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PREVENTION
By: Karen C. Lyon, PhD, APRN, ACNS, NEA
Presented by: Wanda Butaud RN, BSN, WOCN
Spring 2015
http://www.ncbi.nlm.nih.gov/pmc/articles/PMC281019
2/
http://www.justice.gov/opa/pr/2003/June/03_civ_386.
A wound is a disruption of
normal anatomic structure and
function that is usually inclusive
of the skin.
Anatomic continuity
Function
A
C
U
T
E
VS.
C
H
R
O
N
I
C
Shear
Shear is caused by tissue layers sliding
against each other. This results in
disruption or angulation of blood
vessels, usually at the fascia level
Purple areas on sacrum and heels
Ulcers start deep and work outward
Irregularly shaped
Undermined
Moisture
Excessive moisture causes
maceration which acts as a factor in
skin breakdown
Incontinence
Wound drainage
Excessive sweating
Nutritional Compromise
Altered skin integrity
Delayed wound healing
Decreased resistance to infection
Nutritional supplements
Feeding tubes
Anabolic steroids
Contributing Factors
Activity and mobility
Loss of sensation
Age
Disease states
Previous ulcers
Medications
Granulation Tissue
Formation
KEY Cells
Macrophages
Fibroblasts
Endothelial Cells
KEY Elements
Macrophage derived
- PDGF
- TGF-
- FGF-2 (bFGF)
MMPs
- Cell migration
Reference:
Reference: Monaco
Monaco JL,
JL, Lawrence TL. Acute wound healing; an overview.
overview. Clinics
Clinics in
in Plastic
Plastic Surgery 30
30 (2003):
(2003): 1-12.
1-12.
Reepithelialization
Keratinocyte
Keratinocyte Proliferation
Proliferation
Keratinocyte
Keratinocyte Migration
Migration
Relevant
Relevant Growth
Growth Factors
Factors
Keratins
Keratins
injury
Topical
steroids
Local toxins
Trauma, pressure
Foreign bodies
Local malignancy
Connective tissue
disease
Immunological
disorders
Chemotherapeutic
agents
Extremes of age
Systemic steroids
Hepatic failure
NumberofCells
9000
8000
7000
6000
NB
5000
YA
4000
OA
3000
2000
1000
0
10
11
12
13
Day
30
#CellsX104
25
20
15
Diabetic
10
NonDiabetic
5
Days
Pathophysiology
External pressure impedes local
blood flow to skin (20mmHg),
muscle
Muscle and subcutaneous tissue
more susceptible to injury than
skin
Necrosis generally over bony
prominence
Loss of skin moisture
Pressure Ulcers
Stage 2
Stage 1
Stage 3
Stage 4
Heel
8%
Posterior
Calf
Sacrum
23%
Dorsum of
foot and
ankle
Knee Thigh
6%
Iliac
crest 4%
Anterior Cheek
Chest
and ear
Acromion
process
Points of Pressure
Lateral Position
Malleolus 7%
Medial and
lateral
condyles
Greater trochanter
15%
Ribs
Ear
Points of Pressure
Sitting Position
Scapula
Popliteal
Plantar surface of
foot
Heel
Braden Scale
http://education.woundcarestrategies.com/colopla
st/resources/BradenScale.pdf
Pathophysiology
Prolonged venous hypertension
Valvular incompetence, DVT, calf
muscle dysfunction
Enlarged dermal capillaries, reduced
capillary number, microvascular
thrombosis, increased permeability of
microlymphatics
WBC sequestration,
ischemic/reperfusion injury
Pathophysiology
Diabetic Wounds/Amputations
http://www.medicalcriteria.com/site/index.ph
p?option=com_content&view=article&id=114%3Ad
btfoot&catid=49%3Adiabetes&Itemid=80&lang=en
Wound cleansing
Yes
Clean
Supplies (Includes
solutions and dressing
supplies)
Normal saline solution or
commercially prepared
wound cleanser - sterile;
maintain as clean per
care setting policy*
Routine dressing
change without
debridement
Yes
Clean
Dressing change
with mechanical,
chemical, or
enzymatic
debridement
Yes
Clean
Dressing change
with sharp,
conservative
bedside
debridement
Yes
Sterile
Sterile
Sterile
Intervention
Handwashing Gloves
Instruments
Irrigation with sterile device;
maintain as clean per care
setting policy
* "Maintain clean as per care setting policy" means that each care setting must address the parameters
for maintenance,
such as expiration
dates for supplies,
consideration of
cost, and correct
interpretation of
the manufacturer's
Farrow Wraps
NURSING IMPLICATIONS
Nutritional
Skin care
Pressure relief/reduction
Risk assessment
Support
Outcomes
Early
Education
Treatment
Documentation
Tracking