Beruflich Dokumente
Kultur Dokumente
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Phisiology
-Eyelid : Protection Stimulus Blinking 5-10 w/ contact N VIII wo/ contact : aff N V, eff N VII Spread the tear meniscus
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- Precorneal Tear Film ; Lipid, Aqueous,Mucin
nutrients,O2,leucocytes,growth factors,lactoferrin,Ig A,etc. -- Refraksi, Infeksi - Ocular Surface Epitel Kornea : desmosomes,tight junction limbal epitelium
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Epitel konjungtiva mucosal immune defense system dendritic, lymphocytes, pmn,sel plasma, sel mast.
Blood Supply
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Blepharitis Seborrheic Anterior eyelid margin Crusting, oily or greasy on eyelids,eyelashes Redness eyelid, burning, foreign body sensation 15% w/ keratitis or conjunctivitis 1/3 aqueous tear deficiency
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Management
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Chalazion lipogranulomatous inflammation of meibom or zeis glands Spontan Nodul slowly, painless Erythematous 2nd Blurr vision Ca sebaceous
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Management Hot compresses Intralesion steroid injection Incision or drainage Systemic tetracyclines Recurrent Ca meibomian gland
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Hordeolum Eyelid Inflammatory or infections nodul Ext : Zeis or lash follicles Int : Meibomian Localized purulent abscess Sta.aureus Painfull, tender,redness Self limited Internal chalazia
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Management Warm Compresses + massage Topical < Systemic AB Incision and drainage Steroid Injection
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Conjunctivitis Allergic Rx hypersensitive type I Alergen : airborne Symptoms : itching, eyelid swelling, conj.hyperemia, chemosis, mucoid discharge Short lived, episodic
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Management
Avoidance alergen exposure Cold compresses Artificial tears Topical antihistamine and mast cell stabilizers Topical NSAID Topical Steroid Topical Vasokonstrictors
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Vernal Keratokonjuctivitis
Rx hypersensitive type I and IV seasonally, bilateral male children, atopic history itchy, blefarospasme, photophobia, blurred vision, mucoid discharge
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Management Topical antihistamines Mast cell stabilizers Topical NSAID Topical steroid / immunomodulator Supratarsal injection of steroid
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Bacterial
Spontan and epidemic Direct contact, aerogen, Sight threatening : Virulency, Conjunctival inflammmation, purulent discharge
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Onset Slow Severity Mild-moderate Organism Staph. Aureus Moraxella lacunata Proteus sp Enterobacteriaceae Pseudomonas
Acute / subacute
Moderate-severe
Hyperacute
Severe
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Management Topical AB therapy, qid, 5-7 days Gram stained, culture
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Gonococcal conjunctivitis
explosive onset, severe purulent discharge, chemosis untreated corneal infiltrates melting perforation STD, direct contact Neonatal bilateral, 3-5 days postpartus Complication : corneal ulcer, perforation, endophthalmitis
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Management AB systemic, singledose Frequent Topical AB eo Lavages
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Viral
Adenoviral keratokonjunctivitis simple follicular conjunctivitis Pharyngoconjunctival fever Epidemic keratokonjunctivitis Unilateral or bilateral Pseudomembrane/ membran
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Management Supportive Cold compress Artificial tears Topical AB 2nd infection Steroid topical
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Herpes Simplex Direct contact Unilateral blepharoconjunctivitis Follicular inflammatory Palpable preauricular lymph node Cutaneous eyelid margin vesicles
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Management Primer Self limited 2nd AV systemic, topical
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Corneal Ulcer Bacterial Sight threatening Explosive onset, rapidly progressive Untreated corneal perforation Risk factor : Contact lens wear Trauma Contaminated ocular medications Impaired defense mechanism
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Pathogenesis
S aureus adhesins bind to corneal stromal P aeruginosa injured epithelial cellsmolecular receptor proliferation Clinical presentation pain, photophobia, decreased vision sharp epithelial demarcation, dense, supurative,stromal edema
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Fungal Corneal trauma w/ plant or organic material Contact Lens wear Prolonged used of steroid topical & systemic Gray white, dry infiltrat, feathery/filamentous margins Multifocal, satellite infiltrates Endothel plaque, hypopion
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Cause: Candida spp
Aspergillus spp, Fusarium Management Anti fungal, topical & systemic Fusarium Natamycin 5% Aspergillus Amphotericin B Candida Cyclopegic