Beruflich Dokumente
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Overview
Anatomy Review
Chest Trauma
Chest Injuries
2
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Anatomy Review
3
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Chest Trauma
4
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Chest Trauma
5
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Chest Trauma
6
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Chest Trauma
7
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Chest Trauma
8
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Chest Trauma
9
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Chest Trauma
10
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Chest Trauma
Management
– Ensure patient has adequate
oxygenation and perfusion
– Provide high-flow oxygen, ventilating
when necessary
– Halt any obvious bleeding
11
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Chest Trauma
Management
– Support circulation when needed
– Rapidly transport patient to definitive care
12
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Chest Trauma
Transport
– Transport patient to a hospital with the
capability to diagnose and treat serious
traumatic injuries
– Arrange for ALS (advance life support)
intercept as guided
by local protocols
– Notify receiving hospital so staff
can prepare
13
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Chest Injuries
14
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Chest Injuries
15
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Chest Injuries
16
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Chest Injuries
17
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Chest Injuries
18
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Chest Injuries
19
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Open Chest Wound
Watch this animation illustrating an open chest wound.
https://youtu.be/XPjhcCDeBk4
20
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Stop and Review
21
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Chest Injuries
Tension pneumothorax
– Buildup of pressure in pleural space resulting in
decrease in blood pressure
– Potentially life-threatening condition that must be
treated immediately
– Can occur in blunt or penetrating chest trauma
22
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Chest Injuries
23
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Chest Injuries
24
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Chest Injuries
25
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Chest Injuries
Rib fractures
– Local swelling and tenderness may be
the only sign of a broken rib
– Can be very painful
– Patients often present with guarding
and shallow breathing
26
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Chest Injuries
27
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Chest Injuries
28
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Chest Injuries
Flail segment
– When three or more ribs are broken in two or
more places, a rib-cage segment may detach
from the rest
– Flail segment is free floating
29
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Chest Injuries
Flail segment
– Paradoxical movement: movement of flail segment
in opposite direction of the rest of the chest wall
– Paradoxical movement can significantly impair
breathing and cause injury to the underlying lung
30
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31
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Flail Chest Segment
https://youtu.be/uJHfX1RFkF0Watch
32
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Chest Injuries
33
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Chest Injuries
34
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Chest Injuries
Pulmonary contusion
– Bleeding into the lung itself is a
pulmonary contusion
– Bleeding and edema can impair
gas exchange, causing hypoxia
– Soft crackles may be heard over
injury site
– Chest pain, point tenderness, and
localized swelling over area of impact
35
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Chest Injuries
36
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Chest Injuries
Cardiac contusion
– Can impair heart’s ability to pump
– Bleeding into heart tissue can cause heart to
beat irregularly
– Irregular pulse should alert EMT to possibility
of a cardiac contusion
37
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Chest Injuries
38
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Chest Injuries
Aortic injury
– In sudden decelerations such as high-speed head-
on MVCs, body organs are thrown forcefully
against the front of the body
– Most significant tear: aorta
– If tear is complete, patient will die in minutes
• Incomplete tears bleed severely
39
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Chest Injuries
40
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Chest Injuries
Traumatic asphyxia
– Rapid ejection of blood and air out of chest
– Rapid compression of chest increases internal
pressure dramatically
• Blood is immediately forced out of the chest and into the
vessels in the neck, head, and face
41
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Chest Injuries
Traumatic asphyxia
– Neck veins immediately become distended
– Cyanosis is apparent in face
– Bleeding in the eyes’ sclera may occur
42
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Chest Injuries
43
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Stop and Review
44
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Chapter 34
Care of Critically Ill Patients with
Respiratory Problems
Pulmonary Embolism
Collection of particulate matter—solids,
liquids, air—that enters venous circulation
and lodges in pulmonary vessels
Usually occurs when blood clot from a VTE in
leg or pelvic vein breaks off; travels through
vena cava into right side of heart
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Pulmonary Embolus
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Risk Factors
Prolonged immobilization
Central venous catheters
Surgery
Obesity
Advancing age
Conditions that increase blood clotting
History of thromboembolism
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Health Promotion & Illness
Prevention
Smoking cessation
Weight reduction
Increased physical activity
If traveling or sitting for long periods, get up
frequently and drink plenty of fluids
Refrain from massaging/compressing leg
muscles
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Clinical Manifestations
Respiratory
– Dyspnea, tachypnea, tachycardia, pleuritic chest
pain, dry cough, hemoptysis
Cardiac
– Distended neck veins, syncope, cyanosis,
systemic hypotension, abnormal heart sounds,
abnormal ECG
Low grade fever, petechiae, flu-like
symptoms
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Laboratory Assessment
ABGs
Pao2 – Fio2 ratio falls (O2 level in blood
compared to O2 that is breathed)
Pulse oximetry
Imaging assessment
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Nonsurgical Management
Oxygen therapy (nasal cannula, mask)
Continuous patient monitoring
Obtain adequate venous access
Continuous monitoring of pulse oximetry
Drug therapy
– Anticoagulants
– Fibrinolytics
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Interventions
Ensure appropriate antidotes are present on
the nursing unit!
