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& ITS
USE
In men, progesterone lifts the mood and encourage restful sleep. It uses fat
for energy, protect against prostate cancer. Overall, energy and strength is
improved in men who have adequate progesterone levels.
1. Estrogen – bane of male bodybuilders
2. Progestins – used in female contraceptives
3. Mineralocorticoids – control water balance
4. Glucocorticoids – anti-inflammatory compounds
5. Vitamin D
6. Bile acids
Prolactin: This is a protein that promotes milk production in the female body, and even worse,
if you abuse even in the male body. Prolactin causes a decrease in Luteinizing Hormone, and this
lowers testosterone.
Luteinizing Hormone: This stuff (usually called LH, in shorthand) promotes the secretion of
sex hormones. We’re hoping to keep it high (or not let it get too low) so it keeps telling out
testes to secrete testosterone. Both LH as well as testosterone is secreted in pulses between eight
and fourteen times per day, testosterone being preceded by LH by approximately an hour.
Testosterone is, of course, controlled via a negative feedback loop, thus a higher level of
testosterone in your body causes a decrease in LH.
Steroids: These are compounds whose molecules contain fairly complex rings
of Carbon and Hydrogen atoms. Steroid hormones include (but are not limited
to) testosterone and estrogen, and we will be primarily concerned with those
two although we will examine many other hormones. Sometimes we use the
term “steroid” to mean anabolic steroid, which is only one possible type. Steroid
hormones (like testosterone) are soluble in the lipids that make up cell walls.
This means they can get into a cell and mess around with the receptors in the
nucleus, which is exactly what we want.
Receptor: This is a thing in the cell that is basically like a parking spot.When a
steroid hormone comes in, it’s like parking a car in that spot. The steroid
hormone then tells the cell to do something. If the hormone is testosterone, it
may tell the cell to “build more muscle!”
Androgen Receptor: This is the parking spot “reserved” for steroids like
testosterone and such, in other words, androgens.
Prostaglandins: Some of these regulate cellular responses to hormones and stimulate the
secretion of a variety of hormones.
Negative Feedback System (or loop): This is the system by which your body recognizes an
abundance of a particular hormone and consequently stops producing it. In simple terms, if you
are injecting testosterone, your body will sense this and stop producing its own.
Pituitary Gland: The anterior lobe of this secretes a variety of hormones such as growth
hormone, thyroid- stimulating hormone, prolactin, follicle-stimulating hormone, and luteinizing
hormone.You want to keep this thing healthy and happy.
Growth Hormone: Growth Hormone (GH) is a protein that stimulates your body’s cells to
undergo more rapid cell division. It enhances the movement of amino acids through cell
membranes and causes an increase in the rate in which they convert molecules to proteins and
decrease the rate they use carbohydrates and increase the rate they use fats. It is secreted in
rhythmic pulses, especially while you’re asleep and has an important anabolic effect on the body.
Growth Hormone-Releasing Hormone: This stuff is the hormone that releases growth
hormone.
Insulin: Insulin is a protein secreted by the pancreas that acts on the liver to stimulate
the formation of glycogen from glucose and inhibits the conversion of noncarbohydrates
into glucose.
Insulin-Like Growth Factor: Insulin-like growth factor is released from the liver in
response to GH. It has an important anabolic effect on the body.
Glucagon: This is a hormone that is produced in the pancreas and regulates blood
sugar levels. Unlike insulin, glucagon is released when blood sugar levels are low. It
causes the release of glucose from glycogen.
Thryoid Stimulating Hormone: This is a protein bound to a carbohydrate. It
controls the secretion of hormones from the thyroid gland.
Hypothalamus: This releases gonadotropin- releasing hormone, and also controls
most secretions of the pituitary gland, which leads me to the. ...
Pituitary Gland: This is where the Philosopher Rene’ Descartes thought the soul
lived. Actually, it’s much more important because it controls the secretion of LH and
FSH, and thus, the production of testosterone! It also controls secretion of GH and
thyroid stimulating hormone.
Hypothalamic-Pituitary-Testicular-Axis: This is usually called the HPTA,
and it basically regulates all of the hormones that stimulate the production of
testosterone as well as GH and other goodies. Needless to say, keeping your
HPTA in good working order is very important.
