containing venules, arterioles, and smooth- muscle fibers that are located in the anal canal. Three hemorrhoidal cushions are found in the left lateral, right anterior, and right posterior positions. Hemorrhoids are thought to function as part of the continence mechanism and aid in complete closure of the anal canal at rest Schwartz's Principles of Surgery, 2010 Colon and Rectal Surgery, 2005 Functions contribute to anal continence by forming a spongy bolster, which cushions the anal canal and prevents damage to the sphincter mechanism during defecation. This tissue also acts as a compressible lining that allows the anus to close completely
Handbook of Colorectal Surgery, 2003
Incidence Age: Most patients who develop hemorrhoids are between the ages of 30 and 50. Hemorrhoids occur in both sexes. However, pregnancy and childbirth are the prime causes of hemorrhoids in young females. Medical history: Some patients with leukemia or bleeding diathesis will have complications from hemorrhoids. Excessive straining Lack of fiber in diet Spending hours or reading books on the bathroom weight lifters and tennis players practice anorectal intercourse Prolonged sitting and lack of activity Pathophysiology chronic straining secondary to constipation or occasionally diarrhea may result in pathologic hemorrhoids. Eventually, with repeated straining, the hemorrhoids may lose their attachment (Treitzs ligaments) to the underlying rectal wall, leading to the prolapse of the tissue into the anal canal. The engorged tissue becomes more friable, which may contribute to bleeding. These tissues communicate with the superficial subcutaneous venules at the anal verge, which may result in external hemorrhoidal dilation Pathophysiology Current theories : anal "cushions.: aggregations of blood vessels (arterioles, venules, and arteriolar- venular communications), smooth muscle, and elastic connective tissue in the submucosa that normally reside in the left lateral, right posterolateral, and right anterolateral anal canal. disintegration of the anchoring and supporting connective tissue and the terminal fibers of the longitudinal muscle above the hemorrhoids allows these structures to slide distally. Maingot's, 2007 Symptoms Principal : bleeding & prolapse Secondary : mucus discharge, pain, pruritus, anemia, fecal incontinence Symptoms Painless bleeding occurs from internal hemorrhoids, is usually bright red, and is associated with bowel movements. The blood will occasionally drip into the commode and stain the toilet water bright red. After trauma by firm stools or forceful bowel movements, bleeding may continue to occur with bowel movements for severaldays. The bleeding will often then resolve for a variable period of time. Symptoms Prolapse below the dentate line area can occur, especially with straining, and may lead to mucus and fecal leakage and pruritus. Pain is not usually associated with uncomplicated hemorrhoids but more often with fissure, abscess, or external hemorrhoidal thrombosis. Gastrointestinal Bleeding, 2010 Manage hemorrhoid complication
If left untreated, the edema progresses to ulceration
and necrosis. relieving the pain with analgesics (intravenous if necessary); Reducing perianal swelling with either hot soaks, sitzbaths, or ice packs; bed rest; and prevention of constipation Manual reduction, urgent hemorrhoidectomy Diagnostic Rectal examination Proctoscopy/anoscopy It is mandatory that colonoscopy be performed in high-risk patients to exclude other sources of bleeding, such as carcinoma or proctitis (e.g., for patients more than 40 years of age and those with a family history of colorectal neoplasia or a change in bowel habits). Classification
Grade Ibleeding without prolapse.
Grade IIprolapse with spontaneous reduction. Grade IIIprolapse with manual reduction. Grade IVincarcerated, irreducible prolapse. External hemorrhoids are covered with anoderm and are distal to the dentate line; they may swell, causing discomfort and difficult hygiene, but cause severe pain only if actually thrombosed. Internal hemorrhoids cause painless, bright red bleeding or prolapse associated with defecation. Treatment Treatment Early disease (Grade I and early Grade II) is often managed with medications designed to cause vasoconstriction and treat inflammation for the engorged friable hemorrhoid. More advanced disease frequently requires operative management which may include sclerotherapy, cryosurgery, infrared coagulation, rubber band ligation, and various modes of surgical excision. These therapies attempt to remove the redundant tissue and create cicatrices to fix the remaining mucosa within the anal canal once again. Conservative treatment there are three mainstays of conservative management: 1. Bulking agents 2. Sitz baths/warm compresses 3. Local applications Sclerotherapy Rubber band ligation Stapled Hemorrhoidopexy Open hemorrhoidectomy