MALARIA (EPI) schistosomias

1
is
(clinical
-P. falciparum mani)1
predominates
-P. vivax
SHISTOSOMIA
-equal
SIS
-P. malariae
CLINICAL
-P. ovale
FEATURES
-Endemicity
-Three stages,
-children 29
-phase of
years of age
cercarial
as
invasionhypoendemic swimmers
(<10%),
itch
mesoendemic -Cercarial
(1150%),
dermatitis
hyperendemic During worm
(5175%),
maturation
and
acute
holoendemic
schistosomias
(>75%);
is or
In holo- and
Katayama
hyperendemic fever
areas
-chronic

Tyhpoid fever
(etio) 1
-gramnegative
bacilli,family
Enterobacteri
aceae
two species:
S.
choleraesuis6, S. bongori.
-S. enterica
-Salmonella
Typhimurium.
-subspecies
are classified
into
>2400serotyp
es
gram(-), nonsporeforming,
facultatively
anaerobic

Hepatitis (DX)
-Liver
Enzymes
-Antibody
Tests
-Direct Viral
Measures
-imaging: U/S,
CT, MRI
-liver biopsy
-Liver protein
and hepatitis
-Liver
Function Tests

Malaria( epi)
2

schistosomias Tyhpoid fever

is
(etio) 2
(clinical mani)
-stable
2
-Produce acid
transmission.
on glucose
- unstable
-intestinal
fermentation,
transmission. phase- colicky reduce
abdominal pain, nitrates, and
epidemic bloody
results in
do not
diarrhea,
mortality-all
produce
anemia,
age groups.
cytochrome
fatigue, growth
-principal
oxidase.
retardation.
determinants -severity of
-all except S.
of the epi
Gallinarum
intestinal
-transmission schistosomiasis Pullorum are
-hepatosplenic
of malaria
motile
directly_propo phase
all but
rtional to the -subsequent
S.Typhi
phases of
ff:
produce gas
infection,
-Mosquito
(H2S) on
-schistosomal
longevity
sugar
liver disease
-at
fermentation.
-Bleeding from
temperatures esophageal
varices
below 16
-late-stage
18C,