•Any testosterone form will stack well with any anabolic steroid.
•Other anabolic steroids may not stack well together but with testosterone you
can never go wrong.
•Both Steroids & testosterone can make you leaner, harder, increase athletic
performance and simply improve your overall life dramatically.
TESTOSTERONE – THE BASICS
PRODUCTION OF PRODUCTION OF
Stimulation of receptor molecules in muscle cells which activate specific genes to produce proteins
Effectiveness of anabolic steroids is dependent upon unbound receptor sites in muscle
Intense strength training increases number of unbound receptor sites
Research studies demonstrated improved performance in experienced weight lifters
MECHANISM OF ACTION (PART:B)
• Gear
• Juice
• Roids
• Stackers
Steroids are a general class of agents that all have the steroid ring in common.
The steroid ring is comprised of three 6-carbon rings and one 5-carbon ring
joined, of which cholesterol is the most basic form and, indeed, the
precursor.
In general, the goal of altering an AAS is to increase its anabolic characteristics and to
decrease its androgenic features, thus multiplying the compound's desirable, anabolic,
nitrogen-sparing effects and minimizing its generally undesirable, androgenic, virilizing
effects.
To date, however, complete dissociation of the anabolic effects of an AAS from its
androgenic characteristics has not been possible.
Clinically, AASs have been used to treat a host of conditions,
including the following:
• Many forms of anemia
• Acute and chronic wounds
• Protein-calorie malnutrition with associated weight loss
• Severe burns
• Short stature
• Osteoporosis
• Primary or secondary hypogonadism
• Prolonged catabolic state secondary to long-term use of corticosteroids
• Human immunodeficiency virus ( HIV) wasting syndrome
Structure of Testosterone & AAS
TESTOSTERONE DERIVATIVE:
1. Testosterone
2. 4 – hydroxytestosterone
3. Boldenone
4. Clostebol
Pro-hormone like:
1. Dehydroepiandrosterone (DHEA)
2. Exemestane
Prodrugs:
1. Cloxotestosterone
2. QuinboloneTESTOSTERONE- PROPIONATE- PHENYLPROPIONATE –
ISOCAPROATE- DECANOATEne
DIHYDRO-TESTOSTERONE(DHT) DERIVATIVES
DI-HYDRO-TESTOSTERONE DERIVATIVES(DHT):
1. Dihydrostestosterone (DHT; androstanolone, stanolone)
2. Drostanolone
3. Epitiostanol
4. Mesterolone
5. Metenolone (methenolone, methylandrostenolone)
6. Stenbolone
Prodrugs: Ether
1. Mepitiostane
Azine dimers:
1. Bolazine
19 NOR-TESTOSTERONE(nandrolone) DERIVATIVES
Prohormone like:
1. Bolandiol (nor-4-androstendiol)
Prodrugs: Esters
1. Bolmantalate (nandrolone adamantoate
17 a – ALKYLATED TESTOSTERONE DERIVATIVES
Prohormone like:
1. Methyltestosterone 3 – hexyl ether
2. Penmesterol (penmestrol)
17 a – ALKYLATED DIHYDROTESTOSTERONE DERIVATIVES
Prodrugs:
1. Propentandiol
17 a – VINYLATED 19-NOROTESTOSTERONE DERIVATIVES
Esters:
1. Etynodiol diacetate
2. Norethisterone acetate
3. Norethisterone enantate
• Health care providers can prescribe steroids to treat hormonal issues, such as delayed puberty.
• Steroids can also treat diseases that cause muscle loss, such as cancer and AIDS.
• Some athletes and bodybuilders abuse these drugs to boost performance or improve their physical
appearance.
HOW
STEROID
WORKS
STEROIDS WORK, IN PART,
BECAUSE YOU EXPECT THEM TO
WORK
PSYCHOLOGICAL EFFECTS – mechanism of action of steroids
Other factors
• On top of how well they work physiologically, a major factor is how well they
work psychologically – if you do something expecting to get a ton stronger, there’s a
good chance you’ll get a ton stronger. This applies to much more than steroids.
