Beruflich Dokumente
Kultur Dokumente
P wave:
Represents atrial contraction So, become multiple in atrial flutter, branch block, tachycardia Absent in atrial fibrillation Large in atrial hypertrophy Inverted in nodal rhythm Represents ventricular contraction So, become amplitude in ventricular hypertrophy Prolonged in bundle branch block Deep Q (no R wave) in infarction . Why? Infarction is a dead area . So, there will be no contraction in that area
QRS complex:
T wave:
Represents ventricular relaxation So, become amplitude in muscular exercise amplitude or inverted in hypoxia
PR interval:
Represents conduction in AV bundle So, become prolonged in delayed conduction ( HB ) , bradycardia Short in rapid conduction as in tachycardia WPW
Notes :
P wave normally +ve in lead 1 & -ve in AVR RS progression R wave begins small in V1 then gradually in size.
(S in V1 + R in V6) < 7
R=S
Axis :
( lead 1 & AVF ) QRS lead 1 Normal Right axis deviation Left axis deviation Very rare (odd ) AVF
(1)
Rate
By two methods:
(2)
12(1) Type
a)
Rhythm
Type Regularity
Sinus rhythm (normal rhythm) Arises from SA node Normal shape of P wave Nodal rhythm Arises from AV node ( as in cases of heart block ) Inverted P wave
b)
(2)
Regularity
Regular
Regular irregularity
Irregular irregularity
(3) (4)
Axis Waves
* INFARCTION
In infarction, we should answer 3 questions:
1-
Present of Q wave in any site except (V5 & V6) means infarction
2-
Elevated ST segment + inverted T wave Recent Inverted T wave without elevated ST segment Old
3-
The site?
Transmural anterior wall myocardial If in (lead I, AVL, V1 V6)
infarction
infarction
Note:
Elevated ST segments in some leads may be associated with reciprocal ST depression in the other leads.
* Ventricular Hypertrophy
LT vent. Hypertrophy
RT vent. Hypertrophy
S R in V6 5.5
in V1 4
both 7 Left axis deviation Strain in V5 & V6 N.B Strain inverted T wave
Left axis deviation Left vent. Strain N.B RSR` M-shaped like this