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SPECIFIC LARYNGITIS
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• CHRONIC LARYNGITIS IS CHRONIC
INFLAMMATION OF LARYNGEAL STRUCTURES
MOST COMMONLY OF LARYNGEAL MUCOSA
• IT IS MAINLY OF TWO TYPES
1. SPECIFIC
2. NON SPECIFIC
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FOUR POINT GRADATION OF CHRONIC
LARYNGITIS
1. GRADE I - MILD ERYTHEMA,STASIS OF
SECRETIONS, STRING SIGN, PILING UP OF INTER
ARYTENOID MUCOSA
2. GRADE II – DIFFUSE EDEMA AND MUCOSAL
THICKENING BUT WITH LITTLE ERYTHEMA
3. GRADE III – DIFFUSE ERYTHEMA, WITH
GRANULAR FRIABLE MUCOSA OR ULCERATION
4. GRADE IV DISCRETE GRANULOMAS WITH OR
WITHOUT EDEMA AND ERYTHEMA
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NON SPECIFIC LARYNGITIS
IT IS COMMON CONDITION WITH WIDE SPECTRUM
OF SIGN SYMPTOMS AND SEVERITY
PATIENTS PRESENT WITH DYSPHONIA AND
SYMPTOMS SUCH AS THROAT DISCOMFORT,
HALITOSIS OR OTALGIA
COMMON ETIOLOGICAL FACTORS ARE
1. SMOKING
2. VOICE ABUSE
3. GASTRO OESOPHAGAL REFLUX
DISEASE
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TYPES
1. CHRONIC LARYNGITIS WITHOUT
HYPERPLASIA
2. CHRONIC HYPERTROPHIC LARYNGITIS
3. PACHYDERMIA LARYNGIS
4. ATROPHIC LARYNGITIS
5. LARYNGEAL HYPERKERATOSIS
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CHRONIC LARYNGITIS WITHOUT
HYPERPLASIA
IT SYMMETRICALLY INVOLVES WHOLE LARYNX
ETIOLOGY-
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CLINICAL FEATURES
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TREATMENT
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CHRONIC HYPERTROPHIC LARYNGITIS
• VOCAL NODULES
• POLYPUS
• CONTACT ULCER
• REINKE’S EDEMA
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CHRONIC DIFFUSE HYPERTROPHIC
LARYNGITIS
ETIOLOGY IS SIMILAR TO THAT OF LARYNGITIS
WITHOUT HYPERPLASIA
PATHOLOGICAL CHANGES START IN GLOTTIC
REGION AND MAY EXTEND TO BANDS,BASE OF
EPIGLOTTIS ,SUBGLOTTIS AND SUB MUCOSAL
GLANDS
HYPERAEMIA,EDEMA AND CELLULAR
INFILTRATION IN SUB MUCOSA SEEN INITIALLY
THERE WILL BE CHANGE OF RESPIRATORY
EPITHELIUM TO HYPERPLASTIC SQUAMOUS TYPE
AND LATER KERATINISATION OCCUR
MUCOUS GLANDS INITIALLY HYPERTROPHY BUT
LATER UNDERGOES ATROPHY
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CLINICAL FEATURES AND TEATMENT
PATHOLOGY
SINGING IN HIGH NOTES AT END OF BREATH LEADS TO
HYPERKERATOSIS OF FREE EDGES OF VOCAL CORDS
THEY ARE USUALLY BILATERAL
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SIGNS AND SYMPTOMS
TREATMENT
VOICE REST, SPEECH THERAPY
EXCISION IF HOARSENESS PERSISTS
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CONTACT ULCERS
POLYPUS
IT OCCURS AS SMOOTH SESSILE, PEDUNCULATED
SWELLING FROM VOCAL CORD, MAY BE DUE TO
LOCALISED EDEMA
MICROLARYNGIOSCOPIC EXCISION IS PERFORMED
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REINKE’S EDEMA
ETIOLOGY
1. CHRONIC IRRITATION OF VOCAL CORDS
2. HEAVY SMOKING
3. CHRONIC SINUSITIS
4. LARYNGOPHARYNGEAL REFLEX
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CLINICAL FEATURES