• Steroids do provide a substantial advantage for sports that aren’t governed by weight
classes. However, taking too high of a dose right off the bat may actually decrease
performance (increased strength and mass, but decreased relative strength), especially
in sports with weight classes. If you decide to use steroids, you’ll probably get the best
bang for your buck, strength-wise, with very conservative doses initially.
CONTINUTED……
FINALLY HERE ARE SOME TAKEAWAYS
• If you take steroids and then come off of them, you’ll probably lose some of
the size and strength you gained, but you’ll always be at an advantage
relative to a lifetime drug-free athlete.
Anabolic steroids are beneficial in building up the body but, when the steroid course is finished,
the reverse action occurs and there is an increase in catabolism as a result of the corticosteroids
being taken up by the receptors again. This may be one reason for the weight loss that
STEROID
STEP 1 – Steroids first metabolised in the nucleus of the cell
STEP 2 – They are then taken from the cell and degraded in
the liver
• Genetic make-up
• Training
• Illness
• stress
• Diet regulation
SIDE EFFECTS OF ANABOLIC
STEROIDS
• Many but not all of the side effects are of a temporary nature
and will resolve within some weeks of ceasing the drugs.
Vital signs, including heart rate and blood pressure, and basic chemistries, such
as sodium, potassium, hemoglobin, hematocrit, BUN (blood urea nitrogen),
creatinine, hepatic, and lipid profiles, must be monitored carefully.
Monitoring these parameters will help the clinician to determine drug choice,
treatment dose, and duration, and will help to alert the prescriber to potentially
serious adverse effects that necessitate the discontinuation of therapy.
The most common deleterious effects of AAS use on the cardiovascular system
include increased heart rate, increased blood pressure, and changes in lipid
metabolism, including lowered high-density lipoprotein (HDL) and increased
low-density lipoprotein (LDL).
The increase in heart rate is thought to be more profound with the androgens,
especially those resistant to aromatase, and is believed to be due to the inhibition
of monoamine oxidase (MAO).
This effect, when combined with the increased renal recovery of ions, such as
sodium, causing subsequent fluid retention, can lead to dramatic increases in
blood pressure.
Combine this with a tendency to lower HDL and raise LDL, and the stage is set
for untoward atherogenic and cardiac effects.
Anabolic steroid users can have a lower left ventricle ejection fraction.
Ananbolic steroid abuse has been associated with ventriculararrhythmias.
The alanine aminotransferase/aspartate aminotransferase (ALT/AST) can be
seen to rise, usually in a dose-dependent fashion. Levels approaching 2-3 times
baseline are often set as upper limits of reference ranges when administering
oral AASs, but the risk-to benefit ratio must be constantly evaluated.
AAS use also results in suppression of clotting factors II, V, VII, and X, as
well as an increase in prothrombin time.
Another life-threatening, albeit rare, adverse effect that is seen in the liver and
sometimes in The changes made to C-17 to inhibit hepatic degradation make
nearly all oral preparations hepatotoxic.
peliosis hepatitis, which is characterized by the appearance of blood-filled,
cystic structures. These cysts, which may rupture and bleed profusely, have been
found in patients with near-normal liver function test (LFT) values, as well as in
individuals who are in liver failure. Fortunately, drug cessation usually results in
complete recovery.
Primary liver tumors have been reported, most of which are benign, androgen-
dependent growths that regress with the discontinuation of AAS therapy.
Several case reports exist of young, healthy athletes who have died from primary
malignant liver carcinoma, with the only identifiable risk factor being oral AAS
use.
Anabolic steroid abuse has been considered a risk factor for non-alcoholic fatty
liver disease
•The endocrine system has a remarkable array of checks and balances that ensure the
human body is at or near homeostasis at any point in time. Interruption of one feedback
system has been shown to produce changes in other hormone feedback systems via direct
receptor changes, as well as through competition for common enzymes and metabolic
pathways.
•Studies have shown that AASs bind to glucocorticoid, progesterone, and estrogen
receptors and exert multiple effects.
•By suppressing FSH, spermatogenic function should be reduced.
•AASs have also been shown to alter fasting blood sugar levels and decrease glucose
tolerance, presumably due to either a hepatic effect or changes in the insulin receptor.
