Senior lecturer Audiology Prgramme School of Helth Sciences Universiti Sains Malaysia drzuraida@yahoo.com http://bal-exercise.blogspot.com/ *
Elicit history and evaluate dizziness Understand vestibular testing Knows differential diagnosis in dizziness Understand management concepts
3 *What is a balance disorder?
a disturbance that causes an individual to feel unsteady, giddy, woozy, or have a sensation of movement, spinning, or floating. http://www.nidcd.nih.gov/health/balance/balance_disorders.asp
*Dizziness
*Dizziness subtypes
Philip D. Sloane, MD, MPH; Remy R. Coeytaux, MD; Rainer S. Beck, MD; and John Dallara, MD Dizziness: State of the Science Ann Intern Med. 2001;134:823-832.
Dizziness subtype Type of sensation Temporal Characteristics Other Specification Vertigo A feeling one that one or Ones surroundings are Moving (spinning) Episodic vertigo (seconds to days) Continuous vertigo (most of the time for at least a week) Characteristics, duration, and date of the first episode, length of episodes; and exacerbating factors. Presyncope A lightheaded, faint feeling, as though one were about to pass out. Typically occurs in episodes lasting seconds to hours. 1) Has syncope ever occurred during an episode 2) Do episodes occur only when the patient is upright, or do they occur in other positions? 3) Are episodes associated with palpitations, medication meals, bathing, dyspnea, or chest discomfort? Disequilibriu m Unsteadiness: - felt in lower limb - prominent when standing or walking - relieved by sitting or lying down Usually present. Although it may fluctuate in intensity Identify whether symptom occurs in isolation or accompanies another dizziness subtype; describe exacerbating factors. Other dizziness; anxiety- related, ocular, tilting environment , other A feeling not covered by the above definitions, may include swimming or floating sensations, vague lightheadedness, or feeling of dissociation. Present all the time ~ days/weeks/years -Is dizziness a/w anxiety or hyperventilation? - Was change in vision connected with dizziness onset? - Environment is tilting sideways (suggests an otolith problem? http://www.aan.com/go/education/curricula/family/chapter5/section1 VERTIGO History Clinical Examination Investigations Nature Duration Associated symptoms Precipitating factors
OBJECTIVE VNG VEMP (Ocul & Cer.) V-Hit EcohG Posturography Rotating Chair Subjective vertical test SUBJECTIVE Malay Version VSS Malay version Modified VSS
Gen. exam. Eye exam. Aural exam. Neurology exam. Specific test * *Chief complaints *Dizzy !! Lightheadacheness!! Headache!! Floating!! Presyncope!! *Whirling !! Swaying!! Unsteadiness!! *True vertigo or not ? A) Nature *B) Duration of attack: BPPV-seconds TIA-minutes Menieres-hours Vestibular Neuronitis-Days Ototoxins-years (See Hain, 1997) *C) Associated symptom positional related, hearing disturbance, headache, stress D) Precipitating/ provoking factors
Spinning Vestibular Unsteadiness Central lesion Presyncopal/ feeling faint Orthostatic Unspecific (dissociation) Psychology Otoconia exist within a part of the inner ear crystals of calcium carbonate derived from a structure in the ear called the "utricle *
Duration of episode Suggested diagnosis Seconds Peripheral: unilateral loss of vestibular fx, late stage of acute vestibular neuronitis & MD Seconds - minutes BPPV. perilymphatic fistula Minutes one hour Posterior transient ischemic attack; perilymphatic fistula Hours MD; perilymphatic; migraine. Acoustic neuroma Days Early acute vestibular neuronitis*stroke; migraine; Multiple sclerosis Weeks Psychogenic (constant ~weeks w/o Improvement) *-Early acute vestibular neuritis can be two days or as long as one week or more . * Symptom Suggested diagnosis Aural fullness Acoustic neuroma;Menieres disease Ear or mastoid pain Acoustic neuroma; acute middle ear disease (e.g; otitis zoster oticus) Facial weakness Acoustic neuroma; herpes zoster oticus Facial neurologic CPA tumour; CVA; MS Headache Acoustic neuroma; migraine Hearing loss MD; PLF; acoustic neuroma; cholesteatoma;otosclerosis;TIA or stroke involving anterior cerebella artery, herpes zoster oticus Imbalance Acute vestibular neuronitis(usually moderate); CPA tumor (usually severe) Nystagmus Peripheral or central vertigo Phonophobia, photophobia Migraine Tinnitus Acute labyrinthitis; acoustic neuroma; Menieras disease * Provoking Factor Suggested diagnosis Changes in head position Acute labyrinthitis;BPPV; CPA Tumour ;multiple sclerosis (MS); PLF Spontaneous episodes AVN; CVA (stroke or TIA; MD ; migraine; MS Recent URTI Acute vestibular neuronitis (AVN) Stress Psychiatric or psychological causes; migraine Changes in ear press., trauma, excess. straining, loud noises Perilymphatic fistula (PLF) Past medical history -vascular risk factors -ear surgery
Family History -Similar disorder ? -Migraine
Drug History -present and past exposures to ototoxins, antihypertensives.
