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Abdominal Emergencies
Fred W. Wurster III, AAS, NREMT-P
Abdominal Emergencies
Anatomy Review
Non-hemorrhagic abdominal pain Gastrointestinal hemorrhage
Assessments
Treatment modalities
Abdominal boundaries
Peritoneum
Abdominal cavity
Double-walled structure
Visceral and Parietal
Peritoneum
Abdominal anatomy
Abdominal anatomy
GI Structures
Primary
Mouth/Oral Cavity Pharynx Esophagus (digestive tract between pharynx & stomach) Stomach (hollow digestive organ, receives food from
esophagus)
GI Structures
Accessory
Salivary glands (produce/secrete saliva) Liver (solid organ in RUQ; produces/secretes bile, essential
proteins, clotting factors; detoxifies; stores glycogen)
Organs
Solid
Liver Spleen Pancreas Kidneys Ovaries
Organs
Hollow
Stomach Intestines Gallbladder and bile ducts Ureters Urinary bladder Uterus Fallopian tubes
Abdominal quadrants
Spleen
Transverse colon (partial) Descending colon (partial)
Fallopian tube
Fallopian tube
Abdominal Pain
Visceral
Diffuse in nature Stretching of peritoneum of organ capsules by distension or edema Poorly localized Can be perceived at remote locations related to the affected organs sensory innervation
Abdominal Pain
Somatic
Sharp in nature Well localized Inflammation of parietal peritoneum or diaphragm
Referred
Perceived at a distance from the affected organ
Esophagitis
Gastric Ulcer
Greater than 50 years old Employed in positions that require physical activity Pain after eating or when stomach is full Usually no pain at night
PANCREATITIS
Inflammation of the pancreas in which enzymes auto-digest gland
Caused by:
EtOH (80% of cases) Gallstones obstructing ducts Elevated serum triglycerides Trauma Viral or bacterial infections
Pancreatitis
May lead to
Peritonitis, pseudocyst formation, hemorrhage, necrosis, secondary diabetes
Cholecystitis
Gall bladder inflammation, usually secondary to gallstones (90% of cases)
Risk factors (Five Fs)
Fat, fertile, febrile, fortyish, and female Heredity, diet, BCP use
Acalculus cholecystitis
Burns, sepsis, diabetes, multiple organ systems failure
Cholecystitis
Signs and Symptoms
Sudden pain, often severe and cramping, in RUQ that radiates to right shoulder Point tenderness under right costal margin (Murphys sign) Nausea and vomiting Associated with fatty food intake History of similar episodes Can be relieved by nitroglycerin
Cholecystitis
Appendicitis
Inflammation of vermiform appendix
Usually secondary to obstruction by fecalith May occur in older persons secondary to atherosclerosis of appendiceal artery and ischemic necrosis Signs and Symptoms
Classic: Peri-umbilical pain RLQ pain/cramping, guards upon palpation Nausea, vomiting, low-grade fever Patient found right lateral recumbant in fetal position
Appendicitis
Signs and Symptoms:
McBurneys sign pain on palpation of RLQ Aarons sign Epigastric pain upon palpation of RLQ Rovsings sign Pain LLQ upon palpation of RLQ Psoas sign Pain when patient extends right leg while lying on left side and/or flexes legs while supine
Ruptured appendix - true emergency, temporary relief from pain followed by peritonitis
appendicitis
Appendicitis
Bowel obstruction
Blockage of intestine
Caused by
Adhesions (secondary to surgery) hernias, neoplasms volvulus intussusceptions impaction
Bowel Obstruction
Pathophysiology
Fluid, gas, and air collect near obstruction site causing the bowel to distend impeding blood flow/halting absorption. Water and electrolytes collect in bowel lumen leading to hypovolemia. Bacteria from the gas above the obstruction causes further distension and the distension extends proximally. Finally necrosis and/or perforation occur at the site of the obstruction
Bowel Obstruction
Signs and Symptoms
Severe, intermittent, crampy pain High pitched tinkling bowel sounds Abdominal distension Nausea and vomiting Decreased frequency of bowel movements Change in bowel (semi-liquid or pencil-thin stool) If severe enough can have feces in vomitus
Hernia
Protrusion of abdominal contents into groin (inguinal) or through diaphragm (hiatal)
Often secondary to increased intra-abdominal pressure (coughing, lifting, straining) Can progress to ischemic bowel (strangulated hernia) Signs and symptoms:
Pain increased with abdominal pressure Inguinal hernia may be palpable in groin or scrotum
Crohns Disease
Idiopathic inflammatory bowel disease
Occurs anywhere from mouth to rectum 35-45%: small intestine, 40%: colon
Hereditary
High risk groups: caucasian females, Jews, persons under high stress
Crohns Disease
Pathophysiology
Mucosa of GI tract becomes inflamed and granulomas form invading the submucosa. Muscular layer of the bowel become fibrotic and hypertrophied. All of this causes an increased risk for bowel obstruction, perforation, or hemorrhage.