•Thyroxine-binding globulin (TBG) may also be lowered by AASs and result in lowered
total T4 levels, with free T4 levels remaining normal. An up-regulation of sex-hormone
binding globulin, with a concomitant decrease in TBG, is thought to cause the changes in
total T4 levels.
•The aromatization of testosterone/AASs to estradiol and related compounds can render
many adverse estrogenic effects. The most apparent and common adverse effect is the
growth of tender, estrogen-sensitive tissue under the male nipple. This unsightly growth is
termed gynecomastia and can be treated medically or surgically.
The male prostate is very sensitive to androgens, especially those that are reduced in prostatic
tissue to dihydrotestosterone (DHT) or DHT analogs.
In response to this stimulation, the prostate grows in size, potentially causing or exacerbating
benign prostatic hyperplasia (BPH). Worsening BPH may indeed cause severe bladder and
secondary renal damage.
In addition, the use of AASs in patients with underlying carcinoma of the prostate is
absolutely contraindicated due to the potential for hormone-sensitive tumor growth.
Studies have shown no urinary symptoms, urine flow rate, or urine postvoid residual.
Direct clotting factors may be reduced with an increase in prothrombin time. In patients on
concomitant anticoagulant therapy, this increase could cause bleeding.
AASs cause increases in hemoglobin and hematocrit and are used in many cases of
anemia, although the clinician must be aware of the potential for polycythemia.
Skin, especially the face and scalp, has a high degree of androgen receptors and 5AR.
DHT is known to cause increases in sebum production, leading to clinical acne. Also, male
pattern baldness is related to scalp DHT production and binding, along with genetic factors
influencing hair growth.
Male pattern baldness is greatly exacerbated by most AASs in susceptible individuals.
• Self- prescribing habits
• Stacking
• Multiple drugs in a cycle of 12-16 weeks
• Dose 2-8 times higher that the therapeutic range
• Multiple drug use increases side effects and risk to the user
• Decreased or no medical surveillance
• Mood elevator eg. Mesterolone versus amitryphtilline
• Behavior problems
• Addictive
• Withdrawal symptoms includes depression, fatigue, paranoia and
sucidal thoughts
A research in 1983 showed that the pyschoactive effects, withdrawal
symptoms and underlying biological mechanism of AASs appear to be
similar to cocaine, alcohol or opiod abuse.
MEDICAL EXAMINATION BEFORE STARTING
ANABOLIC STEROIDS
RESTING ECG
1.
Physical Examination:
2. TREADMILL STRESS TEST
3. 2D ECHO – OPTIONAL / FOLLOW –UP 1. Skin
4. ANGIO CT – OPTIONAL / FOLLOW-UP
5. BLOOD SUGAR TESTING
2. Temperature
6. HB1 AC 3. Pulse
7. LIPID PROFILES
8. CBC 4. Blood pressure
9. ESR
10. URINE ROUTINE & MICROSCOPIC
5. Pre-existing medical
11. LIVER PROFILE condition
12. RENAL PROFILE
13. SEMEN ANALYSIS
6. Any signs and symptoms of
14. THYROID TESTS steroid side effects
15. USG ABDOMEN AND PELVIS
16. DEXA SCAN
TYPES OF STEROIDS
Doctor Prescribed Steroid
Inject able Steroids Fat Loss Steroids
Oral Steroids
Steroid Cream Horse Steroids
Steroid Pills Illegal Steroids
Steroid Tablets Mexican Steroids
Best Steroids Muscle Building Steroids
British Dragon Steroids Natural Steroids
Bulking Steroids
Cheap Steroids
Oral Anabolic Steroids
Cutting Steroids Real Steroids
Designer Steroids Safe Steroids
There are 32 common types of steroids which represent the anabolic
androgenic steroids that can be used by anyone who supplements with
such hormones for any reason
The form of administration and ester(s) does not change the
hormones specific nature
Example: Testosterone
•These 32 types of steroids can be used in numerous purpose.
•Unlikely anyone will ever use all of them
•Last trait associated with AAS supplementation can be obtained with
just a few.
•Most common steroid will carry a primary purpose; traits that carry
primary purpose.