General Medical condition Blood pressure (lying and sitting) Cardiac arrhythmias Neurological Examination cranial nerve palsies (Multiple sclerosis , acoustic neuroma, advanced brain stem tumor or basilar artery insufficiency Neck examination * *Gait *Cranial nerves *Motor power and reflexes (e.g. Babinski) *Sensory (proprioception)
Cerebellar sign ; a) Finger to nose b) Dysdiadokinesia c) Tandem gait (hell to toe) with eye open and closed *Rombergs test Fall to one side: - Posterior column lesion - Acute ipsilateral vestibular lesion
*Fukuda @ Unterberger test -Walk on the spot for 2 minutes with eye closed -Positive when patient turn > 45 -Ipsilateral peripheral lesion
* l) Spontaneous nystagmus
MD, Vestibular Neuronitis, central disorders, to
rule out Psychiatric (used Frenzel's goggles)
ii) Range of eye movements
Gaze paresis
Ocular paresis
iii) Cover test for strabismus : a
deviation or misalignment eyes.
strabism eye muscle position ~ one or both
eyes may turn in (esotropia), out (exotropia), up
(hypertropia) or down (hypotropia). http://dewa-dony.blogspot.com/2008/10/strabismus.html *
- to detect vestibular neuritis, acoustics, and to rule out psychiatric disturbance
Head-shake test - (Hain et al, 1987) 75% sensitive but wrong side in 1/4 of the time.
- to detect ototoxicity and other bilateral vestibulopathies
Dynamic illegible 'E' test or DIE (Longridge, 87).
* 1. DIX-HALLPIKE TEST
-Rotatory upbeating; Post SCC
-Rotatory downbeating; Ant. SCC
video 1
video 2
video 3 cupulo
Treatment for Post. SCC- Epleys menourve
2. ROLL TEST
- horizontal nystagmus
video 1
Treatment- Barbeque menourve
Video 1 nystagmus *
3) Fistula Test or Valsalva test- Occasionally helpful
4) Hyperventilation test 30 seconds, look for nystagmus. Helpful when nystagmus changes direction compared to vibration or head-shaking nystagmus.
5) Carotid Sinus Compression - for syncope patients.
6) Vertebral artery test - for persons with neck-
position induced vertigo (cervical vertigo). * Feature Peripheral Vertigo Central Vertigo Nystagmus Mix horizontal & tensional; inhib. by fixation of eyes; Fades after a few days; not change direction with gaze to either side Purely vertical , horizontal, or torsional; not inhibited by fixation of eyes ; last weeks to months; change direction With gaze towards fast phase Of Nystagmus Imbalance Mild to moderate; able to walk Severe; unable to stand or walk Nausea, vomiting May be severe Varies Hearing loss, tinnitus Common Rare
Neurologic Sx Rare Common Latency (follow. pro- vocative) Longer (up to 20 seconds) Shorter (up to 5 seconds) *Objective Vestibular Tests Indication; Assess vestibular function Locate the lesion organ/part Causative factor/etiology Vestibular rehabilitation assessment *Videonystagmography (VNG)/Electronystagmograpy (ENG) *Video Head impulsetTest (V-HIT) *Vestibular evoked myogenic potential (VEMP) - Ocular & cervical *Electrocochleargraphy (EcohG) *Rotating chair *Computerized Posturography (CDP) *Subjective vertical test
Reproduced with permission from Luxon LM, Davies RA, eds. Handbook of vestibular medicine. London: Whurr Publishers, 1997. 40 *
Cawthorne cookseey exercise (CCE)
Customised CCE
Bal Ex : Homebased video module for balance exercises = customised CCE +Prayer movement
* 1. Kroenke K, Lucas CA, Rosenberg ML, et al. Causes of persistent dizziness: a prospective study of 100 patients in ambulatory care. Ann Intern Med. 1992;117:898-904.
2. LM Nashner, FO Black, and C Wall, 3d Adaptation to altered support and visual conditions during stance: patients with vestibular deficits J. Neurosci. 1982 2: 536- 544.
3. Shupert CL, Black FO, Horak FB & Nashner LM (1988) Coordination of head and body in response to support surface translations in normals and patients with bilaterally reduced vestibular function. In Amblard B, Berthoz A, Clarac F (eds) Posture and gait: Development, Adaptation and Modulation. New York: Elsevier Science Publishers.
4. Allum, J.H.J., Honegger, F. and Pfaltz, C.R. (1989) The role of stretch and vestibulo-spinal reflexes in the generation of human equilibriating reactions. Progress in Brain Research 80, 399-409
5. Bles W, de Jong JMBV. Uni- and bilateral loss of vestibular function. In: Disorders of posture and gait.Bles W, Brandt T, eds. (1986) Amsterdam: Elsevier, 1986, PP 127-139
6. Fregly AR (1974) Vestibular ataxia and its measurement in man. In: Kornhuber HH (ed) Handbook of Sensory Physiology,. vol VI. Springer, New York, pp 321360
7. Handbook of Balance Function Testing by Gary P. Jacobson (Author), Craig W. Newman (Author), Jack M. Kartush Singular; 1 edition (October 1, 1997)