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POST-REHABILITATION EXERCISE
POST- Course Outline
FOR PERSONAL TRAINERS
Scope of practice
Concepts of injury (pathology, histology,
physiology, physics)
Upper Quadrant – shoulder, elbow, wrist
Lower Quadrant – hip, knee, ankle
Spine
Medical Communications and Post-Rehab
Dean Somerset, BSc. Kinesiology, CEP, CSCS, MEPD Fitness
Rehabilitation & Medical Fitness Coordinator, World Health
Course Objectives
Course Objectives
Develop safe & effective exercise programs Administer & interpret results of fitness
for improving cardiovascular, neurologic, assessments, use parameters set by licensed
metabolic and musculoskeletal health in HCP to give effective exercise programs
clients not requiring medical supervision Understand common mechanisms of
Learn related anatomy/physiology associated injuries and prevention
with specific MSK injuries & medical Communicate with HCPs & insurance
conditions, their response to exercise carriers, knowledge of FOIP &
confidentiality
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Why Post-
Post-Rehab?? What Makes a Post-
Post-Rehab Trainer?
• Able to show •Image
you know presented to
your stuff those you work
with
Perceived Perceived
Competency Professionalism
Work
Referral within
Networks Scope of
Practice
• Physios, • Use your
chiropractors, knowledge,
MDs, etc. refer when
necessary
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BENDING DISTRACTION
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http://www.medicalive.net/319_wound_healing_process DEPENDENT ON
HOMEOSTASIS
RELATIVE RISK
TIME,
ATROPHY
MAGNITUDE,
DEATH
DEATH
INJURY
AND DIRECTION
OF FORCE
APPLICATION
Mueller & Malluf. Phys Ther,Vol. 82, No. 4, April 2002, pp. 383-403
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STRENGTH
TENSILE
No more pain
surrounding tissues Makes individual think
Reduced strength and they can return to
function of surrounding activity
tissues pre-disposes the Pain cessation typically TIME
area to a reduced stability at around 80% of tensile
and greater risk of re-injury strength
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Osteoarthitis
Osteoarthritis
Chronic disease primarily in synovial joints due to
degeneration, previous trauma, or genetic
susceptibility
Changes to articulating structure include erosion
of cartilage, exposure of subchondral bone, and
increased friction in joint
Synovial fluid changes can include increased
enzyme activity and protein concentration
Osteoarthritis
Outer edges of joint develop
osteophytes and increase bone density Examples of
and size, where mid-joint bone
becomes scarred from friction Osteoarthritis
(sclerotic)
Decreased concentration of hyaluronic
acid (fluid forming compound) as well
as reduction in synovial fluid output
Reduced fluid increases friction,
pressure, and scarring of joint surface
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Osteoarthritis Osteoarthritis
May present with visible joint
Pain is always present, and made worse with
deformation (osteophyte), warm
activities requiring involved site
to the touch (inflammation),
Pain is worse following periods of inactivity (early swelling, limited ROM, altered gait
morning, plane rides, end of work day in office) mechanics and possibly crepitus
due to reduction in synovial fluid production (joint noise)
Most often affects 55+ years, but signs of OA can Primary causes are idiopathic (no
be present as young as 45 years known cause), where secondary
Women effected more than men & show greater causes include obesity, repetitive
deleterious effects from OA strain, trauma, infection, genetics,
gout (crystal deposits)
Osteoarthritis Osteoarthritis
Low-impact exercise and weight reduction are Many advanced cases require joint replacement
always recommended for OA, as it reduces surgeries to improve quality of life and functional
loading on affected joint, and increases production capability (many discussed later)
of synovial fluid Typically only done once pain is unbearable and
Muscles around a damaged joint may lose neural function is impeded significantly, replacements have
tone, resulting in atrophy, reduced proprioception, somewhat short lifespan and their own problems
and decreased vascular activity. Exercise helps to Replacements cause immediate pain relief,
regain and maintain these factors increased range of motion, and overall
Cannot be reversed, but can reduce damage and improvements to functional capacity
help control symptoms
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Osteoarthritis Sprains
Ligamental injuries, caused when fibres are stretched
beyond their normal limits.
Rated in degrees based on involved structures, as well
http://video.about.com/arthritis/Osteoarthritis.htm
as extent of damage within the ligament
◦ 1st degree – mild tearing and over-stretched fibres
◦ 2nd degree – partial thickness tear of ligament, accompanied
by pain, swelling, and loss of function to effected area
◦ 3rd degree – complete rupture of ligament, requires surgery
◦ 4th degree – avulsion fracture, rupture with ligamental
attachments dislodging bone segments
Sprains Sprains
Damaged ligaments will Cause reduced stability in effected joint,
produce pain, swelling, reduced strength in associated muscles,
bleeding into the periphery, decreased confidence in functional capabilities
reduced strength/function of until joint is finished healing
area
Healing time is primarily determined by blood
Ligaments with partial- flow to area and level of vascularization
thickness tears will be able to
Bracing/taping is often required to increase
regenerate on their own, while
stability and prevent further injury while
full-thickness tears will require
healing
surgery
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Fractures Fractures
Breaking of bone, result from excessive pressure or rapid
change in force application beyond bone’s capabilities
Fractures in youth can be troublesome if they involve
epiphyseal (growth) plate
Fx’d growth plate lead to premature calcification, and
cessation of bone growth
Salter-Harris Classification
Fractures Fractures
Can be either open or closed, depending on
whether skin is broken
Typically require surgical intervention
May require screwing bone segments together, use
of a plate or rod to provide structural support
during healing, or external bracing
Typically will be isolated in a cast to prevent
movement of end segments that would open the
callus
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Fractures Fractures
5 distinct phases of fracture healing:
1. Fracture occurs, inflammation runs its’
course
2. Formation of granulation tissue at bone ends
3. Replacement of granulation tissue with callus
4. Replacement of callus with lamellar bone
5. Remodelling of bone to normal contours
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