•They will also carry secondary characteristic that can serve another
purpose too.
•Versatile steroids can meet almost any purpose of steroid
supplementation
•Mode of administration can affect compounds versatility
ORAL STEROIDS
There are many oral steroids & with each one of them there are
various traits, benefits and purposes.
In general for most male anabolic steroid users they should not
be the foundation in-which their use is built upon.
Most cycles should be built around exogenous testosterone use.
As a general rule of thumb testosterone should be your base.
Most common concerns is liver damage.
In general oral steroids should only be used for 6 to 8 weeks.
Oral steroids generally has much shorter half-life than the many
injectable steroids; for this reason daily use is generally needed
to receive the maximum benefit.
“Small Doses” spread out over the day is one of the many keys to
successful and responsible anabolic steroid use, even more so
when speaking of oral steroids.
ORAL STEROIDS
COMPOUND TRADE NAME THERAPEUTIC USE PERFORMANCE
OXYMETHOLONE ANADROL Anaemia, muscle wasting Promote mass
disease Fullness in cutting phase
Choose bulking steroids that will add quality mass in the most efficient and effective ways
possible.
Safety should be major concern because each anabolic steroid can carry with it its own various
side-effects as well as level of probability in side-effects occurring and you are encouraged to
seek out the specifics of each one.
For a good bulking cycle most all men will find testosterone to be king.
It is generally very well-tolerated by all healthy adult men it is also by far the most efficient
anabolic steroid of all time. It does not matter which form of testosterone you use.
For a good bulking cycle you are encouraged to always make testosterone your base and for
many this will be the only steroid needed but of course some will want more. Best bulking
steroids of all time include:
1. Testosterone
2. Deca-Durabolin
3. Dianabol
4. Anadrol
5. Trenbolone
Cutting Steroids vs. Bulking Steroids
While every one of these anabolic androgenic steroids serves a multitude of purposes, each
one of the following serves the purpose of cutting to a larger degree than bulking.
1.Winstrol (Stanozolol)
2.Anavar (Oxandrolone)
3.Halotestin (Fluoxymesterone)
4.Masteron (Drostanolone Propionate)
5.Primobolan (Methenolone Acetate)
6.Primobolan Depot (Methenolone Enanthate)
7.Turinabol (4-Chlorodehydromethyltestosterone)
The best Fat-loss steroids
Trenbolone as a powerful muscle building, strength increasing and hardening agent has
also been shown to possess fat reduction qualities.
Trenbolone have been shown to actually reduce stored body fat; while increasing lean tissue,
which leads to more pleasing total of fat versus lean tissue, coupled with actual fat reducing
qualities, this makes Tren more or less the king of this category when we consider the rates in-
which it handles both processes.
Beyond Tren, other well-deserving fat loss steroids would include:
1. Winstrol
2. Primobolan
3. Anavar
4. Masteron
5. Equipoise
Common mistaken Fat-loss steroids
The performance enhancing world is full of many items that are in-fact not anabolic androgenic
steroids.
Human growth Hormone: HGH belongs to a class of hormones known as peptide
hormones; these are not steroids.
Beyond HGH many often labeled fat loss steroids that are not steroids at all include:
Clenbuterol
Cytomel (t-3)
Albuterol
Ephedrine
Muscle building steroids
All the steroids in the Primary Cutting Steroids category and all
the steroids in the Primary Bulking Steroids category can be
effectively used for the opposite purpose and in many cases very
effectively and efficiently.
The key is understanding how each anabolic androgenic steroid
you wish to use functions and reacts.
Understand proper nutritional intake that meets your end goals
and by understanding nutrition and steroid function you will
best understand how to maximize the two and reach optimal
results.
In truth you cannot have an understanding of one and not the
other and expect to reach your best; they work together hand-in-
hand.
WHEN AND HOW TO USE
STEROIDS
produce the protein characteristic of the cell. For Example: In the case of muscle cells, that will
• Whether to use oral or injectable drugs – route of administration is not important factor
• For a second or later course of a drug – using the same anabolic steroid. The body does not
develop tolerance and results can be achieved with the same steroid, providing that the
If you try to do two or more at a time, especially if you try to add mass while trying to cut,
a virtual impossibility at any significant level, you’re going to find it an extremely
frustrating process.
if it’s bulking you’ll be able to add more lean mass with less fat accumulation,
if you’re trying to cut you’ll be able to lose more body-fat with less muscle tissue loss that
often accompanies hard dieting.
