound – bone fragments have broken incomplete separation b/n articulating ends
through the skin. (+) Avulsion
Management
Patient Teaching
1. Heat Illness
Ensure adequate fluid intake
Due to salt loss from sweating
Use sunscreen
DHN ARF
Rest frequently when in hot env’t
Causes spasms in large muscles
Wear light weight, light colored, loose
** decreased Na increased K+ tingling
clothing
Treatment:
Stop activity
Cool environment COLD EMERGENCIES
Balance salt solution Frostbite
Do not rub blisters
2. Heat Exhaustion Assess NVS
Caused by DHN Cause: extreme cold temperature
Stems from heavy perspiration Manage: warm IV, blankets
Poor electrolyte consumption
** decreased Na decreased “Don’t Pray for your grades, Make it Happen!”
Neurotransmission weakness , flu like ~ Anonymous
symptoms, tachycardia, N/V “Study HARD, no matter if it seems impossible, no matter if it
Field Tx: takes time, no matter if you have to up all night, just remember
Stop activity that the feeling of success is the best feeling in the entire
Move to cold place world”
~Anonymous
Cold packs – armpit & groin
Remove constrictive clothing “Trust yourself. You know more than you think you do” ~
Re-hydrate (water, sports drink) Anonymous