Assess for bleeding every 2 hr
Examine all stool, urine, drainage, vomitus for
gross blood; test for occult blood
Measure abdominal girth every 8 hr
Monitor laboratory values
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Surgical Management
Embolectomy
Inferior vena cava filtration
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Acute Respiratory Failure
ABG value of PaO2 <60 mm Hg, SaO2; <90%;
or PaCO2 >50 mm Hg with pH <7.30
Ventilatory/oxygenation failure
Patient is always hypoxemic
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Ventilatory Failure
Physical problem of lungs or chest wall
Defect in respiratory control center in brain
Poor function of respiratory muscles,
especially diaphragm
Extrapulmonary causes
Intrapulmonary causes
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Oxygenation Failure
Insufficient oxygenation of pulmonary blood
at alveolar level
Ventilation normal, lung perfusion decreased
Right to left shunting of blood
V/Q mismatch (ventilation/perfusion)
Low partial pressure of O2
Abnormal hemoglobin
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Combined
Ventilatory/Oxygenation
Failure
Often occurs in patients with abnormal lungs
(e.g., chronic bronchitis, emphysema, asthma
attack)
Diseased bronchioles and alveoli cause
oxygenation failure; work of breathing
increases; respiratory muscles unable to
function effectively
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Dyspnea Interventions
Oxygen therapy
Position of comfort
Relaxation, diversion, guided imagery
Energy-conserving measures
Drugs
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Acute Respiratory Distress
Syndrome (ARDS)
Persisting hypoxia
Decreased pulmonary compliance
Dyspnea
Noncardiac-associated bilateral pulmonary
edema
Dense pulmonary infiltrates seen on x-ray
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Causes of Lung Injury in ARDS
Systemic inflammatory response is common
pathway
Alveolar-capillary membrane injured
– Intrinsic causes—sepsis, shock
– Extrinsic causes—aspiration, inhalation injury
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Diagnostic Assessment
Lower Pao2 value on ABG
Refractory hypoxemia
“Whited-out” (ground glass) appearance to
chest x-ray
No cardiac involvement on ECG
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Interventions
ET intubation, conventional mechanical
ventilation with PEEP or CPAP
Drug and fluid therapy
Nutrition therapy
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Chest tube
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Verifying Tube Placement
Chest x-ray
Assess for breath sounds bilaterally,
symmetrical chest movement, air emerging
from ET tube
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Stabilizing the Tube
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Endotracheal Tubes: Nursing
Care
Assess tube placement, minimal cuff leak,
breath sounds, chest wall movement
Prevent movement of tube by patient
Check pilot balloon
Soft wrist restraints
Mechanical sedation
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Mechanical Ventilation
Ventilator types:
– Negative-pressure
– Positive-pressure
• Pressure-cycled
• Time-cycled
• Volume-cycled
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Modes of Ventilation
Assist-control ventilation (AC)
Synchronized intermittent mandatory
ventilation (SIMV)
Bi-level positive airway pressure (BiPAP)
Others
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Ventilator Controls and
Settings
Tidal volume (Vt)
Rate—breaths/min
Fraction of inspired oxygen (Fio2)
PIP
CPAP
PEEP
Flow and other settings
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Nursing Management
Always assess patient first, ventilator second
Monitor patient response
Manage ventilator system
Prevent complications!
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Complications
Cardiac:
– Hypotension
– Fluid retention
– Valsalva maneuver
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Complications (cont’d)
GI
Nutritional
Infections—ventilator-associated pneumonia
(VAP)
Muscle deconditioning
Ventilator dependence
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Complications (cont’d)
Respiratory:
– Barotrauma
– Volutrauma
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Weaning
Process of going from ventilator dependence
to spontaneous breathing
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Extubation
Hyperoxygenate patient
Thoroughly suction ET and oral cavity
Rapidly deflate ET cuff
Remove tube at peak inspiration
Instruct patient to cough
Monitor patient every 5 min; assess
ventilatory pattern for respiratory distress
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Chest Trauma
About 25% of traumatic deaths result from
chest injuries
– Pulmonary contusion
– Rib fracture
– Flail chest
– Pneumothorax
– Tension pneumothorax
– Hemothorax
– Tracheobronchial trauma
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Pulmonary Contusion
Potentially lethal injury
May be asymptomatic at first, later develop
respiratory failure
Bloody sputum, decreased breath sounds,
crackles, wheezes
Treatment—maintenance of ventilation and
oxygenation
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Rib Fracture
Chest usually not splinted by tape or other
materials
Main focus—decrease pain so adequate
ventilation is maintained
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Flail Chest
Paradoxical chest movement—“sucking
inward” of loose chest area during inspiration,
“puffing out” of same area during expiration
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Tension Pneumothorax
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Tension Pneumothorax
(cont’d)
Assessment findings:
– Asymmetry of thorax
– Tracheal movement away from midline toward
unaffected side
– Respiratory distress
– Absence of breath sounds on one side
– Distended neck veins
– Cyanosis
– Hypertympanic sound to percussion
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Hemothorax
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Tracheobronchial Trauma
Caused by blunt trauma, rapid deceleration
Tracheal lacerations
Upper airway obstruction
Cricothyroidotomy, tracheotomy
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A 65-year-old woman is brought to the ED by
her husband with new onset shortness of
breath. She had an abdominal hysterectomy 5
days ago. Her husband states that she stayed
in bed since she was discharged from her
surgery 48 hours ago, because she feels very
short of breath when she gets up.