As bulking and cutting represent the two primary reasons most people supplement, but
you must ensure you are eating correctly to promote this end and exercising as per your
goals bulking or cutting
ANABOLIC STEROIDS CYCLES & STACKS
An anabolic steroid cycles refers to the time frame anabolic
steroids are being used. This time frame is often referred to as
“On-Cycle.”
When steroids are not being used, this is referred to as “Off-
Cycle.”
For the on-cycle phase, there are countless options and stacks.
“Stacks” refer to the combination of anabolic steroids as well as
non-steroidal items used during the on-cycle phase.
With hundreds of anabolic steroids, varying peptide hormones,
SERM’s, AI’s, thyroid hormones and more, there are truly
innumerable possible stacks
RULES OF CYCLES & STACKS
FIRST STEROID CYCLE
The primary rule of every cycle is that it includes some form of
testosterone. The form of testosterone used is of no consequence. The only
thing that matters is that the body has enough of this essential hormone in
order to function properly.
Exception: The use of essential testosterone does not apply to female
anabolic steroid cycles.
If you’ve never supplemented with anabolic steroids before, it’s
recommended that you keep things as simple as you can. You have no idea
how your body is going to react to supraphysiological doses of a hormone.
Further, you want to start with hormones your body is already familiar
with, such as testosterone.
If you begin with numerous steroids in your cycle, if you have any
problems, it is going to be extremely difficult to pinpoint what’s causing the
problem.
Equally important, you may have a hard time pinpointing which steroids
bring you the greatest results.
Start simple and work your way up.
RULES OF CYCLES & STACKS
ADVANCE CYCLES & STACK
Once you have a few cycles and stacks under your belt, &
have had positive experience, you can now consider moving
to more advanced cycles.
For most men, there may be no need or desire to increase
the number of hormones being used or an increase in doses.
A simple and moderately dosed testosterone cycle may be
all you ever need, and such a cycle will work for you every
single time. Your body isn’t going to magically adapt to
where such a plan will no longer work.
Despite this, many men will inevitably want more if they’ve
enjoyed success with smaller steroid cycles.
However, bigger stacks and cycles come with a word of
caution.
Risk to Reward
All steroid cycles and stacks carry with them a strong risk to reward ratio, and
regardless of your experience this will hold true each and every time.
The more you take the greater the reward, but the more you take the greater the
risk.
As risks increase, so does the need for protective measures.
But there will be a cutoff point; where safety is severely jeopardized and the risk
to reward ratio becomes severely inclined towards risk.
Important factors to consider : steroid being used and genetic
Example: Testosterone (any ester)
Dose – 300 mg /week, maximum being 500 mg / week well tolerated
Some men can go up to 750-1000 mg & can still remain healthy
When we surpass 1g per week, estrogenic issues can often be problematic, and
many men will find controlling them extremely difficult
Duration of Use
As steroid cycles refer to the time in which we are actually supplementing with
anabolic steroids, the obvious question are:
1. What is the acceptable time frame?
2. What is the minimum for positive gains?
3. What is the maximum amount of time in-regards to safety?
The human body does not like change; even if such a change is in its best
interest it will fight it and do all it can to stay at its accustomed normal.
We must allow enough time for this “normal” to change.
We must create a new set normal if we are to hang onto any of the gains
made.
If you are off-cycle for an extended period of time you are going to lose some
of the gains made; without the high influx of hormones present to support
the gains you made they will not last forever.
Duration of Use
Novice to Intermediate:
1. Completed a few novice cycles and done so successfully.
2. Not suffered from severe side effects
3. Have made decent gains but have reached a point where you want a little more.
Intermediate to Advanced:
1. Intermediate cycles are as much as most will ever want, and in truth, most all will ever
need.
2. If you cannot make fantastic gains with such plans, you need to reexamine your diet and
training.