Crohns disease
Diverticulitis
Diverticula
Pouches in the colon wall Typically found in older people Usually asymptomatic Related to diets with inadequate fiber
Causes:
Diverticula traps feces and becomes inflamed Occasionally causes bright red rectal bleeding Rupture of diverticula can lead to peritonitis and sepsis
Diverticulitis
Signs and Symptoms
Usually left-sided pain May localize to LLQ commonly referred to as leftsided appendicitis Alternating constipation and diarrhea Bright red blood in stool Fever
Diverticulitis
Hemorrhoids
Small masses of veins in anus/rectum
Most frequently develop when patients are in 30s to 50s Most are idiopathic, can be associated with pregnancy, portal hypertension, lengthy driving, constipation Bright red bleeding with pain upon bowel movement May become infected and inflamed
Peritonitis
Inflammation of abdominal cavity lining
Signs and symptoms
Generalized pain, tenderness Abdominal rigidity Nausea, vomiting Absent bowel sounds Patient is resistant to movement
Esophageal Varices
Dilated veins in esophageal wall
Occurs secondary to hepatic cirrhosis, common to alcohol abusers Obstruction of hepatic portal blood flow results in dilation, thinning of esophageal veins
Esophageal Varices
Portal Hypertension
Hepatic scarring slows blood flow Blood backs up in portal circulation Pressure rises Vessels in portal circulation become distended
Esophageal varices
Mallory-Weiss Syndrome
Longitudinal tears at the gastroesophageal junction
Occur as a result of prolonged, forceful vomiting or retching Common in alcoholics May be complicated by presence of esophageal varices
Mallory-Weiss Syndrome
Mallory-weiss syndrome
Aortic Aneurysm
Localized dilation due to weakening of aortic wall
Usually older patients with a past history of hypertension, atherosclerosis May occur in younger patients secondary to:
Trauma Marfans syndrome
Usually occurs just above aortic bifurcation and may extend to one or both iliac arteries
Aortic Aneurysm
Signs and Symptoms
Unilateral lower quadrant pain, low back pain or leg pain May be described as tearing or ripping pain/sensation Pulsatile palpable mass usually above umbilicus Diminished pulses in lower extremities Unexplained syncope, often after bowel movement Evidence of hypovolemic shock
Aortic aneurysm
Ectopic Pregnancy
Any pregnancy that takes place outside of uterine cavity
Most common location in fallopian tube Pregnancy outgrows tube, causing tube wall to rupture Hemorrhage into pelvic cavity occurs
Suspect in females of child-bearing age with abdominal pain and/or unexplained shock
When was the patients LMP?
Ectopic Pregnancy
Ectopic pregnancy does NOT necessarily cause a missed period
Ectopic pregnancy
Ectopic pregnancy
History
Where do you hurt?
Try to pinpoint or have patient pinpoint
Onset of pain?
Sudden = perforation or vascular occlusion Gradual = peritoneal irritation, distension of hollow organ
history
Does the pain travel anywhere?
Gallbladder = angle of right scapula Pancreas = straight through to back Kidney/ureter = around flank to groin Heart = epigastrium, neck/jaw, shoulders, upper arms Spleen = left scapula, shoulder Abdominal aortic aneurysm = low back pain, radiating to one or both legs
History
How long have you been hurting?
> 6 hours = increased probability of surgical significance
Urine changes?
Change in frequency, color, or odor; or increased urgency,
History
Bowel movements?
Change in bowel habits? Change in color? Change in odor?
Bright red Melena (black, tarry, and foul-smelling) Dark (suspect bleeding)
History
Last menstrual period?
Abnormal bleeding? In females, lower abdominal pain = gynecological problem until proven otherwise In females of child-bearing age, lower abdominal pain = ectopic pregnancy until proven otherwise
Physical exam
Position and general appearance
Still refusing to move = inflammation or peritonitis Extremely restless = obstruction
Physical exam
Vital Signs
Tachycardia = more important sign of volume loss than a falling blood pressure Rapid shallow breathing = possible peritonitis Consider performing a tilt test
Bowel sounds
Auscultate before palpating Listen for 1 minute in each quadrant Absent sounds= possible peritonitis, shock High pitched tinkling sounds = possible bowel obstruction
Physical exam
Palpation
Palpate each quadrant Palpate area of pain last Do not check rebound tenderness in prehospital setting All abdominal tenderness is significant until proven otherwise
Management
Oxygen by NRM
IV of Lactated Ringers or Normal Saline Solution Keep patient warm
Management
Treat pain per protocols (some believe that masking/treating pain is wrong)
Give a thorough report to receiving facility For aortic aneurysm considering taking patient to hospital that is capable of CT surgery