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RISK FACTORS
Smoking. Obesity Hypertension High fat Diet Physical activity or inactivity Occupation: sedentary or active, and how active? Stress levels; occupational and others.
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Angina
Angina is the temporary chest pain or a sensation of pressure that occurs when the heart muscle is not receiving enough oxygen Not everyone with ischemia experiences angina.ischemia without angina is called silent ischemia
Types of angina
Nocturnal angina that occurs at night during sleep Angina decubitus angina that occurs when the patient is lying down Variant angina angina at rest and not during exertion
Spasm of one of large coronary arteries on surface of heart
Unstable angina is a medical emergency.there exists a varied patterns of symptoms severe pain ,increased frequency of pain
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Angina is felt as a pressure or ache beneath the sternum Either in shoulder or down the inside of either arm,through the back,in the throat,jaw,or teeth Pain is felt in back and shoulders misdiagnosed as arthritis Pain may occur in stomach area misdiagnosed as stomach ulcer
Pulse
Rhythm: The most common irregularities are atrial arrhythmias and extrasystoles (which may disappear on exertion) Character: water hammer - Thready, strong, bounding, collapsing seen in
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Pulse
'pulsus paradoxus' - A pulse that weakens in inspiration is called (as opposed to the normal increase in volume)
constrictive pericarditis pericardial effusion restrictive cardiomyopathy severe asthma.
'Pulsus bigeminus': groups of two heartbeats close together followed by a longer pause. The second pulse is weaker than the first.
premature ventricular contractions after every other beat. hypertrophic obstructive cardiomyopathy
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AUSCULTATORY AREAS
aortic area or right sternal border (RSB) right 2nd intercostal space pulmonic area or left upper sternal border (LUSB) - left 2 nd intercostal space tricuspid area or left lower sternal border (LLSB) is at the left fourth intercostal space mitral area or apex is at the PMI -- the 5 th intercostal space in midclavicular line
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HEART SOUNDS
S1 closure of atrioventricular - mitral and tricuspid Valves S2 - closure of semilunar -- aortic and pulmonic valves S1 and S2 The Lub-dub sound of the heart is S1-S2.
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S1 sound
S1 is the sound made when the mitral and tricuspid (atrioventricular or AV) valves close. It marks the beginning of systole S1 is loudest at apex or left lower sternal border
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S2 sound
S2 is the sound made when the aortic and pulmonic (semilunar) valves close. It marks the beginning of diastole. S2 is loudest at the base. The top of the heart is the base.
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RHYTHM
Rhythm is usually regular. Healthy young people often have a sinus arrhythmia: their pulse is slower in expiration.
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HEART MURMURS
Heart murmurs are produced by turbulence Causes of heart murmurs include: increased blood flow across normal valves for example, in pregnancy or hyperthyroidism, or an innocent murmur), or turbulent flow through abnormal valves: tight valve (stenosis) or leaky valve (regurgitation or insufficiency)
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HEART MURMURS: TIMING Systolic: between S1 and S2 (during systole) Diastolic: after S2
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HEART MURMUR
Mitral stenosis Aortic regurgitation Mitral regurgitation Aortic stenosis Mid-diastolic murmur Diastolic murmur Holosystolic murmur Mid-systolic ejection murmur
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