3. If you have completed several intermediate cycles successfully, have a desire to reach highest
levels of muscularity; an advanced cycle may be in store.
Beyond Advanced:
The advanced steroid cycles listed below are incredibly powerful and carry with them a
significant level of risk. However, some will surpass advanced plans. This is not something we
can ever recommend, we cannot recommend such plans.
HOW TO COME OFF STEROIDS LONG EXTENDED
PERIOD?
It is called PCT (Post cycle Therapy)
SELECTIVE ESTROGEN RECEPTOR MODULATOR – any one
1. Tamoxifen citrate (Nolvadex)
2. Clomiphne Citrate (Clomid)
SERM stimulate the pituatary to release more LH and FSH which
in turn stimulate the testicles to produce more testosterone
HUMAN CHORIONIC GONADOTROPHIN (HCG)
HCG act to stimulate natural testosterone through an LH
mimicking effect; LH is not actually released, but your body
think it is.
HCG use is not always needed but it can be a perfect way to
prepare your body for the SERM therapy to come.
USE OF SERM:
If your steroid cycle is of a simple or moderate nature (upto 12
weeks), you will need SERM for 4 weeks.
Above this you need 5-6 weeks of SERM therapy and 10 days of
HCG therapy preceding it.
CLOMID – 150 – 100 – 50 MG / ED (reducing dose every 2
weeks)
NOLVADEX – 40 – 20 -20 MG / ED ( reducing dose every 2
weeks)
HCG – 500 – 1000 iu for 10 days (never be surpassed in dose and
duration)
WHEN TO START SERM & HCG?
LARGER ESTER & SERM ONLY – like caproate, cypionate,
enanthante, decanoate, heptanoate, hexanoate, isocaproate,
nonanoate, octanoate or undecyclenate
SERM therapy 14-18 days after your last injection
SMALL ESTER & SERM ONLY – acetate, formate,
phenylpropionate or propionate
SERM therapy should start 3 days after your last injection
If you have butyrate or valerate based steroid, you might wait a few
more days to start SERM therapy.
WHEN TO START SERM & HCG?
LARGER ESTER , SERM & HCG ONLY – like caproate,
cypionate, enanthante, decanoate, heptanoate, hexanoate,
isocaproate, nonanoate, octanoate or undecyclenate
HCG therapy – 10 days after your last injection x 10 days
SERM therapy immediately after HCG therapy
SMALL ESTER & SERM ONLY – acetate, formate,
phenylpropionate or propionate
HCG therapy – 2 days after your last injection x 10 days
SERM therapy should start immediately after HCG therapy.
If you have butyrate or valerate based steroid, you might wait a few
more days to start SERM therapy.
AROMATASE I NHIBITOR (AI’s) in PCT PLAN
1. ANASTROZOLE (ARIMIDEX)
2. LETROZOLE (FEMARA)
3. EXEMESTANE(AROMASIN)
Will stimulate LH & FSH in a similar fashion as a SERM But they
are used to reduce estrogen level dramatically to combat
estrogenic and progestin related anabolic steroid side-effect
when on cycle.
HOW TO COME OFF STEROIDS SHORT PERIODS AND BRIDGING ?
If you are going to be off cycle for a short period of time, less than
12 weeks, PCT plan above are not beneficial.
OPTION 1:
Best is stay off everything for a few weeks – you will lose lean
tissue but if you stay consistent with your training and diet it
won’t be much.
OPTION 2:
Low dose of testosterone 200 – 250 mg per week
OPTION 3:
Low dose of Nolvadex and Dianabol (10 mg /day of each for four
to five weeks)
ANABOLIC WORKOUT
muscle.
• Sterile Equipment
• Keep it Clean
• Where to inject
• other sites
• pre-injection
• the injection
• post-injection
COMPLICATIONS OF POOR
INJECTING TECHNIQUES
• Infection
• Muscle Damage
• Haemorrhage
INDIVIDUAL DRUG-PROFILE
ANADROL:
Anadrol has been derived from Dihydrotestosterone
By addition of a hydroxymethylene group to DHT, it becomes the only DHT
that is used to bulk rather than for cutting.
If you want to increase the sizes of your muscles just before a competition,
this is the one to use.
