Beruflich Dokumente
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Group 3
7 jumps
1. Clarifying unfamiliar terms
2. Problem definitions
3. Brainstorm
4. Analyzing problem
5. Formulating learning issues
6. Search journal
7. Reporting
Tutorial group 3
Maarja Vaarsi
Tanel Lepik
Lizz van Duin
Isabelle van der
Lennart Wasmoeth
Eveline Verheij
Hannah Brand
1. Carifying unfamiliar terms
1. Schuffners stipplings? =Characteristics of
P.ovale/P.vivax
2. Liver steatosis? = fatty liver
2. Problem definitions
1. Differential diagnosis?
2. Why does he have liver steatosis? Why does he have high
levels of triglycerides?
3. Why does he have fever?
4. What does parasitaemia of 1.5% mean?
5. Why does he still have malaria although he was treated
with chloroquine?
6. Why was the disease latent for 4 years?
7. What is the mechanism of hematuria?
8. How can previous mild malaria lead to severe malaria?
9. Does he have both P.vivax and P.ovale infection?
3. Brainstorm (1)
1. DD:
Malaria
Dengue
Leptospirosis
Influenza
Hepatitis
Typhoid fever
Leukaemia
Babesiosis
3. Brainstorm (2)
2. Why does he have liver steatosis? Why does he have
high levels of triglycerides?
Spirozytes go into the liver cells and destroy hepatocytes.
As a result fat cells accumulate in the gaps.
KEEP IN MIND!
3. Why does he have a fever?
Infection
Fever rises when red blood cell break and merozytes go
into the bloodstream to infect other cells
KEEP IN MIND!
3. Brainstorm (3)
4. What does parasitaemia of 1.5% mean?
1.5% of all the red blod cells is infected (?)
Normal value for P.vivax/ovale infection