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Prolonged immobility; advancing age; recent
surgery.
88
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(cont’d
)
During triage, the following vital signs and
assessments are noted:
Temp – 99.6° F BP – 80/44 mm Hg
P – 126 (sinus tachycardia) R – 28 and labored
O2 saturation – 84% (room air) Crackles bilaterally
Petechiae across chest and in axillae
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The patient may have a pulmonary embolism.
She could also have pneumonia based on
her recent surgery and immobility. Further
assessment should be performed to ascertain
the specifics of her symptoms.
90
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(cont’d
)
When the ED physician is notified of the
patient’s manifestations, she is moved
immediately to a treatment room. The physician
writes the following orders:
– O2 at 2 L per nasal cannula
– Stat CBC, BMP, d-dimer, aPTT, INR
– Stat CT of the chest
– Start a saline lock
Which order takes priority at this time?
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Based on the patient’s pulse oximetry
reading, the priority order is the
administration of oxygen. Next, the saline lock
should be started. Once the vein is accessed,
blood can also be obtained for the CBC,
BMP, d-dimer, PTT, and INR. After the
laboratory specimens are sent, the radiology
department can be notified to perform the stat
CT of the chest.
92
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(cont’d)
While in the treatment room, the patient says she needs to use the
bathroom. The nursing assistant is delegated this task.
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ANS: A
The nursing assistant should place the patient on a
bedpan and stay with her. She is too short of breath
to ambulate to the bathroom and she should remain
on the oxygen at all times. The nursing assistant
should not ask the provider about an indwelling
catheter because this would only increase the
possibility of a UTI. The patient should never be told
to try to wait, because this could also increase the
risk for UTI.
94
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(cont’d)
Two hours later, the patient is admitted to the medical
unit where she is started on a continuous IV heparin
weight-based protocol.
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ANS: D
When a patient is started on continuous
heparin, the aPTT is drawn before therapy is
started and then every 4 hours until a
therapeutic range of 1.5 to 2.5 times the
control is reached. Thereafter, the aPTT is
checked daily.
96
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(cont’d)
Three days later, the provider prepares to discharge the patient on
warfarin (Coumadin).
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ANS: A, B, D
It will be important for the patient to have follow-up
INR laboratory tests done, reporting any bruising or
bleeding, and use a soft toothbrush and electric razor
while on warfarin therapy. Vitamin K is the antidote
for warfarin, so patients should not consume a great
deal of foods that are high in this vitamin. A skin rash
is a sign of an adverse drug reaction and should be
reported to the provider immediately.
98
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Question 1
Which patient is at greatest risk of developing acute
respiratory distress syndrome (ARDS)?
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Answer: A
Rationale: All patient scenarios create a risk for ARDS.
However, the trauma patient with direct chest injury and known
aspiration is at greatest risk. ARDS risk factors include direct
lung injury (most commonly aspiration of gastric contents),
systemic illnesses, and injuries. The most common risk factor
for ARDS is sepsis. Other risk factors include bacteremia,
trauma with or without pulmonary contusion, multiple fractures,
burns, massive transfusion, near drowning, post-perfusion injury
after cardiopulmonary bypass surgery, pancreatitis, and fat
embolism.
100
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Question 2
A patient is going home on warfarin (Coumadin) therapy
to manage an acute pulmonary embolism. Which
patient response indicates further discharge teaching is
needed?
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Answer: C
Rationale: Patients who experience a
venothromboembolism/pulmonary embolism are
frequently discharged on anticoagulant therapy (e.g.,
warfarin [Coumadin]). The patient should be
educated to understand the risks and monitoring of
this drug to include weekly monitoring for therapeutic
levels, consistency in dosing regimens, and foods to
avoid (e.g., leafy green vegetables, green tea,
alcohol, cranberry juice, etc.).
102
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Question 3
A patient in acute respiratory failure is classified
as having ventilatory failure. A potential cause
of ventilatory failure is:
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Answer: A
Rationale: Acute ventilatory failure is the type of problem in
oxygen intake and carbon dioxide removal (ventilation) and
blood delivery (perfusion) that causes a ventilation-perfusion
(V/Q) mismatch in which perfusion is normal but ventilation is
inadequate. It occurs when chest pressure does not change
enough to permit air movement into and out of the lungs. As a
result, too little oxygen reaches the alveoli and carbon dioxide is
retained. Opioid analgesic overdose is a possible cause of
ventilatory failure. The others listed are related to oxygenation
failure.
104
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