Because Anadrol is oral, it has been modified to be able to pass through the
stomach and the digestive system in one piece.
It has to pass through the liver and get into the muscles without being
eliminated by the liver.
Chemically speaking, it has been made into a 17-alpha alkylated steroid
meaning that the 17th carbon atom has been altered.
One of the best steroid for bulking in short time.
Also used in wasting disease like AIDS due to its strong muscle building
properties
INDIVIDUAL DRUG-PROFILE
SIDE EFFECTS OF ANADROL– due to araomatization
leading to estrogen formation
1. water retention
2. Affects athletic performance
3. Increase in blood pressure – long standing use
4. Gynaecomastia – particularly in man
INDIVIDUAL DRUG-PROFILE
ANAVAR (OXANDR0LONE):
It is mild but powerful
Mild – because of its extemely high threshold of tolerance.
Both men & women can tolerate this steroid fairly well.
Single most female friendly anabolic steroid in the market.
Used to gain weight following surgery or infection or any ailment that
resulted in weight loss.
Useful in osteoporosis by promoting bone density
Also useful in prolonged exposure to corticosteroids
Combats hepatitis
Useful in children to promote growth & development who lack proper
hormone production.
INDIVIDUAL DRUG-PROFILE
ANAVAR 101:
It is a DHT derive AAS.
It is altered form of DHT with an added oxyen atom replacing the carbon-2
in the A-ring.
This structural change prevents metabolic breakdown and enhaces its
anabolic acitivity significantly.
There is also a structural change to the hormone at 17th carbon position
throug addition of methyl group.
This change help to survive oral ingestion.
Hepatotoxic in nature but mild.
High anabolic – anabolic rating is 3-6 times stronger than testosterone.
Anabolic effect more useful in cutting and athletic enhancement cycles.
Low androgenic (rating of 24) effects which makes it more tolerable
compared to other AAS.
INDIVIDUAL DRUG-PROFILE
ANAVAR 101:
Three primary traits
1. Enhance nitrogen balance – anabolic atmosphere and protects the individual
from catabolic state.
2. Ability to significantly reduce SHBG – increase free testosterone
3. Reduction of gluco-corticoids – prevent fat gains
It burns body fat – because of its firm binding to androgen receptors and
reduce thyroid binding globulin and increase thyroxin-binding prealbumin.
Through this action T3 (triiodothyronine)is utilized to a higher degree.
Increase blood count leading to higher oxygenation – promotes muscular
endurance.
FOR MEN – not a good off season steroid for bulking.
FOR WOMEN – best choice as off season steroid
EXCELLENT CUTTING STEROID – preserve muscle mass during dieting
CONDITIONING EFFECT – harder and more defined appearance.
ATLETIC PERFORMANCE – BEST CHOICE
INDIVIDUAL DRUG-PROFILE
SIDE EFFFECTS:
No aromatization – no estrogenic side effect
Androgenic side effects – less likely than other AAS. Hair loss and acne
if sensitive.
For Female – virilisation possible but less likely if doses are
monitored. Genetic will dictate the final outcome.
Virlization symptoms in females – clitoral enlargement, body hair
growth or deepening of voice – use discontinued immediately.
Increases LDL cholesterol and decrease HDL cholesterol.
Low testorsterone levels if exogeneous testosterone is not included.
Hepatotixicity – most mildest hepatotoix AAS. – limit total use to 8
weeks, avoid alcohol and other c17 aa steroids. Supplement with liver
detoxifier.
INDIVIDUAL DRUG-PROFILE
CLENBUTEROL:
Powerful bronchodilator used to treat asthma and other related breathing problems.
Used as thermogenic – fat loss
Very popular fat burner among AAS user and competitive body builders.
Beta-2 receptor stimulator – enhance metaboic rate – increase fat loss.
Not well-suited for Obese or significantly overweight.
Best time to use is when one is already lean – to get rid of stubborn fat.
SIDE EFFECTS: can be strong
Jittery feeling
Shaky hands
Increased sweating
Increased body temperature
Paplitation and high blood pressure in abusers
Headache, nausea, vomiting and Insomnia
Muscle cramps