HOARSENESS
PATIENT USES FALSE CORDS ,THIS GIVES HIM
LOW PITCHED AND ROUGH VOICE
ON IDL VOCAL CORDS APPEAR AS FUSUFORM
SWELLINGS WITH PALE TRANSLUCENT LOOKS,
VENTRICULAR BANDS SHOW HYPERAEMIC AND
BECOME HYPERTROPHIED AND HIDE VIEW OF
TRUE CORDS
TREATMENT
1. DECORTICATION OF VOCAL CORDS
2. VOICE REST
3. SPEECH THERAPY
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PACHYDERMIA LARYNGIS
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CLINICAL FEATURES AND TREATMENT
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ATROPHIC LARYNGITIS
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LARYNGEAL HYPERKERATOSIS
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TREATMENT
VOICE REST
SMOKING SHOULD BE STOPPED
STEAM INHALATION
EXCISION OF LEUCOPLAKIC PATCHES BY
MICROLARYNGOSCOPY
LASER MAY BE USEFUL
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CLINICAL FEATURES AND TREATMENT
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GRANULOMATOUS
CONDITIONS OF
LARYNX
1. TUBERCULOSIS OF LARYNX
2. LUPUS OF LARYNX
3. SYPHILIS OF LARYNX
4. LEPROSY OF LARYNX
5. SCLEROMA OF LARYNX
6. LARYNGEAL MYCOSIS
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LARYNGEAL TUBERCULOSIS
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ETIOLOGY AND PATHOLOGY
AGE – 20 – 40 YEARS
BOTH SEXES ARE AFEECTED EQUALLY
TUBERCLE BACILLI REACHES THE LARYNX
WITH SPUTUM OR IT REACHES BY
LYMPHATIC OR BLOOD VESSELS
TUBERCLE FORMATION IS SEEN INITIALLY
ULCERATION OF THE TUBERCLES OCCURS
SOON
PERICHONDRITIS AND COLD ABSCESS
FORMATION MAY OCCUR IN ADVANCED
CASES
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SIGNS SYMPTOMS
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INVESTIGATIONS
SPUTUM EXAMINATION FOR AFB
RADIOGRAPH OF CHEST
BIOPSY CLINCHES THE DIAGNOSIS
TREATMENT
1. ANTI TUBERCULAR DRUGS
2. VOICE REST IS ADVICED
3. ANALGESICS
LARYNGEAL MYCOSIS
•FUNGAL INFECTIONS SUCH AS CANDIDIASIS , HISTOPLASMOSIS
AND BLASTOMYCOSIS
•DIAGNOSIS MADE ON BIOPSY OR FINDING SIMILAR LESIONS IN
OTHER PARTS OF THE BODY
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SYPHILLITIC LARYNGITIS
IT IS VERY RARE CONDITION NOW
IN TERITIARY STAGE GUMMA MAY BE
ENCOUNTERED, AFFECTING EPIGLOTTIS AND
ANTERIOR 1/3RD OF VOCAL CORDS
HOARSENESS IS COMPLAINT
DIFFUSE INFILTRATION RESEMBLING
HYPERTROPHIC LARYNGITIS
ULCERATION
PERICHONDRITIS
LARYNGEAL STENOSIS DUE TO SCARRING AND
ADHESIONS
TREATMENT INCLUDES ANTI SYPHILLITIC DRUGS,
TRACHEOSTOMY IF REQUIRED, LARYNGOPLASTY
FOR STENOSIS
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•LARYNGEAL LEPROSY IS VERY RARE
•DIFFUSE NODULAR INFILTRATION OF
EPIGLOTTIS,ARYTENOIDS AND FALSE CORD MAY OCCUR
•IT MAY RESULT IN STENOSIS AND DEFORMITY OF LARYNX
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LARYNGEAL SCLEROMA
1. STREPTOMYCIN OR DOXYCYCLINE
2. STEROIDS REDUCE FIBROSIS AND STENOSIS
3. TRACHEOSTOMY IF NECESSARY
4. LARYNGEAL DILATATION
5. LASER EXCISION MAY BE HELPFUL
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